ライフサイエンスコーパス: coagulase

1 .9%; Staphyloslide, 99.1 and 98.9%; and tube coagulase, 99.3 and 100%.

2 ere ectopically expressed, the expression of coagulase, a sae target with low affinity for phosphoryl

3 pigmentation, and decreased hemolysis and/or coagulase activity are periodically isolated by the clin

4 trate phenotypic growth patterns and lack of coagulase activity consistent with SCVs.

5 ency of vWbp or VWF as well as inhibition of coagulase activity reduced S aureus adhesion.

6 gnificantly alter the expression patterns of coagulase and alpha-hemolysin, two well-known sae target

7 pression of major virulence factors, such as coagulase and alpha-hemolysin.

8 P1 did not affect the expression pattern of coagulase and alpha-hemolysin.

9 ting with 125I-fibrinogen, we could identify coagulase and an additional unidentified 52-kDa protein

10 hibitory clindamycin stimulates synthesis of coagulase and fibronectin binding protein B, also at the

11 genes encoding alkaline shock protein 23 and coagulase and have demonstrated SigB and RsbW dependence

12 gy, tests for clumping factor, and tests for coagulase and urease activities and were also tested wit

13 s, assembly of protective fibrin shields via coagulases and protein A-mediated B cell superantigen ac

14 lthough it was deficient in clumping factor, coagulase, and pigment.

15 Protein A and coagulases are distinctive virulence attributes for S. a

16 he increased expression and transcription of coagulase as shown by Western (immunoblot) and Northern

17 ons were regulated by SaeRS and dependent on coagulase-catalyzed conversion of fibrinogen into fibrin

18 Staphylococcus aureus secretes coagulase (Coa) and von Willebrand factor-binding protei

19 protein A (spa), gamma hemolysin (hlg), and coagulase (coa) were also located on the map by PFGE and

20 ulated surface determinants (IsdA and IsdB), coagulase (Coa), and von Willebrand factor binding prote

21 S. aureus secretes coagulase (Coa), which activates host prothrombin and ge

22 nfection, Staphylococcus aureus secretes two coagulases (Coa and von Willebrand factor binding protei

23 the variable prothrombin binding portion of coagulases confer type-specific immunity through the neu

24 tudy, we use a S. aureus mutant lacking both coagulases (Deltacoa/vwb) and dabigatran, a pharmacologi

25 ty and its relationship to the expression of coagulase (encoded by coa) and clumping factor (encoded

26 ureus was identified from all media by slide coagulase, exogenous DNase, and mannitol fermentation as

27 By combining variable portions of coagulases from North American isolates into hybrid Coa

28 Coagulase gene (coa) short sequence repeat region sequen

29 agment length polymorphism (PCR-RFLP) of the coagulase gene and pulsed-field gel electrophoresis (PFG

30 d based on improved PCR amplification of the coagulase gene and restriction fragment length polymorph

31 ally, strains were subtyped according to the coagulase gene in order to strengthen discriminatory pow

32 The coagulase gene PCR products for 24 isolates of MRSA and

33 Coagulase gene polymorphism and agr typing demonstrated

34 Further coagulase gene sequence analysis subtyped those 20 strai

35 the genomes of different S. aureus isolates, coagulase gene sequences are variable, and this has been

36 irulence factors such as alpha-hemolysin and coagulase; however, the molecular mechanism of this sign

37 hese findings emphasize the critical role of coagulase in staphylococcal escape from opsonophagocytic

38 Thus, coagulase is a major binding protein for soluble fibrino

39 Coagulase-mediated biofilms exhibited increased antimicr

40 Adhesion was further enhanced by coagulase-mediated fibrin formation that clustered bacte

41 Nine mauve colonies were slide coagulase negative and were subsequently identified as S

42 ased risk of nosocomial bacterial sepsis and coagulase negative staphylococcal infections, and thus s

43 The dominant isolates were Coagulase negative Staphylococci (CNS) 9(29.0%), Staphyl

44 predominantly isolated pathogen, followed by Coagulase negative staphylococci (CoNS) (33.5%) and Kleb

45 ith another enteric bacteria (n=23), or with coagulase negative staphylococci (CONS) (n=24).

