ライフサイエンスコーパス: chromosome 10

1 , GPLD1 on chromosome 6, and JMJD1C-REEP3 on chromosome 10).

2 utions (Swrl-1 on chromosome 1 and Swrl-4 on chromosome 10).

3 N (phosphatase and tensin homolog deleted on chromosome 10).

4 of phosphatase and tensin homolog deleted on chromosome 10.

5 s lack of COs in the entire proximal half of chromosome 10.

6 nner and the disease gene localizes to mouse chromosome 10.

7 ation and SNPs): 6 on chromosome 18 and 1 on chromosome 10.

8 carcinoma of the lung to a 15.7 Mb region on chromosome 10.

9 on chromosome 1, and rs1509957 near EGR2 on chromosome 10.

10 n SDF-1 gene spans over 88 kilobase-pairs on chromosome 10.

11 arm of wheat consensus chromosome 1 and rice chromosome 10.

12 ibited growth defects and were aneuploid for chromosome 10.

13 le T-DNA insertion in the mutant, located on chromosome 10.

14 titative trait locus (QTL) located on murine chromosome 10.

15 lotype that shows partial homology to normal chromosome 10.

16 ains on 8q and 17q, and deletions on 17p and chromosome 10.

17 QTLs for thalamus volume were identified on chromosomes 10, 11 and 16.

18 Four regions on chromosomes 10, 11, 16, and 17 contained stable fruit As

19 The rs1619661 variant is on chromosome 10, 132 kilobase (kb) downstream from <em>CXC

20 Four additional regions on chromosomes 10, 15 and 16 showed suggestive association

21 enically conserved in three regions on mouse chromosomes 10, 16 and 17.

22 for the early to middle elongation stage on chromosome 10, 3 islands for the middle to late elongati

23 (phosphatase and tensin homologue deleted on chromosome 10), a known tumour suppressor, across tumour

24 N (phosphatase and tensin homolog deleted on chromosome 10), a recently discovered tumor suppressor g

25 N (phosphatase and tenson homolog deleted on chromosome 10), a tumor suppressor that antagonizes the

26 Horn type maps to chromosome 10, a location similar to that previously ide

27 it was possible to map and identify Hrml1 on chromosome 10, a locus responsible for modulating the HR

28 Loss of chromosome 10, a predominant genetic change, was associa

29 phosphatase and tensin homologue deleted on chromosome 10, a tumor suppressor protein that antagoniz

30 phosphatase and tensin homolog deleted from chromosome 10], a phosphatase and critical tumor suppres

31 Maize Abnormal chromosome 10 (Ab10) contains a classic meiotic drive sy

32 A maize chromosome variant called abnormal chromosome 10 (Ab10) converts knobs on chromosome arms i

33 Maize contains the abnormal chromosome 10 (Ab10) drive system that transforms typica

34 The meiotic drive system on maize abnormal chromosome 10 (Ab10) is contained within a terminal doma

35 nd PTEN (phosphatase and tensin homologue on chromosome 10) activities.

36 phosphatase and tensin homologue deleted on chromosome 10 activity.

37 N (phosphatase and tensin homolog deleted on chromosome 10) activity and transcription by palmitic ac

38 N (phosphatase and tensin homolog deleted on chromosome 10) activity was significantly elevated and R

39 hoblastic leukemia-1 (ALL-1)-fused gene from chromosome 10 (AF10), ZFP-1, or tumor suppressor Rb, to

40 se-1/phosphate and tensin homolog deleted on chromosome 10/Akt) and p53 pathways, which converge on t

41 ametric linkage score of 4.27 at 107.2 cM on chromosome 10, although it did not attain genome-wide si

42 a previously unnoticed gene cluster in mouse chromosome 10 and a number of new genes, including mamma

43 ent of single base changes between the human chromosome 10 and chimpanzee sequence revealed nonsense

44 H1, 1p/19q loss, EGFR amplification, loss of chromosome 10 and chromosome arm 10q, gain of chromosome

45 ich lies in close proximity to pten on human chromosome 10 and encodes a 20-kDa nuclear protein.

