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1 -100% with disinhibition and 20.6%-100% with compulsive behavior).
2 direct relative to indirect pathway promotes compulsive behavior.
3 ich has long been implicated in disorders of compulsive behavior.
4  pathway as a potential treatment target for compulsive behavior.
5 ay be attributed to KOR-induced increases in compulsive behavior.
6 n the putamen, which may contribute to their compulsive behavior.
7  seems to be associated with the severity of compulsive behavior.
8 such as drug seeking, social withdrawal, and compulsive behavior.
9 anted electrodes and analyzed its effects on compulsive behavior.
10  behavioral inflexibility and other forms of compulsive behavior.
11 on, which has implications for understanding compulsive behavior.
12 nt interactions emerged primarily limited to compulsive behavior.
13 sconnection lesion led to both impulsive and compulsive behavior.
14 herefore, offers promise in the reduction of compulsive behavior.
15 , a method commonly used to assess obsessive-compulsive behavior.
16 ocaine, and may reflect the establishment of compulsive behavior.
17 apap3 KO mice, including anxiety-related and compulsive behaviors.
18 nin reuptake-inhibitor fluoxetine alleviated compulsive behaviors.
19 o be a beneficial target in the treatment of compulsive behaviors.
20 are often comorbid with the overlap based on compulsive behaviors.
21 OCD treatment modalities focused on reducing compulsive behaviors.
22  glutamate transporter EAAC1 to the onset of compulsive behaviors.
23  be beneficial for decreasing stress-induced compulsive behaviors.
24  (most notably nicotine addiction) and other compulsive behaviors.
25 ity is an endophenotype of vulnerability for compulsive behaviors.
26 right BNST were specifically correlated with compulsive behaviors.
27  beneficial target in treating SPD and other compulsive behaviors.
28  symptom domains: social, communication, and compulsive behaviors.
29  implicated in the pathogenesis of obsessive-compulsive behaviors.
30 hese mice also display highly repetitive and compulsive behaviors.
31  and with symptoms of obsessive thoughts and compulsive behaviors.
32 ve to be useful in the control of a range of compulsive behaviors.
33 itted in families exhibiting increased rigid-compulsive behaviors.
34  may accumulate in response to many types of compulsive behaviors.
35 tal-thalamic loops induce different types of compulsive behaviors.
36 aphe of Cbln2 KO mice largely reversed their compulsive behaviors.
37 usive thoughts (obsessions) and ritualistic (compulsive) behaviors.
38 llion people worldwide suffer from obsessive-compulsive behaviors(1,2), yet our mechanistic understan
39 y disinhibition (79.3%-100% of patients) and compulsive behavior (64.3%-100% of patients), compared w
40 al impact on acute laboratory assessments of compulsive behaviors 90 minutes after TBS.
41 ined phenotypes included social interaction, compulsive behaviors, aggression, hyperactivity, anxiety
42 to the pathophysiology and treatment of both compulsive behavior and eating disorders.
43  new light on which underlying beliefs drive compulsive behavior and highlight the important role of
44 sociated with a symptom dimension comprising compulsive behavior and intrusive thought.
45 isordered eating, and a factor consisting of compulsive behavior and intrusive thoughts.
46 autistic features, mood disorders, obsessive-compulsive behaviors and hetero- and autoaggression.
47 l as a transdiagnostic impairment underlying compulsive behaviors and representing a promising therap
48        In OCD, LOFC is disrupted during both compulsive behaviors and reversal learning, but whether
49 omponent of the neuronal circuitry mediating compulsive behaviors and reward sensitivity.
50 nical research program with severe obsessive-compulsive behaviors and subthreshold symptoms of psycho
51 rallels between the emergence of repetitive, compulsive behaviors and the acquisition of automated be
52 ents with disinhibition and 34.5%-75.0% with compulsive behavior) and GRN carriers (38.2%-100% with d
53 evels of somatization, depression, obsessive-compulsive behavior, and anxiety.
54 viors, such as poor socialization, obsessive-compulsive behavior, and hyperactivity.
55 ast-acting therapeutic effect of ketamine on compulsive behavior, and show that ketamine increases ac
56 ng anxiety-like behavior, social motivation, compulsive behavior, and spatial learning/cognition from
57 e relationships between sensorimotor gating, compulsive behavior, and striatal dopamine receptor leve
58 rbid difficulties, including tics, obsessive-compulsive behaviors, and attention deficit hyperactivit
59 gest synaptic adhesion as a key component in compulsive behaviors, and show that targeted sequencing
60 e to both in vivo striatal hyperactivity and compulsive behaviors, and support a potential role for p
61 ered neuroplasticity associated with chronic compulsive behaviors, anxiety, or compensatory processes
62                                              Compulsive behaviors are a core feature of obsessive-com
63                                              Compulsive behaviors are a hallmark symptom of obsessive
64                                              Compulsive behaviors are central to addiction and obsess
65 Although patients with OCD report that these compulsive behaviors are unproductive and often senseles
66 t understanding highlights three elements of compulsive behavior as it applies to pathological overea
67 hich were also highly susceptible to develop compulsive behavior as measured in a SIP procedure.
68 emerging perspective characterizes obsessive-compulsive behaviors as maladaptive habit learning(3,4),
69 of these changes are causally related to the compulsive behavior associated with drug addiction, and
70  in significantly lower scores for obsessive-compulsive behaviors at 16 weeks.
