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1 s it pertains to ventricular arrhythmias and congenital heart block.
2 provide new insight into the pathogenesis of congenital heart block.
3 lained despite their strong association with congenital heart block.
4 bs, particularly 52Ro, in the development of congenital heart block.
6 (19%) in mothers with a previous child with congenital heart block and in 3 of 74 pregnancies (4%) i
7 Despite the near universal association of congenital heart block and maternal Abs to SSA/Ro and SS
8 ibodies give birth to children with complete congenital heart block and photosensitive skin lesions.
9 ne 400 mg daily to prevent the recurrence of congenital heart block associated with anti-SSA/Ro (anti
10 Rs), as did cardiac tissue from a fetus with congenital heart block (CHB) and an age-matched control.
11 ance in the description and understanding of congenital heart block (CHB) came in the 1970s with the
17 signature lesion of autoantibody-associated congenital heart block (CHB) is fibrosis of the conducti
21 nd recurrence rates of autoimmune-associated congenital heart block (CHB) using information from the
22 IC) may be relevant in autoimmune-associated congenital heart block (CHB) where the obligate factor i
26 -SSB/La are necessary for the development of congenital heart block (CHB), the low frequency suggests
30 ated from 83 children (22 with rash, 35 with congenital heart block [CHB], 26 unaffected siblings) an
31 efficacious after in utero identification of congenital heart block, especially in fetuses with assoc
32 maternal autoantibodies with the genesis of congenital heart block, female BALB/c mice were immunize
33 syndrome, systemic lupus erythematosus, and congenital heart block from anti-Ro52 Ab-positive mother
35 suggest that hydroxychloroquine may prevent congenital heart block in pregnancies exposed to SSA/Ro
40 pathogenesis of neonatal lupus syndrome and congenital heart block reveals important information abo