ライフサイエンスコーパス: chromatin remodeling

1 Repair of UV-induced DNA damage requires chromatin remodeling.

2 immunity, inflammation, protein sorting, and chromatin remodeling.

3 ruit enzymes responsible for DNA readout and chromatin remodeling.

4 activated arginase 1 expression by promoting chromatin remodeling.

5 dentified candidate driver genes involved in chromatin remodeling.

6 pigenetic processes like DNA methylation and chromatin remodeling.

7 on each face of the nucleosome that affects chromatin remodeling.

8 a direct protein interaction independent of chromatin remodeling.

9 activation (ZGA) occurs, accompanied by fast chromatin remodeling.

10 ct with CSB and greatly enhance CSB-mediated chromatin remodeling.

11 low adaptation to new conditions by delaying chromatin remodeling.

12 ociated proteins, including many involved in chromatin remodeling.

13 contribute to transcriptional regulation and chromatin remodeling.

14 nvolved in antifibrotic activity in skin and chromatin remodeling.

15 through promoting glycolysis and subsequent chromatin remodeling.

16 s the key link between T cell activation and chromatin remodeling.

17 omere identity during transcription-mediated chromatin remodeling.

18 ed induction of AP-1 and a failure of proper chromatin remodeling.

19 ) +2, supporting the 'twist defect' model of chromatin remodeling.

20 ole in the regulation of gene expression and chromatin remodeling.

21 es involved in covalent histone modification/chromatin remodeling (34.5%; MEN1, ARID1A, KMT2C, and KM

22 Chromatin remodeling accompanies differentiation, howeve

23 These factors are primarily involved in chromatin remodeling, actin assembly, mRNA 3'-end proces

24 bly of an epigenetic silencing complex, with chromatin remodeling activities that repress transcripti

25 hat HvCEN controlled transcripts involved in chromatin remodeling activities, cytokinin and cell cycl

26 iated mutations in either causing attenuated chromatin remodeling activities.

27 Chromatin-remodeling activities directed by SWI/SNF2 sup

28 te that both motifs stimulate the ATPase and chromatin-remodeling activities upon binding of BRG1 to

29 Many Snf2 enzymes possess chromatin-remodeling activity, requiring a functional AT

30 rofound changes in DNA methylation state and chromatin remodeling, affecting HNF4alpha-dependent gene

31 many sites in nucleosomes without the aid of chromatin-remodeling agents and without irreversibly dis

32 ltiple cellular functions, and they regulate chromatin remodeling, along with transcriptional and pos

33 is improved by small molecules that promote chromatin remodeling and activate the TGFbeta, Shh and W

34 t featuring dysregulation of RNA processing, chromatin remodeling and cell-cycle genes enriched for p

35 gies that have been implicated in human CHD: chromatin remodeling and cilia function.

36 The NuRD chromatin remodeling and deacetylation complex, which in

37 in a context-dependent manner directs local chromatin remodeling and enhancer formation.

38 vement in leukemogenesis by upregulation via chromatin remodeling and enhancer hijacking.

39 of myogenic genes by promoting ATP-dependent chromatin remodeling and formation of transcription-comp

40 s methyl and acetyl), which are critical for chromatin remodeling and gene expression, particularly d

41 nucleosomal response, and acts as a brake on chromatin remodeling and gene regulation.

42 ers SWItch/Sucrose Non-Fermentable (SWI/SNF) chromatin remodeling and global methylation patterns tha

43 n NUPR1 is a multifunctional IDP involved in chromatin remodeling and in the development and progress

44 Hence, glucose fluctuations contribute to chromatin remodeling and may explain persistent vascular

45 ion and localization are tightly linked with chromatin remodeling and might be crucial for transition

46 R, an HDACi, induces histone acetylation and chromatin remodeling and modulates host and HIV gene exp

47 d impairment of nuclear/cytoplasm transport, chromatin remodeling and nuclear lamina formation.

