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1 y, in vitro burst-forming unit-erythroid and colony-forming unit-erythroid assays showed increased er
2 ficant reduction in fetal liver cellularity, colony forming unit-erythroid (CFU-E) progenitors, and a
3 sis of PML+/+ and PML-/- MEFs, and inhibited colony-forming unit erythroid (CFU-E) and CFU granulocyt
5 ype burst-forming unit erythroid (BFU-e) and colony-forming unit erythroid (CFU-e) progenitors in res
6 ously undescribed progenitor population with colony-forming unit-erythroid (CFU-E) activity (stress C
9 red burst-forming unit-erythroid (BFU-E) and colony-forming unit-erythroid (CFU-E) formation compared
11 subsequent increase in the total numbers of colony-forming unit-erythroid (CFU-E) progenitors and er
12 gp55-P induce erythroid differentiation from colony-forming unit-erythroid (CFU-E) progenitors in fet
13 reased in Mll -/- cultures, while numbers of colony-forming unit-erythroid (CFU-E), colony-forming un
14 th antisense oligodeoxynucleotides inhibited colony-forming unit-erythroid (CFU-E)-derived colony gro
18 w erythroid burst-forming units (BFU-Es) and colony-forming units-erythroid (CFU-Es) activities and r
19 cyte-erythrocyte progenitors (PreMegEs), and colony-forming units-erythroid (CFU-Es), as well as myel
20 and myeloid hematopoietic progenitor cells (colony-forming unit-erythroid [CFU-E], burst-forming uni
21 results in a loss of erythroid progenitors (colony-forming units-erythroid, CFU-E) and severe anemia
24 shown that burst-forming units-erythroid and colony-forming units-erythroid from patients with polycy
25 losuppressive therapy in vivo and to enhance colony-forming unit-erythroid generation in vitro may be
26 odysplasia, disorders with excessive heme in colony-forming unit-erythroid/proerythroblasts, explain
27 er erythroid colonies derived from the later colony-forming unit erythroid progenitor erythropoietin
28 table with erythropoietin (Epo), because the colony-forming unit erythroid progenitors (CFU-Es) that
29 TFR2-deficient mice displayed an increase in colony-forming unit-erythroid progenitors and in all ery
30 n and differentiation of committed erythroid colony-forming unit-erythroid progenitors by external si
31 -derived vector, and its capacity to support colony-forming unit-erythroid proliferation and developm
32 opoiesis at the burst-forming unit-erythroid/colony-forming unit-erythroid transition, but without af
33 entiation of burst-forming unit-erythroid to colony-forming unit-erythroid, we hypothesize that this