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1 oral lobe epilepsy and medically intractable complex partial seizures.
2 temporal lobe structures of 21 patients with complex partial seizures.
3 nd SE images in 36 patients with intractable complex partial seizures.
4 ceptibility in a rat model of focally evoked complex partial seizures.
5 onsisted of 56 patients with drug-resistant, complex partial seizures.
6                   Epilepsies were most often complex partial seizures (47%) and more than half were n
7 sed to treat infantile spasms and refractory complex partial seizures and is in clinical trials to tr
8 econdarily generalized seizures, followed by complex partial seizures and lastly simple partial seizu
9 interictal and ictal states in patients with complex partial seizures, and the importance of the tran
10 s during and following seizures that impair (complex partial seizures) but not those that preserve (s
11 tereotyped episodes of epigastric pain), and complex partial seizures consistent with temporal lobe e
12 uding absence, generalised tonic-clonic, and complex partial seizures, converge on the same set of an
13 jections into temporal lobe structures cause complex partial seizures (CPS) and pathological changes
14                               We studied 328 complex partial seizures (CPS) in 63 consecutive patient
15    SWDs have also been proposed as models of complex partial seizures (CPSs) following traumatic brai
16  the patient developed and continues to have complex partial seizures every 2-3 months that are persi
17         Secondarily generalized seizures and complex partial seizures exhibited increased slow waves
18 ionship between seizure type (generalized or complex partial), seizure frequency, age of onset and du
19                                Patients with complex partial seizures had lower GABA levels (1.03 mmo
20     Patients who present with only simple or complex partial seizures have a poorly documented progno
21 mbic motor seizures in animals, analogous to complex partial seizures in humans, result in a consiste
22   If SWDs model mild absence seizures and/or complex partial seizures in humans, then an opportunity
23 nd electroencephalograms in 35 patients with complex partial seizures of temporal lobe origin who wer
24 h independent bilateral temporal lobe (IBTL) complex partial seizures on the intracranial electroence
25 s, reduce the frequency of further simple or complex partial seizures or a combination of both.
26  during seizures and classified as impaired (complex-partial seizures) or unimpaired (simple-partial
27                                Specifically, complex partial seizures require evaluation of the front
28 patients with pharmacologically intractable, complex-partial seizures, surgical excision of the invol
29 zed tonic-clonic seizures (all patients) and complex partial seizures (three patients).
30 he mechanisms of intractable epilepsy of the complex partial seizure type.
31                             All patients had complex partial seizures, underwent amygdalohippocampect
32                    Finally, we observed that complex-partial seizures were somewhat more common with
33  occurred in the same ictal sites as obvious complex partial seizures, were electrographically simila
34 partial seizures and occasional breakthrough complex partial seizures with postictal migraines.