ライフサイエンスコーパス: collapsibility

1 old is a predictor of increased upper airway collapsibility.

2 d tongue and less-severe baseline pharyngeal collapsibility.

3 efulness to non-REM sleep and reduces airway collapsibility.

4 uring sleep and thereby decreases pharyngeal collapsibility.

5 ctility and assessment of inferior vena cava collapsibility.

6 y affect more than just upper-airway anatomy/collapsibility.

7 ut has a minimal effect on pharyngeal airway collapsibility.

8 was not correlated with severity of tracheal collapsibility.

9 l threshold (1.39 0.15 s, P < 0.01), reduced collapsibility (-0.71 0.16 s, P < 0.01), and higher loop

10 Ggg activity and inspiratory flow and airway collapsibility; (3) reflex increases in flow (peak flow

11 geal pathophysiology per se (i.e., increased collapsibility and decreased muscle responsiveness).

12 pnea rely on both more favorable anatomy and collapsibility and enhanced upper-airway dilator muscle

13 iate analysis, baseline passive upper-airway collapsibility and loop gain were independent predictors

14 four phenotypic traits (upper-airway anatomy/collapsibility and muscle function, loop gain, and arous

15 ese patients often have air trapping, airway collapsibility, and a high degree of methacholine hyperr

16 Traits-loop gain, arousal threshold, collapsibility, and muscle compensation-were calculated

17 posteriorly-located tongue plus less-severe collapsibility are the strongest candidates for oral app

18 higher for 50% or greater visually assessed collapsibility (area: 53% +/- 9 and lumen: 52% +/- 10) c

19 Tracheal collapsibility between groups was compared employing one

20 gest that co-activation decreases pharyngeal collapsibility but does not dilate the pharyngeal airway

21 es upper airway muscles, alters upper airway collapsibility by a mechanism similar to tracheal or ton

22 t two basic mechanisms by which upper airway collapsibility can be altered.

23 n pathophysiological contributions of airway collapsibility, chemoreceptive negative feedback loop ga

24 out oral appliance were performed to measure collapsibility (critical closing pressure; Pcrit) and as

25 This flat-ribbon fan balances collapsibility during leg recovery with rigidity during

26 h apnea severity and indices of upper airway collapsibility during NREM sleep.

27 gative epiglottic pressure, and upper airway collapsibility during passive and active conditions were

28 properties of the upper airway determine its collapsibility during periods of muscle hypotonia.

29 erapeutic devices and agents on upper airway collapsibility during sleep.

30 - 10 ms (n.s. vs. awake)) and greater airway collapsibility during the applied pressures (P = 0.043 v

31 iles, including zero initial stiffness, full collapsibility, high power-to-weight ratio, puncture res

32 was a predictor of measures of upper airway collapsibility, including the hypopnea/apnea + hypopnea

33 We found that Pcrit rose (collapsibility increased, p < 0.001) and RN fell (p = 0.

34 Airway collapsibility increases in the injured lung with a sign

35 nterval 0.65-0.89) or the inferior vena cava collapsibility index (area under the curve 0.66; 95% con

36 min)) and end-expiration (IVC(max)), and IVC collapsibility index (IVC(CI)) was calculated.

37 e (R = 0.58), whereas the inferior vena cava collapsibility index and the internal jugular vein aspec

38 The collapsibility index expressed in percentage equaled the

39 The collapsibility index of the inferior vena cava during a

40 To investigate whether the collapsibility index of the inferior vena cava recorded

41 We measured stroke volume index and collapsibility index of the inferior vena cava under a d

42 th a significantly higher inferior vena cava collapsibility index on day 0 than nonacidotic patients

43 venous pressure than the inferior vena cava collapsibility index or the internal jugular vein aspect

44 stic analysis, the area under curve for that collapsibility index was 0.89 (95% CI, 0.82-0.97).

45 Inferior vena cava collapsibility index was not an independent predictor of

46 of the inferior vena cava with inspiration (collapsibility index) by ultrasound.

