ライフサイエンスコーパス: compensatory change

1 hages and induce disease pathology through a compensatory change.

2 reased for the BK current, consistent with a compensatory change.

3 n-functional intermediate state prior to the compensatory change.

4 t appear to further affect the noradrenergic compensatory changes.

5 retained the D441-to-arginine change and no compensatory changes.

6 sion, or recombination, or a series of rapid compensatory changes.

7 genes, suggesting stabilizing selection for compensatory changes.

8 lenced images in combat controls may reflect compensatory changes after trauma exposure that are not

9 mporal lobe epilepsy and provide evidence of compensatory changes after trauma to the hippocampal for

10 ehavioral remediation may be associated with compensatory changes anchored in L.FFG, which reflect at

11 Such models can generate distinct compensatory changes and adaptive changes from direction

12 ing disease process unfolds, with associated compensatory changes and loss of inherent system redunda

13 o influence (1) the time course of acid-base compensatory changes and the respective cerebrovascular

14 orted in other species, such as rodents, but compensatory changes appear to prevent these deleterious

15 These compensatory changes are driven by Akt-mediated negative

16 ar interest are the roles of development and compensatory changes, as well as other factors, such as

17 However, the compensatory changes associated with vitamin A deprivati

18 ERC is often assumed to result from compensatory changes at interaction interfaces (i.e., in

19 e kinetics in H9 cells, contains second-site compensatory changes at MA amino acids 73 (E-->K) and 82

20 Whereas we found no evidence of compensatory changes at the mRNA level of wounded knocko

21 synthesis during synaptic inactivity drives compensatory changes at the presynaptic site.

22 d toward control values driven by reciprocal compensatory changes at two joints.

23 Moreover, we observed that, for some genes, compensatory changes between different rates can result

24 etGC2, and GCAP-1 expression did not undergo compensatory changes, but the absence of GCAP2 affected

25 The data of patient compensatory changes conformed to the following equation

26 The observation that compensatory changes do not involve regeneration of N-li

27 for Cnr1 during development and suggest that compensatory changes during development may mitigate the

28 n cortical EEG activity during REM sleep and compensatory changes following sleep loss.

29 stand why G88R was favored by the virus as a compensatory change for the NLS loss and resultant repli

30 s for disrupting viral functions or impeding compensatory changes for vaccine escape or drug resistan

31 We find that the fixation of compensatory changes has caused the regulation of gene e

32 quences and second-site mutations leading to compensatory changes have been shown in mosaic individua

33 ons to rodent knock-out studies suggest that compensatory changes have limited the understanding of t

34 iferation in crypts of Lieberkuhn, without a compensatory change in basal apoptosis and produces a dr

35 ediate and folded states, but in all cases a compensatory change in entropy results in a small net fr

36 This is probably due to a compensatory change in gait or loading regime, which red

37 There is little or no compensatory change in other proteins or structures rela

38 mechanisms responsible for pathological and compensatory change in Parkinson's disease.

39 ing of synaptic transmission, expressed as a compensatory change in quantal size following chronic ac

40 hat Pum-mediated change in para results in a compensatory change in Shal.

41 ow that chronic activity blockade triggers a compensatory change in the abundance of GLR-1, a Caenorh

42 nce of such a host-pathogen "arms race" is a compensatory change in the host antiviral protein as it

43 d resulted in the selection of a second-site compensatory change in the RDRP palm subdomain.

44 d resulted in the selection of a second-site compensatory change in the RDRP palm subdomain.

45 These include compensatory changes in (1) postsynaptic neurotransmitte

46 ry lymphoid tissue architecture, we examined compensatory changes in ADAM17 and TNF-alpha in ADAM10(B

47 eterious genetic alterations are buffered by compensatory changes in additional genes.

48 the visual network showed responsive and/or compensatory changes in all patients.

49 ver from Mpc1 hypomorphic embryos identified compensatory changes in amino acid and lipid metabolism.

