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1 epidermidis has been considered a beneficial commensal organism.
2 ome-wide transcriptional response of an oral commensal organism.
3 ssociated with the host versus the competing commensal organisms.
4  and maintenance of virulence in recombining commensal organisms.
5 gly, the majority of these species were oral commensal organisms.
6 g pathogens while tolerating colonization by commensal organisms.
7  have been confounded by these contaminating commensal organisms.
8 ogy of numerous Cas9-encoding pathogenic and commensal organisms.
9 cteristics, tissue-resident immune cells and commensal organisms.
10 97% of samples that were distinct from human commensal organisms.
11                      The mechanisms by which commensal organisms affect human physiology remain poorl
12 arbon receptor, Janus kinase inhibitors, and commensal organisms also in trials for topical applicati
13 the JCI, Lei and colleagues demonstrate that commensal organisms also influence the host response to
14         As GBS has the disposition of both a commensal organism and an invasive pathogen, it is impor
15 iae naturally colonizes the nasopharynx as a commensal organism and sometimes causes infections in re
16  while suppressing inflammatory responses to commensal organisms and food Ags.
17 ial cell-free DNA in 62, likely derived from commensal organisms and incidental findings unrelated to
18  can be colonized by many bacteria including commensal organisms and potential pathogens, such as Sta
19            Discrimination between beneficial commensal organisms and potentially harmful pathogens is
20 2 (34.3%) grew a pathogen (with or without a commensal organism) and 143 (30.2%) grew commensal bacte
21 onmental organisms, opportunistic pathogens, commensal organisms, and fungi that can actively and exp
22 ant metabolite, produced by host tissues and commensal organisms, and it represents an important pote
23 gulate immune responses to food antigens and commensal organisms, and protect from pathogens.
24 lth, and it is now clear that the effects of commensal organisms are much broader than previously bel
25 doxically, ZPSs, which are found in numerous commensal organisms, are able to elicit both proinflamma
26 amily of Gram-negative bacteria that include commensal organisms as well as primary and opportunistic
27 n expressed on the surface of S. gordonii, a commensal organism, binds to soluble and immobilized fib
28 Vulvovaginal candidiasis (VVC) caused by the commensal organism Candida albicans remains a significan
29         By applying this method to the model commensal organism Clostridium sporogenes, we knocked ou
30                           The bacterium is a commensal organism colonizing the rectum and the gastroi
31 Enterococcus faecalis, a Gram-positive human commensal organism, depends on a two-component signaling
32  studies suggest that topical application of commensal organisms (eg, Staphylococcus hominis or Roseo
33 n that bacterial pathogens must compete with commensal organisms for the limited territory afforded b
34 t some unique features that distinguish skin commensal organisms from pathogenic microbes.
35            In addition, comparison with oral commensal organisms from the Ann Arbor, Mich., locale co
36 ive cases, patients infected with nonsurface commensal organisms had significantly worse final VA tha
37 ponses are differentially regulated and that commensal organisms have a role in stimulating mucosal e
38  fusion protein from Bacteroides fragilis, a commensal organism implicated in drug-resistant infectio
39                Escherichia coli O157:H7 is a commensal organism in cattle, but it is a pathogen in hu
40  why C. jejuni is more frequently found as a commensal organism in poultry and as the cause of diseas
41                 Because C. albicans is not a commensal organism in rodents and mice are thus naive to
42 rtunistic pathogen Bacteroides fragilis is a commensal organism in the large intestine, where it util
43 obiome, and the dysbalance of pathogenic and commensal organisms in the intestinal microbiome may con
44                        Normally abundant gut commensal organisms, including the Ruminococcaceae and L
45           Pathogenic E. coli, but not normal commensal organisms, increase Gal1-R mRNA synthesis and
46  pathogenic Escherichia coli, but not normal commensal organisms, increase galanin-1 receptor express
47 l pathogens as mutants of otherwise harmless commensal organisms is the challenging new feature of ou
48  wild-type strain, suggesting that competing commensal organisms might be a significant source of gen
49 S2(-/-)) formed massive abscesses containing commensal organisms, mostly enteric bacteria, even when
50 ia meningitidis is also polyploid, while the commensal organism Neisseria lactamica maintains chromos
51 scarring was associated with the presence of commensal organisms (odds ratio [OR], 1.46; 95% confiden
52 used by Candida albicans, a dimorphic fungal commensal organism of the gastrointestinal and lower fem
53  pathogen Chlamydia trachomatis evolved as a commensal organism of the human gastrointestinal (GI) tr
54                    Mannheimia haemolytica, a commensal organism of the upper respiratory tract in cat
55 i) is a gram-negative bacterium and a common commensal organism of the upper respiratory tract in hum
56                              Enterococci are commensal organisms of the gastrointestinal (GI) tracts
57              Lactobacilli are low-virulence, commensal organisms of the gastrointestinal and genitour
58                                They serve as commensal organisms of the gastrointestinal tract and ar
59        Members of the genus Enterobacter are commensal organisms of the gastrointestinal tract and ar
60 cross-reactivity with pathogenic bacteria or commensal organisms of the oropharynx and genital tract,
61 vity with other Candida spp., bacterial skin commensal organisms or commonly encountered viruses.
62 ad to inappropriate immune reactivity toward commensal organisms, perhaps contributing to mucosal inf
63                         Fungal and bacterial commensal organisms play a complex role in the health of
64                                     Although commensal organisms promote health, enteric pathogens, i
65 ection endophthalmitis, virulent, nonsurface commensal organisms remained independently associated wi
66  against pathogens while tolerating harmless commensal organisms remains an important puzzle.
67        Humans are colonized by multitudes of commensal organisms representing members of five of the
68 arkable advances in our understanding of how commensal organisms shape host immunity.
69 To that end, the srtA gene in the human oral commensal organism Streptococcus gordonii was insertiona
70 regulated via TLR2 expression and signaling, commensal organisms such as N. lactamica would benefit f
71         Infections with virulent, nonsurface commensal organisms such as Streptococcus and Enterococc
72 These results show oral delivery of modified commensal organisms, such as LL-CFA/I, may be harnessed
73  to compete with and outgrow other important commensal organisms, suggesting that HOSCN may be a rele
74 ty toward several oral streptococci or other commensal organisms tested.
75 cellular pathogen Staphylococcus aureus is a commensal organism that can cause severe disease when it
76 le Haemophilus influenzae is a gram-negative commensal organism that is commonly associated with loca
77                        Candida albicans is a commensal organism that under certain circumstances can
78 Group B streptococci (GBS) usually behave as commensal organisms that asymptomatically colonize the g
79         Medically important fungi range from commensal organisms that cause opportunistic infections
80 The genus Neisseria comprises pathogenic and commensal organisms that colonize the human nasopharynx.
81                                              Commensal organisms that constitute the skin microbiota
82 our approach, we used it to identify several commensal organisms that induce intestinal expression of
83  of Lactobacillus spp., which are beneficial commensal organisms that thrive in similar locales as En
84 er surfaces are home to a vast population of commensal organisms that together encode millions of pro
85  may facilitate the transition of GBS from a commensal organism to a virulent meningeal pathogen.
86                       The mechanisms used by commensal organisms to circumvent this immune response h
87 nfecting humans and behave as mutualistic or commensal organisms when colonizing arthropod vectors.
88  into the microbiome suggest that modulating commensal organisms within the gut or lung may also be a
89 gastrointestinal or genitourinary tract as a commensal organism, yet it has the potential to cause li