ライフサイエンスコーパス: chromosome 5

1 e to map the disease gene in two families to chromosome 5.

2 a screen for ENU-induced mutations on mouse chromosome 5.

3 at the telomere to a full-length homolog of chromosome 5.

4 approximately 422 kbp, which is one third of chromosome 5.

5 n was mapped to 68.4 +/- 6.0 centimorgans on chromosome 5.

6 e X chromosome, the TAG genes are located on chromosome 5.

7 of H19 and at the alpha-fetoprotein locus on chromosome 5.

8 y to E2-induced mammary cancer on distal rat chromosome 5.

9 ative markers spaced over a 60-cM segment of chromosome 5.

10 y, the human protocadherin alpha cluster, on chromosome 5.

11 -10 gene is located at the 5p12-p13 locus of chromosome 5.

12 a single recessive mutation at the bottom of chromosome 5.

13 d was mapped previously to a region of mouse chromosome 5.

14 f orthologous synteny lies on proximal mouse chromosome 5.

15 receptor 2 (TfR2) were cloned and mapped to chromosome 5.

16 ele because of an interstitial deletion from chromosome 5.

17 xe1-1 to axe1-5) mapped to the same locus on chromosome 5.

18 ion of reduced recombination on B. decumbens chromosome 5.

19 Both mSteap and mPsca map to chromosome 5.

20 dopsis mutant, scarface (sfc), which maps to chromosome 5.

21 ished sequence of a 204-kb region from mouse chromosome 5.

22 y span approximately 40 cM of proximal mouse chromosome 5.

23 ization, we show that Lungkine maps to mouse chromosome 5.

24 east artificial chromosome clone, CIC9E2, on chromosome 5.

25 The MOCO1 locus resides on human chromosome 5.

26 11 and ARC6 map approximately 60 cM apart on chromosome 5.

27 se BACs hybridized to a single site on mouse chromosome 5.

28 compassing the corresponding region on mouse chromosome 5.

29 nant myeloid disorders with abnormalities of chromosome 5.

30 ed to the conserved syntenic region on mouse chromosome 5.

31 K2 probes identified a single locus on mouse Chromosome 5.

32 omologous with those on the domestic horse's chromosome 5.

33 terstitial deletion of the long arm of human chromosome 5.

34 sive D. americana alleles at a single QTL on chromosome 5.

35 n rice chromosomes 11 and 12 and arabidopsis chromosome 5.

36 rated to construct a comprehensive RH map of chromosome 5.

37 of AML/MDS characterized by abnormalities of chromosome 5.

38 Quantitative trait loci on chromosomes 5, 10, 17, 18, and X were found to control a

39 mosome 18, with weaker effects detectable on chromosomes 5, 12, and 14.

40 We report in detail on baboon chromosomes 5, 12, and 18 for which the linkage maps are

41 this disease are located on the short arm of chromosome 5(1H).

42 ive or proliferative retinopathy occurred on chromosomes 5 (2.53 at 11.2 cM), 6 (2.28 at 30.6), and 1

43 islands associated with fiber initiation on chromosome 5, 3 islands for the early to middle elongati

44 with the predicted number of genes on wheat chromosome 5D (4286).

45 hromosome 5 or a deletion of the long arm of chromosome 5, -5/del(5q), is a recurring abnormality in

46 is caused by a deletion in the short arm of chromosome 5 (5p) and has a variable clinical spectrum.

47 ase caused by a deletion in the short arm of chromosome 5 (5p) with a variable clinical spectrum.

48 transcription factor located in a region of chromosome 5 (5p15.3) that has been proposed to contain

49 kage analyses revealed suggestive linkage on chromosome 5 (5q22) with a logarithm of odds (LOD) score

50 ic (heterozygous) markers were identified on chromosome 5, 6 to the left and 12 to the right of the M

51 of BXD strain survival identified linkage on chromosomes 5, 6, 9, 11, and 14.