50 ral occlusion during infancy, which leads to compensatory changes in auditory spatial tuning that ten

51 Thus, compensatory changes in B-class MADS box gene duplicate

52 PNP, and GDP, respectively), associated with compensatory changes in binding entropy and enthalpy.

53 No compensatory changes in Ca2+ regulatory protein expressi

54 hepsin activity in living cells, documenting compensatory changes in cathepsin-deficient cells, and C

55 These findings suggest that compensatory changes in cellular excitability, rather th

56 ates in the epithelial cytoplasm, initiating compensatory changes in cellular gene expression.

57 These findings show that there are dynamic, compensatory changes in cerebral activation during verba

58 ns increased as a function of intake despite compensatory changes in cholesterol and bile acid synthe

59 We evaluated the impacts of compensatory changes in control policies in relation to

60 diated by enhanced CRF receptor signaling or compensatory changes in CRF receptor density within thes

61 Here we show that compensatory changes in demographic rates are buffering

62 gnificantly decreased gene expression and no compensatory changes in DNA methylation or chromatin acc

63 partially denervated striatum without active compensatory changes in dopamine uptake or release.

64 s, leading to seizures, neuronal damage, and compensatory changes in EAAT3 and neuropeptide Y.

65 er ondansetron treatment was not achieved by compensatory changes in eating behaviour such as by a sm

66 ondrial metabolism that feed back and induce compensatory changes in electron transport.

67 Compensatory changes in energy expenditure occur in resp

68 tent alterations in network activity trigger compensatory changes in excitation and inhibition that r

69 in calcium influx, and translates this into compensatory changes in excitatory quantal amplitude.

70 Compensatory changes in expression levels of several bil

71 There was no evidence for compensatory changes in expression of either family memb

72 rine global knock-out approach may result in compensatory changes in expression of other membrane pro

73 tion reduced the amount of PP1cdelta with no compensatory changes in expression of other MYPT1 family

74 esponse to Clcn3 gene deletion, there may be compensatory changes in expression of other proteins tha

75 ether changes in dietary salt intake lead to compensatory changes in expression of the guanylin signa

76 MET activation appears to undergo long-term compensatory changes in expression that may be a hallmar

77 nsensitive to ischaemia, probably because of compensatory changes in extracellular potassium ions.

78 This mechanism of stabilization through compensatory changes in flexibility broadens our underst

79 ates that dietary restriction elicits robust compensatory changes in food consumption.

80 ary macronutrient deprivation, flies undergo compensatory changes in food preference.

81 These compensatory changes in functional brain activity were p

82 These findings suggest that compensatory changes in G alpha q expression occur in mi

83 We observed compensatory changes in G protein accumulation following

84 Additional compensatory changes in Gag permitted efficient virus re

85 tk alter B cell subpopulations and may cause compensatory changes in gene expression, we used B cells

86 cytoplasmic mRNA degradation often leads to compensatory changes in gene expression.

87 although anti-NMDAR antibodies do not induce compensatory changes in glutamate receptor gene expressi

88 Furthermore, no compensatory changes in HMG-14b or histone protein level

89 ons up or down of frataxin expression caused compensatory changes in HSC20 expression inversely, as e

90 s during the prodromal phase of SZ and evoke compensatory changes in inhibition that alter the develo

91 such that a loss of hepatic CPR would cause compensatory changes in intestinal P450 expression and c

92 For these mice, no effective compensatory changes in ion channel gene expression were

93 These compensatory changes in islet vascularization may influe

94 NMR relaxation indicates compensatory changes in loop fluctuations upon binding,

95 Both appear to produce compensatory changes in microtubule assembly that counte

96 Compensatory changes in mRNA expression of GPR120 in GPR

97 l heterogeneity of this receptor and also of compensatory changes in mu-, delta- or kappa-receptors i

98 hat the lack of RLC phosphorylation promotes compensatory changes in MyBP-C and TnI phosphorylation,

99 with selection of reversions and second-site compensatory changes in Nef.