46 al brain tumors--often have both monosomy of chromosome 10 and gains of the epidermal growth factor r

47 -aminotransferase (ALT) (CPN1-ERLIN1-CHUK on chromosome 10 and PNPLA3-SAMM50 on chromosome 22), one l

48 The l2 locus was mapped to the long arm of chromosome 10 and positional cloning revealed the existe

49 Since loss of chromosome 10 and PTEN are common events in cancer, this

50 ling-microarray analysis of the 20 OsWAKs on chromosome 10 and reverse transcription-PCR analysis for

51 the promoter region of the IFN-gamma gene on chromosome 10 and the regulatory regions of the T(H)2 cy

52 The SNPs on chromosomes 10 and 13 lie in or near genes involved in p

53 he mechanism behind it; however, two SNPs on chromosomes 10 and 13 may be useful markers of delayed p

54 an interaction was observed between loci on chromosomes 10 and 19.

55 N (phosphatase and tensin homolog deleted on chromosome 10) and PP1alpha phosphatases.

56 N (phosphatase and tensin homolog deleted on chromosome 10) and SHIP-1 (Src homology [SH2] domain-con

57 N (phosphatase and tensin homolog deleted on chromosome 10) and the androgen receptor (AR) play impor

58 chromosome 11, PHYB gene is in A-sub-genome chromosome 10, and HY5 gene is in D-sub-genome chromosom

59 Loss of 1p36 and 19q13, 17p13, chromosome 10, and p53 mutation were significantly assoc

60 10509852), 400 kb upstream of SORCS1 gene on chromosome 10, and the global trait of abnormal WM micro

61 for chromosomes 8, 15, and 17; monosomy for chromosome 10; and amplification of the distal region of

62 ent genes impacted by broad regional loss of chromosome 10 are tumor suppressors capable of affecting

63 N (phosphatase and tensin homolog deleted on chromosome 10) are associated with the multihamartomatou

64 N (phosphatase and tensin homolog deleted on chromosome 10) are found in sporadic cancers and Cowden

65 al phosphatase and tensin homolog deleted on chromosome 10 as well as increased phosphorylation/inact

66 iRNA production from a second piRNA locus on chromosome 10, as well as pi6 itself.

67 ed genes of interest in an interval on mouse chromosome 10 associated with body mass in muscular dyst

68 Novel regions were identified on chromosome 10 associated with LA (rs10740118; P=8.1x10(-

69 howed a lod score of 3.56 at theta = 0.00 on chromosome 10 at area q25.

70 We also obtained weak evidence of linkage to chromosome 10 at the same location as a previous report

71 Chromosome 10 breakpoints were mapped by fluorescence in

72 hanges in the expression of genes located on chromosome 10 but also with genome-wide differences in g

73 N (phosphatase and tensin homolog deleted on chromosome 10) by cell surface receptors has not been de

74 describe detailed studies of a large knob on chromosome 10 called K10L2.

75 (phosphatase and tensin homology deleted on chromosome 10) cause Cowden and Bannayan-Riley-Ruvalcaba

76 This gene flow has its greatest effect on chromosome 10, causing both structural variation and sin

77 A significant QTL was mapped to chromosome 10 (Chr 10) and confirmed using B6.A<10> mice

78 pes (B6D2, D2B6) with the proximal region of chromosome 10 (Chr10) introgressed into opposing backgro

79 R5 on chromosome 1 (Chr1) and ARMS2/HTRA1 on chromosome 10 (Chr10).

80 Of particular interest is a locus on chromosome 10 containing the autophagy-related protein 1

81 reviously mapped PPCD to a region (PPCD3) on chromosome 10 containing the gene that encodes the two-h

82 gh-density mapping and annotation studies of chromosome 10, contig 395, showed that the De18 locus wa

83 using progeny testing confirmed that sov1 on chromosome 10 controlled obovoid and elongated shape, wh

84 hosphatase and tensin homolog gene (PTEN) on chromosome 10 controls the first step in the phosphatidy

85 KL-40 was significantly associated with both chromosome 10 copy number loss and poorer survival.

86 earance of a clone containing trisomy 12 and chromosome 10 copy-neutral loss of heterogeneity that ma

87 tumor suppressor mechanisms associated with chromosome 10 deletions.