46 Coagulase negative Staphylococci (CoNS) were the most co

47 The proportion of coagulase negative Staphylococci among the Gram positive

48 ssociated with Klebsiella pneumoniae whereas Coagulase negative Staphylococci and Bacillus spp. are c

49 The resistance of S. aureus and coagulase negative staphylococci clinical isolates to te

50 the most frequently isolated species of the coagulase negative staphylococci from human stool.

51 ccus viridians, Streptococcus pneumoniae and Coagulase negative Staphylococci in endophthalmitis diag

52 P. aeruginosa and E. coli in dacryocystitis; Coagulase negative Staphylococci, Pseudomonas aeruginosa

53 s far as this review, Staphylococcus aureus, Coagulase negative Staphylococci, Streptococcus pneumoni

54 ial infection is common, particularly due to coagulase negative staphylococci.

55 les was high and the predominant isolate was coagulase negative Staphylococci.

56 benefit was most pronounced in patients with coagulase negative Staphylococcus bacteremia (5.5d and 4

57 ed among the S. aureus isolates; however, 10 coagulase-negative isolates were MecA+ but oxacillin sen

58 A new definition for complicated coagulase-negative staphylococcal (CoNS) bacteremia was

59 Fewer coagulase-negative staphylococcal (CoNS) contaminants gr

60 validated by correctly identifying 36 of 37 coagulase-negative staphylococcal (CoNS) isolates identi

61 caprae was the cause of 6 of 18 episodes of coagulase-negative staphylococcal bacteremia, was the mo

62 ive quantitative blood cultures and definite coagulase-negative staphylococcal bloodstream infection.

63 occus aureus rather than those caused by its coagulase-negative staphylococcal counterparts.

64 Differentiating true coagulase-negative staphylococcal infection from contami

65 ntries showed a trend of increasing risk for coagulase-negative staphylococcal infection.

66 has been implicated in a large proportion of coagulase-negative staphylococcal infections in very-low

67 re comorbidity and a rise in enterococci and coagulase-negative staphylococcal infections.

68 The presence of icaA in a coagulase-negative staphylococcal isolate associated wit

69 ed non-prosthetic joint infection-associated coagulase-negative staphylococcal isolates were icaA pos

70 A total of 46% (20 out of 44) of coagulase-negative staphylococcal prosthetic joint infec

71 postimplementation period was also noted for coagulase-negative staphylococci (1.9% preimplementation

72 mong which filamentous fungi (25, 39.1%) and coagulase-negative staphylococci (14, 21.9%) comprised a

73 (18.6%), Staphylococcus aureus (15.6%), and coagulase-negative staphylococci (14.5%).

74 56%), Staphylococcus aureus (28%), and other coagulase-negative staphylococci (16%).

75 ms causing CLABSI in oncology locations were coagulase-negative staphylococci (16.9%), Escherichia co

76 Staphylococcus epidermidis (MSSE) (9), other coagulase-negative staphylococci (19), Streptococcus sal

77 episodes, including Escherichia coli (24%), coagulase-negative staphylococci (21%), and P. aeruginos

78 Common pathogens were coagulase-negative staphylococci (21%, 10/48) and methic

79 The most frequent causal microorganisms were coagulase-negative staphylococci (24%), followed by Stap

80 Coagulase-negative staphylococci (36%) were the most com

81 37.2% vs 23.8%; P = .009) but lower rates of coagulase-negative staphylococci (4.7% vs 20.0%, P = .00

82 The most common pathogens were coagulase-negative staphylococci (68%) and Staphylococcu

83 organism (128 patients [23.0%]), followed by coagulase-negative staphylococci (94 patients [16.9%]).

84 , (ii) distinguish between S. aureus and the coagulase-negative staphylococci (CNS) (based on amplifi

85 ut not for Streptococcus spp. (P = 0.85) and coagulase-negative staphylococci (CNS) (P = 0.88).

