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1 ial arterial occlusion, and moderate-to-good collateral circulation.
2 eral extent for use in investigations of the collateral circulation.
3 the best technique to quantify the degree of collateral circulation.
4 ients with extensive versus poorly developed collateral circulation.
5  to unravel the pathways leading to improved collateral circulation.
6 se of the possible interruption of important collateral circulation.
7  of the luminal diameter, and the absence of collateral circulation.
8 w-up, which may be related to development of collateral circulation.
9 bility, especially in patients with coronary collateral circulation.
10 esis in rat hearts, which have little native collateral circulation.
11 tenoses and obstructions and to evaluate the collateral circulation.
12 le in small vessel ischemic disease or brain collateral circulation.
13 n test occlusion (BTO) is used to assess the collateral circulation and cerebrovascular reserve in pa
14                No association between patent collateral circulation and favorable outcome was found.
15 of the final infarct volume are the grade of collateral circulation and the clot burden score, especi
16  score to record the grade of leptomeningeal collateral circulation and the extension of the thrombus
17 uding organogenesis, wound healing, ischemic collateral circulation, and solid tumor growth.
18 gated the relationship between ischemia, the collateral circulation, and symptoms in stable coronary
19 ctors, including the grade of leptomeningeal collateral circulation, as well as the length, density,
20 icrovessels resulting in the re-formation of collateral circulation associated with enhanced neuronal
21 eatment remains undefined, and the status of collateral circulation at presentation may be a key feat
22 ns vascular remodeling, a key determinant of collateral circulation (CC) in ischemic disease, yet the
23 tion of the palmar arch and forearm arterial collateral circulation during transradial access.
24 l hypertension and the dynamic physiology of collateral circulation, gastric variceal classification
25  of neovascularization in the development of collateral circulation in coronary artery disease.
26                       We compared the native collateral circulation in healthy tissue and collateral
27  mechanisms responsible for formation of the collateral circulation in healthy tissues.
28  infarct core, salvageable brain tissue, and collateral circulation in patients suspected of acute st
29  underlines the importance of being aware of collateral circulation in patients with chronic aortoili
30 ostatin) related to the presence of coronary collateral circulation in patients with significant coro
31                                              Collateral circulation is a potent determinant of stroke
32 ed adult coarctations, particularly when the collateral circulation is in doubt.
33                        Although the coronary collateral circulation is known to react to several vaso
34                                          The collateral circulation is tissue- and life-saving in obs
35 ng cardiomyocytes, suggesting that increased collateral circulation may provide an important source o
36                                     Coronary collateral circulation may sustain ischemic, dysfunction
37 n of brain perfusion because, theoretically, collateral circulations may restore it.
38     In an unusual situation of bleeding from collateral circulation near the pseudocyst wall during p
39   Patients with a large infarct core or poor collateral circulation on computed tomography (CT) and C
40 ts with BAO and to evaluate the influence of collateral circulation on outcome.
41 he level of the subclavian vein and abnormal collateral circulation over the chest wall consistent wi
42  correlated with the grade of leptomeningeal collateral circulation (p=0.03) and with the clot burden
43                           The development of collateral circulation plays an important role in protec
44                    A well-developed coronary collateral circulation provides a potential source of bl
45 , a small infarct core, and moderate-to-good collateral circulation, rapid endovascular treatment imp
46 ew microvessels and leads to re-formation of collateral circulation, reperfusion and better recovery.
47                In this review, we define the collateral circulation, review methods of its measuremen
48 enous imaging-based biomarkers with grade of collateral circulation, the ischaemic penumbra and clini
49 the brain is sufficiently reperfused through collateral circulation to allow reemergence of conscious
50                          The capacity of the collateral circulation to lessen injury in occlusive vas
51 s ischemic brain injury in SHR by increasing collateral circulation to the ischemic region and allowi
52 tenotic plaques are more likely to stimulate collateral circulation to the post-stenotic segment, pla
53                            The growth of the collateral circulation was functional in the RI group be
54 lumped parameter model of the human coronary collateral circulation was used to simulate normal and a
55  who received nitroglycerin and had coronary collateral circulation were more likely to exhibit impro
56 he vexing dilemma that an effective coronary collateral circulation, which is determined primarily by
57               In humans, suboptimal cerebral collateral circulation within the circle of Willis (CW)