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1  are at the crux of these toxic effects, or "collateral damage".
2 omiscuous cleavage of nearby RNAs (so-called collateral damage).
3 eter of 6.62 mm but retain the potential for collateral damage.
4 rmful to the host, but can also cause severe collateral damage.
5 infection, which led to economic crises as a collateral damage.
6 chinery to protect against and tolerate this collateral damage.
7 endent apoptosis of individual cells without collateral damage.
8 he efficacy of cancer therapy while limiting collateral damage.
9 f lymphocytes, while minimizing the risk for collateral damage.
10  a thermally confined manner without causing collateral damage.
11 that downregulate hyperinflammation to avoid collateral damage.
12 g structures, thereby minimizing the risk of collateral damage.
13 he penetration force is important to prevent collateral damage.
14 ng cellular functions and protecting against collateral damage.
15 a key role of the immune system, but without collateral damage.
16 ollagen vibrational modes results in minimal collateral damage.
17 human corneoscleral rim tissues, with little collateral damage.
18 ined volumes of soft tissue with very little collateral damage.
19 s, optimizing host protection and minimizing collateral damage(1,2).
20 hs showed similar features, i.e., 2 zones of collateral damage, a zone generally < 10 mm of extensive
21 dormancy, suggesting insignificant levels of collateral damage against host RNA or DNA.
22 sed for transfection, but causes significant collateral damage and a high rate of cell death, especia
23 h danger signals, are needed to protect from collateral damage and are of therapeutic interest.
24  quell overzealous immune signaling to limit collateral damage and enable inflammation resolution.
25 am to prevent excessive inflammation, repair collateral damage, and restore tissue homeostasis, and f
26  of off-target cleavage events and potential collateral damage are still lacking.
27 t a major role of tolerance is in minimizing collateral damage associated with inflammation.
28 y impact on trafficker behavior and the many collateral damages associated with the militarized war o
29 s study was to compare lesion durability and collateral damage between focally delivered unipolar/bip
30 Lysis of bystanders is viewed as acceptable "collateral" damage, but the persistent presence of activ
31 ated immunosuppression protects tissues from collateral damage by antipathogen immune cells.
32  rapid journal turnaround times, may produce collateral damage by incentivizing lower-quality researc
33  that protects normal tissues from excessive collateral damage by overactive immune cells and their p
34 by enabling an antimicrobial defense without collateral damage by the adaptive immune system.
35 lated with target RNA expression levels, and collateral damage can be observed even after reducing Rx
36 e ongoing and new strategies to overcome the collateral damage caused by antibiotics and to limit the
37 oduced by effector Th1 cells helps limit the collateral damage caused by exaggerated inflammation.
38  fibrin deposition protects host tissue from collateral damage caused by the immune system as it comb
39 ning to insert foreign DNA and HDR to repair collateral damage caused by the microprojectiles.
40 mpaction, aggregates blood cells, and causes collateral damage due to leukocyte activation.
41 hus the host can benefit from suppression of collateral damage during parasite infection and from red
42 e to accumulating insights, and it may incur collateral damage (e.g., impairing cognitive processes a
43 ) hepatectomy (eHx) was modified to minimize collateral damage; effects were compared with those of s
44 plications create transmural lesions without collateral damage experimentally and whether they can sa
45 els of SBS2/SBS13 in the small intestine are collateral damage from APOBEC1 fulfilling its physiologi
46 n dogs with sDM and sDMPanc, suggesting that collateral damage from inflammation in the exocrine panc
47  injury in vivo as well as greatly decreased collateral damage from inflammation.
48 deling following I/R injury secondary to the collateral damage from sustained myocardial inflammation
49 that these broad events are a consequence of collateral damage from targeting single loci.
50 rance of pathogenic organisms while limiting collateral damage from the host inflammatory response, k
51 mpts to explain the wavelength-dependence of collateral damage have invoked a wavelength-dependent lo
52                       To prevent detrimental collateral damage, IL-1beta release is tightly controlle
53 anding the observed wavelength-dependence of collateral damage in mid-infrared laser ablation.
