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1 gulation and other analytical limitations of coagulation tests.
2 Ab1 showed anticoagulant activity by global coagulation tests.
3 tivity, but demonstrate inhibition of global coagulation tests.
4 f normal cFIX antigen levels, which improved coagulation tests.
5 bin 16 g/dl), with normal liver function and coagulation testing.
6 etry panels, parathyroid hormone assays, and coagulation testing.
7 The method could extend and improve current coagulation testing.
9 anding of TSOA pharmacology, their effect on coagulation tests and, hence, a correct interpretation o
10 nificant coagulopathy (as defined by routine coagulation tests) and is used to justify preprocedure u
11 itory effect on PL-restricted in vitro blood coagulation tests, and are comprised mainly of Ab agains
12 val, expression levels, in vitro and in vivo coagulation tests, and histopathology for up to 16 month
13 s been reinforced by the fact that screening coagulation tests (APTT, prothrombin time--INR) are ofte
16 ally ill patients with deranged conventional coagulation tests are often perceived to have an increas
19 with TBI have abnormalities on conventional coagulation tests at admission to the emergency departme
20 lation has been difficult using conventional coagulation tests, but thrombocytopenia, fibrin polymeri
21 , although some of the more widely available coagulation tests can provide information that is potent
31 chest tube bleeding, whereas platelet count, coagulation tests, heparin dose, and thrombotic events w
32 thromboelastometry (ROTEM) and conventional coagulation testing in patients with Crimean-Congo haemo
33 coagulation assessment was based on standard coagulation tests in 8 centers (50%), on viscoelastic as
34 nicopathological characteristics and routine coagulation tests including prothrombin time, and intern
36 ma transfusion in either correcting abnormal coagulation tests or reducing perceived risk of hemorrha
37 1% (10 of 123) of patients with any abnormal coagulation test results and 9.7% (85 of 877) of patient
39 clinicians in the setting of mildly abnormal coagulation test results, and there is no evidence that
42 ancer range from asymptomatic basic abnormal coagulation tests to massive clinical thromboembolism, w
44 ted and blood samples for ROTEM analysis and coagulation testing were drawn at admission and during h
48 tometry/platelet aggregometry), conventional coagulation tests, whole blood counts, and platelet flow
49 dministered to address abnormal preoperative coagulation tests, with the hope to mitigate bleeding co