86 Coagulase-negative staphylococci (CNS) are important cau

87 Coagulase-negative staphylococci (CNS) are the most comm

88 odstream infections (BSI-SA) and noninvasive coagulase-negative staphylococci (CNS) isolates were use

89 s and 100 isolates of erythromycin-resistant coagulase-negative staphylococci (CNS) were examined by

90 Coagulase-negative staphylococci (CNS) were the most com

91 lococcus epidermidis, Staphylococcus aureus, coagulase-negative staphylococci (CNS), Peptostreptococc

92 as among mecA(+) strains of other species of coagulase-negative staphylococci (CNS).

93 and was mostly attributed by a reduction in coagulase-negative staphylococci (CoNS) (17% vs 2%, P <

94 2021, with Staphylococcus aureus (n = 621), coagulase-negative staphylococci (CoNS) (n = 234), Pseud

95 The most common organisms identified were Coagulase-negative Staphylococci (CoNS) [65.9% (91/138)]

96 ) to accurately differentiate S. aureus from coagulase-negative staphylococci (CoNS) and other Gram-p

97 investigating issues related to isolation of coagulase-negative staphylococci (CoNS) and other skin m

98 Coagulase-negative staphylococci (CoNS) and Staphylococc

99 gh horizontal gene transfer originating from coagulase-negative staphylococci (CoNS) and through clon

100 Coagulase-negative staphylococci (CoNS) are an increasin

101 Coagulase-negative staphylococci (CoNS) are important pa

102 Coagulase-negative staphylococci (CoNS) are the main cau

103 microdilution and disk diffusion testing of coagulase-negative staphylococci (CoNS) by using a PCR a

104 Recent reports suggest that skin-dwelling coagulase-negative staphylococci (CoNS) can prime the sk

105 Coagulase-negative staphylococci (CoNS) form a thick, mu

106 he presence of secreted cytotoxic factors of coagulase-negative staphylococci (CoNS) from bovine clin

107 Differentiating Staphylococcus aureus from coagulase-negative staphylococci (CoNS) is important in

108 used to determine the clinical importance of coagulase-negative staphylococci (CoNS) is isolation of

109 P67 card for detecting methicillin-resistant coagulase-negative staphylococci (CoNS) is not known.

110 sis is an aggressive, virulent member of the coagulase-negative staphylococci (CoNS) that is responsi

111 Unbiased species-level identification of coagulase-negative staphylococci (CoNS) using matrix-ass

112 .01 for detection of oxacillin resistance in coagulase-negative staphylococci (CoNS) was compared to

113 s for detection of Staphylococcus aureus and coagulase-negative staphylococci (CoNS) were 99.5% (217/

114 inical isolates of Staphylococcus aureus and coagulase-negative staphylococci (CoNS) were determined

115 Five hundred isolates of coagulase-negative staphylococci (CoNS) were recovered f

116 spersal of S. aureus, a 1.4-fold increase in coagulase-negative staphylococci (CoNS), and a 3.9-fold

117 methicillin-resistant S. aureus (MRSA), and coagulase-negative staphylococci (CoNS), including methi

118 and 53 strains of Staphylococcus aureus and coagulase-negative staphylococci (CoNS), respectively.

119 it ocular isolates of Staphylococcus aureus, coagulase-negative staphylococci (CoNS), Streptococcus p

120 dical Center for the rapid identification of coagulase-negative staphylococci (CoNS).

121 es-specific differences are identified among coagulase-negative staphylococci (CoNS).

122 abilities to detect oxacillin resistance in coagulase-negative staphylococci (CoNS).

123 se-positive S. aureus (CoPS) isolates and 38 coagulase-negative staphylococci (CoNS).

124 lar for infections caused by enterococci and coagulase-negative staphylococci (CoNS; adjusted SHR, 0.

125 BSIs were caused by coagulase-negative staphylococci (n = 27), enterococci (

126 ), followed by Enterobacteriaceae (n=22) and coagulase-negative staphylococci (n=6).

127 ruginosa (2 of 4), streptococci (2 of 5), or coagulase-negative staphylococci (none of 8) (P = 0.02).

