戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 piration, demonstrated in a paradigm of mild closed head injury.
2 very of a fallen patient who has sustained a closed head injury.
3 ese lesions evolve following relatively mild closed head injury.
4  volunteers and 20 patients within 6 days of closed head injury.
5 tic potential of CN-105 in a murine model of closed head injury.
6 was performed on anesthetized mice following closed head injury.
7 e rates of depressive illness 50 years after closed head injury.
8 ve declines in mice exposed to repeated mild closed head injury.
9       MR images were obtained 3 months after closed-head injury.
10 trations peaked at 30 to 42 hrs after severe closed-head injury.
11 e, injury severity, and outcome after severe closed-head injury.
12 lity, symptoms, and objective findings after closed-head injury.
13 es C for 24 hrs) on neurologic outcome after closed-head injury.
14 ds to establish the presence and severity of closed head injuries.
15  and normothermia in 82 patients with severe closed head injuries (a score of 3 to 7 on the Glasgow C
16 tonic saline administration to children with closed head injury appears to be a promising therapy for
17 erms traumatic brain injury, brain injuries, closed head injury, blunt head trauma, concussion, atten
18 fficult to reproduce TAI in animal models of closed head injury, but in vitro stretch injury models r
19  this study, based on advantages of both the closed head injury (CHI) and controlled cortical impact
20                This review will examine mild closed head injury (CHI) and the current evidence on hea
21 Edition (ICD-9) codes denoting open (OHI) or closed head injury (CHI) in isolation or in combination
22 eporter mice were also exposed to repetitive closed head injury (CHI) or i.p. kainic acid.
23 l TBI, controlled cortical impact (CCI), and closed head injury (CHI).
24 al models have been developed to mimic human closed head injury (CHI).
25 ynaptic proteins in an experimental model of closed head injury (CHI).
26 ead-specific model for TBI termed Drosophila Closed Head Injury (dCHI), where well-controlled, nonpen
27                                       Severe closed head injury in mice induced abundant MAC depositi
28                                              Closed-head injury-induced lesions in the right putamen
29    Our results suggest that mild-to-moderate closed head injury may increase PD risk decades later.
30                                We utilized a closed head injury model to induce TBI in mice, followed
31                                      Using a closed-head injury model of TBI in mice, we showed by MR
32                             In patients with closed head injury, neither the administration of erythr
33        Clinical evaluation of patients after closed-head injury, particularly mild head trauma, must
34           Standards for management of severe closed head injury should help to establish a foundation
35 nsequences of a clinically relevant model of closed head injury, the lateral fluid percussion injury
36  induced by a weight drop model to produce a closed head injury to mice and the effect of inhaled nit
37                                              Closed head injury to the developing rat brain causes an
38                                       Severe closed head injury was induced in rats by the impact of
39 core of < or = 7) adult patients with severe closed-head injury were studied during the prospective,
40 o 65 years of age with coma after sustaining closed head injuries who were randomly assigned to be tr
41 ythropoietin, n = 102; placebo, n = 98) with closed head injury who were unable to follow commands an