ライフサイエンスコーパス: coaptation

1 d annulus; or between TV leaflets, improving coaptation.

2 weeks, correlating to a compromised leaflet coaptation.

3 act and mitral annulus that enhanced leaflet coaptation.

4 ective tissues in the urethral wall also aid coaptation.

5 mia by reducing leaflet tethering to improve coaptation.

6 ormal valve morphology or incomplete leaflet coaptation.

7 te coaptation, and decreased the duration of coaptation.

8 was the result of a lack of complete leaflet coaptation.

9 ther posterior MVP (12) or abnormal anterior coaptation (5).

10 al leaflet closure (IMLC: apically displaced coaptation) also have restricted diastolic leaflet openi

11 ly compatible donor with primary optic nerve coaptation and conventional postoperative immunosuppress

12 cal interference with TV leaflet mobility or coaptation and is amenable to lead extraction or valve r

13 minimum number of basal chordae can improve coaptation and reduce ischemic MR.

14 to the correct length to restore MV leaflet coaptation and secured at the epicardium.

15 I+PNT, suggesting that urethral mucosal seal coaptation and tissue elasticity also contribute to cont

16 systolic regurgitant volume before complete coaptation, and decreased the duration of coaptation.

17 ameter (TAD), apical displacement of leaflet coaptation, and estimated PA systolic pressure were dete

18 lation (size), the mode of tricuspid leaflet coaptation, and tricuspid leaflet tethering-factors ofte

19 on reserve of >3.0 mm (P<0.001), addressable coaptation area of >=52 mm(2) (P<0.001), and coaptation

20 Addressable coaptation area was calculated using the physical bounda

21 n reserve, its augmentation, and addressable coaptation area were 3.7 (2.8-4.5) mm, 7.3 (5.2-9.5) mm,

22 n reserve, its augmentation, and addressable coaptation area were strong predictors of MR reduction (

23 coaptation area of >=52 mm(2) (P<0.001), and coaptation augmentation of >=4.7 mm (P<0.001).

24 s annular dilatation, which hindered leaflet coaptation by drawing the individual scallops apart.

25 pheral innervations and control, and mucosal coaptation contribute to maintenance of continence.

26 +/- 16% vs. 42 +/- 16%; p = 0.0001) and less coaptation deficiency (1.06 +/- 0.24 for repaired and 1.

27 y (change in systolic to diastolic area) and coaptation deficiency (conjoint and reference cusp heigh

28 al anterior leaflet angle (P<0.001), greater coaptation depth and tenting area (P<0.001), larger left

29 ral valve area, type IIIa morphology, larger coaptation depth, and shorter posterior leaflet.

30 let angle (ALAtip), posterior leaflet angle, coaptation depth, tenting area, mitral annular dimension

31 erity, annular dilation, and mode of leaflet coaptation (extent of tethering).

32 Mitral regurgitation occurs from leaflet coaptation failure that is either primary (a problem wit

33 postprocedure residual TR of >moderate were coaptation gaps >=8 mm (OR: 1.67; 95% CI: 1.03-2.72; P =

34 al anterior coaptation phenotype (n=10, with coaptation height >40% of the annulus similar to posteri

35 had greater leaflet displacement, thickness, coaptation height, and mitral regurgitation jet height (

36 terior leaflet projections onto the annulus, coaptation height, and mitral regurgitation jet height i

37 rea (-0.57 +/- 1.1 cm(2)) and increase in MV coaptation length (0.13 +/- 0.2 cm).

38 tenting height (P<0.01) were decreased, and coaptation length was increased (P<0.05) after TAVR.

39 with statistically significant decreases in coaptation length.

40 ral regurgitation without addressing reduced coaptation lengths and thus increased leaflet surface ar

41 he boundary zone near the annulus and at the coaptation line, with reduced strain concentration in th

42 a result of leaflet tethering with impaired coaptation, most commonly from apical and lateral distra

43 ons that aimed for functional restoration by coaptation of all main available motor and sensory nerve

44 red to cause chronic AR by preventing proper coaptation of the 2 aortic valve cusps.

45 After coaptation of the severed nerve ends, fine wire electrod

46 resulting from LV remodelling that prevents coaptation of the valve leaflets.

47 1), longer lengths from papillary muscles to coaptation (P<0.0001), and more frequent chordal rupture

48 isplacement (n=50) and the abnormal anterior coaptation phenotype (n=10, with coaptation height >40%

49 g was measured from the annulus plane to the coaptation point and tethering area by tracing the leafl

50 no change in the distance between the mitral coaptation point and the septum, as measured in two plan

51 his reflects a more anterior position of the coaptation point in those who developed SAM/ LVOTO.

52 The position of the coaptation point of the mitral leaflets is dynamic and a

53 rior (PL) leaflets and the distance from the coaptation point to the septum (C-Sept) were measured be

54 8 (80%) participants with abnormal anterior coaptation progressed to posterior MVP; 17 (34%) subject

55 Coaptation reserve and its augmentation are simple, inde

56 he best values for optimal MR reduction as a coaptation reserve of >3.0 mm (P<0.001), addressable coa

57 Coaptation reserve was measured as the distance of conti

58 Coaptation reserve, its augmentation, and addressable co

59 er mild or no residual MR, median values for coaptation reserve, its augmentation, and addressable co

60 MR in TIC resulted from decreased leaflet coaptation secondary to annular dilatation in the septal

61 ng angle (P<0.001) despite a similar leaflet coaptation status compared with patients with left-sided

62 minimal annular dilatation distorts leaflet coaptation sufficiently to produce severe mitral regurgi

63 nted, with recent trends to preserve leaflet coaptation surfaces if possible.

64 ce implantation to improve tricuspid leaflet coaptation, thereby reducing TR.

65 gned to provide a surface for native leaflet coaptation to reduce tricuspid regurgitation (TR) by occ

66 n in the control group; furthermore, leaflet coaptation was displaced more apically (5.6+/-2.2 mm, P<

67 reate a double orifice with improved leaflet coaptation was introduced in the early 1990s.

68 Augmentation in coaptation was measured as the total amount of leaflet i

69 ent displaying sufficient leaflet motion and coaptation with only minor paravalvular leakage in some

70 hest strain at the commissures, annulus, and coaptation zones.