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1 e measure was the frequency of an incomplete circle of Willis.
2 ximal contrast-to-noise ratios (CNRs) in the circle of Willis.
3 light (TOF) acquisition was performed in the circle of Willis.
4 teral proximal middle cerebral artery of the circle of Willis.
5 aving or not having occlusive disease at the circle of Willis.
6 icating artery (PcomA) hypoplasticity in the circle of Willis.
7 omatic acute occlusions of the ICA below the circle of Willis.
8 mulation was demonstrated, especially in the Circle of Willis.
9 er characterized by occlusive lesions of the circle of Willis.
10 sonance angiography was performed to examine circle of Willis anatomy and arterial spin labeled perfu
13 tically classify important structures of the Circle of Willis and extract biomarkers from cerebrovasc
15 aneurysms are particularly vulnerable in the circle of Willis and pose a significant concern due to t
16 n individualised 3D representation of a full circle of Willis and then construct two further domains
18 ble, segmental, cerebral vasoconstriction of circle of Willis arteries and normal or near-normal resu
20 ith Alzheimer's disease had grossly apparent circle of Willis atherosclerosis, a percentage that was
21 nts than controls, while vascular pathology, Circle of Willis atherosclerosis, Lewy body pathology, l
22 hology and lacunar infarcts, large infarcts, Circle of Willis atherosclerosis, or cerebral amyloid an
23 the internal carotid artery (ICA) below the circle of Willis can cause a variety of stroke symptoms,
24 n revealed dolichoectasia of arteries of the circle of Willis coexisting with a fusiform aneurysm of
25 e, presence of intraplaque hemorrhage (IPH), circle of Willis collaterals, and the presence and sever
26 ng that all ChAT-I fibers in the MCA and the circle of Willis contain NOS, and that these fibers orig
27 osis can cause emboli that travel across the Circle of Willis (CoW) which can potentially lead to amb
28 vestigated VSMC differentiation on zebrafish circle of Willis (CoW), comprising major arteries that s
30 l cerebral collateral circulation within the circle of Willis (CW) predisposes to ischemia and stroke
32 in three common structural variations of the circle of Willis during and in the moments after a block
33 re Pd is intracranial inflow pressure in the circle of Willis, ICP-intracranial pressure; FFR = Pd/Pa
34 on diverging waves can accurately render the circle of Willis in 3D and minimize human errors during
35 d severity of atherosclerotic plaques in the circle of Willis in Alzheimer's disease and multiple oth
37 r results indicate how, and how quickly, the circle of Willis is able to redistribute flow following
40 hat the middle cerebral artery (MCA) and the circle of Willis received dense ChAT-immunoreactive (I)
42 he brainstem and cerebellum but can, via the Circle of Willis upon stenosis of the internal carotid a
43 Using a quantitative score of the burden of circle of Willis variants, migraine with aura subjects h
47 pproach for categorizing and quantifying the Circle of Willis vascular anatomy using magnetic resonan
53 all 102 participants, atherosclerosis of the circle of Willis was strongly associated with lacunar an
54 tery hypoplasia, and an incomplete posterior circle of Willis, which were coupled with increased cere