ライフサイエンスコーパス: cinerea

1 ughout the brain of the green treefrog (Hyla cinerea).

2 using the parameters of a Grey Heron (Ardea cinerea).

3 with berries infected by the fungus Botrytis cinerea.

4 in driving time-of-day susceptibility to B. cinerea.

5 e radicals, and reduced susceptibility to B. cinerea.

6 o the necrotrophic fungal pathogen, Botrytis cinerea.

7 ant wrky33 is highly susceptible to Botrytis cinerea.

8 1 than in wild-type plants in response to B. cinerea.

9 of the most fungitoxic compounds against B. cinerea.

10 n infect and cause hypovirulence in Botrytis cinerea.

11 P13 was induced in leaves challenged with B. cinerea.

12 ses hypovirulence in Sclerotinia spp. and B. cinerea.

13 of the necrotrophic fungal pathogen Botrytis cinerea.

14 ion against the necrotrophic fungus Botrytis cinerea.

15 s plant host and the plant pathogen Botrytis cinerea.

16 DF1.2, and confers enhanced resistance to B. cinerea.

17 to the necrotrophic fungal pathogen Botrytis cinerea.

18 than wild-type plants to the fungus Botrytis cinerea.

19 sclerotiorum and the related fungus Botrytis cinerea.

20 us niger, Trichoderma harzianum and Botrytis cinerea.

21 lity of the ERF6-EAR transgenic plants to B. cinerea.

22 c acid is essential for the resistance to B. cinerea.

23 t the necrotrophic fungal pathogen, Botrytis cinerea.

24 the response of the transgenic plants to B. cinerea.

25 psidis, and the necrotrophic fungus Botrytis cinerea.

26 lings were infected with the fungus Botrytis cinerea.

27 by the necrotrophic fungal pathogen Botrytis cinerea.

28 ense toward the necrotrophic fungus Botrytis cinerea.

29 rks mediating the Arabidopsis response to B. cinerea.

30 mato DC3000 and the fungal pathogen Botrytis cinerea.

31 rd the necrotrophic fungal pathogen Botrytis cinerea.

32 nduced by the necrotrophic pathogen Botrytis cinerea.

33 oth exhibited decreased susceptibility to B. cinerea.

34 by the necrotrophic fungal pathogen Botrytis cinerea.

35 nd the necrotrophic fungal pathogen Botrytis cinerea.

36 enhanced susceptibility of ssi2 plants to B. cinerea.

37 are attenuated in ripe fruit infected by B. cinerea.

38 ally epistatic to rst1, for resistance to B. cinerea.

39 erexpressing line inhibited tip growth of B. cinerea.

40 bility to the necrotrophic pathogen Botrytis cinerea.

41 B1 overexpression conferred resistance to B. cinerea.

42 disease symptoms when infected with Botrytis cinerea.

43 r resistance to the fungal pathogen Botrytis cinerea.

44 to necrotrophic pathogens, such as Botrytis cinerea.

45 camalexin production upon infection with B. cinerea.

46 three displayed an enhanced resistance to B. cinerea.

47 sis resistance to the necrotrophic fungus B. cinerea.

48 abiotic stress and in the pathogenesis of B. cinerea.

49 to a necrotrophic fungal pathogen, Botrytis cinerea.

50 nd the necrotrophic fungal pathogen Botrytis cinerea.

51 more susceptible to both P. syringae and B. cinerea.

52 on against the fungal phytopathogen Botrytis cinerea.

53 ingae and the necrotrophic pathogen Botrytis cinerea.

54 induced resistance in Arabidopsis against B. cinerea.

55 ty against the fungal phytopathogen Botrytis cinerea.

56 of the inclusion complexes against Botrytis cinerea.

57 ut not to the necrotrophic pathogen Botrytis cinerea.

58 e grape (Vitis vinifera) berries by Botrytis cinerea.

59 abscisic acid (ABA) in resistance towards B. cinerea 2100.