48 rans-activator to bridge innate sensing with chromatin remodeling and potentiate robust antiviral res

49 ons of coding and non-coding mutations, with chromatin remodeling and proliferation pathways altered

50 hanol exposure rescued CREB activation, Arc, chromatin remodeling and Rac1 expression, spatial memory

51 Complete loss of chromatin remodeling and related proteins such as TP2, P

52 eraction with CHD9 and BRM, thereby blocking chromatin remodeling and robust GR binding at GR-binding

53 regulatory elements of these genes result in chromatin remodeling and transcription activation.

54 biophysical basis for methylation-dependent chromatin remodeling and transcription regulation by NuR

55 ed by adenosine triphosphate (ATP)-dependent chromatin remodeling and transcription.

56 n, BRD9, was previously reported to regulate chromatin remodeling and transcription.

57 tablished important regulatory mechanism for chromatin remodeling and transcription.

58 and caused histone acetylation by p300/CBP, chromatin remodeling, and cohesin loading to establish M

59 dysfunction, mitochondrial oxidative stress, chromatin remodeling, and genomic instability.

60 and identified genes involved in metabolism, chromatin remodeling, and neurogenesis-related functions

61 uding DNA methylation, histone modification, chromatin remodeling, and noncoding RNA expression, and

62 n, RNA processing and transport, DNA repair, chromatin remodeling, and nucleocytoplasmic transport.

63 DNA methylation-mediated gene activation and chromatin remodeling, and provides a general framework t

64 lation by SNF5 is separable from its role in chromatin remodeling, and that reintroduction of SNF5 in

65 involved in gene regulation, DNA repair and chromatin remodeling, and though it is known to be essen

66 AMP activates EPAC signaling, CTCF-dependent chromatin remodeling, and transcriptional activation of

67 d in regulating gene expression, DNA repair, chromatin remodeling, apoptosis, and cell proliferation;

68 of the shortest mlonRNA (mlonRNA-c) induces chromatin remodeling around a transcription factor-bindi

69 immediate early genes (IEGs) and longer-term chromatin remodeling around secondary response genes (SR

70 ell lineages, loss of proliferative control, chromatin remodeling as well as extensive morphological

71 that are regulated by SOX10 and MITF and for chromatin remodeling at distal and proximal regulatory s

72 9 and BRM were required for GR occupancy and chromatin remodeling at GR-binding regions associated wi

73 y of Mef2c and Tbx5 to independently promote chromatin remodeling at previously inaccessible sites.

74 x2 and Brg1 occupancy occurs coincident with chromatin remodeling at some of these loci.

75 s accompanied by increased transcription and chromatin remodeling at specific endogenous retroviral s

76 whereby T-BET serves to promote appropriate chromatin remodeling at specific gene loci that underpin

77 ng auxin perception, which revealed that the chromatin remodeling ATPase BRAHMA is implicated in auxi

78 nvariable core subunits of the ATP-dependent chromatin remodeling BAF (BRG1-associated factor) comple

79 Multiple subunits of the chromatin remodeling BAF complex, including ARID1A, play

80 the gene SMARCB1, a conserved subunit of the chromatin remodeling BAF complex, which has known contri

81 transcallosal dysconnectivity via repressive chromatin remodeling by the SETDB1 repressor complex.

82 nd gene networks, our study demonstrates how chromatin remodeling can dictate gene expression distrib

83 mage site, initiation of signaling cascades, chromatin remodeling, cell-cycle checkpoint activation,

84 ings identify a direct link between aberrant chromatin remodeling, cellular senescence, and accelerat

85 Upon recruitment via PARP1-triggered chromatin remodeling, CHD7 stimulates further chromatin

86 ns were predominantly in genes that regulate chromatin remodeling, chromosome alignment, and stabilit

87 Here we show that Mot1, Ino80 chromatin remodeling complex (Ino80C), and NC2 co-locali

88 anied by release of the repressive nucleolar chromatin remodeling complex (NoRC) from rDNA, together

89 one variant H2A.Z by the SWI2/SNF2-Related 1 chromatin remodeling complex (SWR1-C) is important for g

90 Subunits of the SWI/SNF chromatin remodeling complex are frequently mutated in h

91 romodomain-containing subunit of the SWI/SNF chromatin remodeling complex BAF.

