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1 s the hyperdirect and indirect basal ganglia circuitry.
2 or evolution, and the rewiring of regulatory circuitry.
3 in neurons across Drosophila's visual motion circuitry.
4 mories might be implemented in insect neural circuitry.
5 ts microstructure and large-scale functional circuitry.
6 ne and posttreatment to examine brain reward circuitry.
7 ltifunctional players in the midbrain reward circuitry.
8 S effects of hormones on non-endocrine brain circuitry.
9 ween the insula cortex and the corticolimbic circuitry.
10 n for piriform cortex supported by recurrent circuitry.
11 e expression in the offspring's dopaminergic circuitry.
12 f inhibition in brainstem sound-localization circuitry.
13 long-lasting adaptations in reward and habit circuitry.
14 urons as the key component of hyperexcitable circuitry.
15  contributions to functional changes in this circuitry.
16 nd corresponding alterations in frontolimbic circuitry.
17 s the structure and function of brain reward circuitry.
18 linkage in raphe to hippocampus serotonergic circuitry.
19 rstanding of the complexities of spinal itch circuitry.
20 ing will require knowledge of the underlying circuitry.
21  found extensive graft integration with host circuitry.
22 en the visual cortices and mesolimbic reward circuitry.
23 nd therapeutic manipulation of acquired fear circuitry.
24 nses that are guided by nociceptive neuronal circuitry.
25 rafast and quantum optical signal processing circuitry.
26 potent repulsive barriers to construct novel circuitry.
27  PNs and thus the output from the cerebellar circuitry.
28 report to directly engage higher-order brain circuitry.
29 ing's end along with a capacitance measuring circuitry.
30  hippocampus only captures a mere 5% of this circuitry.
31 possible roles of SIN function in the tectal circuitry.
32 RNA expression throughout the brain's reward circuitry.
33 n kept separate from the pattern recognition circuitry.
34  and occurred independently of basal ganglia circuitry.
35  recruit mesocorticolimbic incentive-related circuitry.
36 ion in this input relates to preverbal brain circuitry.
37 ineate specific changes to dorsal horn input circuitry.
38  dissection of their function in spinal itch circuitry.
39 serves as a hub for thermo- and hygrosensory circuitry.
40 l modeling to tap into the underlying neural circuitry.
41 ult affective disorders involving the reward circuitry.
42 icularly for genes of high-relevance for DMN circuitry.
43 c interneurons are an essential node of such circuitry.
44 the need for photonic and electronic control circuitry.
45 onnections and functions of different neural circuitries.
46 he complex interactions of stress and reward circuitries.
47 mediated by neuroadaptations in brain reward circuitry, a comprehensive framework of how stress-induc
48 enervation in male and female mice to detect circuitry alterations underlying interhemispheric cortic
49                 To determine how dorsal horn circuitry alters to facilitate recovery post-injury, we
50 h altered activity across emotion regulation circuitry and a higher risk of developing anxiety disord
51 e maturation and synchronization of cortical circuitry and alterations in these inhibitory neurons, e
52 (F5) and (M1) provide direct input to spinal circuitry and are critical for skilled movement control.
53 r serotonin as a modulator of vocal-acoustic circuitry and behavior in midshipman fish, consistent wi
54 , how unconscious information impacts neural circuitry and behavior remains unknown.
55 ogabine (also known as retigabine) on reward circuitry and clinical symptoms in patients with MDD.
56        Despite marked changes in this neural circuitry and extinction learning throughout development
57 d crystal waveguides attractive for photonic circuitry and for studying hybrid light-matter states.
58 ical players in the regulation of cerebellar circuitry and function.
59 unctionally integrated into local excitatory circuitry and have heightened activity compared to adult
60 trols the flow of signals through interareal circuitry and how this communication might be modulated
61  MDD has been associated with altered reward circuitry and increased brain glutamate in relation with
62 ntified in critical components of the reward circuitry and interacting stress circuits.
