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1 new genes: Gpr65, Plzp, Toso, and Cd5l (in a companion paper).
2 ) and food as unconditional stimulus (US) [ (companion paper)].
3 brovascular risk factors is the subject of a companion paper.
4 chromatography (IC), which is discussed in a companion paper.
5 as based on studies described in a preceding companion paper.
6 article is part of a Special Issue entitled Companion Paper.
7 sional cellular microscopy, described in the companion paper.
8 ta-analysis papers are discussed in the next companion paper.
9 comparison of arms A and B is described in a companion paper.
10 mer's Disease Genetic Consortium (ADGC) in a companion paper.
11 comparison of arms A and C is described in a companion paper.
12 hes, EMG sites and motor units reported in a companion paper.
13 ctivity of KatG is addressed here and in our companion paper.
14 investigated and discussed in the following companion paper.
15 nd high-hroughput AMS measurement are in the companion paper.
16 e algorithms and parameters discussed in the companion paper.
17 for the preparation of AHA is presented in a companion paper.
18 of the DURS2 phenotype, as described in the companion paper.
19 n alternative route to AHA is presented in a companion paper.
20 of the selected sequences is described in a companion paper.
21 d in vitro, and the data are reported in the companion paper.
22 entatively identified as cytokeratin 18 in a companion paper.
23 ucture of wild-type HPPK as described in the companion paper.
24 ribution of ejecta blocks are discussed in a companion paper.
25 ies of the 'ponds' are discussed in a second companion paper.
26 and propriospinal input was examined in the companion paper.
27 g the forward and backward potential sweeps (companion paper 1), the semiempirical treatment here, wh
28 agenic activity results are presented in our companion paper.1 The results show that ELCR values for
29 tatistical analysis approach, presented in a companion paper (10.1021/acs.analchem.9b01325), to relia
30 in complex with both enzymes described in a companion paper, (34) provides a rationale for the obser
33 re variation across genes and species in our companion paper, and here we explore variation within in
35 o be a source of confusion.Dual Perspectives Companion Paper: Are the Neural Correlates of Consciousn
46 based on traditional views.Dual Perspectives Companion Paper: Diverse Spatiotemporal Scales of Cholin
49 anding of ACh in the brain.Dual Perspectives Companion Paper: Forebrain Cholinergic Signaling: Wired
52 ype method for amino acids, developed in the companion paper in this issue of the Journal, are used.
54 of aging-associated variables reported in a companion paper in this issue) and targeted replication
56 xperiments complement those presented in the companion paper in which binding and protonation of CH3-
58 lyse new data, presented in more detail in a companion paper, in which BrdU/IdU cell-labelling experi
59 ations for its utilization.Dual Perspectives Companion Paper: Integrating CRISPR Engineering and hiPS
60 matic studies reported in this article and a companion paper of theoretical calculations of the nanog
62 ng therapies in the future.Dual Perspectives Companion Paper: Parkinson's Disease Is Not Simply a Pri
65 ence that Slit is the midline Robo ligand; a companion paper presents biochemical evidence that Slit
68 detergent or chaotropic conditions, but our companion paper provides strong evidence to the contrary
69 ut the etiopathology of PD.Dual Perspectives Companion Paper: Prying into the Prion Hypothesis for Pa
71 lation of gene expression was described in a companion paper published in this journal, using flux-ba
74 f our genetic humanization approach, and the companion paper reports that the humoral immune systems
75 promising research avenues.Dual Perspectives Companion Paper: Should a Few Null Findings Falsify Pref
76 and an antibody-stabilized active structure (companion paper) shows how binding events at both the ex
77 progress in drug discovery.Dual Perspectives Companion Paper: Studying Human Neurodevelopment and Dis
78 er results, some of which are presented in a companion paper, suggest that DspA acts as a global regu
79 een GluR6Q and GluR2Q(flip), reported in the companion paper, suggests that at a glutamate concentrat
83 p and a YAC-based physical map reported in a companion paper, the fundamental maps required for mouse
86 consistent with bioaccumulation results in a companion paper (this issue) observed for amphipods.
87 consistent with bioaccumulation results in a companion paper (this issue) using a microbial bioreport
89 chimeric mice with deletion of Tssk1 and 2 (companion paper) this centriolar kinase/substrate pair i
90 and the gp32 monomer-binding results of the companion paper to propose a detailed model for how gp32
98 ms." Their contributions are summarized in a companion paper we have written to introduce the Special
118 ysical properties (with the exception of the companion paper where Tapia et al. demonstrate a direct