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1 lin is expressed by immature osteoblasts and chondroblasts.
2  into osteoblasts, adipocytes, myoblasts, or chondroblasts.
3 n to myoblasts, adipocytes, osteoblasts, and chondroblasts.
4                                        These chondroblasts and myoblasts lie close together within ea
5 ms of derived connective tissue cells (e.g., chondroblasts and osteoblasts).
6  profile of MPCs induced to differentiate to chondroblasts and osteoblasts, significant differences i
7               These cells are specified into chondroblasts before ear unfolding and then undergo exte
8 evelopment of a highly proliferating pool of chondroblasts between the epiphyses and metaphyses of fu
9 ss of BMP5 does not prevent specification of chondroblasts, but does impair chondroblast proliferatio
10 duction of c-Fos in vitro in osteoblasts and chondroblasts caused an increase in Fgfr1 RNA and FGFR1
11 everal molecular markers required for normal chondroblast differentiation (&agr; 1(II) collagen, Fgfr
12 tion factors that act redundantly to promote chondroblast differentiation in all cartilages of the mo
13 ption factors that redundantly promote early chondroblast differentiation.
14 ha in limb development and the regulation of chondroblast differentiation.
15 hypertrophic chondrocyte markers, epiphyseal chondroblasts ectopically activate hypertrophic chondroc
16 Sox6 are thus redundant, potent enhancers of chondroblast functions, thereby essential for endochondr
17  apoptosis, and decreased differentiation of chondroblasts in Meckel's cartilage.
18                                        While chondroblasts in the center of cartilages ultimately act
19 these cells with the ability to give rise to chondroblasts in vivo.
20 Ps) induce differentiation of osteoblast and chondroblast lineage cells from uncommitted mesenchymal
21 any other genes occurs within 3 days, before chondroblast phenotypic genes are detectable.
22                     In these double mutants, chondroblasts poorly differentiate.
23 on of growth plate columnar zones by keeping chondroblasts proliferating and by delaying chondrocyte
24                                     Finally, chondroblast proliferation remains impaired in the adult
25 cification of chondroblasts, but does impair chondroblast proliferation.
26 fferentiation was at the late-blastema/early-chondroblast stage.
27 of other cell types such as preadipocytes or chondroblasts, the effect of hypoxia on myogenesis is in
28           In contrast, adhesion of auricular chondroblasts to fibrillin-1 was only partially inhibite
29                     While differentiation of chondroblasts to pre-hypertrophic chondrocytes (IHH expr