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1 CD2 repair factor, and a higher frequency of chromosome aberrations.
2 pendent, RAD51-independent BIR and extensive chromosome aberrations.
3 way is required to prevent DNA mutations and chromosome aberrations.
4 reases in both spontaneous mutation rate and chromosome aberrations.
5 ads to aneuploidy, translocations, and other chromosome aberrations.
6 mples while enhancing the detection of fetal chromosome aberrations.
7 henotypes are due to genetic inheritance and chromosome aberrations.
8 ical advances that have come from studies of chromosome aberrations.
9 which showed a nonrandom pattern of multiple chromosome aberrations.
10  cells, which continue proliferating despite chromosome aberrations.
11             Ionizing radiation produces many chromosome aberrations.
12 cell killing, sister chromatid exchange, and chromosome aberrations.
13 cies by analysing 15 cases with unidentified chromosome aberrations.
14 A double-strand break repair [3] and develop chromosome aberrations [4].
15  of BRIP1 leads to a substantial increase in chromosome aberrations, a characteristic phenotype of ce
16                                              Chromosome aberration analysis reveals numerous incomple
17 eterogeneity, we performed exome sequencing, chromosome aberration analysis, and ploidy profiling on
18                 Our results provide detailed chromosome aberration and cell death dynamics in (137)Cs
19  as evidenced by the presence of aneuploidy, chromosome aberration and centrosome amplification.
20 y biological endpoints (DNA repair kinetics, chromosome aberration and mutation formation, survival)
21  irradiation of these cells led to increased chromosome aberrations and aneuploidy when compared with
22  susceptible to 3MeA-induced S phase arrest, chromosome aberrations and apoptosis, but it is not know
23 resent about 1/20 of all balanced structural chromosome aberrations and can lead to disease by gene d
24 his study, we present time-course studies of chromosome aberrations and cell death in zebrafish embry
25 RD reduced the frequency of both G1 and S/G2 chromosome aberrations and enhanced the disappearance of
26              Cells from A-T patients exhibit chromosome aberrations and excessive spontaneous apoptos
27 sion of Myc resulted in increased numbers of chromosome aberrations and gammaH2AX foci in non-transfo
28               Using the Mitelman Database of Chromosome Aberrations and Gene Fusions in Cancer, we de
29 emonstrated a marked increase in spontaneous chromosome aberrations and HPRT mutations, indicating a
30 e-negative breast cancers continue to evolve chromosome aberrations and maintain a reservoir of subcl
31 ible for male infertility, sex and autosomal chromosome aberrations and possible mental, physical and
32 y, and suggest mechanisms by which T induces chromosome aberrations and promotes the immortalization
33 onsidered by workers in the field concerning chromosome aberrations and psychoses etiology.
34  including transposon activation and various chromosome aberrations and rearrangements, that unfolded
35 s spontaneous and replication stress-induced chromosome aberrations and tumor predisposition.
36 on, severe G(2)/M cell cycle arrest, massive chromosome aberration, and defects in ionizing radiation
37 e complex proteins, (4) elevated MMC-induced chromosome aberrations, and (5) sensitivity to MMC and c
38 eased number of centrosomes, acquire complex chromosome aberrations, and lack Rad51 nuclear foci in t
39 ypersensitivity to DNA cross-linking agents, chromosome aberrations, and reduced FANCD2 monoubiquitin
40 imizing the generation of DNA strand breaks, chromosome aberrations, and the ensuing apoptotic respon
41                                      Complex chromosome aberrations are characteristically induced af
42                                              Chromosome aberrations are common outcomes of exposure t
43                      Microscopically visible chromosome aberrations are due to mutations that distort
44 Mutations in genes on the X chromosome rival chromosome aberrations as a cause of mental retardation.
45 ion is a new approach to identify structural chromosome aberrations associated with cancer.
46 , there were significantly more BPDE-induced chromosome aberrations at the 3p21.3 locus in cases (51.
47                                     Over 650 chromosome aberration breakpoints map to this chromosome
48                                 Induction of chromosome aberrations by DNA damage generates cells wit
49 opoisomerase I has been suspected of causing chromosome aberrations by mediating illegitimate recombi
50 rosophila, mutations in the dPdxk gene cause chromosome aberrations (CABs) and increase glucose conte
51 This order was consistent with the extent of chromosome aberrations (caMCF > bcMCF >>> trMCF).
52  other cytogenetic-based information such as chromosome aberrations can be linked to this framework.
53 nizing radiation is an established source of chromosome aberrations (CAs).
54 in mitochondria and nucleus, accumulation of chromosome aberrations, cell cycle delay, and elevated s
55                      Thus, whether numerical chromosome aberrations dictate disease progression in pr
56 e subcultured strains had very high rates of chromosome aberrations: duplications, deletions, and tra
57 cohesion, which selectively eliminates gross chromosome aberrations during mitosis.
58 f both Rad52 and BRCA2 resulted in extensive chromosome aberrations, especially chromatid-type aberra
59 t there was a significant increase in stable chromosome aberrations evaluated by multicolor fluoresce
60 ated levels of G4 structures, DNA damage and chromosome aberrations following treatment with PhenDC3,
61 se observations advance our understanding of chromosome aberration formation and have implications fo
62 c instability was exemplified by aneuploidy, chromosome aberrations, gene amplification and centrosom
63 ll as genetic and epigenetic alterations and chromosome aberrations, have been shown to contribute to
64 ocated at 11q23, are among the most frequent chromosome aberrations in acute myeloid leukemia (AML).
65 ion and assay design of inversions and other chromosome aberrations in diverse taxa.