100 In adolescent females, compensatory changes in Netrin-1 protect against the del

101 n maintenance, sufficient cognitive reserve, compensatory changes in network function, or some combin

102 Neuroadaptation theories predict that compensatory changes in neurochemical systems that are a

103 sis and biophysical properties, and possible compensatory changes in other channels that contribute t

104 te a disruptive change in one ion channel by compensatory changes in other channels.

105 uptive change in one channel to be offset by compensatory changes in other channels.

106 rsistent activity of variant channels, or to compensatory changes in other conductance(s) in response

107 rned by the nature of the death stimulus and compensatory changes in other forms of autophagy.

108 the possibility of two different patterns of compensatory changes in other ion-translocating transpor

109 e and body weight remained normal because of compensatory changes in other meal parameters.

110 clean and are apparently not accompanied by compensatory changes in other muscarinic receptors.

111 IV-1 restriction, in some cases depending on compensatory changes in other nearby charged residues.

112 ctable xyloglucan does not cause significant compensatory changes in other polysaccharides, although

113 whether there is a kidney phenotype and any compensatory changes in other renin angiotensin system e

114 have led to the hypothesis that correlated, compensatory changes in particular parameters can at lea

115 on emission tomography study, we demonstrate compensatory changes in PD in another midbrain dopamine

116 ckbone atomic rms shifts (<0.5 A) because of compensatory changes in phi and psi backbone torsion ang

117 rom 50% to 150% normal accompanied by marked compensatory changes in plasma angiotensin II and renin

118 decreased PLC-alpha mRNA that may represent compensatory changes in PLC expression.

119 scent LCN manipulation may have sex-specific compensatory changes in PNN structure and LCN output to

120 surprisingly survive into adulthood without compensatory changes in protein expression levels.

121 stresses, each of which results in specific compensatory changes in protein expression that can be a

122 Compensatory changes in protein levels of SERCA1, TRP an

123 sponds to oxygen level changes by undergoing compensatory changes in reduced electron carrier levels.

124 onal and expression studies did not indicate compensatory changes in relevant transporters.

125 Compensatory changes in remaining normal myocardium with

126 Several of these are likely to be compensatory changes in response to Purkinje cell injury

127 g LC noradrenergic neurons in PD demonstrate compensatory changes in response to the neuronal loss, a

128 phenotypically normal, and have no apparent compensatory changes in ROCK2.

129 The compensatory changes in sensorimotor control improved tr

130 nstem, and hypothalamus, possibly reflecting compensatory changes in serotonergic innervation precedi

131 alpha*q expression are maintained in part by compensatory changes in signal transduction and other pa

132 ensing changes in tubular flow and eliciting compensatory changes in single nephron GFR (SNGFR).

133 Compensatory changes in SPAK/OSR1-independent phosphoryl

134 ce show an absence of tonic currents without compensatory changes in spontaneous IPSCs (sIPSCs), sEPS

135 ions in wdr-20 transcription and homeostatic compensatory changes in surface GLR-1 levels that are de

136 nal activity (over a period of days) trigger compensatory changes in synaptic function that seem to c

137 Homeostatic plasticity is characterized by compensatory changes in synaptic strength and intrinsic

138 l activity is reduced over a period of days, compensatory changes in synaptic strength and/or cellula

139 in a network is perturbed for hours to days, compensatory changes in synaptic strength are triggered

140 n a physiologically functional range through compensatory changes in synaptic strength or intrinsic c

141 soleus burst amplitude and may have induced compensatory changes in the activity of other muscles.

142 eceptor alpha1 subunit expression can induce compensatory changes in the affected areas.

