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1 ted percutaneous ablation may induce greater coagulation necrosis.
2 o an electrode cluster induced 4.5-7.0 cm of coagulation necrosis.
3 nnel with only a minor degree of surrounding coagulation necrosis.
4 and significantly larger zones of RF-induced coagulation necrosis.
5 t in site-specific differences in RF-induced coagulation necrosis.
6                                          The coagulation necrosis achieved for a standardized RF appl
7                              The diameter of coagulation necrosis achieved with optimal RF deposition
8  grade, endotheliitis, transplant arteritis, coagulation necrosis, acute pancreatitis, presence of in
9  Blood flow was correlated with both induced coagulation necrosis and tissue temperatures.
10 ons at 4 weeks, slightly variable margins of coagulation necrosis, and occasional bacterial infection
11 crophages and myofibroblasts surrounding the coagulation necrosis area (42.9 and 113.6 vs 7.3 and 46.
12 ast agent enabled the immediate detection of coagulation necrosis as a region devoid of contrast enha
13 fty-five tissue specimens were classified as coagulation necrosis (CN), thermal artifact only, or tum
14  (external) lased areas, investigators found coagulation necrosis covered by fibrin and coagulated bl
15 rhage; higher doses of endotoxin resulted in coagulation necrosis, hemorrhage, areas of fibrin deposi
16 tion therapy increases the extent of induced coagulation necrosis in rat breast tumors, as compared w
17 - 0.1, 6.2 cm +/- 0.1, and 7.0 cm +/- 0.2 of coagulation necrosis, respectively.
18                                    Resultant coagulation necrosis was compared between treatment grou
19               At hematoxylin-eosin staining, coagulation necrosis was observed in all ablation zones,
20                                              Coagulation necrosis was observed only within treated tu
21    In the subacute animal, there was diffuse coagulation necrosis with neutrophilic infiltrates at th