128 I]: 1.27-3.41; P = .003) and IE secondary to coagulase-negative staphylococci (ORadj, 2.71; 95% CI: 1

129 More staphylococci (P < 0.05), especially coagulase-negative staphylococci (P < 0.05), and yeasts

130 < 0.001), Staphylococcus aureus (P = 0.003), coagulase-negative staphylococci (P = 0.008), gram-negat

131 N anaerobic bottle, while significantly more coagulase-negative staphylococci (P = 0.01), Streptococc

132 and streptococci slightly declined, whereas coagulase-negative staphylococci and enterococci consist

133 enced recurrence in this study (isolation of coagulase-negative staphylococci and inadequate duration

134 Coagulase-negative staphylococci and other gram-positive

135 Coagulase-negative staphylococci and Staphylococcus aure

136 ganisms; the species cultured were typically coagulase-negative staphylococci and were associated wit

137 rococcus faecalis, Staphylococcus aureus, or coagulase-negative staphylococci and who were being trea

138 xplanation for the clinical observation that coagulase-negative staphylococci are a highly prominent

139 Coagulase-negative staphylococci are by far the most com

140 Coagulase-negative staphylococci are common nosocomial p

141 Coagulase-negative staphylococci are dominant human skin

142 Coagulase-negative staphylococci are prominent skin comm

143 Approximately 75% of coagulase-negative staphylococci are resistant to methic

144 Coagulase-negative staphylococci are the most frequently

145 species identification of blood isolates of coagulase-negative staphylococci as a predictor of the c

146 ); (ii) controlled cultural findings showing coagulase-negative staphylococci as the most common isol

147 s more likely to have poor final acuity than coagulase-negative staphylococci cases (adjusted OR, 11.

148 All Staphylococcus aureus and coagulase-negative staphylococci clinical isolates cultu

149 Cure proportions were highest for coagulase-negative staphylococci followed by gram-negati

150 temic infections is usually required because coagulase-negative staphylococci have become increasingl

151 Coagulase-negative staphylococci have long been regarded

152 8), Candida albicans in 5.8% (26/443), other coagulase-negative staphylococci in 6.0% (27/448), Propi

153 ferentiate between Staphylococcus aureus and coagulase-negative staphylococci in blood cultures growi

154 or determining beta-lactam susceptibility in coagulase-negative staphylococci in our laboratory.

155 Other important infections due to coagulase-negative staphylococci include central nervous

156 days for S. aureus infection vs. 14 days for coagulase-negative staphylococci infection [p < 0.001]).

157 olonisation of indwelling medical devices by coagulase-negative staphylococci is a prevalent risk in

158 iated with prosthetic joint infection and 23 coagulase-negative staphylococci isolated from noninfect

159 R/ESI-MS showed the following sensitivities (coagulase-negative staphylococci not included): Gram-pos

160 Clinical isolates of coagulase-negative staphylococci often elaborate a biofi

161 eliability of routine sensitivity testing in coagulase-negative staphylococci often lead to the use o

162 Isolation of coagulase-negative staphylococci only in broth, in the a

163 oximately 60% of the samples (730) contained coagulase-negative staphylococci or nonstaphylococci as

164 species of Acinetobacter, Enterobacter, and coagulase-negative staphylococci recovered from the hand

165 monas aeruginosa, Staphylococcus aureus, and coagulase-negative staphylococci strains.

166 Of 118 strains of coagulase-negative staphylococci tested, 81 were oxacill

167 As it has been shown for coagulase-negative staphylococci that some autolysins ar

168 resistant rates of Staphylococcus aureus and coagulase-negative staphylococci were 45% and 43%, respe

169 Similar numbers of consecutive patients with coagulase-negative staphylococci were analyzed for compa

170 Coagulase-negative staphylococci were identified in 7 ey

171 nstitutional rates of 73% contamination when coagulase-negative staphylococci were identified, 67.6%

172 Coagulase-negative staphylococci were more frequently as

173 cocci, and 11/42 (26%) methicillin-resistant coagulase-negative staphylococci were mupirocin resistan

174 Strains from 11 species of coagulase-negative staphylococci were selected such that

175 In Gram-positive isolates, coagulase-negative staphylococci were the most commonly

176 Coagulase-negative staphylococci were the only pathogens

177 n pathogens were associated with device use: coagulase-negative staphylococci with central lines, P.