88 ely, PTEN (phosphatase and tensin homolog on chromosome 10) dephosphorylates PtdIns(3,4,5)P3 and nega

89 Moreover, phosphate tensin homolog on chromosome 10 depletion in human kidney-2 cells resulted

90 oid maize lines that contain three copies of chromosome 10 derived from inbred lines B73 and H99.

91 N (phosphatase and tensin homolog deleted on chromosome 10) during neurite outgrowth of human embryon

92 omplement factor H (CFH) gene and a locus on chromosome 10 encompassing the HTRA1/LOC387715/ARMS2 gen

93 tumor (Ept)1], chromosome 3 (Ept2 and Ept6), chromosome 10 (Ept9), and chromosome 1 (Ept10 and Ept13)

94 omosome 10 from H99 pair preferentially over chromosome 10 from B73 in different stages of prophase I

95 demonstrate that the two identical copies of chromosome 10 from H99 pair preferentially over chromoso

96 nergy function using the Hi-C contact map of chromosome 10 from human GM12878 lymphoblastoid cells.

97 of phosphatase and tensin homolog deleted on chromosome 10 function.

98 king PTEN (phosphatase and tensin homolog on chromosome 10) function were relatively resistant to dru

99 N (phosphatase and tensin homolog deleted on chromosome 10) function, increased growth factor signali

100 by phosphatase and tensin homolog deleted on chromosome 10 functional status.

101 hosphatase and tensin homologue deleted from chromosome 10) functions as a tumour suppressor by oppos

102 tified a common good prognostic signature of chromosome 10 genes from two gene expression datasets (T

103 re (BP) quantitative trait loci (QTL) on rat chromosome 10 has been clearly demonstrated by linkage a

104 and allelic association in the IDE region of chromosome 10 have been reported in families with late-o

105 s with several biological candidate genes on chromosome 10 have been reported, these findings have no

106 The parental and recombinant chromosome 10 in B73 x Mo17 F(1) hybrids and F(2) progen

107 bular inflammation included SNP rs1227756 on chromosome 10 in COL13A1 (P = 2.0 x 10(-7)), rs6591182 o

108 ichment of differentially regulated genes on chromosome 10 in the course of validating RNA-Seq result

109 tion between type 2 diabetes and variants on chromosome 10 in the Framingham SHARe data.

110 identify the parental origin of each copy of chromosome 10 in the materials using oligonucleotide-bas

111 N (phosphatase and tensin homolog located on chromosome 10) in cancer has surpassed all predictions a

112 Loci on chromosome 10 include MSMB, which encodes beta-microsemi

113 PTEN (phosphatase/tensin homolog on chromosome 10)-induced putative kinase 1 (PINK1), a puta

114 we report the discovery of a large region on chromosome 10 involved in adaptation or domestication th

115 Thus, phosphate tensin homolog on chromosome 10 is an upstream regulator of renal PPM1A de

116 mutation in a second gene, Slc35d3, on mouse chromosome 10 is the basis of this discrepancy.