63 s the structure and function of brain reward circuitry and is an important risk factor for developing
64 ng, engages prefrontal cortex (PFC)-striatal circuitry and is impaired in both manifest and premanife
65 rates adaptations in insula-ventral striatal circuitry and metabolic regulatory hormones that warrant
66 ntifying the effect of ketamine on the brain circuitry and networks is becoming increasingly critical
67 s of ketamine and their effects on the brain circuitry and networks.
68  that prefrontal cortical GABAergic synaptic circuitry and Npas4 are strongly implicated in 16p11.2 d
69 e basic organization of area postrema nausea circuitry and provide a framework toward understanding a
70 nula plays an important role in brain reward circuitry and psychiatric conditions.
71  activation of the brain's mesolimbic reward circuitry and release of the neurotransmitter dopamine t
72 nted to understand differences in the neural circuitry and subsequent influence on volumetrics after
73 tion of complex cutaneous receptor-emulating circuitry and the lack of an appropriate protocol to dis
74 d contribution of coupling motifs in retinal circuitry and the necessity of their detection in connec
75 omputational processes in auditory brainstem circuitry and to a more holistic understanding of modula
76 em matures to connect and shape the neuronal circuitry and to keep it functional in humans for decade
77 nctional connectivity of the stress response circuitry and variations in cardiovagal activity [normal
78 mportant for the formation of precise neural circuitry, and its disruption has been linked to neurode
79 hat upends current models of thalamocortical circuitry, and that might as well illuminate the functio
80  the maturation and operations of the reward circuitry, and the potential underlying mechanisms, rema
81             Although some components of this circuitry are known and conserved across animals, how th
82                              Defects in this circuitry are linked to neuropsychiatric diseases, inclu
83 lecularly defined pathways within the reward circuitry are particularly susceptible to early-life adv
84 Several fundamental aspects of motion vision circuitry are prevalent across flies and mice.
85 gdala perfusion, a key hub of the OT central circuitry, are explained entirely by OT increases in sys
86 s in the frontal-executive and corticolimbic circuitries as those with aMCI or AD, suggesting distinc
87  microbiome alterations on the fronto-limbic circuitry as a convergence hub for emotional dysregulati
88  The results also point to galanin-dependent circuitry as a potential substrate for the evolution of
89 nowledge on the role of BDNF within the fear circuitry, as well as address mounting evidence whereby
90  between disruptions in reward system neural circuitry associated with a history of depression (parti
91 we studied the adult Drosophila melanogaster circuitry associated with antennal thermo- and hygrosens
92 rganoid hair follicles, mimicking the neural circuitry associated with human touch.
93 ions between frontoparietal and limbic brain circuitry at rest, which may reflect a potential mechani
94  the regulated development of complex neural circuitry, but the underlying molecular mechanisms are u
95 natomically connected components of thalamus circuitry, but uncoupling from most other brain regions
96                                 However, the circuitry by which the BLA modulates ARC in multiple com
97                            Here, we describe circuitry by which these two are integrated into a compl
98   Our phosphoproteomic signatures of network circuitry can identify kinase topologies associated with
99                 We also show that adding RNA circuitry can invert responses, reduce crosstalk and imp
100 hat rely on the basal ganglia and associated circuitry) can explain numerous brain and behavioral cha
101 elective chemogenetic inactivation of DG-CA3 circuitry completely and reversibly abolishes the CA1 re
102                            Dysregulations of circuitries controlling hedonic feeding may disrupt home
103 ely studied, knowledge about transcriptional circuitries controlling human NK cell development and ma
104 e1 homolog that is part of a transcriptional circuitry controlling hyphal growth.
105 lly relevant rates, we find that the genetic circuitry controls the average final complexoform assemb
106 es reporting structural and functional brain circuitry correlates of STBs.
107                              Core regulatory circuitry (CRC)-dependent transcriptional network is cri
108  constitutes core transcriptional regulatory circuitry (CRC).