52 lusion, we determined regions of interest on chromosomes 5, 6, and 10 where imprinted genes might be

53 Three other regions, on chromosomes 5, 6, and 15, that were nominally significan

54 The structural maintenance of chromosomes 5/6 complex (Smc5/6) is a host restriction f

55 ion.IMPORTANCE The structural maintenance of chromosomes 5/6 complex (Smc5/6) is a host restriction f

56 The host structural maintenance of chromosomes 5/6 complex (Smc5/6) suppresses hepatitis B

57 dation of the host structural maintenance of chromosomes 5/6 complex (Smc5/6) that inhibits HBV trans

58 Abnormalities of chromosome 5, 7, or both accounted for 76% of all cases

59 Chromosomes 5, 7, 10, and 14 were found to contain signi

60 ed linkage disequilibrium (LD) maps of human chromosomes 5, 7, 17, and X.

61 iking were the increases in abnormalities of chromosomes 5, 7, and 17 among the elderly.

62 andom interstitial deletions and monosomy of chromosomes 5, 7, and 17 in refractory myelodysplasia (M

63 enetic abnormalities, including deletions of chromosomes 5, 7, or both.

64 inkage to autism-spectrum disorders (ASD) on chromosomes 5, 8, 16, 19, and X and showing nominal evid

65 , and population-specific GWS for markers on chromosomes 5, 9 and 19.

66 nificant and suggestive evidence for loci on chromosomes 5, 9, 11, 15, 16 and 19, which influence hyp

67 We observed clustered loss of chromosomes 5, 9, and 16, orthologous to a similar patte

68 somes 1, 3, 11, 14, 17, and 18 and losses on chromosomes 5, 9, and 16.

69 3 open reading frames (ORFs) on one circular chromosome (5,928,787 bp) and two plasmids (131,950 bp a

70 e epiregulin gene (Ereg) is located on mouse chromosome 5 adjacent to three other epidermal growth fa

71 Although partially fertile, these chromosome 5 and 10 addition plants have not yet transmi

72 ause cytogenetic studies frequently identify chromosome 5 and 17 deletions within a single clone, we

73 Unlike in primary MDS, aberrancies of chromosome 5 and 20 were infrequent.

74 a marker of adverse prognosis independent of chromosome 5 and 7 deletions.

75 ealed that a single copy of the mdr1 gene on chromosome 5 and a novel amplification of the plasmepsin

76 lenalidomide for patients with alteration of chromosome 5 and anemia.

77 alization of genes on the short arm of human chromosome 5 and as a framework for both generating and

78 etween wheat consensus chromosome 1 and rice chromosome 5 and between the proximal portion of the lon

79 entation of four contigs of Candida albicans chromosome 5 and determined the sizes of three gaps betw

80 ional candidate genes in a linkage region on chromosome 5 and genotyped selected variants in several

81 e is enhanced by a second locus that maps to chromosome 5 and heightened by additional genetic modifi

82 the conservation of synteny between macaque chromosome 5 and human chromosome 4.

83 mposed of 31 exons spanning >100 kb on mouse chromosome 5 and is located between Cyln2 and Gtf2i.

84 hat are trisomic for 90% of the short arm of chromosome 5 and monosomic for a small distal portion of

85 d in close proximity to gp130 genes on human chromosome 5 and mouse chromosome 13.

86 7/Q7Z4C4 on chromosome 5, rs1445898/CAPSL on chromosome 5 and rs2302188/NM_033543 on chromosome 19),

87 es showed significant evidence of linkage to chromosome 5 and suggestive evidence of linkage to chrom

88 k2beta that are encoded by distinct genes on chromosome 5 and that are ubiquitously expressed in matu

89 between genes linked to the targeted CD38 on Chromosome 5 and the ART2 complex on Chromosome 7.

90 nalysis of the GABA(A) gene cluster on mouse chromosome 5 and the corresponding region on human chrom

91 been identified from the cytokine cluster on chromosome 5 and the MHC on chromosome 6.

92 The UVH1 locus was mapped to chromosome 5 and the position of the UVH1 gene was furth

93 apped two additional mutations to the distal chromosome 5 and the proximal chromosome 10 in a second

94 ies]: HR, 2; P < .001), and abnormalities of chromosome 5 and/or 7 (HR, 1.89; P < .001).

95 The gene was mapped to the short arm of chromosome 5D and mediated recognition of the effector S

96 minor effect was mapped on the short arm of chromosome 5S and was designated QPh.ucd-5S.

