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1 n reduced viral DNA accumulation and no leaf chlorosis.
2 s such as reduced stature and development of chlorosis.
3 ampers photosynthesis and is associated with chlorosis.
4  grapevines that leads to leaf scorching and chlorosis.
5 iliverdin concentrations-a phenomenon called chlorosis.
6 r fresh weight, reduced root elongation, and chlorosis.
7 'h1 mutant plants from Fe deficiency-induced chlorosis.
8 uch as accumulation of carbohydrates or leaf chlorosis.
9 s in tobacco (Nicotiana benthamiana) induced chlorosis.
10 rce's disease of grape and citrus variegated chlorosis.
11 oved fertility and also reversed interveinal chlorosis.
12 , leading to accumulation of sugars and leaf chlorosis.
13 ssion of AvrB in rpm1 plants results in leaf chlorosis.
14  Fe deficiency symptoms, such as interveinal chlorosis.
15 ase symptoms, with plants showing no visible chlorosis.
16 racterized by necrotic lesions surrounded by chlorosis.
17 nts with reduced CpNifS expression exhibited chlorosis, a disorganized chloroplast structure, and stu
18 r the tic40 and hsp93-V mutations, exhibited chlorosis, aberrant chloroplast biogenesis, and ineffici
19 plants were found to have significantly less chlorosis after treatment with the superoxide-generating
20           Other phenotypes, however, such as chlorosis along the leaf veins, are likely caused by thi
21 ns 16% pRBR binding activity, only developed chlorosis along the veins, and viral DNA, AL1 protein an
22 t for the tolerance to Fe deficiency-induced chlorosis, also on soil substrate.
23 tically low Fe concentrations and, hence, Fe chlorosis, although the transcriptional Fe deficiency re
24 of silencing from germination rapidly caused chlorosis and a strong developmental phenotype that led
25 hereas ethylene insensitivity led to reduced chlorosis and ABA deficiency to reduced anthocyanin accu
26 ive28 (cpd28) and cpd47 mutants exhibit leaf chlorosis and accumulation of starch and soluble sugars.
27 ough leaves and cotyledons continued to show chlorosis and altered chloroplasts.
28  we found that GPA feeding induced premature chlorosis and cell death, and increased the expression o
29 in alkaline soil, fro7 seedlings show severe chlorosis and die without setting seed unless watered wi
30 ight promoted the development of interveinal chlorosis and growth inhibition in the transgenic plants
31                  Furthermore, development of chlorosis and growth inhibition was dependent on growth
32 a was supplemented with ribose, which led to chlorosis and growth inhibition.
33  iron deficiencies, measured as reduced leaf chlorosis and improved maintenance of the photosynthetic
34 seedlings showed severe growth defects, leaf chlorosis and leaf shrinkage.
35 ection of Brachypodium with PMV+SPMV induced chlorosis and necrosis of leaves, reduced seed set, caus
36 measurements of leaf arching, increased leaf chlorosis and necrosis, and altered UV-B regulation of s
37  while S. albescens suffered reduced growth, chlorosis and necrosis, impaired photosynthesis, and hig
38 sical defense marker, and symptoms including chlorosis and necrosis.
39 multiplication, and delays the onset of leaf chlorosis and necrosis.
40 showed a distinct phenotype characterized by chlorosis and reduced plant size, as well as hypersensit
41 cript levels by RNA interference resulted in chlorosis and reduced Pst sporulation.
42 mpact crop yield and food quality by causing chlorosis and reduced root and shoot growth.
43                                         Both chlorosis and ribose accumulation were abolished upon th
44 cluding Arabidopsis to actinonin resulted in chlorosis and severe reductions in plant growth and deve
45                    The infection causes leaf chlorosis and stimulates the plant to produce nutrient-r
46                          We suggest that the chlorosis and stunting in P6-transgenic and CaMV-infecte
47 for mutants that suppressed the phenotype of chlorosis and stunting.
48 anisms underlying the onset of Fe-deficiency chlorosis and the maintenance of photosynthetic function
49 ression of this gene in rice alleviates leaf chlorosis and wilting under cold stress.
50 II), to reduce photosynthesis, regulate leaf chlorosis, and confer Pst resistance.
51 thesize any nicotianamine, shows strong leaf chlorosis, and is sterile.
52 ere obvious, including impaired growth, leaf chlorosis, and necrosis and curling of leaf margins.
53 ymptoms such as developmental abnormalities, chlorosis, and necrosis.
54  predominantly in female tissues, as well as chlorosis, and the accumulation of anthocyanins in cepr1
55 biochemical basis, biology, and evolution of chlorosis are poorly understood.
56  coronatine, a major determinant of the leaf chlorosis associated with DC3000 pathogenesis.
57 umber of mutants exhibiting photorespiratory chlorosis at ambient CO(2), including several with defec
58 tudies have shown that mild to moderate iron chlorosis can have positive effects on grape quality pot
59 ed for optimal activity in tomato, including chlorosis, changes in chloroplast structure, cell wall t
60 nglongbing, citrus canker, citrus variegated chlorosis, citrus tristeza virus, citrus sudden death, s
61  at 22 degrees C but showed chilling-induced chlorosis, confirming that the gene is essential for low
62 ees C exhibits a pattern of chilling-induced chlorosis consistent with a disruption of chloroplast de
63                                 An improved, chlorosis-corrected, cytoplasmic male sterile Brassica j
64      Two Xylella diseases, citrus variegated chlorosis (CVC) and Pierce's disease (PD) of grapevines,
65 recently showed that iron deficiency-induced chlorosis depends on phosphorus availability.