60 reased resistance toward gray mold (Botrytis cinerea), a pathogen responsible for major losses in agr

61 n of oxi-mC across the genome of Coprinopsis cinerea, a basidiomycete that encodes 47 TET/JBP paralog

62 However, reduced susceptibility to Botrytis cinerea, a major postharvest fungal pathogen of tomato,

63 ae Inhibition of EXO70 by ES2-14 in Botrytis cinerea also reduces its virulence in Arabidopsis (Arabi

64 V1/WF-1 virions into strain KY-1 of Botrytis cinerea also resulted in reductions in virulence and myc

65 to several plant pathogens (namely Botrytis cinerea, Alternaria brassicicola and Golovinomyces oront

66 economically important necrotrophic fungi B. cinerea, Alternaria brassicicola, Fusarium graminearum,

67 ptosis in females of the cockroach Nauphoeta cinerea, an insect with reproductive cycles.

68 ected better against infections with both B. cinerea and A. brassicicola.

69 ptibility to the necrotrophic fungi Botrytis cinerea and Alternaria brassicicola as well as the gener

70 lity to the necrotrophic fungal pathogens B. cinerea and Alternaria brassicicola based on increased p

71 o the necrotrophic fungal pathogens Botrytis cinerea and Alternaria brassicicola concomitant with red

72 o the necrotrophic fungal pathogens Botrytis cinerea and Alternaria brassicicola in the Nossen-0 back

73 o the necrotrophic fungal pathogens Botrytis cinerea and Alternaria brassicicola, whereas HUB1 overex

74 o the necrotrophic fungal pathogens Botrytis cinerea and Alternaria brassicicola.

75 o the necrotrophic fungal pathogens Botrytis cinerea and Alternaria brassicicola.

76 o the necrotrophic fungal pathogens Botrytis cinerea and Alternaria brassicicola.

77 ceptibility to the pathogenic fungi Botrytis cinerea and Alternaria brassisicola Both PAMPs and osmot

78 responses to the fungal necrotrophs Botrytis cinerea and Alternaria solani, bacterial pathogen Pseudo

79 tibility to the necrotrophic fungus Botrytis cinerea and an increased tolerance to the biotrophic Pse

80 resistance to the fungal pathogens Botrytis cinerea and Bipolaris sorokiniana but not to the bacteri

81 stance to the necrotrophic pathogen Botrytis cinerea and contributes to basal defense induced by flg2

82 -type locus of the model species Coprinopsis cinerea and encodes two tightly linked pairs of homeodom

83 Botrytis cinerea and Erysiphe necator are among the most relevant

84 s against the necrotrophic pathogen Botrytis cinerea and for the shade-triggered increased susceptibi

85 duced and assayed for their resistance to B. cinerea and glucose transport activity.

86 The genomes of C. cinerea and Laccaria bicolor, a symbiotic basidiomycete,

87 ceptible to the necrotrophic fungus Botrytis cinerea and less tolerant to salt stress.

88 lium italicum, Rhizopus stolonifer, Botrytis cinerea and Monilinia fructicola, all of which cause rel

89 The shared function of fHbp in N. cinerea and N. meningitidis and cross-reactive responses

90 Nonpathogenic Neisseria strains (Neisseria cinerea and Neisseria flavescens) do not activate NLRP3

91 s has the potential to affect carriage of N. cinerea and other commensal species.

92 s a negative impact on resistance against B. cinerea and P. carotovorum.

93 m pathogens (phytopathogenic fungus Botrytis cinerea and pathogenic bacteria, respectively).

94 of defense genes and resistance to Botrytis cinerea and Pectobacterium carotovorum infection.

95 Slshn3-RNAi plants are more sensitive to B. cinerea and produce more hydrogen peroxide than wild-typ

96 t defenses against foliar pathogens Botrytis cinerea and Pseudomonas syringae, which normally result

97 of differing surface properties (Coprinopsis cinerea and Pythium ultimum) were employed.

