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1 e displacement of a ligand in an equilibrium competition binding experiment.
2 ystems lacking GABAB receptors and performed competition binding experiments.
3 increased affinity for neuropilin-1 based on competition binding experiments.
4 ity of nonphoto-CIDNP-polarizable ligands in competition binding experiments.
5 o human ACKR3, as measured in [(125)I]CXCL12 competition binding experiments.
6 ation spectroscopy assay for saturation- and competition-binding experiments.
9 u-NeoBOMB1 were also synthesized and used in competition binding experiments against [(125)I-Tyr(4)]B
10 at CXCR4, we furthermore report their use in competition binding experiments and confocal microscopy
11 s, was further characterized by quantitative competition binding experiments and DNA mutational analy
19 determining the DNA binding affinities, and competition binding experiments further characterized th
20 mobility shift assays, together with ligand competition binding experiments, have demonstrated the i
22 -1alpha to bind either F-actin or aa-tRNA in competition binding experiments is also consistent with
28 ly binding at the catalytic site, and ligand competition binding experiments revealed no competition
34 erein described is applicable to equilibrium competition binding experiments such as radioligand assa
39 -delta1 to evaluate the feasibility of using competition binding experiments to identify specific lip
43 and-receptor binding in living HEK293 cells, competition binding experiments using commercially avail
49 orinated spy molecules allows one to perform competition binding experiments with high sensitivity wh
52 in separate heterocomplexes with hsp90, and competition binding experiments with the PP5 TPR domain