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1 iferation of Itpkb(-/-) HSC without rescuing colony forming activity.
2 hese cells lacked radioprotection and spleen colony-forming activity.
3 itself in rescuing signaling, survival, and colony-forming activities.
4 nd microRNA-196b inhibited in vitro leukemic colony forming activity and depleted in vivo leukemia-in
5 MLL(DNMT1 CXXC)-AF9 shows robust in vitro colony forming activity and in vivo leukemogenesis, simi
6 ted ROS levels, exhibiting up to 4-fold less colony-forming activity and 4-fold less capability for d
7 em cells from Pbx1(-/-) embryos have reduced colony-forming activity and are unable to establish mult
10 splenomegaly, an increase in splenic myeloid colony-forming activity, and marked granulocytosis in th
11 rleukin-3 (IL-3), has enhanced hematopoietic colony-forming activity as compared with individual or e
12 M domain was required for SAMD14 to increase colony-forming activity, c-Kit signaling, and progenitor
13 ineage differentiation capacity, and possess colony-forming activity, despite constituting a small fr
14 ficient to abrogate the observed increase in colony forming activity implying a direct role for strom
16 ed several defects in hematopoiesis, reduced colony-forming activity in vitro, decreased expression o
17 3 and OVCAR-3 ovarian cancer cells decreases colony-forming activity, increases apoptosis, and decrea
19 ation was demonstrated by the suppression of colony forming activity of v-Crk NIH 3T3 cells when a do
24 nontransfected parental cells decreased the colony-forming activity of the parental cells, compatibl
25 referentially depleted myeloid and erythroid colony-forming activity rather than CD4-bearing thymocyt
27 and a unique mouse Runx1bEx6e showed higher colony-forming activity than the full-length Runx1b (Run