98 Moreover, B. cinerea and S. sclerotiorum mycelial growth was reduced

99 BOI expression was enhanced by B. cinerea and salt stress but repressed by the plant hormo

100 virulence of the necrotrophic fungi Botrytis cinerea and Sclerotinia sclerotiorum.

101 rongest immunoreactivity in CA2 and fasciola cinerea and sporadic immunoreactivity in CA1; labeling i

102 diated resistance to the necrotroph Botrytis cinerea and susceptibility to the hemibiotroph Pseudomon

103 ce of a chemical cross-regulation between B. cinerea and T. arundinaceum and contributes to understan

104 ression conferred increased resistance to B. cinerea and T. ni.

105 to the necrotrophic foliar pathogen Botrytis cinerea and the biotrophic bacterial pathogen Xanthomona

106 sistance to the necrotrophic fungus Botrytis cinerea and the caterpillar Mamestra brassicae In additi

107 abundance of cospin in fruiting bodies of C. cinerea and the lack of trypsin-like proteases in the C.

108 e plant fungal pathogens, including Botrytis cinerea and three Fusarium spp.

109 tibility to the necrotrophic fungus Botrytis cinerea and to feeding by larvae of tobacco hornworm (Ma

110 saic virus as well as to the fungus Botrytis cinerea and to P. syringae.

111 e as the recipient Br-0 to the necrotroph B. cinerea and to the biotroph Hyaloperonospora arabidopsid

112 PA production in response to necrotrophic B. cinerea and virulent Pst DC3000 infection, but contribut

113 hal growth of most germinating conidia of B. cinerea and was eventually lethal to infected hyphae, si

114 two American species (Arizonica Texas, Vitis cinerea) and two interspecific crosses.

115 trains of the generalist necrotroph Botrytis cinerea, and have decreased camalexin production upon in

116 tibility to the necrotrophic fungus Botrytis cinerea, and increased sensitivity to salt and oxidative

117 tin matrix is the main CW target of Botrytis cinerea, and pectin methylesterification status is stron

118 Pathogens such as Monilinia spp., Botrytis cinerea, and Penicillium expansum are important fungi th

119 mutant exhibits reduced susceptibility to B. cinerea, and the B. cinerea dcl1 dcl2 double mutant that

120 sponses against the fungal pathogen Botrytis cinerea, and thus we conclude that the regulation of sym

121 the ripening-associated genes induced by B. cinerea are LePG (for polygalacturonase) and LeExp1 (for

122 The evaluation of Botrytis cinerea as noble rot on withered grapes is of great impo

123 DC3000 and the necrotrophic fungus Botrytis cinerea As pldgamma1 mutant plants responded with elevat

124 several PGs from the plant pathogen Botrytis cinerea as well as one from the saprotroph Aspergillus n

125 d the susceptibility of ripening fruit to B. cinerea, as measured by fungal biomass accumulation and

126 ulated, the nanoemulsions inhibited Botrytis cinerea at 110ppm of thymol.

127 cutin-defective mutants for resistance to B. cinerea: att1 (for aberrant induction of type three gene

128 Trichoderma arundinaceum (Ta37) and Botrytis cinerea (B05.10) produce the sesquiterpenoids harzianum

129 astingly affects resistance against Botrytis cinerea between the two species.

130 erium (BNCIN) harbored by the host Nauphoeta cinerea (Blaberidae).