92 ral genes encoding components of the SWI/SNF chromatin remodeling complex cause neurodevelopmental di

93 associated factors-containing complex (PBAF) chromatin remodeling complex component BRG1-associated f

94 well as other members of the LSD1-containing chromatin remodeling complex CoREST, is rapidly polyubiq

95 Mutations in the chromatin remodeling complex could thus mitigate the pro

96 The SWItch/Sucrose non-fermentable (SWI/SNF) chromatin remodeling complex has also emerged as a criti

97 The SWI/SNF chromatin remodeling complex has been found mutated in a

98 TF NANOG is highly dependent on the SWI/SNF chromatin remodeling complex in individual blastocysts b

99 the role of the mammalian SWI/SNF (mSWI/SNF) chromatin remodeling complex in NEPC.

100 Here we show that the INO80 chromatin remodeling complex is required for oncogenic t

101 cleosome remodeling and deacetylation (NuRD) chromatin remodeling complex opposes this transcriptiona

102 The BAF (SWI/SNF) chromatin remodeling complex plays a crucial role in mod

103 d properties potentially by interacting with chromatin remodeling complex PRC2 and downregulation of

104 , the Arabidopsis homolog of the yeast INO80 chromatin remodeling complex subunit IES6 (INO EIGHTY SU

105 d the phosphorylated FgSR interacts with the chromatin remodeling complex SWI/SNF at the target genes

106 ors is BRG1, the major ATPase subunit of the chromatin remodeling complex SWI/SNF, establishing a rel

107 ew set of genes encoding subunits of the BAF chromatin remodeling complex that exhibited Ras-mediated

108 which EBF2 cooperates with a tissue-specific chromatin remodeling complex to activate brown fat ident

109 acts with and recruits a tissue-specific BAF chromatin remodeling complex to brown fat gene enhancers

110 l enhancers and recruiting the SWI/SNF (BAF) chromatin remodeling complex to establish accessible chr

111 l deletion of Swi3, a subunit of the SWI/SNF chromatin remodeling complex, and partial loss of functi

112 utations in ARID1A, a subunit of the SWI/SNF chromatin remodeling complex, are the most common altera

113 that Arid1a, a key component of the SWI/SNF chromatin remodeling complex, controls liver regeneratio

114 ched association with components of the NuRD chromatin remodeling complex, especially RBBP4.

115 ch/sucrose non-fermentable (mSWI/SNF or BAF) chromatin remodeling complex, including its core catalyt

116 rs with mutations in subunits of the SWI/SNF chromatin remodeling complex, inclusive of most epitheli

117 ADP-ribosylation of BAF170, a subunit of BAF chromatin remodeling complex, is critical for activation

118 Atxn7, a subunit of SAGA chromatin remodeling complex, is subject to polyglutamin

119 of SMARCB1, encoding a member of the SWI/SNF chromatin remodeling complex, is the hallmark genetic ab

120 n BRG1, the catalytic subunit of the SWI/SNF chromatin remodeling complex, results in a meiotic arres

121 principle, the nucleosome preference of the chromatin remodeling complex, RSC.

122 nd pathways, including HMGB1 and the SWI/SNF chromatin remodeling complex, that are SARS lineage and

123 Smarca4 -encoded BRG1 subunit of the SWI/SNF chromatin remodeling complex, we employed in vitro model

124 (Pygo) and the Osa/ARID1A subunit of the BAF chromatin remodeling complex, which could synergize with

125 , 2 missense) in the BICRA (BRD4 interacting chromatin remodeling complex-associated protein) gene, a

126 ge (>300 kD) multidomain subunit of the NURF chromatin remodeling complex.

127 hromatin in budding yeast depends on the RSC chromatin remodeling complex.

128 wo mutually exclusive ATPases of the SWI/SNF chromatin remodeling complex.

129 y interacts with the Imitation SWItch (ISWI) chromatin remodeling complex.