66 unctional evidence of recurrent evolution of chlorosis, describe a biliverdin-binding protein in vert
67 d with the modified viral vectors manifested chlorosis due to silencing of either ChlI or PDS in appr
68 ron uptake, resulting in impaired growth and chlorosis during iron limitation.
69 nd produced phenotypes of starvation-induced chlorosis during short-day growth conditions and extende
70 ormal phenotype characterized by interveinal chlorosis, growth inhibition and weakening of stems and
71 to promote further growth (HopM1 and HopE1), chlorosis (HopG1), lesion formation (HopAM1-1), and near
72 ic studies of abiotic stress iron deficiency chlorosis (IDC) of soybean is reported.
73 rmination rates, slow growth rates, moderate chlorosis, impaired fertility and reduced long term seed
74 mechanisms underlying pharmaceutical-induced chlorosis in a model microalgae species and demonstrates
75  screen for mutants that lack AvrB-dependent chlorosis in an rpm1 background, we isolated TAO1 (targe
76  Significantly, starch accumulation precedes chlorosis in cells that will become a yellow sector.
77 lence of Pst DC3000 and for the induction of chlorosis in host plants.
78 in wild-type plants but strongly exacerbated chlorosis in irt1 plants, indicating that manganese anta
79 abnormalities, ranging from a characteristic chlorosis in leaves to a necrosis and large inhibition o
80 s germination and leads to rapid wilting and chlorosis in mature plants.
81 on of ascorbate occurred before the onset of chlorosis in Mn-stressed plants, especially in cv ZPV-29
82 tors (14 of 63 tested) induced cell death or chlorosis in N. benthamiana.
83  molecular mechanisms that may contribute to chlorosis in plants when exposed to metals.
84 diated Pst resistance is accompanied by leaf chlorosis in Pst-infected regions, but the underlying me
85 in urban waters, has been observed to induce chlorosis in Raphidocelis subcapitata close to environme
86             The cpd33 mutants also exhibited chlorosis in the leaf blades, greatly diminished plant g
87 ing type III effectors; however, it promotes chlorosis in the model plant Nicotiana benthamiana in a
88 ic delivery of plant micronutrients to treat chlorosis in tomato plants and crop biofortification thr
89                                              Chlorosis incidence was associated to improvements in wi
90 S in transgenic plants also resulted in leaf chlorosis, increased light sensitivity, and dwarfism due
91 y metals mimics iron (Fe) deficiency-induced chlorosis, indicating a disturbance in Fe homeostasis.
92 expression lines are slightly chlorotic, and chlorosis is rescued by exogenous iron.
93 l response that occurs prior to the onset of chlorosis, namely the disconnection of the LHCI antenna
94 t not limited to severe dwarfing appearance, chlorosis, nearly complete reduction of internodes and a
95 tially complements rosette dwarfing and leaf chlorosis of irt1-2, as well as root-to-shoot Fe partiti
96 h eventually led to drooping, yellowing, and chlorosis of leaves.
97 Pierce's disease of grape, citrus variegated chlorosis, olive quick decline, and numerous leaf scorch
98 esponse and low sulfur levels activating the chlorosis or bleaching response.
99 en to yellow-green, a process referred to as chlorosis or bleaching.
100 ivity (approximately 90% or more), developed chlorosis or necrosis on some of their lower leaves.
101 nd development, like stunting, leaf curling, chlorosis, or necrosis, which we recognize as disease sy
102 enotype at 22 degrees C, it has a pronounced chlorosis phenotype at 8 degrees C that is correlated wi
103 the chlorosis phenotype, suggesting the leaf chlorosis phenotype is caused by a deficiency of Ni(2+)
104  with Ni(2+) or Co(2+) partially rescued the chlorosis phenotype, suggesting the leaf chlorosis pheno
105 omes within a QTL region for iron deficiency chlorosis resistance.
106                   The symptoms included leaf chlorosis, restriction of root elongation, and eventual
107                                              Chlorosis resulting from application of F. oxysporum cul
108 ra lesion (zl1) mutant characterized by leaf chlorosis, severe defects in chloroplast development, an
109 d Fe content in chloroplasts (1.2-1.5-fold), chlorosis, structural damage to chloroplasts and a high
110 ngs for soybean sudden death syndrome foliar chlorosis suggested that STAY-GREEN genes with loss-of-s
111 d to a severe reduction in growth and strong chlorosis symptoms.
112 ed delay of transition to flowering and mild chlorosis symptoms.
113  iscA and sufA mutant strains exhibited less chlorosis than the wild type.
114 nd the double mutant (k1 k3) displayed rapid chlorosis upon high light stress.
115 l regions of the bipartite genome of Lettuce chlorosis virus (LCV), a member in the genus Crinivirus
116                          Infection by tomato chlorosis virus (ToCV) inhibited ABA content significant
117                                         Host chlorosis was associated with virulence, whereas necroti
118                                 This induced chlorosis was dependent on ENHANCED DISEASE RESISTANCE1,
119  IF response was retained in NahG leaves and chlorosis was more pronounced than in the wild-type.
120      Anthocyanin accumulation, stunting, and chlorosis were common symptoms.
121 eir ability for reduction of iron deficiency chlorosis were explored.
122                      Reduced development and chlorosis were observed for plants exposed to highly neg
123  necrosis (the D192K mutant), or an atypical chlorosis with necrotic flecking (the L194A mutant).
124 20 results in extensive necrosis and limited chlorosis within 5-6 days post-inoculation (d.p.i.), whi
125 bserved two distinct syndromes: one included chlorosis ('yellows-fragariae') and the other did not ('

 
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