131 activity and induced expression of Botrytis cinerea BOT genes, although their total antagonistic pot

132 olerance to the necrotrophic fungus Botrytis cinerea but susceptibility to the hemibiotrophic bacteri

133 tance to the necrotrophic fungal pathogen B. cinerea, but a negative role in the SA-dependent signali

134 to the necrotrophic fungal pathogen Botrytis cinerea, but showed normal responses to virulent and avi

135 (1) was assigned to a metabolite of Botrytis cinerea, but the spectra of several synthetic analogues

136 roteomics analyses have been performed in B. cinerea, but they cover only 10% of the total proteins p

137 13 contributes to the basal resistance to B. cinerea by limiting symptom development and points out t

138 rom the necrotrophic plant pathogen Botrytis cinerea, catalyzes the multistep cyclization of farnesyl

139 Pst-AvrRpt2, Dickeya dadantii, and Botrytis cinerea Characterization of the redox status demonstrate

140 hogenic fungi, the "phosphomembranome" of B. cinerea, combining the two most important signal transdu

141 SEP4 in the plant grey mould fungus Botrytis cinerea completely blocked IFS formation and abolished t

142 sistance to the necrotrophic fungus Botrytis cinerea, consistent with substantial upregulation of the

143 ced susceptibility to B. cinerea, and the B. cinerea dcl1 dcl2 double mutant that can no longer produ

144 We recently discovered that Botrytis cinerea delivers small RNAs (Bc-sRNAs) into plant cells

145 lants show differential susceptibility to B. cinerea depending on the time of day of inoculation.

146 cate that Sep4 plays pleiotropic roles in B. cinerea development and specifically facilities host inf

147 NA effectors are mostly produced by Botrytis cinerea Dicer-like protein 1 (Bc-DCL1) and Bc-DCL2.

148 and could detect as little as 2 copies of B. cinerea DNA.

149 we show that the commensal species Neisseria cinerea expresses functional fHbp on its surface and tha

150 N. cinerea fHbp binds CFH with affinity similar to that of

151 s, which is required for full immunity to B. cinerea Finally, we present a structural model of MOS7 a

152 The decreased susceptibility to B. cinerea following inoculation at subjective dawn was ass

153 udomonas syringae and to the fungus Botrytis cinerea Furthermore, bsk5 mutant plants were impaired in

154 0 mg/L against Rhizopus stolonifer, Botrytis cinerea, Fusarium oxysporum and Colletotrichum gloeospor

155 arately and together to down-regulate the B. cinerea genes analyzed.

156 esults in an up-regulation of most of the B. cinerea genes involved in virulence yet the presence of

157 Critically, we identified a subset of B. cinerea genes where allelic variation was linked to alte

158 the lack of trypsin-like proteases in the C. cinerea genome, these results suggest that cospin and it

159 nables genome-wide association mapping of B. cinerea Genome-wide association mapping of the pathogen

160 quantified variation in lesion size of 97 B. cinerea genotypes (isolates) on six domesticated tomato

161 Botrytis cinerea had a positive impact on fruity and floral notes

162 hronology of the defense response against B. cinerea, highlighting the times at which signaling and m

163 th OGs and macerozyme-induced immunity to B. cinerea in Col-0, only OGs also induced immunity in gae1

164 ibited germination in quiescent spores of B. cinerea In germlings, it breached the fungal plasma memb

165 ningococcal fHbp and promotes survival of N. cinerea in human serum.

166 inst Pectobacterium brasiliense and Botrytis cinerea in more than one plant species.

167 se symptoms caused by the fungal pathogen B. cinerea in tomato and tobacco plants, and postharvest pr

168 induced by the necrotrophic fungus Botrytis cinerea, including the genes that encode the transcripti

169 rabidopsis thaliana with the fungus Botrytis cinerea Indeed, in contrast to previous reports, most OG

170 oph H. schachtii and the necrotroph Botrytis cinerea, indicating a potential suppression of defenses

171 coi1) and ethylene-insensitive2 (ein2) to B. cinerea, indicating that ELP2 is an important player in

172 During noble rot, B. cinerea induced the expression of key regulators of ripe

173 g a four-amino acid deletion, compromises B. cinerea-induced activation of the key immunoregulatory M

174 t18:0-P appear as key players in Pst- and B. cinerea-induced cell death and reactive oxygen species a

175 pression (EAR) motif, strongly suppresses B. cinerea-induced defense gene expression, leading to hype

176 d-cultured seedling system, we found that B. cinerea-induced ethylene biosynthesis was greatly compro

177 eam substrates of MPK3/MPK6, also reduced B. cinerea-induced ethylene production.

