ライフサイエンスコーパス: cognitive deterioration

1 a possible role for Deltatau314 proteins in cognitive deterioration.

2 mild cognitive impairment and aging-related cognitive deterioration.

3 of Casp2-mediated Deltatau314 production to cognitive deterioration.

4 y any statistically significant predictor of cognitive deterioration.

5 ncreases amyloid burden, although preventing cognitive deterioration.

6 t epilepsy, and progressive neurological and cognitive deterioration.

7 er TAVI, despite the high intrinsic risk for cognitive deterioration.

8 inant disorders characterized by progressive cognitive deterioration.

9 oligomeric peptides, known to be involved in cognitive deterioration.

10 uvignon modulates AD-type neuropathology and cognitive deterioration.

11 hese findings as an independent mechanism of cognitive deterioration.

12 flammation and, in turn, may lead to further cognitive deterioration.

13 urosurgery carries the risk of postoperative cognitive deterioration.

14 Only a small percentage of patients had cognitive deterioration after radiotherapy.

15 esults confirm that some patients experience cognitive deterioration after TAVI.

16 cantly slower rate of functional, motor, and cognitive deterioration (all p < 0.001), independent of

17 RS alone compared with WBRT resulted in less cognitive deterioration among long-term survivors.

18 genetic effects on the processes leading to cognitive deterioration and Alzheimer's disease.

19 richment may reduce the risk of experiencing cognitive deterioration and dementia in later-life.

20 nt was associated with an increased risk for cognitive deterioration and dementia.

21 al stage of Alzheimer's disease and predicts cognitive deterioration and disease-associated changes i

22 score can be used to increase vigilance for cognitive deterioration and for selection of high-risk p

23 disorder characterized by motor impairment, cognitive deterioration and psychiatric disturbances fol

24 inetic and/or hypokinetic movement disorder, cognitive deterioration and psychiatric disturbances.

25 al-AZ (MN), significantly attenuated AD-type cognitive deterioration and reduced cerebral amyloid dep

26 clinical stage of AD and is associated with cognitive deterioration and structural brain changes ove

27 enetic liability for ADHD is associated with cognitive deterioration and the development of AD pathop

28 to neuroregeneration are twofold: arresting cognitive deterioration (and directing the brain toward

29 Intracranial tumor control, toxic effects, cognitive deterioration, and QOL.

30 The disease results in blindness, motor and cognitive deterioration, and seizures.

31 human loci that protect against age-related cognitive deterioration arising from neurodegenerative d

32 loric restriction (CR) may delay age-related cognitive deterioration as well as the onset of neurolog

33 There was less cognitive deterioration at 3 months after SRS alone (40/

34 ith SRS combined with WBRT, resulted in less cognitive deterioration at 3 months.

35 [HR] 0.47 [95% CI 0.35-0.63]; p<0.0001), and cognitive deterioration at 6 months was less frequent in

36 gelastic seizures are often associated with cognitive deterioration, behavioral problems, and poor r

37 d no major impact on the disease or delaying cognitive deterioration, but lowering the risk of vascul

38 copolamine revealed that ovariectomy-induced cognitive deterioration coincided with a compromised cho

39 preparation significantly attenuates AD-type cognitive deterioration coincidentally with reduced HMW

40 ssion who experienced clinically significant cognitive deterioration compared with baseline was stabl

41 The primary end point was cognitive deterioration (decline >1 SD from baseline on

42 attenuated the development of Abeta-mediated cognitive deterioration, even when delivered at a dose a

43 The co-primary endpoints were cognitive-deterioration-free survival and overall surviv

44 Cognitive-deterioration-free survival was longer in pati

45 nce and Kruskal-Wallis analysis; the odds of cognitive deterioration (> or =3-point decline on the mo

46 ify risk factors associated with significant cognitive deterioration (>10% decline).

47 n hippocampal neurogenesis may contribute to cognitive deterioration, however, the fate of neurogenes

48 ilepsy and epileptiform activity can precede cognitive deterioration in AD by years, and its presence

49 brocortical regions to the earliest signs of cognitive deterioration in AD.

50 An optimised threshold of ferritin predicted cognitive deterioration in mild cognitive impairment sub

51 ) visible on MRI are associated with SVD and cognitive deterioration in older individuals is unknown.

52 tigated functional hub load and postsurgical cognitive deterioration in patients undergoing lesion re

53 We confirmed the presence of a widespread cognitive deterioration in PP-MS patients, with main inv

54 p-tau181), clinical staging and longitudinal cognitive deterioration in subjects from the BioFINDER c

55 ssociated with a significant acceleration of cognitive deterioration in the absence of any overt chan

56 nce activity rhythms will reduce the risk of cognitive deterioration in the elderly.

57 e, whether their presence is associated with cognitive deterioration in the general population remain

58 incorrect assumptions that neurotoxicity or cognitive deterioration may be avoided.

59 Furthermore, the behavioral and cognitive deterioration observed in late life did not co

60 Cognitive deterioration occurred in 1 patient and electr

61 der from the psychosis and severe social and cognitive deterioration of chronic schizophrenia.

62 Cognitive deterioration or improvement was defined as ch

63 No significant cognitive deterioration or loss of language occurred, an

64 Whereas some contribute to progressive cognitive deterioration, others lower the level of cogni

65 A 72-year-old man sought care for a cognitive deterioration over the past 5 years.

66 roportion of high-grade glioma patients with cognitive deterioration over time is stable, most consis

67 In view of the minimal cognitive deterioration overall in this group, this sugg

68 e present study, intracranial tumor control, cognitive deterioration, QOL, and cognitive outcomes wer

69 lications both for the spread of tau and the cognitive deterioration that may occur over an 18-month

70 AD is characterized by gradual cognitive deterioration, the buildup of misfolded amyloi

71 ment (MCI), an early stage of age-associated cognitive deterioration, underwent cognitive testing, in

72 Thus, cortical iron might act to propel cognitive deterioration upon the underlying proteinopath

73 Odds ratios for cognitive deterioration using 0 to 2 symptoms as the ref

74 Cognitive deterioration was found in 29 patients (12.7%)

75 For long-term survivors, the incidence of cognitive deterioration was less after SRS alone at 3 mo

76 Cognitive deterioration was less frequent with SRS (37%-

77 high ADHD-PRS and brain Abeta deposition on cognitive deterioration was more significant than each i

78 asingly recognized as a general mechanism of cognitive deterioration with aging.

79 The association of late cognitive deterioration with WBRT was clinically meaning