109 een different components of intrahippocampal circuitry derived from experimental pathway-tracing expe
110       Little is understood about dorsal horn circuitry, despite the fact that this region loses most
111                           In the hippocampal circuitry, detection fidelity is thought to depend on th
112 example of amygdala-medial prefrontal cortex circuitry development to illustrate a principle of human
113 ance of early amygdala and extended amygdala circuitry development to the emergence of anxiety.
114 sence of abnormalities in the frontostriatal circuitry, differences in synaptic ultra-structures by e
115 nstay paradigm for characterizing the neural circuitry driving object-centric actions.
116  tissue/organ-specific temperature signaling circuitry either downstream of photobody dynamics or ind
117 transfer to a variety of host substrates for circuitry fabrication has been among the most popular su
118 e hypothesis that the organization of neural circuitry favors computational flexibility: that it make
119 he proper stimulation target dependent brain circuitry for a DBS outcome.
120 ed among corticocortical and thalamocortical circuitry for the ability to learn new visuospatial asso
121 he basic architecture of cerebellar neuronal circuitry for their ability to acquire pOKR.
122 riptional regulator Nolz1 in establishing DA circuitry formation.
123 f the striatum in orchestrating dopaminergic circuitry formation.
124          Next-generation photonics envisions circuitry-free, rapidly reconfigurable systems powered b
125 tive relationships between markers of reward circuitry function and affective symptom trajectories.
126 fluences relationships between reward neural circuitry function and the course of future affective an
127                     Markers of reward neural circuitry function, specifically activation to reward pr
128 cted networks, i.e. mimicking both local and circuitry functionality of the brain.
129 n at cellular and molecular scales (neuronal circuitry, gene expression, and other properties).
130 ct the role of galectins in shaping cellular circuitries governing each hallmark of tumors, illustrat
131 es is central to understanding the molecular circuitry governing cellular processes.
132  AMPK signaling pathway and neuropeptidergic circuitry governing feeding behavior.
133  Hhex-Tle3 in regulating the transcriptional circuitry governing MBC differentiation.
134 RPR neurons and thus map an exquisite spinal circuitry hard-wired for converting innocuous touch to i
135            Our results suggest that neuronal circuitry has a substructure at far higher resolution th
136 lphaproteobacteria, the VtlR/LsrB regulatory circuitry has diverged in order to accommodate the uniqu
137                       The discovery of MISTR circuitry highlights the use of evolution-guided studies
138 espond to rod/cone input through the retinal circuitry, however, a small population of RGCs are in ad
139 arget engagement favorably impacts the brain circuitry hypothesized to mediate clinical effects.
140 mood- and sex-dependent alterations in brain circuitry implicated in the regulation of the stress res
141  PIK3CA wild-type cells adopt MAPK-dependent circuitries in breast cancer cells and that the kinase T
142 atter of frontal-executive and corticolimbic circuitries in five groups of older adults putatively at
143 phasize the involvement of mesocorticolimbic circuitries in mediating the effects of PPARgamma agonis
144 erstanding the impact of mesolimbic dopamine circuitry in acute and chronic pain.
145 ed to integrate long-range inputs with local circuitry in frontal cortex to implement top-down attent
146 pecific prenatal influence over social brain circuitry in humans and may be relevant towards explaini
147 highlight the potential importance of reward circuitry in ketamine's mechanism of action, which may b
148 e achievable via small adjustments of neural circuitry in key brain areas.
149 EMENT Lateral inhibition is a key feature of circuitry in many sensory systems including vision, audi
150 nderstanding of the central thermoregulatory circuitry in non-torpid mammals.
151 ity and impairments in hippocampal GABAergic circuitry in Ophn1 mouse model of X-linked intellectual
152 nd pathophysiological degeneration of neural circuitry in Parkinson's disease.
153 l functional connectivity of fronto-striatal circuitry in pre-HD.
154 strates a balance between pro- and anti-fate circuitry in single cells.
155 ow grafts functionally repair damaged neural circuitry in the adult brain is not known.
156 sm regulating formation of the glutamatergic circuitry in the amygdala.