178 asmonic acid (JA) and increased basal and B. cinerea-induced expression of the plant defensin PDF1.2

179 idopsis Elongator subunit 2 (ELP2) alters B. cinerea-induced transcriptome reprogramming.

180 Additionally, on unripe fruit, B. cinerea induces the expression of genes also expressed a

181 uiterpenes that were relatively higher in B. cinerea infected samples.

182 ne expression is similar in both types of B. cinerea-infected plants but is repressed in Atdpl1-1 aft

183 Further analysis revealed that B. cinerea-infected Slshn3-RNAi plants are more sensitive t

184 charides (PDOs) in three regions of Botrytis cinerea-infected tomato fruit tissue is described.

185 ssion of SlSHN3 resulted in resistance to B. cinerea infection and to X. campestris pv. vesicatoria,

186 DNA concentrations in the wine and Botrytis cinerea infection before harvest.

187 this study was to identify biomarkers of B. cinerea infection in sweet wines with a focus on laccase

188 thogens in vitro and significantly reduce B. cinerea infection in vivo.

189 ction transgenic plants or in response to B. cinerea infection increases ERF6 protein stability in vi

190 ated during abiotic stresses during Botrytis cinerea infection or after benzothiadiazole and methyl j

191 genic plants were more resistant to Botrytis cinerea infection than wild type, possibly as a conseque

192 direct inhibition, P. aphidis may inhibit B. cinerea infection via induced resistance in a manner ind

193 d more damage than wild type plants after B. cinerea infection, and pretreatment of plants with methy

194 exhibited reduced susceptibility to Botrytis cinerea infection, confirming AA signaling in other plan

195 P. aphidis also reduced B. cinerea infection, locally and systemically, in Arabidop

196 in a jasmonate resistant1-1 mutant, after B. cinerea infection, suggesting that P. aphidis can bypass

197 duced stronger activation of PDF1.2 after B. cinerea infection.

198 ential transcriptional reprogramming upon B. cinerea infection.

199 by MPK3/MPK6 in vivo in response to Botrytis cinerea infection.

200 t is transcriptionally regulated by Botrytis cinerea infection.

201 nce of Arabidopsis thaliana against Botrytis cinerea infection.

202 Arabidopsis to resist CW degradation upon B. cinerea infection.

203 Arabidopsis (Arabidopsis thaliana) during B. cinerea infection.

204 are dynamically modulated by PMEIs during B. cinerea infection.

205 cted Arabidopsis from S. sclerotiorum and B. cinerea infections.

206 ienoic acid (13-HPOT), 2 days after Botrytis cinerea inoculation.

207 JA-related defense gene expression after B. cinerea inoculation.

208 response networks may control A. thaliana-B. cinerea interaction in this population.

209 ond faster and in a more effective way to B. cinerea invasion.

210 The mushroom Coprinopsis cinerea is a classic experimental model for multicellula

211 Cospin (PIC1) from Coprinopsis cinerea is a serine protease inhibitor with biochemical

212 Botrytis cinerea is a well-studied necrotrophic fungus taken as a

213 We demonstrate that B. cinerea is able to actively absorb glucose and fructose

214 The chromosome assembly of C. cinerea is an essential resource in understanding the ev

215 g infection is higher and the immunity to B. cinerea is compromised in pmei10, pmei11, and pmei12 mut

216 Resistance to B. cinerea is compromised in the sib1 and sib2 mutants but

217 sistance to the necrotrophic fungus Botrytis cinerea is conferred by ethylene via poorly understood m

218 We show that reduced susceptibility to B. cinerea is dependent specifically on the accumulation of

219 uggest that PA-mediated susceptibility to B. cinerea is linked to interference with the functions of