117 showed diminished paternal transmission, and chromosome 10 is transmitted to offspring only as a shor

118 N (phosphatase and tensin homolog deleted on chromosome 10) is a potent tumor suppressor gene frequen

119 N (phosphatase and tensin homolog deleted on chromosome 10) is a proven critical tumor suppressor.

120 (phosphatase and tensin homologue deleted on chromosome 10) is a tumor suppressor that is mutated or

121 PTEN (phosphatase and tensin homolog on chromosome 10) is a tumor suppressor whose cellular regu

122 phosphatase and tensin homologue, deleted on chromosome 10) is a tumor suppressor with dual phosphata

123 (phosphatase and tensin homologue deleted on chromosome 10) is frequently mutated or deleted in vario

124 N (phosphatase and tensin homolog deleted on chromosome 10) is the underlying cause of PTEN hamartoma

125 n (phosphatase and tensin homolog deleted on chromosome 10) is thought to mediate the majority of pro

126 N (phosphatase and tensin homolog deleted on chromosome 10) is well characterized for its role in ant

127 (phosphatase and tensin homologue deleted on chromosome 10) levels are frequently found reduced in hu

128 Furthermore, LRRTM3 maps to a region of chromosome 10 linked to both LOAD and elevated plasma Ab

129 hat the genetic linkage of AD in this set of chromosome 10-linked AD families may be the result of sy

130 12 affected and unaffected members of three chromosome 10-linked AD pedigrees in the National Instit

131 therto unknown association between SNPs at a chromosome 10 locus and the severity of NASH fibrosis.

132 The chromosome 10 locus is particularly promising given that

133 investigations on the ATG18 and genes at the chromosome 10 locus may provide an important lead for a

134 s (Ho) to a approximately 7.4 cM interval on chromosome 10 (LOD=8.78).

135 On chromosome 10 maximal logarithm of odds (LOD) scores of

136 onserved with three regions located on mouse chromosome 10 (Mmu10), Mmu16 and Mmu17.

137 egions in the mouse genome, located on mouse chromosome 10 (Mmu10), Mmu16, and Mmu17.

138 phosphatase and tensin homologue deleted on chromosome 10 mutations or deletions were also resistant

139 pe I (Ab10-I) does not recombine with normal chromosome 10 (N10) over an approximately 32-cM terminal

140 Three SNPs on chromosome 10 near the CHUK (conserved helix-loop-helix

141 me 16 (the top meta-analysis SNP) and one on chromosome 10 (not reported by RHM or in the meta-analys

142 Oligonucleotides (oligos) specific to chromosome 10 of maize inbreds B73 and Mo17, respectivel

143 Four SNPs on chromosomes 10 (one), 13 (two), and 14 (one) were signif

144 (phosphatase and tensin homologue deleted on chromosome 10), one of the most important tumor suppress

145 ites was observed on chromosome 9 but not on chromosome 10 or 19.

146 Starting from a single donor locus on chromosome 10, over 1500 elements were distributed throu

147 N (phosphatase and tensin homolog deleted on chromosome 10) phosphatase represents a novel switch bet

148 N (phosphatase and tensin homolog deleted on chromosome 10) plays important roles in tumor developmen

149 A single SNP, rs10824026 (chromosome 10: position 73661450), was found to signific

150 phosphatase and tensin homologue deleted on chromosome 10 protein, whereas the mass of phosphorylate

151 he tumor suppressor ANXA7 due to monosomy of chromosome 10 provides a clinically relevant mechanism t

152 ncRNA gene lincRNA-Tnfaip3, located at mouse chromosome 10 proximal to the tumor necrosis factor alph

153 phosphatase and tensin homolog deleted from chromosome 10 (Pten) (either as heterozygotes or by cond

154 1 and/or ptase and tensin homolog deleted on chromosome 10 (PTEN) (such as Jurkat, CEM, Molt) and, co

155 phosphatase and tensin homologue deleted on chromosome 10 (PTEN) abnormal HNSCC cell line FaDu, inhi

156 phosphatase and tensin homologue deleted on chromosome 10 (PTEN) activity.

157 phosphatase and tensin homologue deleted on chromosome 10 (PTEN) and activation of the downstream pr

158 es phosphatase and tensin homolog deleted on chromosome 10 (Pten) and androgen upregulated gene 19 (U

159 or phosphatase and tensin homolog deleted on chromosome 10 (PTEN) and decreases its phosphorylation.