143 channels, however, may be complicated by the compensatory changes in the animals in response to the t

144 -Tsr* suppression most likely occurs through compensatory changes in the conformation or dynamics of

145 networks, indicating frequent coevolutionary/compensatory changes in the context of protein structure

146 n addition to determining the effects of the compensatory changes in the context of the original muta

147 No compensatory changes in the distribution of group-III mG

148 Evidence for compensatory changes in the evolved mechanism sets the s

149 port metabolon is likely one of the multiple compensatory changes in the exocrine parotid gland of Nh

150 pact on F-actin levels and did not result in compensatory changes in the expression of endogenous ADF

151 igh levels of CaMKII-Asp286 have reversible, compensatory changes in the expression of genes associat

152 ese alterations could partly be explained by compensatory changes in the expression of matrix-related

153 Compensatory changes in the expression of mRNA for other

154 ciency in Bcl-x(L) expression did not induce compensatory changes in the expression of other Bcl-2 pr

155 cardiac function compared with controls, or compensatory changes in the expression of other DYRK iso

156 n in the hBCRP mice and the absence of major compensatory changes in the expression of other genes in

157 tion of TRPC6 protein without any detectable compensatory changes in the expression of other TRPC cha

158 ed perturbed epiboly with low penetrance and compensatory changes in the expression of terpenoid/ster

159 mmatory cell recruitment was associated with compensatory changes in the expression profiles of CCL2,

160 ation of histopathologic findings because of compensatory changes in the eyes over time (before enucl

161 d with an internal exon can be suppressed by compensatory changes in the first two positions of a con

162 erving the deprived sense, but also produces compensatory changes in the functionality of other senso

163 cells and are not accompanied by detectable compensatory changes in the level of expression of other

164 L-Myc deficiency by other Myc oncoproteins, compensatory changes in the levels of c- and/or N-myc tr

165 confirmed gene ablation and demonstrated no compensatory changes in the levels of other FABP mRNA in

166 of morphogenic scaling that have focused on compensatory changes in the morphogen gradient itself.

167 MICU1 loss causes substantial compensatory changes in the mtCU composition and abundan

168 se data suggest that previously unrecognized compensatory changes in the nigrostriatal system occur i

169 Compensatory changes in the number of inhibitory and exc

170 ither improved nor degraded, suggesting that compensatory changes in the outputs of the spinal circui

171 ciated with these structural alterations are compensatory changes in the physiology and ultrastructur

172 ocytosis were followed in less than 0.7 s by compensatory changes in the rate of endocytosis.

173 evelopmental resources to one trait produces compensatory changes in the relative sizes of other trai

174 However, compensatory changes in the serotonergic system that are

175 in iput1 mutants is complicated by the vast compensatory changes in the sphingolipidome; however, ou

176 in young-adult mice results in wide-ranging compensatory changes in the structure and dynamics of th

177 ion of preproenkephalin mRNA expression, and compensatory changes in the synthesis and metabolism of

178 nd that target specificity can be changed by compensatory changes in the target site and the donor in

179 t not position 1, of the VSV peptide induced compensatory changes in the TCR in both the amino acid r

180 Compensatory changes in the two parameters were observed

181 ese changes may be related to lesion-induced compensatory changes in the use of the non-impaired (ips

182 Dynamic compensatory changes in this network and interconnected

183 Moreover, microarray analyses revealed no compensatory changes in transcripts encoding ion channel

184 lear how the 5' intron finds the 3' introns, compensatory changes in U1 snRNA rescue trans-splicing o

185 3-Int3 3' splice site mutants by introducing compensatory changes in U1 snRNA.

186 ogress, but rarely take account of potential compensatory changes in wall polymers that may accompany

187 dation of the mutant tubulin does not elicit compensatory changes in wild-type tubulin synthesis or a

188 In addition, compensatory changes independent of striatal dopamine ha

189 tadpole to metamorphose, it may result from compensatory changes initiated by de novo growth of axon

190 These data demonstrate unexpected and compensatory changes involving the TM in the NHP model o

191 These data suggest that short- and long-term compensatory changes maintain dopaminergic control over

192 lts of the present study indicate that early compensatory changes may be taking place within the neur

193 can diminish viral replicative fitness, but compensatory changes may explain the limited impact of n

194 tructural integrity, and suggest sites where compensatory changes may stabilize the mutant structure.