220 e against the necrotrophic pathogen Botrytis cinerea is primarily quantitative and genetically comple

221 e that oxalate production in A. niger and B. cinerea is solely dependent on the hydrolytic cleavage o

222 Botrytis cinerea is the causing agent of the grey mold disease in

223 Botrytis cinerea isolates showed differing sensitivity to purifie

224 Resistance to specific Botrytis cinerea isolates was also compromised in gae1 gae6 doubl

225 Testing multiple B. cinerea isolates, we identified 23 separate QTL in this

226 to the susceptibility of wrky33 plants to B. cinerea, it is insufficient for WRKY33-mediated resistan

227 trait loci influencing plant response to B. cinerea, measured as expansion of necrotic lesions on le

228 d B. terrestris amplified Bd abundance on H. cinerea more so in the absence than presence of G. carol

229 , G. carolinensis reduced Bd abundance on H. cinerea more so in the presence than absence of B. terre

230 Examination of the constituents of a B. cinerea mutant that overproduces polyketides gave suffic

231 nd transcriptomes of leaves infected with B. cinerea mutants with reduced pectinolytic activity but w

232 ins (OT) were investigated in relation to B. cinerea negative effects in grapes and musts.

233 Below, we report the cloning of the Botrytis cinerea oahA gene and the demonstration that the disrupt

234 lementation we have shown that the intact B. cinerea oahA gene restores oxalate production in an Aspe

235 accumulation, and susceptibility to Botrytis cinerea, one of the most important postharvest pathogens

236 potentiated by RLs following challenge by B. cinerea or P. syringae pv tomato.

237 Genetic disruption of the B. cinerea oscillator by mutation, overexpression of BcFRQ1

238 e proteins play a significant role in the B. cinerea pathogenic cycle.

239 Here we used the Arabidopsis-B. cinerea pathosystem to test how plant host and fungal pa

240 ore resistant to the phytopathogens Botrytis cinerea, Pectobacterium carotovorum, and Pseudomonas syr

241 grape berries were inoculated with Botrytis cinerea, Penicillium expansum, Aspergillus niger or A. c

242 ing influences the course of infection by B. cinerea, perhaps by changing the structure or the access

243 Infiltration of B. cinerea PGs into Arabidopsis accession Columbia induced

244 pecially necrotrophic fungi such as Botrytis cinerea, produce high levels of ethylene.

245 , including resistance to P. syringae and B. cinerea, production of reactive oxygen species, callose

246 lturally important plant pathogens (Botrytis cinerea, Pseudomonas syringae, and Fusarium oxysporum) w

247 Adult green treefrogs, Hyla cinerea, received injections of BrdU and were sacrificed

248 Macerozyme treatment or infection with B. cinerea released less soluble uronic acid, likely reflec

249 Thus, to infect fruit, B. cinerea relies on some of the processes and events that

250 lysis revealed flg22-induced PTI to Botrytis cinerea requires BIK1, EIN2, and HUB1 but not genes invo

251 sms by which PP2A-B'gamma regulates Botrytis cinerea resistance and leaf senescence in Arabidopsis (A

252 RELATED GENES (BRGs), which contribute to B. cinerea resistance and the suppression of disease-associ

253 YC2 fail to restore PDF1.2 expression and B. cinerea resistance in elp2, suggesting that ELP2 is requ

254 ating that ELP2 is an important player in B. cinerea resistance.

255 aps with COI1 and is additive to EIN2 for B. cinerea resistance.

256 icola resistance but additive to HUB1 for B. cinerea resistance.

257 response to the necrotrophic fungus Botrytis cinerea revealed decreases in the levels of phosphatidyl

258 oes with this coating and inoculated with B. cinerea showed a significant decrease in fungal growth a

259 Here, we show that some B. cinerea small RNAs (Bc-sRNAs) can silence Arabidopsis an

260 Germination of B. cinerea spores on sma4 mutant leaves was inhibited, and

261 st obtained by inoculation of grapes with B. cinerea strain 213.