97 ric duplicated segments shared between mouse chromosomes 5 and 1.

98 Monosomic additions for maize chromosomes 5 and 10 to a haploid complement of oat (n =

99 Two of these loci, Hpi1 and Hpi2 on mouse chromosomes 5 and 13, were mapped to the significant and

100 ifiers of L. monocytogenes susceptibility to chromosomes 5 and 13.

101 ethod to detect interactions between SNPs on chromosomes 5 and 15 on lung cancer data.

102 Cosegregating deletions of chromosomes 5 and 17 were found to specifically include

103 Survival mapped to chromosomes 5 and 17, anti-chromatin Ab to chromosomes 4

104 een ancestral chromosomes, syntenic to human chromosomes 5 and 17, respectively.

105 L contributing to variation in gut length on chromosomes 5 and 18.

106 18, and 20, and secondary loci were found on chromosomes 5 and 19.

107 cloned and assigned to conserved regions of chromosomes 5 and 2, respectively.

108 alized HBECs to have duplication of parts of chromosomes 5 and 20.

109 acid identity) are encoded by genes on mouse chromosomes 5 and 6 and, together with the Tmhs-encoded

110 that the duplications associated with mouse chromosomes 5 and 6 are recent and that the resulting du

111 Twenty microsatellite markers spanning chromosomes 5 and 6 at an average distance of 20 cM were

112 In this study, we determined allelic loss on chromosomes 5 and 6 in 29 primary early-stage epithelial

113 pping and sequencing of the regions of mouse chromosomes 5 and 6 involved in this chromosome-fission

114 enes are located on five distinct regions on chromosomes 5 and 6, i.e., 5p15.2, 5q13-21, 6p24-25, 6q2

115 hromosome 5 (n = 63), chromosome 7 (n = 85), chromosomes 5 and 7 (n = 66), recurring balanced rearran

116 ells showed that only one of the two HORs on chromosomes 5 and 7 incorporate CENP-A, an organization

117 in offspring and identify loci on offspring chromosomes 5 and 7 that modify maternal behaviour.

118 ultiple cytogenetic aberrations, commonly on chromosomes 5 and 7, suggesting an association with prev

119 omplex, with abnormalities in either or both chromosomes 5 and 7.

120 togenetics, especially abnormalities in both chromosomes 5 and 7.

121 pattern was principally in abnormalities in chromosomes 5 and 7.

122 cant genomewide P values <.05 for markers on chromosomes 5 and 8 and with suggestive linkage evidence

123 inkage studies that have reported linkage to chromosomes 5 and X.

124 mia patients who had clonal abnormalities of chromosomes 5 and/or 7, 7 (16%) were homozygous for the

125 h radiotherapy, and in patients with loss of chromosomes 5 and/or 7, acquired abnormalities associate

126 , in particular, complete or partial loss of chromosomes 5 and/or 7.

127 among those with t-AML or an abnormality of chromosomes 5 and/or 7.

128 ave a rough map position on the upper arm of chromosome 5, and deep sequencing of DNA from these 13 l

129 complex etiology with significant linkage to chromosome 5, and marginal linkage was observed to five

130 NRH1 maps on chromosome 5, and NRH2 and NRH3 are less than 48kb apart

131 s, 3 SNPs (rs2540317 in MFSD9, rs10515353 on chromosome 5, and rs2242400 in BCAT1 were associated wit

132 tly localized to 1.0, 7.5, and 4.5 Mb of rat chromosome 5, and the orthologous regions are on human c

133 Nonrandom, complete and partial deletions of chromosome 5 are common anomalies in AML.

134 Complete loss or interstitial deletions of chromosome 5 are the most common karyotypic abnormality

135 (GENDEP) identified an intergenic region on chromosome 5 associated with early improvement after 2 w

136 .27), chromosome 4 at 135.3 cM (LOD = 2.63), chromosome 5 at 139.3 cM (LOD = 0.84), chromosome 6 at 1

137 n situ hybridization to the proximal half of chromosome 5 at bands q2.2-q2.3.

138 ion analysis localized the FYB gene to human chromosome 5 at position p13.1.

