1 ssion of retinal responses via activation by cholinergic agents.

2 contributes to the antinociceptive action of cholinergic agents.

3 ng the antinociceptive action of intrathecal cholinergic agents.

4 dent or voltage-independent calcium channels.Cholinergic agents (ACh, nicotine, muscarine, bethanecho

5 aneous retinal waves of action potentials by cholinergic agents altered the subsequent eye-specific l

6 nergic agent, we verified that delivery of a cholinergic agent by means of topical application can le

7 served that nicotine (2 mg/kg) and the novel cholinergic agent CAP55 (12 mg/kg) inhibit endothelial c

8                 Nitric oxide (NO) donors and cholinergic agents decrease intraocular pressure, in par

9                             In contrast, the cholinergic agent donepezil did not measurably affect ar

10 s have suggested the potential usefulness of cholinergic agents for ADHD.

11                                        Since cholinergic agents have already demonstrated antiacantho

12                          Treatment with this cholinergic agent improved heart remodeling manifested b

13 in the antinociceptive action of intrathecal cholinergic agents in normal rats and in a rat model of

14 lgesic mechanisms and the site of actions of cholinergic agents in the spinal cord are not fully unde

15     However, the mechanisms of the action of cholinergic agents in the spinal cord are not fully unde

16                  There is also evidence that cholinergic agents may have disease-modifying effects.

17 several types of cells, we hypothesized that cholinergic agents might have similar effects on keratin

18 rect inhibitory effects of acetylcholine and cholinergic agents on tumor necrosis factor (TNF)-induce

19                                     For each cholinergic agent, we verified that delivery of a cholin

20 oM nicotine) and impermeant (5 mM carbachol) cholinergic agents were used to define specific [3H]epib