262 The B. cinerea strain was also grown in a liquid medium for lac

263 een hydrophobic T4 phages and hydrophobic C. cinerea surfaces.

264 e in necrotropic pathogen susceptibility, B. cinerea susceptibility was assessed in transgenic fruit

265 We analyzed the role of HA and Asp in the B. cinerea-T. arundinaceum interaction, including changes i

266 nic tomato lines were more susceptible to B. cinerea than the wild-type plants; however, responses to

267 nt to P. syringae but more susceptible to B. cinerea than wild-type plants.

268 ree mushroom TET homologues from Coprinopsis cinerea that can mediate 5mC oxidation.

269 tify only in infected berries proteins of B. cinerea that represent potential markers of the presence

270 ce against the necrotrophic fungus, Botrytis cinerea The induced resistance was enhanced in the P2K1

271 immunity to the necrotrophic fungus Botrytis cinerea The mos7-1 mutation, causing a four-amino acid d

272 When transformed into C. cinerea, the C. disseminatus A and B homologs elicited s

273 Botrytis cinerea, the causative agent of gray mold disease, is an

274 ection with Pseudomonas syringae or Botrytis cinerea, the expression of genes regulated by both the s

275 e, we demonstrate that Jar1/KDM5 in Botrytis cinerea, the grey mould fungus, plays a crucial role in

276 arkedly increased plant susceptibility to B. cinerea; the effect of low R:FR was (1) independent of t

277 d by an increased susceptibility to Botrytis cinerea This process was accompanied by an overexpressio

278 cadian system of the plant pathogen Botrytis cinerea to assess if such oscillatory machinery can modu

279 copyranoside (Q3G) isolated from Echinophora cinerea to protect PC12 cells from H2O2-induced cytotoxi

280 ne silent and diseased (infected by Botrytis cinerea) tomato leaves.

281 ar dialogue between Arabidopsis cells and B. cinerea triggers major changes in host metabolism, inclu

282 aliana with the necrotrophic fungus Botrytis cinerea using millicell culture insert, that enables mol

283 ility of the ySpdSyn transgenic tomato to B. cinerea was associated with down-regulation of gene tran

284 Induction of ATG18a and autophagy by B. cinerea was compromised in the wrky33 mutant, which is h

285 and all the virulence genes analyzed when B. cinerea was grown alone.

286 ultimum and the filamentous fungus Botrytis cinerea was inhibited.

287 The 37-megabase genome of C. cinerea was sequenced and assembled into 13 chromosomes.

288 tion of these pathways to defence against B. cinerea was validated through the use of multiple Arabid

289 ial impact of fHbp-containing vaccines on N. cinerea We found that immunization with Bexsero elicits

290 pulation of the generalist pathogen Botrytis cinerea We quantified variation in lesion size of 97 B.

291 uired for Arabidopsis resistance to Botrytis cinerea were isolated.

292 expression and basal resistance to Botrytis cinerea were restored.

293 genic fungi, Fusarium oxysporum and Botrytis cinerea, were chosen to examine the antifungal activity

294 highly susceptible to A. brassicicola and B. cinerea, whereas T-DNA insertion alleles are embryonic l

295 sis for tadpoles of Bufo terrestris and Hyla cinerea, whereas tadpoles of B. terrestris (an obligate

296 icits serum bactericidal activity against N. cinerea, which is primarily directed against fHbp.

297 ow the existence of a functional clock in B. cinerea, which shares similar components and circuitry w

298 to infection by the fungal pathogen Botrytis cinerea, which was associated with much stronger inducti

299 f the coprophilous basidiomycete Coprinopsis cinerea with different bacterial species and identified

300 rhythmic susceptibility of Arabidopsis to B. cinerea with the enhanced susceptibility to this pathoge