178 eudomonas with piperacillin (1 of 28, 3.6%), coagulase-negative staphylococci with oxacillin (2 of 74

179 th higher abundance of Bacilli (specifically coagulase-negative Staphylococci) and a lack of anaerobi

180 isolate each of Propionibacterium acnes and coagulase-negative staphylococci) in FAN bottles, wherea

181 aureus and 56 [31.6% {95% CI, 24.9%-39.0%}] coagulase-negative staphylococci); and a high prevalence

182 For the coagulase-negative staphylococci, 150 were MecA+ and 111

183 tive bacteria, 40 Staphylococcus aureus, 152 coagulase-negative staphylococci, 28 streptococci, 22 en

184 6.2% of cases (Staphylococcus aureus, 26.6%; coagulase-negative staphylococci, 9.7%).

185 ssociated infections are currently caused by coagulase-negative staphylococci, a pathogen that incite

186 isms that are often considered contaminants (coagulase-negative staphylococci, aerobic and anaerobic

187 isolates, 3/16 (19%) methicillin-susceptible coagulase-negative staphylococci, and 11/42 (26%) methic

188 Propionibacterium species, coagulase-negative staphylococci, and Corynebacterium sp

189 lates were Staphylococcus epidermidis, other coagulase-negative Staphylococci, and Corynebacterium sp

190 d isolates, including Staphylococcus aureus, coagulase-negative staphylococci, Enterococcus faecalis,

191 Although coagulase-negative staphylococci, including Staphylococc

192 increased isolation of Enterococcus species, coagulase-negative staphylococci, intrinsically antibiot

193 epidermidis and a series of closely related coagulase-negative staphylococci, most of which are oppo

194 -1), enterotoxin, and other superantigens by coagulase-negative staphylococci, no associated pathogen

195 49 (53.3%), 29 (31.5%), and 13 (14.1%) were coagulase-negative staphylococci, other gram-positive, a

196 negative bacterial rods and three species of coagulase-negative staphylococci, recovered from both th

197 in-susceptible S. aureus, and 14 and 35% for coagulase-negative staphylococci, respectively.

198 Contrary to other coagulase-negative staphylococci, S. lugdunensis bound t

199 Coagulase-negative staphylococci, such as Staphylococcus

200 methicillin-resistant Staphylococcus aureus, coagulase-negative staphylococci, vancomycin-resistant e

201 Coagulase-negative staphylococci, with the leading speci

202 . aureus and 100% sensitive and specific for coagulase-negative staphylococci.

203 less accurate for methicillin resistance in coagulase-negative staphylococci.

204 osed in vitro to opsonized S. pneumoniae and coagulase-negative staphylococci.

205 r internalization of either S. pneumoniae or coagulase-negative staphylococci.

206 er contained biofilms comprised primarily of coagulase-negative staphylococci.

207 Standards breakpoint of 4 micrograms/ml for coagulase-negative staphylococci.

208 including endocarditis, compared with other coagulase-negative staphylococci.

209 ugh cloth and paper gowns was performed with coagulase-negative staphylococci.

210 The most prevalent pathogens were coagulase-negative staphylococcus (39.4%), followed by S

211 The prevalence of coagulase-negative Staphylococcus (40.9% in group A and

212 ost frequent germs found in the samples were coagulase-negative Staphylococcus (48.6%).