54  of patients with cancer, but it also causes collateral damage in the body that can lead to treatment
55 lation resulted in decreased cellularity and collateral damage in the tissue during viral infection.
56 , when it is drawn into battle, it can cause collateral damage in tissues.
57  in the protection of cells and tissues from collateral damage induced by inflammatory responses.
58                                          The collateral damage induced on healthy tissues during radi
59 ither destroy foreign DNA directly or elicit collateral damage inducing death of infected cells.
60 cts in the Arab world are much more than the collateral damage inflicted on civilians, infrastructure
61              How the immune response to this collateral damage influences brain maturation and functi
62                                        Such 'collateral damage' is prohibited by international laws b
63                                     To avoid collateral damage leading to tissue injury and organ dys
64                                 Such hepatic collateral damage may be a general consequence of expand
65  conditions, potentially as a means to limit collateral damage of bacterial mutualists.
66                                          The collateral damage of early-life antibiotic treatment and
67                             To mitigate this collateral damage of macrolides and tetracyclines, we sc
68 ute to protection of commensal bacteria from collateral damage of plant glucosinolate-based defense r
69 exerts on an individual's own health via the collateral damage of the drug on bacteria that normally
70  T cells while simultaneously minimizing the collateral damage of their potentially lethal actions an
71 ether IL-10 is also involved in limiting the collateral damage of vigorous T cell responses, has not
72 in the liver, making this a pure example of "collateral damage" of the liver.
73 , the repression of CMT3 prevents epigenetic collateral damage on endogenous genes.
74 rget cancer cells have the ability to reduce collateral damage on normal tissue due to pan-toxic effe
75  are far from perfect and impose significant collateral damage on the fragile endothelial cell (EC),
76 heir therapeutic benefits, antibiotics exert collateral damage on the microbiome and promote antimicr
77 fects harmful to hermaphrodites appear to be collateral damage rather than the goal.
78 o a more complete explanation of the reduced collateral damage resulting from infrared laser irradiat
79 rable safety profile by avoiding much of the collateral damage seen with conventional thermal ablatio
80 d (5) hospital personnel or those considered collateral damage shot, injured, or arrested (5 individu
81 ble to it, the pandemic has caused extensive collateral damage that has led to losses of lives and li
82 x with aspect ratio of 12:1, and without the collateral damage that is observed in unmodulated CW dri
83 tems of several countries in addition to the collateral damage that societies will face in the next y
84                      Despite this well-known collateral damage, the activity spectrum of different an
85 r, recent studies have shed new light on the collateral damage they impart on the indigenous host-ass
86 n overly exuberant immune response can cause collateral damage through immune effectors and because o
87  and/or infected cells while also minimizing collateral damage to adjacent noninfected tissues.
88 ssary chemoradiation treatment can result in collateral damage to adjacent vital structures causing a
89 se lesions focally ablated PRs, with minimal collateral damage to cells above and below the plane of
90 t unfolding activity is prevented from doing collateral damage to cellular proteins are not well unde
91 ion of antimicrobial-resistant organisms and collateral damage to commensal microbes.
92 inst MRE colonization, but antibiotics cause collateral damage to commensals and open the way to colo
93 bundance target bacteria without significant collateral damage to complex microbial communities.
94 f the hippocampus (HIPP) or hippocampus plus collateral damage to extrahippocampal structures (HCX) w
95 . cancer cells or parasites) without causing collateral damage to healthy or to host cells is complic
96  catalysis is a promising strategy to reduce collateral damage to healthy tissues and organs.
97 contribute to the inactivation of pathogens, collateral damage to host proteins can also occur and ha
98  is a host response to infection that limits collateral damage to host tissues while having a neutral
99 acterium tuberculosis in vivo but also cause collateral damage to host tissues.
100 sponses counteract infections but also cause collateral damage to hosts.
101 e efficiency of targeted killing and prevent collateral damage to neighboring healthy cells.
102 ion of post-operative quality of life due to collateral damage to neighboring structures.
