ライフサイエンスコーパス: collateral blood flow

1 followed by an ischemia-induced increase in collateral blood flow.

2 with no evidence of preinfarction angina or collateral blood flow.

3 surgical instrumentation and measurement of collateral blood flow.

4 PAF caused a decrease in coronary collateral blood flow.

5 actor (PAF) can cause a decrease in coronary collateral blood flow.

6 a from the perfusion circuit, mainly through collateral blood flow.

7 without evidence of preinfarction angina or collateral blood flow.

8 g an inflammatory response, does not improve collateral blood flow.

9 t intraarterial vasodilators actually reduce collateral blood flow acutely, by preferentially dilatin

10 devascularized segment maintained viable by collateral blood flow after 48 hours.

11 emia, it has also been advocated to increase collateral blood flow after mesenteric vascular occlusio

12 The improved collateral blood flow and increased muscle capillary den

13 se findings suggest that factors that affect collateral blood flow and metabolism account for approxi

14 the Circle of Willis are a central source of collateral blood flow and play an important role in path

15 in the ischemic penumbra, thereby increasing collateral blood flow and significantly reducing ischemi

16 ic stroke transferred for thrombectomy, poor collateral blood flow and stroke clinical severity are t

17 o test this hypothesis, we measured coronary collateral blood flow and VEGF expression in myocardial

18 est that an accurate noninvasive estimate of collateral blood flow can be provided by an intravenous

19 ia (DI) is principally due to the effects of collateral blood flow (CollBF).

20 f NO and control hypertension, respectively, collateral blood flow did not increase and reactive hype

21 O contributes to the maintenance of coronary collateral blood flow during exercise.

22 Collateral blood flow during occlusion was measured with

23 ously administered VEGF was shown to augment collateral blood flow in animals and patients with exper

24 re of the severity of ischemia) decreased as collateral blood flow increased.

25 moderate plasma elevations of ET-1, whereas collateral blood flow is unchanged.

26 tory) and protective (eg, increased coronary collateral blood flow) mechanisms may operate, but remai

27 In controls, collateral blood flow (microspheres) progressively incre

28 unds studied affected systemic hemodynamics, collateral blood flow, or AAR.

29 ed, presence and extent of prior infarction, collateral blood flow, plaque characteristics, microvasc

30 tine ganglion stimulation increased cerebral collateral blood flow, stabilised the blood-brain barrie

31 fibroblast growth factor (bFGF) to increase collateral blood flow to dependent tissue was quantified

32 The adequacy of leptomeningeal collateral blood flow was rated as no or poor, decreased

33 Collateral blood flow was significantly higher in the ca