139 e carbamoyl phosphate synthetase I locus, on chromosome 5 at the alanine-glyoxylate aminotransferase

140 Linkage analysis mapped this gene on chromosome 5D at intervals of 5.1 cM and 3.4 cM in the t

141 The KLHL3 gene is mapped to human chromosome 5, band q31, contains 17 exons, and spans app

142 h-resolution map of a 6-Mb interval of human chromosome 5, band q31, incorporating 175 sequence tagge

143 The RAB6KIFL gene is mapped to human chromosome 5, band q31, spans approximately 8.5kb of gen

144 et identified a novel variant (rs7726475) on chromosome 5 between the SUB1 and NPR3 genes, as being a

145 zed to a 0.8-cM interval on the short arm of chromosome 5, between the polymorphic microsatellite mar

146 struct a map of 54 markers located on bovine chromosome 5 (BTA5).

147 T1: dormancy cycling) is physically close on chromosome 5, but is distinct from DOG1.

148 elphia chromosome (Ph) positive (4 cases: 44 chromosomes; 5 cases: 40-43 chromosomes) and were not co

149 genomic orientation of this cluster on mouse chromosome 5: cen-D5Mit151-Gabrg1-Gabra2-Gabrb1-D5Mit58-

150 rce L-sorbose, by the reversible loss of one chromosome 5 (Ch5).

151 We examined CNV in a region of chromosome 5 (chr5) in haploid and diploid strains of Sa

152 LDL with significant linkage is detected on chromosome 5, chr5-104 Mbp (LOD 3.7).

153 f murine I-TAC (gene symbol Scyb11) to mouse chromosome 5 close to the genes for monokine induced by

154 In reciprocal chromosome 5 congenic mice, introgressed D2 alleles incr

155 t clones, suggesting that certain regions of chromosome 5 contain haploinsufficient developmental gen

156 ongenic strain, in which a smaller region of chromosome 5 containing the CYP4A genes from a Lewis rat

157 Transfer of chromosome 5 containing the CYP4A genes responsible for

158 The short arm of human chromosome 5 contains approximately 48 Mb of DNA and com

159 ximately 209-kbp portion of the right arm of chromosome 5 contains at least five spatially separated,

160 aucoma and provides additional evidence that chromosome 5 contains susceptibility loci for glaucoma i

161 features, which shows a promising signal on chromosome 5 corresponding to SERINC5, a protein involve

162 Interstitial deletion of the long arm of chromosome 5 (del(5q)) is the most common structural gen

163 A heterozygous deletions of the long arm of chromosome 5 [del(5q)], observed in 40% of patients, is

164 Deletions of the long (q) arm of chromosome 5 [del(5q)]occur in patients with myelodyspla

165 Interstitial deletion or loss of chromosome 5, del(5q) or -5, is a frequent finding in my

166 itial loss of all or part of the long arm of chromosome 5, del(5q), is a hallmark of myelodysplastic

167 An interstitial deletion of chromosome 5, del(5q), is the most common structural abn

168 e 'CNA-devoid' subgroup and a basal-specific chromosome 5 deletion-associated mitotic network.

169 The frequency of unbalanced chromosome 5 deletions has led to the idea that 5q harbo

170 ltiple dosage-dependent modifier loci on PWD chromosome 5 each contributed to tumor suppression.

171 The human MACROH2A1 gene, on chromosome 5, encodes two MACROH2A subtypes, MACROH2A1.1

172 me (BAC)-based map of a 5-Mb region on mouse Chromosome 5, encompassing three gene families: receptor

173 ukemic cell line, ML3, is diploid for all of chromosome 5, except for an inversion-coupled translocat

174 ultiple redundant regulators scattered along chromosome 5 explain, in a simple, elegant way, why the

175 QTLs were discovered near the centromere of chromosome 5 (Eye1: genomewide P < 0.005) and on proxima

176 d delineation of a 1.1-Mb critical region on chromosome 5 for the gene mutated in CdLS.

