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1 liferative response to specific alloantigen (clonal anergy).
2 his may be integrated with other features of clonal anergy.
3 e progression and promote the development of clonal anergy.
4 genes involved in pathways leading to T cell clonal anergy.
5 kade do not escape the ultimate induction of clonal anergy.
6  (Mls-1(a)) induces tolerance to Mls-1(a) by clonal anergy.
7 T lymphocytes that prevents the induction of clonal anergy.
8 stemic tolerance by both clonal deletion and clonal anergy.
9  factor interleukin-2 (IL-2), a state called clonal anergy.
10  contributes to defective IL-2 production in clonal anergy.
11 ls through a combination of the induction of clonal anergy and anti-inflammatory cytokines.
12 is state resulted from a combination of both clonal anergy and cytokine-mediated immunosuppression.
13 ) nondeletional T-cell tolerance mediated by clonal anergy and dependent on the structure, location,
14 hus, Ndrg1 contributes to the maintenance of clonal anergy and inhibition of T-cell-mediated inflamma
15 ral tolerance has generally been ascribed to clonal anergy and/or active immunoregulatory states.
16 ce mechanism in addition to clonal deletion, clonal anergy, and receptor editing.
17 duced T-cell hyporesponsiveness as a form of clonal anergy, and they supported an important role for
18 monstrate that T cell adaptive tolerance and clonal anergy are distinct biochemical states, possibly
19 s by tumor cells to T lymphocytes may induce clonal anergy as a mechanism of escape from immune surve
20  ligand-mediated apoptosis and the resulting clonal anergy as the mechanisms of high dose AChR alpha
21  proposed to inhibit the induction of T cell clonal anergy by either directly antagonizing negative s
22                                        Thus, clonal anergy can contribute to the development of Ag-sp
23                                   Therefore, clonal anergy can interfere with the delivery of helper
24 c cells, but may instead induce a persistent clonal anergy capable of blocking subsequent immunity.
25 ctivation in the steady state include T cell clonal anergy, deletion, and conversion of peripheral re
26 ession by Th2 or TGF-beta-secreting cells or clonal anergy/deletion, depending on the Ag dose used, w
27 inhibition: bystander immune suppression and clonal anergy/deletion.
28                     CTLA-4 molecules promote clonal anergy development only indirectly by limiting ce
29              Nevertheless, this induction of clonal anergy did not interfere with the capacity of nai
30 reactive B cells functionally silent through clonal anergy has long posed the question of why fill th
31                             The induction of clonal anergy in a T cell inhibits IL-2 secretion becaus
32                                              Clonal anergy in Ag-specific CD4+ T cells is shown in th
33  progression and facilitate the induction of clonal anergy in nearby responder CD25(-)CD4(+) T cells.
34        This state was contrasted with T cell clonal anergy in which the RAS/MAPK pathway was preferen
35 ith reduced Ikaros activity are resistant to clonal anergy induced by TCR ligation in the absence of
36 ction signaling defect as the sole basis for clonal anergy induction and document the presence of a d
37  therefore explored the relationship between clonal anergy induction and the avoidance of autoimmune
38                                       T cell clonal anergy induction in lymphopenic nu/nu mice was fo
39  been shown to be a barrier to CD4(+) T cell clonal anergy induction.
40                          Induction of T-cell clonal anergy involves serial activation of transcriptio
41                                              Clonal anergy is an enigmatic self-tolerance mechanism b
42                                         One, clonal anergy, is principally a growth arrest state, whe
43 vented from autoantibody secretion by B cell clonal anergy marked by downregulation of surface IgM, i
44                             In CD4+ T cells, clonal anergy occurs when the T-cell receptor is activat
45                                              Clonal anergy of autoreactive B cells is a key mechanism
46   T-cell hyporesponsiveness can be caused by clonal anergy or adaptive tolerance, but the pathophysio
47  T and B cells, perhaps, via CTLA4Ig-induced clonal anergy or deletion.
48 ory mechanisms, such as receptor editing and clonal anergy, preventing the activation of B cells expr
49 s and the development and maintenance of the clonal anergy state.
50  responses were independent of an ability of clonal anergy to block T cell clonal expansion.