160 phosphatase and tensin homologue deleted on chromosome 10 (PTEN) and glycogen synthase kinase beta (

161 phosphatase and tensin homologue deleted on chromosome 10 (PTEN) and p53 tumor suppressors.

162 ted phosphatase and tensin homolog delete on chromosome 10 (PTEN) and promoted the phosphoinositide 3

163 ne phosphatase and tensin homolog deleted on chromosome 10 (Pten) are associated with multiple cancer

164 in phosphatase and tensin homolog deleted on chromosome 10 (PTEN) are implicated in neuropsychiatric

165 ty phosphatase and tensin homolog deleted on chromosome 10 (PTEN) blocks PI3K signaling by dephosphor

166 nd phosphatase and tensin homolog deleted on chromosome 10 (PTEN) by all 3 HexAbs and the notable dif

167 of phosphatase and tensin homolog deleted on chromosome 10 (PTEN) confers sensitivity to N-methyl-N'-

168 in phosphatase and tensin homolog deleted in chromosome 10 (PTEN) contributes to renal cell hypertrop

169 phosphatase and tensin homologue deleted on chromosome 10 (PTEN) down-regulates phosphorylation of p

170 or phosphatase and tensin homolog deleted on chromosome 10 (Pten) elicits a senescence response that

171 hosphatase and tensin homologue deleted from chromosome 10 (Pten) encodes a lipid phosphatase control

172 Phosphatase and tensin homolog deleted on chromosome 10 (PTEN) encodes a tumor-suppressor phosphat

173 in phosphatase and tensin homolog deleted on chromosome 10 (PTEN) experience autoimmunity and lymphoi

174 nd phosphatase and tensin homolog deleted on chromosome 10 (PTEN) function to block growth factor-ind

175 he phosphatase and tensin homolog deleted on chromosome 10 (PTEN) gene in a p38-dependent manner, but

176 he phosphatase and tensin homolog deleted on chromosome 10 (PTEN) gene is often altered in prostate c

177 sphatase and tensin homologue deleted on the chromosome 10 (PTEN) gene was significantly up-regulated

178 phosphatase and tensin homologue deleted on chromosome 10 (PTEN) has inhibitory effects on the PI3K/

179 phosphatase and tensin homologue deleted on chromosome 10 (PTEN) in AD pathogenesis have not been ex

180 he phosphatase and tensin homolog deleted on chromosome 10 (PTEN) in AMs, we attenuated the inhibitor

181 -phosphatase and tensin homologue deleted on chromosome 10 (PTEN) in Fc gamma R-induced macrophage fu

182 ator phosphate and tensin homolog deleted on chromosome 10 (PTEN) in mural cells.

183 phosphatase and tensin homologue deleted on chromosome 10 (PTEN) in repressing the protumorigenic ef

184 phosphatase and tensin homologue deleted on chromosome 10 (PTEN) in the regulation of PDGF expressio

185 nd phosphatase and tensin homolog deleted on chromosome 10 (PTEN) induces activation of the phospho-5

186 ic phosphatase and tensin homolog deleted on chromosome 10 (PTEN) inhibitor SF1670.

187 Phosphatase and tensin homologue deleted on chromosome 10 (PTEN) is a dual-function phosphatase with

188 Phosphatase and tensin homolog deleted on chromosome 10 (PTEN) is a lipid phosphatase implicated i

189 Phosphatase and tensin homologue on chromosome 10 (PTEN) is a lipid phosphatase that, by ant

190 Phosphatase and tensin homologue deleted on chromosome 10 (PTEN) is a lipid phosphatase.

191 Phosphatase and tensin homologue deleted on chromosome 10 (PTEN) is a phosphatidylinositol phosphate

192 Phosphatase and tensin homolog located on chromosome 10 (PTEN) is a tumor suppressor gene and one

193 Phosphatase and tensin homolog deleted on chromosome 10 (PTEN) is a tumor suppressor that antagoni

194 Phosphatase and tensin-homolog deleted on chromosome 10 (PTEN) is a tumor-suppressor protein that

195 Phosphatase and tensin homolog deleted on chromosome 10 (PTEN) is an important negative regulator

196 Phosphatase and tensin homolog deleted on chromosome 10 (PTEN) is an important negative regulator

197 hosphatase and tensin homologue deleted from chromosome 10 (Pten) is expressed aberrantly in non-smal