177 use cases: (i) 5-kb resolution Hi-C data of chromosome 5 from GM12878 (a human lymphoblastoid cell l

178 lines identified a dominant genetic locus on chromosome 5 from the wild relative that affected total

179 largest factor (0.30 variance) included the chromosome 5 gene cluster that encodes the most common G

180 ned the position of tlt relative to 17 mouse chromosome 5 genes with orthologous loci in the human 4p

181 Although chromosome 5 has been implicated in previous linkage stu

182 eterogeneity lod = 2.36, dominant model) and chromosome 5 (heterogeneity lod = 2.23, recessive model)

183 ide p < 0.005, between 65 and 100 cM) and to chromosome 5 (Hipp5a, p < 0.05, between 15 and 40 cM).

184 or homozygosis in one offspring, loss of one chromosome 5 homolog followed by duplication of the reta

185 combination, gene conversion, or loss of one chromosome 5 homolog, followed by duplication of the ret

186 spring, which arise primarily by loss of one chromosome 5 homologue followed by duplication of the re

187 of the heterozygosity of non-MTL genes along chromosome 5, however, results in larger decreases in vi

188 dered gene maps for equine homologs of human chromosome 5 (HSA5), viz., horse chromosomes 14 and 21 (

189 Crohn's disease have now been identified on chromosomes 5 (IBD5/5q31 risk haplotype) and 16 (IBD1 lo

190 This reduction in chromosome 5-imprinted Mkrn1-p1 transcripts was proposed

191 mouse, cappuccino (cno), that maps to mouse chromosome 5 in a region conserved with human 4p15-p16.

192 In mice, the homologs of these genes map to chromosome 5 in a region of conserved synteny with human

193 ly due to a gene(s) closely linked to Opn on chromosome 5 in conjunction with other randomly assortin

194 Preb maps to the proximal end of chromosome 5 in mouse, near the Hmx1 homeobox gene.

195 association (prs58502974 = 2.11 x 10(-8)) on chromosome 5 in the ADCY2 gene.

196 previously unidentified while a major QTL on chromosome 5 in the BTx623/BTx642 RIL population corresp

197 redicts the presence of a gene at the top of chromosome 5 in this subset of patients that is importan

198 Three QTL were identified--a major one on chromosome 5 in which the Col parent contributed the sup

199 The region of chromosome 5 including the osteopontin (OPN) gene (Opn)

200 ent cells had unique abnormalities involving chromosome 5, including amplicons centered on the target

201 e, which was mapped to the central region of chromosome 5, indicated that prosysA and prosysB transcr

202 A deletion of the long arm of chromosome 5 is a recurring abnormality in malignant mye

203 Interstitial deletion or loss of chromosome 5 is frequent in malignant myeloid disorders,

204 Complete loss or deletion of the long arm of chromosome 5 is frequent in myelodysplastic syndrome (MD

205 es in the conserved syntenic region of mouse chromosome 5 is inverted relative to the human map.

206 The distal portion of the long arm of chromosome 5 is linked to hypertension and contains func

207 Chromosome 5 is one of the largest human chromosomes and

208 nation frequencies across a large portion of chromosome 5 is the first large-scale analysis of mitoti

209 he discovery that deletion of all or part of chromosome 5 is the most common genetic aberration in my

210 ing of 3 dominant NZB contributions (Nba4 on chromosome 5, Lbw4 on chromosome 6, and Nba5 on chromoso

211 The strong signal on chromosome 5 lies in the region in which a novel gene, W

212 NCMD (MCDR1) and 2 individuals affected with chromosome 5-linked NCMD (MCDR3).

213 showed evidence for interactions between the chromosome 5 locus and region on chromosome 12 containin

214 (+/-), including a region upstream of Eln on chromosome 5 (LOD 4.5).

215 76 cM of chromosome 4 (lod = 2.7), 143 cM of chromosome 5 (lod = 2.0), 7 cM of chromosome 9 (lod = 2.

216 ewly discovered loci for absence seizures on chromosome 5 (lod scores 3.8/2.8 and 3.4/1.9).

217 I to maternally derived alleles was found on chromosome 5 (LOD(MO) = 1.7) and to paternally derived a

218 TLs influencing urine arsenic metabolites on chromosomes 5 (LOD = 2.03 for %iAs), 9 (LOD = 2.05 for %

219 diabetes with maternally derived alleles on chromosomes 5 (LOD(MO) = 1.5) and 14 (LOD(MO) = 1.6).

