ライフサイエンスコーパス: cholesterol sulfate

1 The third composition consisted of no cholesterol sulfate.

2 nd fatty acids (15%) with varying amounts of cholesterol sulfate (0-15%) and cholesterol (15-30%).

3 third component of the migratory pheromone), cholesterol sulfate (6), and squalamine (8)] and model c

4 Cholesterol sulfate, a liver X receptor (LXR) antagonist

5 change in spatial distribution of the sterol cholesterol sulfate, a membrane stabilizing lipid.

6 Here, we show that cholesterol sulfate, a molecule present in relatively hi

7 Cholesterol sulfate, a SC membrane lipid, is synthesized

8 ion in cholesterol, we assessed here whether cholesterol sulfate accumulation and/or cholesterol defi

9 Together, these results suggest that cholesterol sulfate accumulation rather than cholesterol

10 d further suggesting a cellular location for cholesterol sulfate action.

11 ide, and steroid sulfatase, which desulfates cholesterol sulfate, also increased with air exposure.

12 In agreement, topical skin application of cholesterol sulfate, an activator of PKCeta, significant

13 first identify a highly specific metabolite, cholesterol sulfate, an endogenous steroid.

14 olesterol sulfate and 25% cholesterol or 15% cholesterol sulfate and 15% cholesterol.

15 e lipid interaction than LUVs with either 5% cholesterol sulfate and 25% cholesterol or 15% cholester

16 We found that the LUVs with no cholesterol sulfate and 30% cholesterol exhibited a more

17 rongly inhibit it (coprostanol, androstenol, cholesterol sulfate, and 4-cholestenone).

18 er, with only one of two leaflets containing cholesterol sulfate, and stabilization of the external l

19 Cholesterol sulfate appears to combine the advantages of

20 tial distribution and relative abundances of cholesterol sulfate are reported and correlated with the

21 studies of Adx binding to substrate-free and cholesterol-sulfate-bound CYP46A1 revealed changes in th

22 amide proton exchange in substrate-free and cholesterol-sulfate-bound P450.

23 Doping DMPC/DHPC bicelles with cholesterol sulfate broadens the temperature range over

24 yosis display not only a 10-fold increase in cholesterol sulfate, but also a 50% reduction in cholest

25 Mincle activation by cholesterol sulfate causes the secretion of a range of p

26 exhibited reduced gastrointestinal levels of cholesterol sulfate (Ch-S) compared with wild-type B. th

27 precursors, plant sterols, some oxysterols, cholesterol sulfate, cholesterol acetate, and 5-alpha-ch

28 ft phosphatidylethanolamine PE(O-16:1/18:2))/cholesterol sulfate common to both mucosal maturation an

29 Bt_0416 showed altered host cholesterol and cholesterol sulfate compared with wild-type mice, identi

30 es with this being the area with the highest cholesterol sulfate content suggesting that the physiolo

31 is study was to examine changes in levels of cholesterol sulfate (CS) and activity of its biosyntheti

32 Here we show that cholesterol sulfate (CS), a naturally occurring analog o

33 ol% sterol when cholesterol is replaced with cholesterol sulfate (CS).

34 ulfoconjugation of cholesterol to synthesize cholesterol sulfate (CS).

35 sequence of SSase deficiency, its substrate, cholesterol sulfate (CSO4), accumulates in the epidermis

36 These studies suggest that induction of the cholesterol sulfate cycle enzymes during SC ontogenesis

37 Cholesterol and cholesterol sulfate have been suggested to be RORalpha l

38 The inclusion of 5-15% cholesterol sulfate helps to prevent the collapse of fus

39 we identify that cholesterol is converted to cholesterol sulfate in a Bacteroides-specific manner via

40 Additionally, 88% of cholesterol sulfate in NHEK was membrane-associated furt

41 The presence of cholesterol sulfate in prostate tissues might serve as a

42 Additionally, cholesterol sulfate is a constituent of human platelets,

43 Cholesterol sulfate is a highly amphipathic molecule tha

44 Cholesterol sulfate is a multifunctional sterol metaboli

45 nt suggesting that the physiologic action of cholesterol sulfate is likely carried out in this region

46 Recent evidence suggests that cholesterol sulfate may be an important second messenger

47 Profiles obtained from lamellae with cholesterol sulfate partially substituted for cholestero

48 Yet, topical cholesterol sulfate produced both a barrier abnormality

49 that was reflected in enzymatic activity and cholesterol sulfate production.

50 e for the synthesis of glucosylceramides and cholesterol sulfate, respectively, were accelerated furt

51 nflammatory mediators, and s.c. injection of cholesterol sulfate results in a Mincle-mediated inducti

52 ializing skin is characterized by diminished cholesterol sulfate signal along the stratum corneum tow

53 Cholesterol sulfate, the most important sterol sulfate i

54 Finally, addition of exogenous cholesterol sulfate to explants in vitro did not acceler

55 Despite the apparent importance of cholesterol sulfate-to-cholesterol processing for normal

56 esults demonstrate both a potential role for cholesterol sulfate-to-cholesterol processing in normal

57 Cholesterol sulfate was identified as a differentiating

58 , sphingomyelin, lysophosphatidylcholine and cholesterol sulfate were higher in the confirmed virus i