198 f phosphatase and tensin homology deleted on chromosome 10 (PTEN) is linked to increased PI3K-AKT sig

199 Phosphatase and tensin homologue deleted on chromosome 10 (PTEN) is mutated or lost in 60% to 70% of

200 or phosphatase and tensin homolog deleted on chromosome 10 (PTEN) is strongly correlated with human l

201 on phosphatase and tensin homolog deleted on chromosome 10 (PTEN) is the second most frequently mutat

202 phosphatase and tensin homologue deleted on chromosome 10 (PTEN) lacking lipid (G129E) or lipid and

203 phosphatase and tensin homologue deleted on chromosome 10 (PTEN) levels, or the downregulation of PI

204 phosphatase and tensin homologue deleted on chromosome 10 (PTEN) localization in the nucleus and cyt

205 phosphatase and tensin homologue deleted on chromosome 10 (PTEN) loss regulated protein kinase B (AK

206 nd phosphatase and tensin homolog deleted on chromosome 10 (PTEN) negatively regulate this insulin si

207 Phosphatase and tensin homolog deleted on chromosome 10 (PTEN) negatively regulates phosphatidylin

208 phosphatase and tensin homologue deleted on chromosome 10 (PTEN) or have reduced PTEN expression thr

209 he phosphatase and tensin homolog deleted on chromosome 10 (PTEN) overexpression down-regulated PH do

210 Phosphatase with TENsin homology deleted in chromosome 10 (PTEN) phosphatase and a PI3K activating p

211 or phosphatase and tensin homolog deleted on chromosome 10 (PTEN) plays a critical role in regulating

212 he phosphatase and tensin homolog deleted on chromosome 10 (PTEN) plays a critical role in the mainte

213 phosphatase and tensin homologue deleted on chromosome 10 (PTEN) plays a pivotal role in various cel

214 re phosphatase and tensin homolog deleted on chromosome 10 (PTEN) protein when compared with CD56(dim

215 he phosphatase and tensin homolog deleted on chromosome 10 (PTEN) regulates innate immune responses i

216 he Phosphatase And Tensin Homolog Deleted On Chromosome 10 (PTEN) regulates the phosphoinositol-3-kin

217 sor gene phosphatase and tensin homologue on chromosome 10 (PTEN) result in elevated levels of phosph

218 or phosphatase and tensin homolog deleted on chromosome 10 (PTEN) through its interaction with a hist

219 phosphatase and tensin homologue deleted on chromosome 10 (PTEN) tumor suppressor gene, one of the m

220 he phosphatase and tensin homolog deleted on chromosome 10 (PTEN) tumor suppressor is a phosphatase t

221 he phosphatase and tensin homolog deleted on chromosome 10 (PTEN) tumor suppressor is frequently phos

222 phosphatase and tensin homolog deleted on chromosome 10 (PTEN) tumor suppressor is the major brake

223 he phosphatase and tensin homolog mutated on chromosome 10 (PTEN) tumor-suppressor gene.

224 of phosphatase and tensin homolog deleted on chromosome 10 (PTEN) was observed both in fluorescence a

225 phosphatase and tensin homologue deleted on chromosome 10 (PTEN) were coordinately expressed, with c

226 phosphatase and tensin homologue deleted on chromosome 10 (PTEN) were detected in ULTH cells.

