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3 taneous valve interventions has emerged as a closed-chest alternative for all patients, but particula
4 te that under physiological conditions, with closed-chest and phasic negative intrathoracic pressure
5 ty-four measurements were performed in eight closed-chest anesthetized greyhounds at baseline and dur
6 analysis in Langendorff perfused hearts and closed-chest anesthetized mice also indicated depressed
7 was performed by cardiac catheterization in closed-chest anesthetized mice, by pressure-volume-loop
8 recorded from the apical 4-chamber view in 7 closed-chest anesthetized mongrel dogs during 5 differen
11 artery was perfused at constant pressure in closed-chest, anesthetized dogs, and exogenous adenosine
13 s study was to quantitatively evaluate, in a closed-chest animal model, a noninvasive method of measu
17 ted molecular signatures, a porcine model of closed-chest balloon MI was used along with serial cardi
18 dium content changes over time in an in vivo closed-chest canine model (n=13) of myocardial infarctio
19 nary arterial muscularization as assessed by closed-chest cardiac catheterization and anti-alphaSMA s
22 ght mongrel dogs underwent an acute 2-minute closed-chest coronary artery occlusion while continuous
23 or using porcine models of MI created with a closed-chest coronary artery occlusion-reperfusion techn
26 MA sites (DT-A) was measured in nine normal closed-chest dogs by use of surgically implanted radiopa
31 as studied using radiopaque markers in eight closed-chest dogs with acute posterior left ventricular
42 4796 can be visualized on in vivo imaging in closed chest large animal model after intravenous inject
43 ht to show the feasibility of catheter-only, closed-chest, large-vessel anastomosis (superior vena ca
44 n a Yucatan swine model of heart failure via closed chest left anterior descending coronary artery (L
45 protocol before and 1-month after undergoing closed chest left anterior descending coronary artery (L
46 ion) at distinct times of the day, using the closed-chest left anterior descending coronary artery oc
47 We examined 7 dogs 2 days after a 90-minute closed-chest left anterior descending coronary artery oc
48 to apex in seven dogs 2 days after a 90-min closed-chest left anterior descending coronary occlusion
49 inal echocardiography, rats underwent either closed-chest left ventricular (LV) catheterization or La
50 went transthoracic echo Doppler studies, and closed-chest left ventricular pressures with direct left
51 ested the hypothesis that when compared with closed-chest manual compressions at 80 chest compression
53 ers and physiological assay results (ECG and closed-chest Millar catheter) were within the normal ran
54 disruption and antibody neutralization in a closed-chest model of reperfused murine myocardial infar
58 re well-established protocols for creating a closed-chest myocardial infarction (MI) as well as proto
59 exposed to VA-ECMO and used a swine model of closed-chest myocardial ischemia/reperfusion injury to e
60 ection in normal canines with open (n=3) and closed chest (n=5) and in a pig model with features of h
65 were recorded simultaneously in anesthetized closed-chest pigs (n=5) during sinus rhythm, and epicard
84 multaneous biplane videofluoroscopy in seven closed-chest, sedated sheep before and during asynchrono
87 stained atrial fibrillation was induced in 8 closed-chest sheep with burst pacing and continuous peri
88 stained atrial fibrillation was induced in 8 closed-chest sheep with burst pacing and maintained with
94 : Closed-chest resuscitation with opened and closed chest tubes and medical and fluid interventions w
96 ower a microimplant (2 mm, 70 mg) capable of closed-chest wireless control of the heart that is order
97 (open-chest) or modified four-chamber view (closed-chest) with 1:4 end systolic electrocardiogram tr