103 ate tissue with negligible heat transfer and collateral damage to neighboring tissue.
104 and often severe side effects as a result of collateral damage to normal cells.
105 is specifically in cancer cells with minimal collateral damage to normal cells.
106 or removal of damaged neurons, but can cause collateral damage to normal neurons located close to def
107 nciple, yet it is accompanied by significant collateral damage to normal tissue and unwanted side eff
108 ruction of pathogens may result in excessive collateral damage to normal tissues, and the failure to
109 ted to infect only myeloma cells to minimize collateral damage to normal tissues: viral binding to it
110  effective chemotherapy treatments with less collateral damage to ocular tissues and may allow reduce
111 an promote genome evolution while minimizing collateral damage to overall chromosome architecture and
112 ced peripheral neuropathy (CIPN) arises from collateral damage to peripheral afferent sensory neurons
113 ontrolled oxidant bursts could cause serious collateral damage to phagocytes or other host tissues, p
114 djunctive neuroprotective therapy may reduce collateral damage to photoreceptors and improve visual o
115 ole target of FALI and provide evidence that collateral damage to proximal proteins occurs following
116 lled death of cells that occurs with minimal collateral damage to surrounding cells or tissue during
117 while effective against cancer, often causes collateral damage to surrounding healthy tissues, leadin
118 o acts non-autonomously to limit ROS-induced collateral damage to surrounding tissues.
119 wed for unprecedented precision with minimal collateral damage to surrounding tissues.
120 ness of the A1 pulley release and detect any collateral damage to the A2 pulley, interdigital nerves,
121 es were dissected by laser axotomy, avoiding collateral damage to the adjacent dendrite and the forma
122  while minimizing their potential to inflict collateral damage to the adjacent lung tissue and indica
123                                      Causing collateral damage to the B-cell genome during CSR and SH
124  therapies in some cases, but issues such as collateral damage to the commensal microbiota and consis
125 s is 43.1% lower, highlighting a decrease in collateral damage to the environment.
126  as a general treatment, potentially causing collateral damage to the gut microbiome of the patient.
127  viral persistence and survival with minimal collateral damage to the healthy host.
128 o tune the immune response, thereby limiting collateral damage to the host and the risk for sepsis.
129 mune system controls inflammation and limits collateral damage to the host during acute bacterial inf
130  in order to optimize responses and to avoid collateral damage to the host, immune responses must be
131 ective antimicrobial protection with minimal collateral damage to the host.
132 , to prevent excessive inflammation to limit collateral damage to the host.
133 d downregulates Th1 responses that may cause collateral damage to the host.
134 as the eradication of pathogens with minimal collateral damage to the host.
135 nt antimicrobials, however, exert tremendous collateral damage to the human microbiome through overus
136 ive papers emerged this past year concerning collateral damage to the liver in extrahepatic infection
137 lance of initiating immunity without causing collateral damage to the lungs because of an exaggerated
138 lusively within the tumors, and there was no collateral damage to the neighboring hepatic parenchyma.
139 ffects pathologic vessels, it can also cause collateral damage to the overlying retina.
140 to target a specific pathogen can also cause collateral damage to the patient's resident microbial po
141 ding histology sections exhibit only minimal collateral damage to the surrounding tissue and the dept
142 s and can treat microvessels without causing collateral damage to the surrounding tissue.
143                                              Collateral damage to the vagal nerve and the upper gastr
144                       In contrast, mice with collateral damage to the ventromedial hypothalamus and p
145 easy-to-implement techniques without causing collateral damage to true memories.
146 these nasty invaders while also constraining collateral damage to vital tissue.
147                          Finally, no sign of collateral damage was observed in any of the groups.
148                                     However, collateral damage was observed with both catheters in 17
149 e empiric therapy against toxicity risks and collateral damage when selecting antibiotic therapy for
150  this beneficial activity comes at a cost of collateral damage when the immune system overreacts to i
151  Our data suggest that T6SS toxins may avoid collateral damage within a complex ecosystem by recogniz

 
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