227 Phosphatase and tensin homologue deleted on chromosome 10 (PTEN) with shRNA in three estrogen recept

228 Phosphatase and tensin homolog deleted on chromosome 10 (PTEN), a multifunctional tumor suppressor

229 of phosphatase and tensin homolog deleted on chromosome 10 (PTEN), a negative PI3K regulator that tau

230 phosphatase and tensin homolog deleted from chromosome 10 (PTEN), a negative regulator of Akt activa

231 hosphatase and a tensin homolog deleted from chromosome 10 (PTEN), a negative regulator of the phosph

232 phosphatase and tension homologue deleted on chromosome 10 (PTEN), a negative regulator of the phosph

233 of phosphatase and tensin homolog deleted on chromosome 10 (PTEN), a negative regulator of the PI3K/A

234 phosphatase and tensin homologue deleted on chromosome 10 (PTEN), a phosphatase that dampens phospha

235 of phosphatase and tensin homolog deleted on chromosome 10 (PTEN), a phosphatase that inactivates Akt

236 of phosphatase and tensin homolog deleted on chromosome 10 (PTEN), a phosphatidylinositol 3-phosphata

237 Phosphatase and tensin homologue deleted on chromosome 10 (PTEN), a tumor suppressor phosphatase tha

238 of phosphatase and tensin homolog deleted on chromosome 10 (Pten), activation of AKT, fast proliferat

239 r, phosphatase and tensin homolog deleted on chromosome 10 (PTEN), alters the invasive potential of m

240 regulates phosphatase and tensin deleted on chromosome 10 (PTEN), AMP-activated protein kinase (AMPK

241 hosphatase and tensin homologue deleted from chromosome 10 (PTEN), and this action of oncogenic Ras i

242 phosphatase and tensin homologue deleted on chromosome 10 (PTEN), but also targets Fas ligand (FasL)

243 atase, phosphatase tensin homolog deleted on chromosome 10 (PTEN), has been reported to block insulin

244 ll phosphatase and tensin homolog deleted on chromosome 10 (PTEN), increased cell survival with down-

245 y, phosphatase and tensin homolog deleted on chromosome 10 (PTEN), is primarily localized in nonraft

246 phosphatase and tensin homologue deleted on chromosome 10 (PTEN), leading to activation of the epide

247 Phosphatase and tensin homologue, deleted on chromosome 10 (PTEN), the major tumor suppressor and key

248 r, phosphatase and tensin homolog deleted on chromosome 10 (PTEN), was identified as a bona fide targ

249 by phosphatase and tensin homolog deleted on chromosome 10 (PTEN)-dependent activation of PI 3-kinase

250 1 (phosphatase and tensin homolog deleted on chromosome 10 (PTEN)-induced kinase 1), a Parkinson's di

251 osphatase, phosphatase and tensin homolog on chromosome 10 (Pten).

252 phosphatase and tensin homologue deleted on chromosome 10 (PTEN).

253 phosphatase and tensin homologue deleted on chromosome 10 (PTEN).

254 hosphatase and tensin homologue deleted from chromosome 10 (PTEN).

255 by phosphatase and tensin homolog deleted on chromosome 10 (PTEN).

256 of phosphatase and tensin homolog deleted on chromosome 10 (PTEN).

257 phosphatase and tensin homologue deleted on chromosome 10 (PTEN)/MMAC1/TEP gene in human cancer cell

258 phosphatase and tensin homologue detected on chromosome 10 (PTEN)/phosphatidylinositol 3-kinase/AKT/T

259 phosphatase and tensin homologue deleted on chromosome 10 (PTEN); assessed apoptosis; and determined

260 phosphatase and tensin homologue deleted on chromosome 10 (PTEN; eg, phosphatidylinositol-3-kinase [

261 et phosphatase-and-tensin-homolog-deleted-on-chromosome-10 (PTEN).

262 s "phosphatase and tensin homolog deleted on chromosome 10" (PTEN), a key regulator of Akt activation

263 phosphatase and tensin homologue deleted on chromosome 10, PTEN.