130 the switch-sucrose non-fermentable (SWI-SNF) chromatin remodeling complex.

131 the disease-specific recruitment of a major chromatin remodeling complex.

132 of the polymorphic BRG/BRM-associated factor chromatin remodeling complex.

133 nteraction of PRDM3 and PRDM16 with the NuRD chromatin remodeling complex.

134 of the BRAF-histone deacetylase (HDAC) (BHC) chromatin-remodeling complex (LSD1, RCOR1, HMG20A, HMG20

135 connection between mutations in the SWI/SNF chromatin-remodeling complex and the tumor suppressor ge

136 nsin D interacts with a putative member of a chromatin-remodeling complex during development.

137 This decondensation is mediated through chromatin-remodeling complex PBAP, as PBAP is both robus

138 minichromosome maintenance (MCM) protein and chromatin-remodeling complex SMARCB1 and SMARCC2 to be L

139 1, a subunit of the evolutionarily conserved chromatin-remodeling complex SWI/SNF.

140 of BRG1-associated factors (BAF), an SWI/SNF chromatin-remodeling complex with known repressive funct

141 re known to alter the composition of the BAF chromatin-remodeling complex, causing ejection and degra

142 lta) forms of Swi1, a subunit of the SWI/SNF chromatin-remodeling complex, confer dramatically distin

143 ed by BRG1, an ATPase subunit of the SWI/SNF chromatin-remodeling complex.

144 eterminant is Swi1, a subunit of the SWI/SNF chromatin-remodeling complex.

145 modeling factor (NURF), a member of the ISWI chromatin-remodeling complex.

146 phosites in SMARCC2, a member of the SWI/SNF chromatin-remodeling complex.

147 ammalian SWI/SNF complexes are multi-subunit chromatin remodeling complexes associated with an ATPase

148 Among them, the ATP-dependent chromatin remodeling complexes control the chromatin arc

149 In addition, chromatin remodeling complexes have an impact on the abu

150 an switch/sucrose non-fermentable (mSWI/SNF) chromatin remodeling complexes have been widely implicat

151 Chromatin remodeling complexes instruct cellular differe

152 gulatory switch for other PTMs, and connects chromatin remodeling complexes into gene transcription a

153 ammalian SWI/SNF complexes are ATP-dependent chromatin remodeling complexes that regulate genomic arc

154 nucleosome arrays, which are repositioned by chromatin remodeling complexes to control DNA accessibil

155 et is associated with its failure to recruit chromatin remodeling complexes to the Ifng gene promoter

156 gh interactions with RNA-binding proteins in chromatin remodeling complexes, and modulate dynamic and

157 SWI/SNF-family chromatin remodeling complexes, such as S. cerevisiae RS

158 recently identified subunit of SWI/SNF(BAF) chromatin remodeling complexes, yet its function is poor

159 ved from human DNA repair proteins and yeast chromatin remodeling complexes.

160 r, there is no known link between MUC1-C and chromatin remodeling complexes.

161 ARCA4 and SMARCA2, key components of SWI/SNF chromatin remodeling complexes.

162 ranscription factors that recruit activating chromatin remodeling complexes.

163 rum of human disorders caused by ablation of chromatin remodeling complexes.

164 sine signaling kinases and components of the chromatin remodeling complexes.

165 onfigurations such as histone chaperones and chromatin remodeling complexes.

166 or members of the SWI/SNF (or BAF) family of chromatin remodeling complexes.

167 ian Switch/Sucrose-Nonfermentable (mSWI/SNF) chromatin-remodeling complexes (also called BAF complexe

168 Genes encoding subunits of SWI/SNF (BAF) chromatin-remodeling complexes are collectively mutated

169 Switch/sucrose-nonfermentable (SWI/SNF) chromatin-remodeling complexes are critical regulators o

170 The Swi/Snf (PBAF and BAF) chromatin-remodeling complexes contribute to DNA damage-

171 ttractive strategy to target mutated SWI/SNF chromatin-remodeling complexes in cancer.