195 Compensatory changes occur in the structure of the activ

196 ntral alpha7 expression, it is possible that compensatory changes occur that confound the results obt

197 onal, meaning a change in a food may imply a compensatory change of other foods.

198 of 5-lipoxygenase metabolites was not due to compensatory changes of 5-lipoxygenase or 5-lipoxygenase

199 herapeutics for chronic pain and ask whether compensatory changes or development of pain generators w

200 restoration of KYNA levels, suggesting that compensatory changes or ontogenetic expression of anothe

201 d neuropathological changes and suggest that compensatory changes or ontogenic expression of another

202 utations in SIV Gag p11C was coincident with compensatory changes outside of the epitope.

203 eases in intake from larger portions without compensatory changes over time.

204 to paradoxical functional facilitation, with compensatory changes particularly in the right posterior

205 base-pairing potential of the pseudoknot by compensatory changes restores telomerase activity to ess

206 nformative, they can be problematic, because compensatory changes sometimes occur during development.

207 rturbed directly and the neural basis of the compensatory changes studied in detail.

208 erable capacity of lactating dairy goats for compensatory changes such as feed intake and tissue mobi

209 ral replicative fitness that was restored by compensatory changes such as L50F and T21I.

210 ess of interspecies hybrids suggest that the compensatory change that makes a CPD fit usually occurs

211 inhibition leads to previously unanticipated compensatory changes that affect cytoplasmic Ca(2+) home

212 at limits further ATP depletion and promotes compensatory changes that maintain cellular ATP levels.

213 Sdc1-/- mecs show compensatory changes that maintain the number of HS chai

214 mice, we evaluated hepatic transporters for compensatory changes that might circumvent the profound

215 g-term absence of NHE1 does not elicit major compensatory changes that might negate the cardioprotect

216 c lack of NPY during development may lead to compensatory changes that normalize regulation of food i

217 es have aimed at differentiating the initial compensatory changes that occur within the heart with ag

218 We identified compensatory changes that pointed at convergent evolutio

219 e direct effects of Parkinson's disease from compensatory changes that reconfigure the functional sig

220 an be explained by the presence of secondary compensatory changes that render these mutations phenoty

221 portant for its regulatory function and that compensatory changes that restored base pairing partiall

222 Disruption of TAR reduced processing, while compensatory changes that restored the RNA structure als

223 perturbation and triggering several distinct compensatory changes that should help to recover and mai

224 these same sites, rather than by second-site compensatory changes, the more frequently observed mecha

225 Because of these and other compensatory changes, the population growth rates of sou

226 nal recovery is due to a more complex set of compensatory changes throughout the spinal network.

227 In fact, this decline may reflect a compensatory change to help reverse the decline of angio

228 constraint on epitope sequences that require compensatory changes to go to fixation.

229 in this ratio between groups, indicative of compensatory changes to keep the cholesterol/phospholipi

230 e; their population is dynamic and undergoes compensatory changes to maintain euglycemia.

231 ed food intake by reducing meal size without compensatory changes to metabolic rate.

232 dependent upon T3 or MCT8 and there were no compensatory changes to promote T3 uptake in a T3-deplet

233 The introduction of compensatory changes to restore base pairing potential l

234 This insensitivity derives from compensatory changes to the pattern of local and long-ra

235 bly because selection drove the evolution of compensatory changes to these lever systems that amelior

236 The most common compensatory change was the acquisition of thymidines im

237 to increase firing rates independent of any compensatory changes was validated by numerical simulati

238 When U-box mutants did replicate detectably, compensatory changes were consistently observed in the v

239 When these compensatory changes were eliminated by a 20-hour fast,

240 ence to interfere with ribozyme binding, and compensatory changes were generated in the ribozyme reco

241 terious effect on gene regulation, even when compensatory changes were included.

242 No compensatory changes were seen in the expression of othe

243 ns during innervation of the same muscle, or compensatory changes when the output of one motoneuron i

244 These lesions appear to lead to compensatory changes, with N/OFQ mRNA levels approximate

245 Because compensatory changes within species may obscure differen