220 6 cM on chromosome 4; lod = 1.7 at 146 cM on chromosome 5; lod = 3.5 at 160 cM on chromosome 9; lod =

221 ve evidence for linkage was found for BMI on chromosome 5 (logarithm of odds [LOD] score = 1.9) and P

222 ritical region of loss in chromosome 5q31.1 (chromosome 5, long arm, region 3, band 1, subband 1) in

223 uman chromosome 13q12-13 and telomeric mouse chromosome 5 (MMU5).

224 282 (92%) had clonal abnormalities involving chromosome 5 (n = 63), chromosome 7 (n = 85), chromosome

225 d to hypergammaglobulinemia; an NZB locus on chromosome 5 (Nba4; peak at 15 cM), linked to IgG anti-s

226 We identified one strong association on chromosome 5 near a previously characterized disease res

227 Of particular interest was the region on chromosome 5 near D5S1480, where a reasonable candidate

228 D1S3462; chromosome 4, near marker D4S2361; chromosome 5, near marker D5S1505; and chromosome 17, ne

229 omain protein encoded by a gene that maps to chromosome 5, near the chromosomal location of the cop8

230 RPS4 is a disease-resistance locus on chromosome 5 of Arabidopsis thaliana specifying resistan

231 ng-time regulator FLC, located at the top of chromosome 5 of Arabidopsis thaliana.

232 to the Frost resistance-2 (Fr-Am2) locus on chromosome 5 of diploid wheat (Triticum monococcum) usin

233 Integration of EnPV into chromosome 5 of rats was confirmed by PCR cloning and se

234 a second form of the TFIIA small subunit on chromosome 5 of rice.

235 FC lyase is encoded by a gene on chromosome 5 of the Arabidopsis genome (FCLY, At5g63910)

236 d with the allelic state of the major QTL on chromosome 5, oligonucleotide array expression patterns

237 variant rs26232, in the first intron of the chromosome 5 open reading frame 30 (C5orf30) locus, has

238 unfavorable abnormalities (rearrangement of chromosome 5 or 7, or > or = 3 abnormalities) had a poor

239 g patients with partial/complete deletion of chromosome 5 or 7, or abnormalities of chromosome 3.

240 or gene lesions, and 18 had abnormalities of chromosome 5 or 7.

241 Loss of a whole chromosome 5 or a deletion of the long arm of chromosome

242 Loss of a whole chromosome 5 or a deletion of the long arm, del(5q), is

243 SNPs in the subset of patients with loss of chromosomes 5 or 7 or both, acquired abnormalities assoc

244 Abnormalities involving chromosomes 5 or 7 were found in 10 cases, which suggest

245 ncidence of TP53 mutations, abnormalities of chromosomes 5 or 7, complex cytogenetics and a reduced r

246 Loss of a whole chromosome 5, or a del(5q), are recurring abnormalities

247 Loss of a whole chromosome 5, or a del(5q), is a recurring abnormality i

248 arise by multiple mitotic cross-overs along chromosome 5 outside of the MTL region.

249 Tgfbm1 (chromosome 5, P = 8 x 10(-5)) and Tgfbm3 (chromosome 12,

250 ed the protocadherin gene family clusters on chromosome 5 (PCDHA, PCDHB, and PCDHG).

251 d strong evidence for susceptibility loci on chromosome 5 (per allele OR = 1.37 (95% CI = 1.19-1.58),

252 y, including a cluster of genes localized on chromosome 5 (PHT1;1, PHT1;2, and PHT1;3).

253 The gene located on chromosome 5 (PIMT2) produces two proteins differing by

254 ng-type (MTL) locus, not associated genes on chromosome 5, provides a competitive advantage.

255 a conventional backcross but also resolved a chromosome 5 QTL identified in the backcross into two QT

256 The identification of the chromosome 5 quantitative trait locus through linkage to

257 al chromosome clones from the syntenic mouse chromosome 5 region that contains Gtf2ird1 and Gtf2i as

258 A recessive mutation in the sim locus on chromosome 5 results in clusters of adjacent trichomes t

259 /NM_144715 on chromosome 3, rs9127/Q7Z4C4 on chromosome 5, rs1445898/CAPSL on chromosome 5 and rs2302

260 de significant locus was found in ANKRD55 on chromosome 5 (rs159572; odds ratio [OR], 1.62; 95% CI, 1

261 bon source, l-sorbose, by reversible loss of chromosome 5, serves as a model system.