157 e been advances in understanding the central circuitry in the brain that facilitates urination(1-3),
158 upport the functional organization of neural circuitry in the brain via regulation of synaptic gene e
159  further demonstrate specific roles for this circuitry in the daily control of heart rate and cortico
160 hese hypotheses, we reconstructed the midget circuitry in the fetal human fovea by serial electron mi
161  has formed the foundation for understanding circuitry in the human brain.
162  reveal a causal and specific role of DG-CA3 circuitry in the maintenance of latent information withi
163 ions lead to specific predictions for neural circuitry in the superior colliculus.
164 how ankyrin-G and GABARAP regulate GABAergic circuitry in vivo.
165 amma/delta), was engineered to form a sensor circuitry, in which a target-specific monolayer of thiol
166 utamate receptor (mGluR) 5 in regional brain circuitries, including striatum, hippocampus, and raphe
167  for other conditions linked to pathological circuitry, including major depressive disorder and Alzhe
168 ence has revealed that the mesocorticolimbic circuitry, including the prefrontal cortex (PFC) and mes
169                     Light responsive genetic circuitry initiated by the IL1B regulator induces a high
170 vers' inferred causal structure, the FEF-IPS circuitry integrates auditory and visual spatial signals
171  have provided a firmer understanding of the circuitry involved in major depression, providing potent
172 oid receptors; how opioid receptors modulate circuitry involved in processes such as pain, respiratio
173 evelopmental trajectory than fronto-amygdala circuitry involved in traditional extinction learning, s
174 n ASD, it remains elusive how the cerebellar circuitry is altered and whether the cerebellum can serv
175                             The potentiostat circuitry is described along with its working principles
176  and these are also abolished when recurrent circuitry is eliminated.
177                                         This circuitry is extensively modulated by serotonin.
178  the continued development of the underlying circuitry is highly indicative of an adolescent sensitiv
179                  Understanding this changing circuitry is important for designing targeted therapies.
180 s what we do each day, and understanding its circuitry is important for health.
181 t an adaptive mechanism by which hippocampal circuitry is modified in response to environmental deman
182 ex has led to the common assumption that the circuitry is modular and performs stereotyped computatio
183 altered in MDD and given that reward-related circuitry is modulated by dopamine and serotonin, we exa
184 idely acknowledged, the understanding of its circuitry is not complete.
185 evidence shows that, even at a time when its circuitry is not yet mature, the infant hippocampus is a
186 se systems, but the importance of electronic circuitry is often overlooked by microbial electrochemis
187 ionally integrated into stroke-injured brain circuitry is poorly understood.
188 known and conserved across animals, how this circuitry is regulated at the genetic and developmental
189 ccumbens (NAc), considered the hub of reward circuitry, is comprised of two medium spiny neuron (MSN)
190                    However, the polysynaptic circuitry linking the brain areas involved in feeding be
191              Here we show that an inhibitory circuitry located in the Botzinger complex is an importa
192 ketamine affects the brain's reward and mood circuitry located in the corticomesolimbic structures in
193 ession across the brain's emotion regulation circuitry may underlie individual differences in trait a
194        Thus, the underlying normative neural circuitry may, in part, explain the propensity of partic
195                                         This circuitry, mediated by GABAergic and glycinergic synapse
196 ic-yet nontrivial-function which neocortical circuitry must satisfy is the ability to maintain stable
197 al changes across the basal ganglia-thalamic circuitry occur early after SCI and progress over time;
198                                 The habenula circuitry of anuran amphibians, decedents of the first l
199  insight into the pathobiological signalling circuitry of C. neoformans.
200 and BZDs share similar effects on the neural circuitry of fear is unclear.
201  caused by homeostatic changes in the neural circuitry of reward, further establishes the course of a
202 member of an EET-encoding clade, the genetic circuitry of taxis to insoluble acceptors may be conserv
203 gration of newborn neurons in the functional circuitry of the DG.
204                   Here, we review the visual circuitry of the mouse, describing projections from reti
205 o quantitative alterations in the inhibitory circuitry of this layer.