213 olate was Propionibacterium acnes (11/26) vs coagulase-negative Staphylococcus (57/92); and 4) patien

214 The most common isolate overall was coagulase-negative Staphylococcus (CNS) and the most com

215 ely 30 Staphylococcus aureus isolates and 20 coagulase-negative staphylococcus (CoNS) isolates in a p

216 disk and MIC breakpoints, the CLSI M100-S25 coagulase-negative Staphylococcus (CoNS) oxacillin MIC b

217 eriophages from human skin swabs that infect coagulase-negative Staphylococcus (CoNS) species, which

218 and 9 (2.5%) were identified by StaphPlex as coagulase-negative Staphylococcus (CoNS), methicillin-re

219 teremia and blood cultures contaminated with coagulase-negative Staphylococcus (CoNS).

220 significantly worse final VA than those with coagulase-negative Staphylococcus (mean logMAR 2.14 vs 0

221 thicillin-sensitive S. aureus (n = 134), and coagulase-negative Staphylococcus (n = 176).

222 Coagulase-negative staphylococcus accounted for 41 of th

223 monly identified organisms from the eye were coagulase-negative Staphylococcus and Candida.

224 culture results were positive in 41.6%, with coagulase-negative Staphylococcus being the most common

225 on at both DEI (70%) and LAC+USC (68%), with coagulase-negative Staphylococcus being the most common

226 tes, the most commonly isolated organism was coagulase-negative Staphylococcus both at baseline (73%)

227 organism (30.6%), but the number of isolated coagulase-negative Staphylococcus cases increased signif

228 significantly decreased the number of nasal coagulase-negative Staphylococcus compared with saline c

229 re gram-positive bacteria (91.3%), including coagulase-negative Staphylococcus in 78.3%.

230 Coagulase-negative Staphylococcus isolates were not cons

231 For 155 Staphylococcus aureus and 261 coagulase-negative Staphylococcus isolates, the bDNA ass

232 aphylococcal bacteremia, was the most common coagulase-negative staphylococcus recovered from the nar

233 Coagulase-negative staphylococcus remains the most frequ

234 llin-resistant Staphylococcus aureus (4.4%), coagulase-negative Staphylococcus species (15.3%), Strep

235 vestigations revealing no microorganism or a coagulase-negative Staphylococcus species (CNSP), and fu

236 acillin BMD and cefoxitin DD tests using the coagulase-negative Staphylococcus species (CoNS) breakpo

237 Certain coagulase-negative Staphylococcus species appeared more

238 Propionibacterium acnes and coagulase-negative Staphylococcus species were the most

239 Acinetobacter , Enterococcus, Candida , and Coagulase-negative Staphylococcus species, and more meth

240 d methicillin-susceptible S. aureus species, coagulase-negative Staphylococcus species, and other cli

241 Gram-positive bacteria, particularly coagulase-negative Staphylococcus spp, remain the leadin

242 gnificantly more Bacillus spp. (P < 0.0001), coagulase-negative Staphylococcus spp. (P < 0.0001), and

243 es of Staphylococcus aureus (P = 0.0113) and coagulase-negative Staphylococcus spp. (P = 0.0029) than

244 tream infections (septic episodes) caused by coagulase-negative Staphylococcus spp. (P = 0.0146).

245 ty-nine clinical staphylococcal isolates (58 coagulase-negative Staphylococcus spp. [CoNS] and 41 Sta

246 ion, using breakpoints for human isolates of coagulase-negative Staphylococcus spp., had low sensitiv

247 fied as 10 MRSA strains, 10 MSSA strains, 12 coagulase-negative Staphylococcus strains, and 8 Microco

248 Staphylococcus lugdunensis is a coagulase-negative staphylococcus that has several simil

249 Staphylococcus caprae, a hemolytic coagulase-negative staphylococcus that is infrequently a

250 Coagulase-negative Staphylococcus was isolated in 39 of

251 tious endophthalmitis (2 Candida glabrata, 2 coagulase-negative Staphylococcus, 1 Streptococcus pneum

252 d in 22 of 46 children (48%) with LOM due to coagulase-negative Staphylococcus, 43 of 67 (64%) due to

253 For coagulase-negative Staphylococcus, an increased inoculum

254 easts, Actinobacillus actinomycetemcomitans, coagulase-negative Staphylococcus, Campylobacter rectus,

255 nd II, and vitreous cultures for infections (coagulase-negative Staphylococcus, Candida, Fusarium, an

256 Escherichia coli, Staphylococcus aureus, coagulase-negative Staphylococcus, Klebsiella pneumoniae

257 ye preparations demonstrated the presence of coagulase-negative Staphylococcus, Streptococcus type al

258 in 4 of 5 cases; all positive cultures grew coagulase-negative Staphylococcus.

