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1 epigenetic and 3D connectivity landscape of Clear Cell Sarcoma, an aggressive cancer driven by the E
2 and EWS/FLI1 fusion proteins associated with Clear Cell Sarcoma and Ewing's Sarcoma, respectively, we
3 otein in maintaining tumor cell viability of Clear Cell sarcoma and indicate that intracellular antib
4 a, salivary-duct cancer, ovarian cancer, and clear-cell sarcoma and among patients with colorectal ca
5 al sarcomas, round-cell/myxoid liposarcomas, clear-cell sarcomas and gastrointestinal stromal tumors
7 chromosomal translocation t(12;22) found in Clear Cell sarcoma (CCS) fuses the genes for Ewing's sar
12 81 inhibited the growth of a patient-derived clear cell sarcoma cell line whose oncogenic potential i
13 ions in primary melanocytes and melanoma and clear cell sarcoma cells through hypoxia inducible facto
15 in children, including renal-cell carcinoma, clear-cell sarcoma, (congenital) mesoblastic nephroma, r
16 rous histiocytoma (AFH) and gastrointestinal clear cell sarcoma (GI-CCS), although both fusions lead
18 of chemotherapy (including doxorubicin) for clear cell sarcoma improves recurrence-free survival.
20 or stages III to IV/FH WT or stages I to IV/clear cell sarcoma of the kidney (high-risk [HR] group)
21 combination chemotherapy for LR or HR WT or clear cell sarcoma of the kidney have equivalent 2-year
22 osis of rare cancers, including Wilms tumor, Clear Cell Sarcoma of the Kidney, Neuroblastoma, Osteosa
23 terminus of BCOR in 23 of 27 (85%) pediatric clear cell sarcomas of the kidney (CCSK) from two indepe
24 ), stages III to IV FH WT, or stages I to IV clear-cell sarcoma of the kidney (high-risk[HR]) were ra
25 heterogeneous group of tumours that includes clear-cell sarcoma of the kidney, congenital mesoblastic
26 al translocation associated with soft tissue clear cell sarcoma results in a chimeric protein EWS-ATF
27 ignant melanoma of soft parts or soft tissue clear cell sarcoma which shares t(12;22) chromosome tran