172 Eukaryotes encode a plethora of chromatin-remodeling complexes, histone-modification enz

173 e show that BAF60a, a subunit of the SWI/SNF chromatin-remodeling complexes, serves an indispensable

174 eiosis has been extensively studied, yet how chromatin remodeling contributes to this process is larg

175 are controlled by the ATP-dependent Swi/Snf chromatin remodeling coregulatory complex that physicall

176 this spectrum overlaps with that of several chromatin-remodeling developmental disorders.

177 These genes are involved in chromatin remodeling, DNA binding, cell survival, and ce

178 hylation, histone acetylation/deacetylation, chromatin remodeling, DNA methylation, and microRNAs in

179 igenetic regulation, including long-distance chromatin remodeling, DNA methylation, posttranslational

180 MORC2 encodes an ATPase that plays a role in chromatin remodeling, DNA repair, and transcriptional re

181 F1) is a histone H3/H4 chaperone involved in chromatin remodeling during cell division, which we have

182 cell stage-specific roles for CHD7-mediated chromatin remodeling during cell lineage acquisition.

183 e for developmental mechanisms, particularly chromatin remodeling, during learning or in response to

184 Here the authors show that the chromatin remodeling enzyme ATRX is a regulator of TIS a

185 Here, we demonstrate that the chromatin remodeling enzyme ATRX is required for therapy

186 , we report that conditional knockout of the chromatin remodeling enzyme Chd4 in granule neurons of t

187 The chromatin remodeling enzyme CHD4 is a component of the N

188 i2/snif2-related 1 (SWR1), the ATP-dependent chromatin remodeling enzyme that deposits H2A.Z into chr

189 bunit of the mammalian SWI/SNF ATP-dependent chromatin remodeling enzyme.

190 Human ALC1 is an oncogene-encoded chromatin-remodeling enzyme required for DNA repair that

191 stone variant isoforms and the action of the chromatin-remodeling enzyme SRCAP.

192 m, exploiting gene-specific requirements for chromatin remodeling enzymes to selectively influence DN

193 leosome displacement, exchange or removal by chromatin remodeling enzymes.

194 ation factor TFIIS, and that NPH-I resembles chromatin remodeling enzymes.

195 ATP-dependent chromatin-remodeling enzymes control accessibility, nucl

196 riptional regulation is modulated in part by chromatin-remodeling enzymes that control gene accessibi

197 a catalytic subunit of the mammalian SWI/SNF chromatin-remodeling enzymes, is required for both myobl

198 bout the function of the conserved motifs in chromatin-remodeling enzymes.

199 inhibition serving as signaling platform for chromatin-remodeling enzymes.

200 were characterized by dramatic and congruent chromatin remodeling events affecting multiple neighbori

201 g their respective DNA binding or downstream chromatin-remodeling events have shown promise in precli

202 nges in the histone modification profile and chromatin-remodeling events leading to Sp7 gene expressi

203 Treg-specific deletion of the chromatin remodeling factor Brg1 impairs Treg cell activ

204 t also reveal the mechanism underlying how a chromatin remodeling factor can regulate oocyte meiosis.

205 is response and shows how a highly conserved chromatin remodeling factor has a distinct role in anti-

206 is activated to reduce proline levels by the chromatin remodeling factor lymphoid-specific helicase (

207 Chromatin remodeling factor metastatic tumor protein 1 (

208 egulator(s) and relationship with the master chromatin remodeling factor MTA1, continues to be poorly

209 Here, we demonstrate that the ATP-dependent chromatin remodeling factor Snf2h (also known as Smarca5

210 Smarcad1 is a conserved chromatin remodeling factor with a poorly understood tis

211 CHD7 encodes an ATP-dependent chromatin remodeling factor.

212 ethyltransferase 1 (CARM1) methylates Pontin chromatin-remodeling factor under glucose starvation, an

213 nd tyrosine kinases and their interplay with chromatin remodeling factors in cancer cells.