262 y are related to rice genes on syntenic rice chromosome 5 short arm (5S).

263 on lines have regular transmission; however, chromosome 5 showed diminished paternal transmission, an

264 on chromosome 1 and a recessive CBA locus on chromosome 5; significantly, there was an epistatic inte

265 ide significant signal (p=1.12 x 10(-10)) on chromosome 5 spanning three genes: MSH3, DHFR, and MTRNR

266 panning approximately 5.8 cM) and another on chromosome 5 (spanning approximately 25.6 cM) resulted i

267 e report that CYP3a13 gene, located on mouse chromosome 5, spans 27.5 Kb and contains 13 exons.

268 in situ hybridization (FISH) studies using a chromosome 5-specific whole chromosome painting probe an

269 o completely sequenced versions of the large chromosome-5-specific internal duplications.

270 l-sorbose, was determined by copy number of chromosome 5, such that monosomic strains utilized l-sor

271 remained moderate in intestines carrying PWD chromosome 5, suggesting that PWD variants restrict aden

272 n site was localized to the region of distal chromosome 5 syntenic to the region on human chromosome

273 eport the finding of another gene, Slit2, on chromosome 5 that also accounts for variation in HSC num

274 emory task and identified a genetic locus on chromosome 5 that contributes to a substantial proportio

275 y the analysis of a 180 kb interval on human chromosome 5 that encodes for the kinesin family member

276 region on Arabidopsis (Arabidopsis thaliana) chromosome 5 that includes 106 genes.

277 on chromosome 18 and suggest a new locus on chromosome 5 that was not identified using traditional l

278 CDH)-alpha, -beta and -gamma gene cluster on chromosome 5 that were associated with nicotine withdraw

279 re constructed by the loss of one homolog of chromosome 5, the site of the MTL loci, MTLa and MTLalph

280 ations from the ancestral homolog of macaque chromosome 5 to macaque chromosomes 1 and 6.

281 of a major female heterogametic ZW locus on chromosome 5, two separate male heterogametic XY loci on

282 ed and characterized a gene, Mospd3 on mouse chromosome 5 using gene trapping in ES cells.

283 Suggestive evidence of linkage to chromosome 5 was also found for rMBP, to chromosome 16 f

284 sition disease, a region on the short arm of chromosome 5 was found to be genetically linked to the p

285 knox10, which is located on the short arm of chromosome 5, was shown to be ectopically expressed in d

286 To identify a leukemia-related gene on chromosome 5, we previously delineated a 970-kb segment

287 High frequencies of loss on chromosome 5 were identified at loci D5S428 (48%), D5S42

288 f modifier candidates and DNA methylation on chromosome 5 were predominantly cis controlled and large

289 imately 80 kb and was used for sequencing of chromosome 5, were arbitrarily selected for analysis.

290 ochromosome composed of the two left arms of chromosome 5, were associated with azole resistance.

291 ent signals within the TERT-CLPTM1L locus on chromosome 5, which has previously been associated with

292 evidence that this formation of monosomy of chromosome 5, which is apparently a result of nondisjunc

293 e gene within the homologous region on human Chromosome 5, which is linked to systolic and diastolic

294 ossesses a single mating-type (MTL) locus on chromosome 5, which is normally heterozygous (a/alpha).

295 s to isolate the effects of the major QTL on chromosome 5 while holding the minor QTL constant.

296 o families with 46,XY DSD to the long arm of chromosome 5 with a combined, multipoint parametric LOD

297 rovided "suggestive" evidence of a region on chromosome 5 with pleiotropic effects on both traits (ML

298 ized the disease interval to the long arm of chromosome 5, with a maximum two-point parametric LOD sc

299 sociations of ABR thresholds with markers on chromosome 5, with a peak lod score of 5.5 for D5Mit309.

300 accepted threshold for suggestive linkage on chromosomes 5, X, and 19.