47 ke volume index, and high inferior vena cava collapsibility index, which improved with subsequent rea

48 nder the logistics model, called "absence of collapsibility," is noted in motivating VanderWeele and

49 Theory also suggests that collapsibility itself could be a physical restriction on

50 Inferior vena cava (IVC) size and collapsibility (IVC dynamics) are used for estimating ri

51 s into three groups: 50% or greater tracheal collapsibility, less than 50% collapsibility, or fixed s

52 Pharyngeal anatomy/collapsibility, loop gain (LG), upper-airway muscle resp

53 scopy) exhibit a preferential improvement in collapsibility (lowered critical closing pressure) with

54 ryngeal physiology was assessed by examining collapsibility (lowered ventilation at eupneic drive) an

55 ctive sleep apnea (OSA), abnormal pharyngeal collapsibility may be offset by increased mechanoreflex-

56 l min(-1); P < 0.05) and improved pharyngeal collapsibility (mean +/- s.d. 3.4 +/- 1.4 l min(-1) vs.

57 rovides important information on both airway collapsibility (mechanics) and ventilatory control, we c

58 threshold, circulatory delay, and pharyngeal collapsibility).Methods: Data were analyzed from 1,546 p

59 formation on ventilatory features and airway collapsibility not captured by the AHI.

60 helped identify more patients with tracheal collapsibility of 50% or greater than did bronchoscopy,

61 essment of 4D CT detected more patients with collapsibility of 50% or greater than paired CT, and con

62 4D CT, 25 of 52 (48%) patients had tracheal collapsibility of 50% or greater, 20 of 52 (38%) less th

63 he pathophysiology of increased upper airway collapsibility of DS and to evaluate the efficacy of the

64 To determine the factors that influence the collapsibility of the hypotonic airway, the critical pre

65 The collapsibility of the isolated UA was examined in older

66 A concomitant increase in the rigidity and collapsibility of the mixed MLF was observed.

67 emodynamic profiles: fluid loading (index of collapsibility of the superior vena cava>/=36%), inotrop

68 al area change<45% without relevant index of collapsibility of the superior vena cava), or increased

69 order would vary depending on the underlying collapsibility of the upper airway.

70 gnized as major determinants of the size and collapsibility of the upper airway.

71 Pathological collapsibility of the upper airways, caused by many diff

72 Mean collapsibility of tracheal lumen area and volume at 4D C

73 Collapsibility of tracheal lumen area and volume did not

74 Crows that had learned about the mechanism (collapsibility) of the platform without the use of stone

75 eater tracheal collapsibility, less than 50% collapsibility, or fixed stenosis.

76 associated with decreased upper airway (UA) collapsibility (p < 0.05), unchanged maximum flow, and i

77 teriorly-located tongue (p = 0.03) and lower collapsibility (p = 0.04) at baseline were significant d

78 , P = 0.004), and lower loop gain (in milder collapsibility, per significant interaction, P = 0.003).

79 h posteriorly-located tongue and less severe collapsibility (predicted responder phenotype) exhibit g

80 erotonin(2A/2C) receptor agonist improves UA collapsibility predominantly, but not exclusively, via s

81 ral appliance therapy (i) reduces pharyngeal collapsibility preferentially in patients with posterior

82 here was a smaller decrease (p < 0.05) in UA collapsibility that was also associated with increased u

83 Upper airway collapsibility (UAC) is increased in children with sleep

84 iven by improvements in upper-airway anatomy/collapsibility under passive (1.9 +/- 0.7 vs. 4.7 +/- 0.

85 st that OA therapy improves the upper-airway collapsibility under passive and active conditions.

86 d not significantly alter pharyngeal anatomy/collapsibility, upper-airway gain, or arousal threshold.

87 Purpose To quantify tracheal collapsibility using low-dose four-dimensional (4D) CT a

88 However, there was no effect of REM sleep on collapsibility (ventilation at eupneic drive), baseline

89 r correlation coefficient was calculated for collapsibility versus pulmonary function tests.

90 p gain, arousal threshold (ArTH), pharyngeal collapsibility (Vpassive), and pharyngeal muscle compens

91 Upper airway collapsibility was also reduced with desipramine compare

92 Pharyngeal collapsibility was quantified as the ventilation at CPAP

93 in 'CPAP pressure drops': pharyngeal anatomy/collapsibility was quantified as the ventilation at CPAP

94 of arousal, apnea severity and upper airway collapsibility were ascertained during NREM sleep.

95 The LVIDs and IVC collapsibility were independent predictors for mortality