259 (P < 0.001), S. aureus isolates (P < 0.001), coagulase-negative staphyococci (P = 0.003), and total o

260 For coagulase-negative strains at 24 h of incubation, breakp

261 h enhanced expression of clumping factor and coagulase on immunoblots.

262 Confirming coagulase on pink isolates, the sensitivity and specific

263 d-phase fibrinogen, clumping factor, but not coagulase, plays a major role in binding to immobilized

264 ositive S. agnetis isolates were found to be coagulase positive at 4 h.

265 Overall, 88% (37/42) of coagulase-positive S. agnetis isolates were found to be

266 The herd-level prevalence of coagulase-positive S. agnetis ranged from 0 to 2.17%.

267 pecies isolates were recovered, including 27 coagulase-positive S. aureus (CoPS) isolates and 38 coag

268 on that had previously been characterized as coagulase-positive S. hyicus based on phenotypic species

269 Although Staphylococcus intermedius and coagulase-positive species of staphylococci are reported

270 Staphylococcus pseudintermedius is a coagulase-positive species that colonizes the nares and

271 ection control decisions requires that these coagulase-positive staphylococci are accurately identifi

272 s intermedius group (SIG) is a collection of coagulase-positive staphylococci consisting of four dist

273 All coagulase-positive staphylococci produced a single PCR a

274 Coagulase-positive Staphylococcus aureus (S. aureus) is

275 , partially controls exoprotein synthesis in coagulase-positive Staphylococcus aureus by modulating t

276 nrichment method was optimum for recovery of coagulase-positive Staphylococcus spp.

277 phenotypic properties of beta-hemolysis and coagulase positivity allowed the clinical isolates to ma

278 acing between promoter elements in P1 or the coagulase promoter was altered to the optimal spacing of

279 polysaccharide (CP8), nuclease, alpha-toxin, coagulase, protease, and protein A.

280 and nuclease but a repressor of alpha-toxin, coagulase, protease, and protein A.

281 losest match being the Staphylococcus aureus coagulase protein.

282 ified 56/56 S. aureus isolates identified by coagulase/protein A latex agglutination.

283 The secretion of coagulases, proteins that associate with and activate th

284 S. aureus secretes the coagulases staphylocoagulase and von Willebrand factor-b

285 an S. aureus strain with genetic deletion of coagulases, survival of mice improved, highlighting the

286 tested using the BNSA test and a direct tube coagulase test (DTCT).

287 h (API) (bioMerieux, Durham, N.C.), the tube coagulase test (TCT) read at 4 h, and peptide nucleic ac

288 The assays were compared to the tube coagulase test and latex agglutination (LA) (Sanofi Diag

289 Staphaurex Plus, Staphyloslide, and the tube coagulase test for the identification of staphylococcal

290 nd definitive treatment compared to the tube coagulase test interpreted at 24 h (TCT24).

291 Data suggest that the tube coagulase test may be interpreted at 4 h (TCT4) with lit

292 o difference in the performance of the slide coagulase test or in susceptibility testing performed on

293 on TSA II was confirmed by Gram staining, a coagulase test, and a cefoxitin disk test.

294 coccus aureus by Gram stain morphology and a coagulase test.

295 Isolates were coagulase tested and identified to the species level usi

296 Direct tube coagulase testing for identification of Staphylococcus a

297 uding VWF-binding protein (vWbp), a secreted coagulase that activates the host's prothrombin to gener

298 protection against challenge with different coagulase-type S. aureus strains in mice was derived.

299 5 pulsed-field gel electrophoresis types, 4 coagulase types, and 2 ribotypes.

300 us hyicus and Staphylococcus agnetis are two coagulase-variable staphylococcal species that can be is