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1 e measure was the frequency of an incomplete circle of Willis.
2 ximal contrast-to-noise ratios (CNRs) in the circle of Willis.
3 light (TOF) acquisition was performed in the circle of Willis.
4 teral proximal middle cerebral artery of the circle of Willis.
5 aving or not having occlusive disease at the circle of Willis.
6 icating artery (PcomA) hypoplasticity in the circle of Willis.
7 omatic acute occlusions of the ICA below the circle of Willis.
8 mulation was demonstrated, especially in the Circle of Willis.
9 er characterized by occlusive lesions of the circle of Willis.
10 sonance angiography was performed to examine circle of Willis anatomy and arterial spin labeled perfu
11 bally (over the whole brain) and locally (at circle of Willis and below).
12  of mean curvature) globally and locally (at circle of Willis and below).
13 tically classify important structures of the Circle of Willis and extract biomarkers from cerebrovasc
14                              Analysis of the Circle of Willis and its variants can shed light on opti
15 aneurysms are particularly vulnerable in the circle of Willis and pose a significant concern due to t
16 n individualised 3D representation of a full circle of Willis and then construct two further domains
17              The arterial connections in the Circle of Willis are a central source of collateral bloo
18 ble, segmental, cerebral vasoconstriction of circle of Willis arteries and normal or near-normal resu
19         The association between age-adjusted circle of Willis atherosclerosis and Alzheimer's disease
20 ith Alzheimer's disease had grossly apparent circle of Willis atherosclerosis, a percentage that was
21 nts than controls, while vascular pathology, Circle of Willis atherosclerosis, Lewy body pathology, l
22 hology and lacunar infarcts, large infarcts, Circle of Willis atherosclerosis, or cerebral amyloid an
23  the internal carotid artery (ICA) below the circle of Willis can cause a variety of stroke symptoms,
24 n revealed dolichoectasia of arteries of the circle of Willis coexisting with a fusiform aneurysm of
25 e, presence of intraplaque hemorrhage (IPH), circle of Willis collaterals, and the presence and sever
26 ng that all ChAT-I fibers in the MCA and the circle of Willis contain NOS, and that these fibers orig
27 osis can cause emboli that travel across the Circle of Willis (CoW) which can potentially lead to amb
28 vestigated VSMC differentiation on zebrafish circle of Willis (CoW), comprising major arteries that s
29                                          The circle of Willis (cW) is a major arterial collateral str
30 l cerebral collateral circulation within the circle of Willis (CW) predisposes to ischemia and stroke
31         We conclude that the pathobiology of circle of Willis disease in the child with sickle cell a
32 in three common structural variations of the circle of Willis during and in the moments after a block
33 re Pd is intracranial inflow pressure in the circle of Willis, ICP-intracranial pressure; FFR = Pd/Pa
34 on diverging waves can accurately render the circle of Willis in 3D and minimize human errors during
35 d severity of atherosclerotic plaques in the circle of Willis in Alzheimer's disease and multiple oth
36 the lower prevalence of abnormalities of the circle of Willis in MF1 mice.
37 r results indicate how, and how quickly, the circle of Willis is able to redistribute flow following
38                                An incomplete circle of Willis is more common in migraine with aura su
39           MF1 mice had significantly greater circle of Willis plasticity (more PcomAs) than C57Bl/6J
40 hat the middle cerebral artery (MCA) and the circle of Willis received dense ChAT-immunoreactive (I)
41 he middle cerebral artery from distal to the circle of Willis to deep regions of the midbrain.
42 he brainstem and cerebellum but can, via the Circle of Willis upon stenosis of the internal carotid a
43  Using a quantitative score of the burden of circle of Willis variants, migraine with aura subjects h
44 emplate rating system was used to categorize circle of Willis variants.
45                      The association between circle of Willis variations and cerebral blood flow was
46                                 Furthermore, circle of Willis variations predispose to aneurysm forma
47 pproach for categorizing and quantifying the Circle of Willis vascular anatomy using magnetic resonan
48                         The integrity of the circle of Willis was examined by carbon black perfusion
49                                   A complete circle of Willis was present in only one of 10 mice.
50                        The plasticity of the circle of Willis was recorded using carbon black perfusi
51                                An incomplete circle of Willis was significantly more common in the mi
52 ansfer through jugular venous return and the circle of Willis was simulated.
53 all 102 participants, atherosclerosis of the circle of Willis was strongly associated with lacunar an
54 tery hypoplasia, and an incomplete posterior circle of Willis, which were coupled with increased cere