264 (phosphatase and tensin homologue deleted on chromosome 10) reduces PIP3 levels and leads to fatty li

265 constructed and characterized to fine-map a chromosome 10 region (QTL1) linked to blood pressure.

266 putative familial late-onset AD gene in this chromosome 10 region.

267 N (phosphatase and tensin homolog deleted on chromosome 10) regulates the peripheral homeostasis of T

268 lated retroelements surrounding the locus on Chromosome 10 responsible for double minute chromosome f

269 The Mcs7 region on rat chromosome 10 (RNO10) is orthologous to human 17q, a com

270 to mtDNA levels: one within the TFAM gene on chromosome 10 (rs11006126, p value = 8.73 x 10(-28), var

271 e maternal genotype for an intergenic SNP on chromosome 10 (rs11008222, combined P = 6.3 x 10(-7); re

272 leotide polymorphism in the CYP2C cluster on chromosome 10 (rs12777823) and warfarin dose requirement

273 dence interval=1.05-1.11; P=2.86x10(-8)) and chromosome 10 (rs7920721; closest gene, ECHDC3; odds rat

274 A locus carried on SWR/J chromosome 10 seems to be particularly important in AR f

275 A secondary structure formation on the human chromosome 10 sequence, and utilize this analysis to com

276 (phosphatase and tension homolog deleted on chromosome 10), Ser(380)/Thr(382/383) PTEN phosphorylati

277 We performed a chromosome 10-specific association study with 1,412 gene

278 We identified a locus on mouse chromosome 10 that accounts for 60% of the genetic varia

279 ocus (QTL) mapping identified a QTL on mouse chromosome 10 that accounts for greater than 50% of the

280 on at 16p12, and a novel 1.2-Mb inversion on chromosome 10 that is supported by the presence of two d

281 variant in a kelch domain-containing gene on chromosome 10 that may epistatically modulate artemisini

282 (phosphatase and tensin homolog deleted from chromosome 10), the antagonist of the phosphosphoinosito

283 s identified one locus near the NEK9 gene on chromosome 10 to have a significant effect on crossover

284 N (phosphatase and tensin homolog deleted on chromosome 10) tumor suppressor gene functions that elud

285 N (phosphatase and tensin homolog deleted on chromosome 10) tumor suppressor protein and phosphate-de

286 N (phosphatase and tensin homolog deleted on chromosome 10) tumor suppressor regulates a variety of c

287 (phosphatase and tensin homologue deleted in chromosome 10) tumour suppressor has been associated wit

288 verse strand of the last 11.2 Mb sequence of chromosome 10 was analyzed in detail based on a mathemat

289 de polymorphisms (SNPs) in a novel region on chromosome 10 was genome-wide significant (lowest P=1.3E

290 2 near 60 cM, proximal chromosome 6, and mid-chromosome 10 were distinct from BW, lean tissue, and bo

291 gments containing noncontiguous sequences of chromosome 10, whereas, in the other two families, the b

292 allele of the gene Columnar (Co) located on chromosome 10 which can appear in a heterozygous (Co/co)

293 Cia5 is a locus on rat chromosome 10 which regulates the severity of collagen-

294 pitulating the fact that loss of one copy of chromosome 10 (which harbors the tumor suppressor PTEN)

295 de phosphatase and tensin homolog deleted on chromosome 10, which has been suggested to mediate S1P(2

296 lzheimer disease (LOAD) has been observed on chromosome 10, which implicates a wide region and at lea

297 these, one major QTL was mapped to bin 10 of chromosome 10, which is 29.7 kb in size and harbors thre

298 hosphatase and tensin homologue deleted from chromosome 10), whose inactivation would shift the equil

299 netic mapping associated a large deletion on chromosome 10 with a quantitative change in seed composi

300 A peak for %DMA on chromosome 10 within 2 Mb of AS3MT had an LOD of 1.80.