66 anding in this study and resolved additional chromosome aberrations in every patient studied except t
67                                              Chromosome aberrations in human solid tumors are hallmar
68 hort similarly showed a greater influence of chromosome aberrations in light-eyed individuals.
69 ach by demonstrating that elevated levels of chromosome aberrations in lymphocytes are associated wit
70 ing the International Space Station program, chromosome aberrations in lymphocytes of astronauts have
71 ed clustered damage manifested a spectrum of chromosome aberrations in mitosis.
72                                              Chromosome aberrations in peripheral blood lymphocytes h
73 performed three-color FISH to detect numeric chromosome aberrations in testicular tissue samples from
74 lutionary bottleneck, and further identified chromosome aberrations in the persistent subclones that
75 ammed but may result from the segregation of chromosome aberrations in the postirradiation generation
76         Cytogenetic analysis revealed clonal chromosome aberrations in three of the five cell lines.
77 f the G-rich telomeric strand contributes to chromosome aberrations in WS cells, demonstrating a link
78 alysis demonstrated the presence of numerous chromosome aberrations including dicentric chromosomes,
79 rs or days after exposure, and appearance of chromosome aberrations (including dicentrics and ring fo
80 retrovirus suffered numerical and structural chromosome aberrations, including increases in aneuploid
81   Loss of both helicases exacerbates TDs and chromosome aberrations, indicating that BLM and WRN func
82                              We found clonal chromosome aberrations involving 2p23 upon metaphase ana
83 ing is not sensitive enough to detect subtle chromosome aberrations (<5-10 Mb).
84    Human tumors frequently display recurrent chromosome aberrations, many of which are hallmarks of p
85 th the hypothesis that detection of specific chromosome aberrations may be a powerful approach to ide
86                                              Chromosome aberrations may cause cancer and many heritab
87                                   Therefore, chromosome aberration might be used as a prognostic fact
88                           We hypothesize how chromosome aberrations might cause disease but the gene
89 transposons (Ty or delta-repeats), and these chromosome aberrations nonrandomly involved chromosome I
90 mulation of DNA strand breaks and results in chromosome aberrations observed in mitosis, ultimately r
91                 This mutation, named chaos1 (chromosome aberration occurring spontaneously 1), was ge
92                                  chaos1 (for chromosome aberrations occurring spontaneously 1) is a r
93 ouse chromosome instability mutation Chaos3 (chromosome aberrations occurring spontaneously 3), isola
94 to cancer are known to result in large-scale chromosome aberrations, our results suggest that small-
95               Genetic analysis revealed that chromosome aberrations, particularly combined monosomy 3
96                            Two new recurring chromosome aberrations previously not reported in T-ALL
97 n of 1 alpha-particle/cell) and analyzed the chromosome aberrations produced by using 24-color multip
98 p'99, Davis Human-Mouse Homology Map, Cancer Chromosome Aberration Project (CCAP) pages, Entrez Genom
99 e, HomoloGene, ProtEST, dbMHC, dbSNP, Cancer Chromosome Aberration Project (CCAP), Entrez Genomes and
100 ms (dbSNP), Human/Mouse Homology Map, Cancer Chromosome Aberration Project (CCAP), Entrez Genomes and
101  exclamation markVMouse Homology Map, Cancer Chromosome Aberration Project (CCAP), Entrez Genomes, Cl
102 GeneMap'99, Human-Mouse Homology Map, Cancer Chromosome Aberration Project (CCAP), Entrez Genomes, Cl
103 l rearrangements within cancer cells (Cancer Chromosome Aberration Project) are also being cataloged
104                                    Recently, chromosome aberrations provided new insights into pathog
105          Exposure to IR led to more residual chromosome aberrations, radioresistant DNA synthesis (a
106  the ex vivo dose-response of the pre-flight chromosome aberration rate (CAR), contributes to the pre
107 udies revealed an unusually high spontaneous chromosome aberration rate, contrasting with other FA su
108           The authors identify a hotspot for chromosome aberrations reminiscent of fragile sites in h
109 ng System (INRGSS) and incorporate segmental chromosome aberrations (SCA) as an additional genomic bi
110 , relapses occurred independent of segmental chromosome aberrations (SCAs); only 1p loss decreased EF
111 ent thymocytes are susceptible to developing chromosome aberrations that predispose to malignant tran
112 ID1A inactivation is selective against gross chromosome aberrations through causing defects in telome
113 P1 leads to enhanced sensitivity to and also chromosome aberrations upon DNA damage, demonstrating a
114 ye color SNPs with tumor characteristics and chromosome aberrations using Pearson's chi-square test a
115                                              Chromosome aberrations were enumerated during in vitro a
116                      Interestingly, although chromosome aberrations were evident, aneuploidy was not
117 be to telomeric DNA, spontaneously occurring chromosome aberrations were examined for telomere signal
118                                 In contrast, chromosome aberrations were seen in 79% of early MDS sam
119          F96-224 cells also display elevated chromosome aberrations when compared with control cells.
120   All samples showed complex karyotypes with chromosome aberrations which, in most cases, were not fu
121  biological dosimeters (e.g., cell survival, chromosome aberrations), which can be used to measure th
122 cytes give rise to regenerative nodules with chromosome aberrations, which are enhanced by ploidy red
123 diploid or near diploid but exhibit multiple chromosome aberrations with an average of 4 abnormal chr
124  evaluating the CometAssay, micronuclei, and chromosome aberrations with multicolor fluorescent in si
125            Conclusion: An increase in stable chromosome aberrations with respect to baseline is detec
126 ities of all agents were tested by examining chromosome aberration yields in first-division metaphase

 
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