214 ATP-dependent chromatin remodeling factors of SWI/SNF2 family includin

215 ally, NAP1 histone chaperones, ATP-dependent chromatin remodeling factors, and some histone-modifying

216 ny and carries motifs for binding of certain chromatin remodeling factors, including insulator protei

217 Mutations in specific chromatin-remodeling factors appear to be key determinan

218 ration events, which inappropriately recruit chromatin-remodeling factors to elicit the aberrant tran

219 ial response to IFNalpha is characterized by chromatin remodeling, followed by changes in transcripti

220 inactivating mutations of ARID1A, a SWI/SNF chromatin remodeling gene, are prevalent in human endome

221 mor suppressor SMARCA4 (BRG1), a key SWI/SNF chromatin remodeling gene, is frequently inactivated in

222 Alterations in chromatin remodeling genes have been increasingly implic

223 Chromatin remodeling genes were frequently mutated, wher

224 ion from mutations affecting cilia genes and chromatin remodeling genes; however, the mechanism(s) co

225 ifferentiation involves rapid but reversible chromatin remodeling, glial fibrillary acidic protein (G

226 Gene mutations in the chromatin remodeling groups were relatively more frequen

227 l differentiation and myelination depends on chromatin remodeling, histone acetylation, and methylati

228 that mlonRNA-initiation effectively induces chromatin remodeling in a limited distance within 290 bp

229 ation advance a mechanistic understanding of chromatin remodeling in disease states.

230 es renal inflammation by several mechanisms: chromatin remodeling in promoter regions of specific gen

231 s in mice is accompanied by lineage-specific chromatin remodeling in regions enriched with binding mo

232 The molecular basis of chromatin remodeling in response to IL-4 stimulation in

233 Collectively, our findings define a role for chromatin remodeling in the control of genome architectu

234 expected dichotomous role of BAF60a-mediated chromatin remodeling in transcriptional control of brown

235 Stimulation caused widespread chromatin remodeling, including response elements shared

236 process of lung carcinogenesis is linked to chromatin remodeling, inflammation, and tumor microenvir

237 cleosomes at these sites, we demonstrate how chromatin remodeling is initiated during GG-NER.

238 In addition, chromatin remodeling is required to generate a nucleosom

239 3, 5.7% vs. 2.3%; NOTCH4, 3.6% vs. 0.6%) and chromatin-remodeling (KDM6A, 15.2% vs. 3.4%; KMT2C/MLL3,

240 lly, we provide evidence for PRDM9-dependent chromatin remodeling leading to increased accessibility

241 Dysregulation of chromatin remodeling leads to aberrant cell proliferatio

242 This chromatin remodeling likely facilitates de novo methylat

243 ore the connections between circadian clock, chromatin remodeling, lncRNAs, and CRFH and how these im

244 RNA silencing, epigenetic modification, and chromatin remodeling machineries.

245 The molecular cues that recruit the chromatin remodeling machinery are not well characterize

246 velopment and implicate another component of chromatin remodeling machinery in brain disease.

247 Our study sheds light onto the role of the chromatin remodeling machinery in intestinal epithelial

248 ith variations in ACTL6B, a component of the chromatin remodeling machinery including the BAF complex

249 n concert with the general transcription and chromatin remodeling machinery to regulate gene expressi

250 The chromatin-remodeling machinery that governs genome acces

251 While chromatin remodeling mediated by post-translational modi

252 enome is modulated by the ATP-driven SWI/SNF chromatin remodeling multiprotein complexes BAF (BRG1/BR

253 d within 48 h after MS1 induction; out of 14 chromatin-remodeling mutants studied, expression of clus

254 cording to endogenous yeast DNA sequence and chromatin-remodeling network, as judged by a yeast-like

255 stinct H1 subtypes may mediate the extensive chromatin remodeling occurring during epigenetic reprogr

256 tch1 by BMPR2, to coordinate metabolism with chromatin remodeling of genes that enable EC regeneratio

257 strate that Pax3 molecular function involves chromatin remodeling of its bound elements through an in

258 own transcription by limiting BRD4-mediated chromatin remodeling of its locus.