206 ng greater understanding of their underlying circuitry offers better opportunity to control these sei
207 st both the quantum hardware and its control circuitry on the same chip, providing a scalable approac
208 emicals regulate healthy and diseased neural circuitry, one needs to measure their spatiotemporal dyn
209 uronal activity, can affect the entire brain circuitry or remain more focal in the specific brain reg
210 rd and stimulate neurons to understand brain circuitry or restore lost functions.
211  to result from the rewiring of brain reward circuitries, particularly ventral tegmental area (VTA) d
212 , its precise functional roles within the BG circuitry, particularly in comparison to the adjacent ex
213 ood about the reorganization of the neuronal circuitry, particularly within the dorsal horn.
214 ecurrent neural circuits, the role recurrent circuitry plays in implementing these processes remains
215     In this study, we examined whether brain circuitry previously implicated in suppressing negative
216 nlargements, central pattern generator (CPG) circuitry produces the rhythmic output necessary for lim
217 n in the ventral striatum (VS), a key reward circuitry region, moderated differential response to ser
218               BDNF signaling in hypothalamic circuitries regulates mammalian food intake.
219  food, demonstrating that this glutamatergic circuitry regulates aspects of feeding.
220  rodents, but the mechanisms by which HC-PrL circuitry regulates this process remain poorly understoo
221                                       Neural circuitry regulating urine storage in humans has been la
222 organization of the accessory olfactory bulb circuitry remain unclear.
223 tinely observed, indicating that the retinal circuitry remained intact during this period.
224  Degeneration of basal forebrain cholinergic circuitry represents an early event in the development o
225  normal adult brain, but also its underlying circuitry, requirements for development, susceptibility
226 vestigations into the spinal and supraspinal circuitry responsible for the sensation of itch.
227 ins unclear what long-term changes in neural circuitry result from disruptions in these genes, and ho
228       Interrogation of the underlying neural circuitry revealed that the MeApv mediates aggression pr
229 s on the architecture of the corticostriatal circuitry.SIGNIFICANCE STATEMENT Projections from the ip
230 P1 and LASP2 as novel regulators of neuronal circuitry.SIGNIFICANCE STATEMENT Proper regulation of th
231 rstanding of the complexities of spinal itch circuitry.SIGNIFICANCE STATEMENT The spinal cord is a cr
232 fferent connections often bypass feedforward circuitry, suggesting that a different GABAergic mechani
233  evidence for disrupted central sensorimotor circuitry suggests that there may be unrecognized behavi
234 nce-dependent 4 Hz resonance across BLA-mPFC circuitry supports post-extinction fear memory retrieval
235 g aggression are mediated by hardwired brain circuitries that specialize in processing certain sensor
236         Our results establish an inter-organ circuitry that alerts arterial macrophages to regulate v
237                   Here we study the neuronal circuitry that allows larval Drosophila melanogaster of
238 and the intraparietal sulcus (IPS0-4) form a circuitry that concurrently encodes spatial (auditory an
239 ctors forming the adrenergic core regulatory circuitry that controls the malignant cell state in neur
240                                   The neural circuitry that determines visual acuity begins in the re
241 H) neurons lie within a complex hypothalamic circuitry that engages distinct hindbrain regions and is
242 rons are an important component of the brain circuitry that generates brief arousals from sleep in re
243              Here we characterize the neural circuitry that implements mating decisions in the brain
244 ye field and the intraparietal sulcus form a circuitry that integrates auditory and visual spatial si
245                  Here we identify the neural circuitry that links egg laying to mating status in Dros
246 Thus, we present a characterization of brain circuitry that may promote overeating and contribute to
247 al target for alcohol-induced changes is the circuitry that modulates negative affect and stress, two
248 for the future exploration of mechanisms and circuitry that regulate extreme hypothermic and hypometa
249 ections are critical components of forebrain circuitry that regulate motivated behaviors.
250  genomic instability, uncovering a signaling circuitry that safeguards the genome against R loops.