71  functioning and quality of life, as well as compulsive behavior, but only subjects with OCD reported
72 al role of the orbitofrontal cortex (OFC) in compulsive behaviors, but human studies have largely bee
73  OCPD are both impairing disorders marked by compulsive behaviors, but they can be differentiated by
74 gether, our results show that Cbln2 controls compulsive behaviors by regulating serotonergic circuits
75 ission does not negate it, and how seemingly compulsive behaviors can co-exist with the sensitivity t
76              Prominent theories suggest that compulsive behaviors, characteristic of obsessive-compul
77 ted males exhibited reduced anxiety-like and compulsive behavior compared to controls, whereas female
78 d food test, although they exhibit increased compulsive behavior, determined by marble burying test.
79 r a baseline assessment, 69 individuals with compulsive behavior disorders were randomly assigned, in
80 ation of targeted stimulation treatments for compulsive behavior disorders.
81 al regions in the pathology and treatment of compulsive behavior disorders.SIGNIFICANCE STATEMENT The
82                                              Compulsive behaviors (e.g., addiction) can be viewed as
83 ordered quantitative variation in social and compulsive behaviors established linkage to two loci for
84    Striatal hyperactivity has been linked to compulsive behavior generation in correlative studies in
85 sociated with intolerance of uncertainty and compulsive behaviors; however, little is known about the
86 specific pattern of reward-seeking impulsive-compulsive behaviors (ICBs), as well as more subtle chan
87 ymptoms, some patients develop impulsive and compulsive behaviors (ICBs), manifested as repetitive an
88 an optogenetic approach to block repetitive, compulsive behavior in a mouse model in which deletion o
89 re over 5 days robustly attenuates obsessive-compulsive behavior in a non-clinical population for 3 m
90 he specific beliefs that trigger and sustain compulsive behavior in a simple symptom-provoking scenar
91 lation as a strategy to reduce impulsive and compulsive behavior in addiction.
92 ia, catatonia, encephalopathy, and obsessive-compulsive behavior in otherwise healthy individuals wit
93 between impulsivity and the development of a compulsive behavior in rats, which captures the hallmark
94 tes to individual vulnerability to impulsive-compulsive behavior in rats.
95 eference, and it had no measurable effect on compulsive behavior in the schedule-induced polydipsia t
96 bit circuit, and the severity of ritualistic/compulsive behaviors in adults and adolescents with AN.
97 Hyperactivity in striatum is associated with compulsive behaviors in obsessive-compulsive disorder (O
98 l may underlie deficits in stopping unwanted compulsive behaviors in the disorder.
99                                        These compulsive behaviors included episodes of perseverance o
100  mice, but not Cbln1 KO mice, display robust compulsive behaviors, including stereotypic pattern runn
101  neurocomputational account of the nature of compulsive behaviors induced by dopaminergic drugs.
102                                              Compulsive behavior is a debilitating clinical feature o
103                                              Compulsive behavior is a defining feature of disorders s
104 pulations to the generation and treatment of compulsive behavior is unknown.
105  the orbitofrontal cortex is associated with compulsive behaviors, its disruption may contribute to c
106 lness Scale; and scored at least moderate on compulsive behaviors measured with the Children's Yale-B
107 and scored moderate or more than moderate on compulsive behaviors measured with the modified Children
108 yses revealed a positive correlation between compulsive behavior (measured with the Repetitive Behavi
109 multiple tics [CMT], and five with obsessive-compulsive behaviors [OCB]).
110 ing onto dorsal raphe neurons reproduced the compulsive behaviors of Cbln2 KO mice.
111 ents were conducted of target engagement and compulsive behaviors performed in response to an idiogra
112 unction that may underlie the inflexible and compulsive behaviors present in psychiatric disorders.
113             Substance use as assessed by the Compulsive Behaviors Questionnaire.
114 e, withdrawal (physiological dependence), or compulsive behavior related to alcohol use.
115 ved in disruption of sensorimotor gating and compulsive behavior relevant to OCD.
116 ferentially drive goal-directed and habitual/compulsive behaviors, respectively, and are implicated i
117  control over stereotyped and repetitive and compulsive behaviors, respectively.
118 ptive processes to complementary facets of a compulsive behavior resulting from operant reward learni
119  new insight into the biobehavioral basis of compulsive behaviors, revealing differential noradrenerg
120 asure of distress associated with preventing compulsive behaviors significantly correlated with TSPO
121 ages of onset, in locomotor and anxiety-like compulsive behaviors, spatial learning and memory, visua
122 biology of motivation might help in reducing compulsive behaviors such as drug addiction or eating di
123 uD's to neuropsychiatric disorders involving compulsive behaviors, such as Tourette syndrome, attenti
124 l theories of reward, learning and obsessive-compulsive behavior, suggest a unifying functional role
125 re communication abnormalities and obsessive-compulsive behaviors than controls.
126 t of treatment in this study was repetitive, compulsive behavior that commonly occurs in persons with
127 uch cues also appear to trigger the harmful, compulsive behaviors that characterize addiction and oth
128 (+/-) mice manifested tic-like behaviors and compulsive behaviors that could be rescued by the tic-re
129  indeed the pathological process that drives compulsive behavior, then compulsivity should be apparen
130 ide training, affected short-term markers of compulsive behavior vulnerability.
131             The presence of perseverative or compulsive behavior was more common in the TDP-B and TDP
132 In the marble-burying task, the anxiety-like compulsive behavior was normal in IDUA(-/-) mice at almo
133  Sapap3 knock-out (Sapap3-KO) mouse model of compulsive behaviors, which also exhibits hyperactivity

 
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