259 Cell differentiation is accompanied by chromatin remodeling of specific loci to permanently sil

260 -specific enhancer that was found to mediate chromatin remodeling of the EDC in an AP-1 dependent man

261 nd sirtuin 1 (SIRT1), which were involved in chromatin remodeling of the RARbeta and nerve growth fac

262 idate risk genes with predicted functions in chromatin remodeling or neurodevelopment, including ACTL

263 Four of these genes encode members of the chromatin remodeling Osa-containing Brahma (BAP) complex

264 ers of BRD7 and BRD9, which form part of the chromatin remodeling PBAF and BAF complexes, respectivel

265 enes involved in Wnt/beta-catenin signaling, chromatin remodeling, PIK3CA/AKT/mTOR signaling, and ang

266 Chromatin remodeling plays important roles in gene regul

267 ied include genes involved in transcription, chromatin remodeling, post-translational modifications,

268 idues to regulate cellular processes such as chromatin remodeling, pre-mRNA splicing, and signal tran

269 influences the successful completion of the chromatin remodeling processes that facilitate homologou

270 Thus, rewiring of the HR network, coupled to chromatin remodeling, promotes context-specific control

271 Mechanistically, ZNF367 interacted with chromatin remodeling protein BRG1 and transcriptionally

272 tion, is caused by haploinsufficiency of the chromatin remodeling protein gene CHD7 (Chromodomain hel

273 s growth inhibitory in vivo In addition, the chromatin remodeling protein INO80C was identified as a

274 Mutations of the chromatin remodeling protein Lsh/HELLS can cause the hum

275 nriched expression was CHD2, which encodes a chromatin remodeling protein mutated to cause human epil

276 tes degradation of maternal RNA encoding the chromatin remodeling protein Smarca2, and that clearance

277 stablish an in vivo function of CHD Type III chromatin remodeling proteins in this process, and revea

278 How chromatin remodeling proteins influence the activity of

279 Chromatin remodeling proteins of the chromodomain DNA-bi

280 here SIRT6 mono-ADP-ribosylates and recruits chromatin remodeling proteins to mediate the formation o

281 Although LSH belongs to a family of chromatin remodeling proteins, it remains unknown how LS

282 - and adult-ventricular(V) CM, two candidate chromatin remodeling proteins, SMYD1 and SMARCD1 were fo

283 Chromatin remodeling provides the foundation for the cel

284 ngs linking REST to differentiation-specific chromatin remodeling, PTCH1 silencing, and AKT activatio

285 Their chromatin-remodeling rates were impaired accordingly, bu

286 ong intergenic non-coding RNAs (lncRNAs) and chromatin remodeling remains unclear.

287 ated haploinsufficiency of ARID1B, a SWI/SNF chromatin-remodeling subunit, in short stature, autism s

288 her SWI/SNF (SWItch/Sucrose Non-Fermentable) chromatin remodeling subunits are also observed in endom

289 The multi-subunit chromatin-remodeling SWI/SNF complex determines gene exp

290 te that interplay of p300-HDAC2-Sin3A in the chromatin remodeling system is involved in HIF-1alpha de

291 r by fine-tuning the activity of other major chromatin remodeling TFs such as TCF1.

292 uncovers the different steps of CSB-mediated chromatin remodeling that can be regulated by NAP1L1.

293 genes; however, the mechanism(s) connecting chromatin remodeling to CHD is unknown.

294 loops and require histone modifications and chromatin remodeling to ensure appropriate timing and am

295 n is a result of a complex interplay between chromatin remodeling, transcription factors, and signali

296 on factors to shared targets mediated stable chromatin remodeling upon T cell activation.

297 n that can be relieved through ATP-dependent chromatin remodeling via complexes such as the switch-su

298 Previously, SWI/SNF chromatin remodeling was found to define the kinetics an

299 T cell activation induced major chromatin remodeling, while the presence of cytokines fi

300 ing the components and mechanisms connecting chromatin remodeling with transcriptional regulation by