251                  (2020) examine the detailed circuitry that supports a flexible integration of head a
252                Reduced recruitment of neural circuitry that translates taste stimulation to motivated
253 ortant regulatory components of the neuronal circuitry that underlies female aggressive social intera
254 maintenance of mechanical allodynia, but the circuitry that underlies this clinically important form
255 at the neuronal networks formed a functional circuitry that was successfully cultured in MEMO for up
256 n two key areas of the brainstem respiratory circuitry (the Kolliker-Fuse nucleus (KF) and pre-Botzin
257 avioral expression of anxiety and its neural circuitry, the ethical and technical limitations in expl
258 s ability to modify addiction-related neural circuitry through the activation of neurotrophic factor
259                          However, the neural circuitry through which the brain integrates external se
260 ed in software or via auxiliary compensation circuitry thus constraining real-time operation and inte
261                                         This circuitry thus coordinates the two key events in female
262 holine can act directly on cortical premotor circuitry to adaptively shape behavior.
263 ucidate the contributions of the PFC and its circuitry to depression- and anxiety-like behavior.
264 ohol drinking has been shown to modify brain circuitry to predispose individuals for future alcohol a
265  postsynaptic signaling pathways, and neural circuitry to propose a multilevel model that addresses t
266 ing external signals and internal regulatory circuitry to secure organismal integrity.
267 or imitation and possess a specialized brain circuitry to support this behavior.
268                          SCI disrupts neural circuitry to vital organs in the body.
269 zes the competence regulon, a quorum-sensing circuitry, to acquire antibiotic resistance genes and in
270  leading to enhanced sensitization of reward circuitries towards food reward.
271 e it a potential tool for anterograde neural circuitry tracing.
272 arning and suggest that the canonical neural circuitry typically associated with fear learning requir
273    Here we examine the formation of synaptic circuitry under cellular heterotopia in hippocampal CA1,
274 e modes for motivation via mesocorticolimbic circuitry under the control of CeA activation.
275            Disrupted operation of the reward circuitry underlies many aspects of affective disorders.
276 he feasibility of investigating the neuronal circuitry underling complex movement control in rodents,
277  reveals strong similarity in the regulatory circuitry underlying bovine and human EGA compared to mo
278    Serotonin (5-HT) is a modulator of neural circuitry underlying motor patterning, homeostatic contr
279 tunity to study the molecular mechanisms and circuitry underlying non-homeostatic eating.
280  distinct matrix versus core thalamocortical circuitry underlying the generation of a higher-order br
281 table predictions that may shed light on the circuitry underlying the neural computation of predictio
282                                          The circuitry underlying the surround of the AII, however, r
283 molecular marker to interrogate the neuronal circuitry underlying thermoregulation.
284                     To gain insight into the circuitry underlying this response, we used an intersect
285 ation, it is crucial to understand the basic circuitry underpinning brain-wide activity patterns.
286 genetic development, the primordial habenula circuitry underwent various evolutionary adaptations and
287  a fundamental restructuring of dopaminergic circuitries upon time-restricted altered maternal nutrit
288 vide a synaptic-resolution connectome of the circuitry upstream of all DANs in a learning center, the
289                 We provide insights into the circuitry used to integrate MB outputs, connectivity bet
290      Spatial and genetic targeting of HC-PrL circuitry was used for RNA sequencing (n = 31), chemogen
291 In order to better understand the underlying circuitry, we performed retrograde injections of modifie
292  changes implicate the brainstem's breathing circuitry which we confirm by locally eliminating the u-
293 require better understanding of serotonergic circuitry, which has been hampered by our inability to m
294 rons into the stroke-affected brain's neural circuitry, which raises the possibility that such repair
295   By simultaneously imaging fine-scale brain circuitry while measuring large-scale brain connectivity
296 odes embedded within a distributed defensive circuitry whose goal is to determine and realize the bes
297 hip-based integrated photonic and electronic circuitry with those from optogenetics.
298 tory activity, suggesting complex inhibitory circuitry within the BotC.
299              Recent seminal advances in itch circuitry within the nervous system have intersected wit
300 ith functional adaptations within the reward circuitry, within which the nucleus accumbens (NAc) is a

 
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