ライフサイエンスコーパス: colorectal neoplasm

1 ndent risk of developing CVD, T2DM, CKD, and colorectal neoplasms.

2 and 1,699 had multiple adenomas or advanced colorectal neoplasms.

3 nomas >/=5 mm, multiple adenomas or advanced colorectal neoplasms.

4 odifiable, preneoplastic risk biomarkers for colorectal neoplasms.

5 ND1 A870G polymorphism may increase risk for colorectal neoplasms.

6 cid to prostaglandins and is up-regulated in colorectal neoplasms.

7 n mediating the association between diet and colorectal neoplasms.

8 non that has previously been demonstrated in colorectal neoplasms.

9 ts a new approach for the early detection of colorectal neoplasms.

10 tyrosine kinase inhibitors for treatment of colorectal neoplasms.

11 that the above recommendations apply only to colorectal neoplasms.

12 ry pattern is associated with higher risk of colorectal neoplasms.

13 sensitivity and specificity for detection of colorectal neoplasms 10 mm and larger.

14 owel habits (7 patients, 64%), screening for colorectal neoplasm (3 pts, 27%) and lower gastrointesti

15 Advanced colorectal neoplasms (ACNs), including colorectal cancer

16 Colorectal neoplasms (adenomatous polyps) missed at OC b

17 The risk of colorectal neoplasms among siblings of patients with adv

18 mutations in stool samples for detection of colorectal neoplasms and compared this test with other a

19 ntributed to the elevated risk of breast and colorectal neoplasms and possibly ocular melanoma.

20 Our study shows that FAS is expressed in all colorectal neoplasms and there is a concomitant increase

21 vitamin D3 as chemopreventive agents against colorectal neoplasms, and CaR, VDR, CYP27B1, and CYP24A1

22 In contrast, approximately 15-20% of colorectal neoplasms arise through a distinct genetic pa

23 ease test sensitivity for detecting advanced colorectal neoplasms at 2 predefined cutoffs of a quanti

24 Regular use of aspirin reduces the risk of a colorectal neoplasm, but the mechanism by which aspirin

25 is a new-generation technique for detecting colorectal neoplasms by using volumetric CT data combine

26 The development of colorectal neoplasms can be characterized by an ordered

27 Colorectal neoplasms [colorectal cancer (CRC) and colore

28 Persons with no prior history of colorectal neoplasms completed comprehensive questionnai

29 Nonpolypoid colorectal neoplasms containing carcinoma were smaller i

30 overy phase (n = 39) and individuals without colorectal neoplasms (controls, n = 39).

31 t and indicate that risk of serrated pathway colorectal neoplasms could be reduced with lifestyle cha

32 We assessed colorectal neoplasm detection by a next-generation sDNA

33 The overall rates of colorectal neoplasm detection by quartiles of cecal inse

34 ion on the impact of cecal insertion time on colorectal neoplasm detection is limited.

35 association between cecal insertion time and colorectal neoplasm detection rate in colonoscopy screen

36 Although the vast majority of colorectal neoplasms develop as a consequence of somatic

37 Three different macroscopic subtypes of colorectal neoplasms display distinct carcinogenetic pat

38 associated with the development of advanced colorectal neoplasms during surveillance.

39 Both early and late colorectal neoplasms exhibit defective expression of sev

40 expression of the inducible COX-2 isoform in colorectal neoplasms from patients with hereditary nonpo

41 rocyclic amine (HCA) exposure in the risk of colorectal neoplasm has been suggested but not yet estab

42 The role of DPC4 inactivation in colorectal neoplasms has not been fully characterized.

43 t blood, has a low sensitivity for detecting colorectal neoplasms in asymptomatic patients at average

44 ound to be associated with a reduced risk of colorectal neoplasms in observational studies.

45 tions of circulating vitamin D with risk for colorectal neoplasms in observational studies.

46 showing the existence of flat and depressed colorectal neoplasms in Western countries.

47 a suggest that the enhanced level of CD44 in colorectal neoplasms is asynchronous with cell replicati

48 iation of type 2 diabetes mellitus (DM) with colorectal neoplasms is contradictory, and African Ameri

49 Keywords: Colorectal Neoplasms, Liver, Angiography, CT, Incidental

50 A detailed case study on breast neoplasms, colorectal neoplasms, lung neoplasms, and 32 other disea

51 The appreciation that colorectal neoplasms may present as flat or depressed le

52 ns when screening for cancer, and many early colorectal neoplasms may therefore be missed.

53 There is concern that flat or depressed colorectal neoplasms might be easily missed during a col

54 e limited on the significance of nonpolypoid colorectal neoplasms (NP-CRNs).

55 Enhanced expression of CD44 in colorectal neoplasms occurred not only in epithelial cel

56 e identified 4,249 (33.5%) participants with colorectal neoplasms, of whom 1,956 had small single ade

57 e is to review the principles for diagnosing colorectal neoplasms on double-contrast images and the s

58 Enhancement of 29 consecutive colorectal neoplasms on pre- and postcontrast CT colonog

59 not exclude the effects of secular trends in colorectal neoplasm prevalence, the observed increase wa

60 and its expression is markedly increased in colorectal neoplasms, suggesting that expression is link