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1 romosomes at the metaphase plate (chromosome congression).
2 cruitment is important for proper chromosome congression.
3 eir stability and interfered with chromosome congression.
4 is spatially regulated to achieve chromosome congression.
5 ble self-organizing mechanism for chromosome congression.
6 n, spindle pole organization, and chromosome congression.
7 sponsive signal transduction, and chromosome congression.
8 lk1 at the kinetochore to promote chromosome congression.
9 on at the kinetochore and rescued chromosome congression.
10 ay contribute to Xorbit's role in chromosome congression.
11 heckpoint control and for correct chromosome congression.
12 l wall breakdown is not required for nuclear congression.
13 he cell, and correlates with slow chromosome congression.
14 ort results in chromosome bi-orientation and congression.
15 d2 became undetectable soon after chromosome congression.
16 llation, but is not essential for chromosome congression.
17  reattachment/detachment cycle that prevents congression.
18 owards the metaphase plate during chromosome congression.
19 microtubule motors play an important role in congression.
20 lines, which is due to defects in chromosome congression.
21 y contrast, this module is unable to support congression.
22 r proper spindle architecture and chromosome congression.
23 olar spindle capable of efficient chromosome congression.
24 rotubule plus end-tip positioning to mediate congression.
25 nsduction but are dispensable for chromosome congression.
26 lation are required for efficient chromosome congression.
27 indle equator, a process known as chromosome congression.
28 early mitosis promoting efficient chromosome congression.
29 e of cell fusion, causing a block in nuclear congression.
30  may be important for its role in chromosome congression.
31 acological agents and for mitotic chromosome congression.
32 ochore-microtubule attachment and chromosome congression.
33 er that is required for efficient chromosome congression.
34 in the vicinity of the spindle poles without congression.
35 in spindle assembly and metaphase chromosome congression.
36 n prophase facilitates subsequent chromosome congression.
37 romoting checkpoint signaling and chromosome congression.
38 tached kinetochores to facilitate chromosome congression,(11)(,)(12)(,)(13)(,)(14)(,)(15)(,)(16) inte
39 duced centromere stretch, delayed chromosome congression, alignment defects, and severe missegregatio
40 ment is an indicator of defective chromosome congression and aberrant kinetochore-microtubule attachm
41 spindle is required for efficient chromosome congression and accurate chromosome segregation to ensur
42         Errors in the fidelity of chromosome congression and alignment can lead to improper chromosom
43 his was accompanied by defects in chromosome congression and alignment of the maternal and paternal m
44 as a protein that is required for chromosome congression and alignment.
45 forces that contribute to mitotic chromosome congression and alignment.
46 nesin-10 family) are required for chromosome congression and alignment; Kif4A and Kif4B (members of t
47 tachment, resulting in failure of chromosome congression and an increased propensity for lagging chro
48 ional centrosomes have a delay in chromosome congression and anaphase onset, which can be explained b
49 ole in turning off the SAC, in promoting the congression and bi-orientation of bivalents on meiosis I
50 educes CSV and facilitates timely chromosome congression and correct segregation.
51 tors implicating small GTPases in chromosome congression and cytokinesis.
52 yeast mating, cell fusion is followed by the congression and fusion of the two nuclei.
53            Chromosome biorientation promotes congression and generates tension that stabilizes kineto
54 g, we captured the precise timing of nuclear congression and identified two minus end-directed motors
55 of two sister kinetochores during chromosome congression and imply that vertebrate kinetochores consi
56  the bipolar spindle and promotes chromosome congression and interkinetochore tension during mitosis.
57 sis during metaphase, even though chromosome congression and metaphase alignment do not appear to be
58 zygotic division, PLK-1-dependent chromosome congression and metaphase plate alignment are necessary
59 arms extend toward spindle poles during both congression and metaphase.
60 detachment, and impairs metaphase chromosome congression and mitotic Golgi fragmentation, without aff
61 al component that monitors proper chromosome congression and mitotic timing during cell division.
62 lly condensed chromosomes, failed chromosome congression and multiple centrosomes.
63  losses in Plk1 activity impaired chromosome congression and produced severe anaphase dysfunction cha
64 ctivity of CENP-E serves to power chromosome congression and provides a flexible, motile tether linki
65 stability relies upon efficacious chromosome congression and regulation by the spindle assembly check
66                   Chromosome dynamics during congression and segregation are controlled by opposing f
67         In the absence of Nup358, chromosome congression and segregation are severely perturbed.
68 trosome maturation and results in chromosome congression and segregation defects during mitosis.
69                                   Chromosome congression and segregation have been widely known to be
70 oped system for observing meiotic chromosome congression and segregation in living maize cells, we sh
71 ion and is required for efficient chromosome congression and segregation in mammalian cells.
72 he spindle is a driving force for chromosome congression and segregation in mitosis.
73  Coordination of cytokinesis with chromosome congression and segregation is critical for proper cell
74 sights into how kinetochores robustly ensure congression and segregation of chromosomes during mitosi
75                                   Chromosome congression and segregation require the proper attachmen
76 tive Astrin:PP1-delivery disrupts chromosome congression and segregation, revealing a dynamic mechani
77 rs to coordinate cytokinesis with chromosome congression and segregation.
78 ndle microtubules and drives both chromosome congression and segregation.
79 and CENP-F, which are involved in chromosome congression and spindle assembly checkpoint signaling.
80 ulation significantly compromises chromosome congression and the deposition of HJURP-CENP-A complexes
81  they control distinct aspects of chromosome congression and the SAC.
82 ses defects in both prometaphase chromosomal congression and the spindle checkpoint; however, the mec
83 n of microtubule dynamics, proper chromosome congression, and correction of improper kinetochore-micr
84 romosome arm orientation, delayed chromosome congression, and sensitized cells to nocodazole.
85 ed roles in kinetochore assembly, chromosome congression, and spindle checkpoint signaling.
86 ed NDC80 complex are required for chromosome congression, and their disruption results in mitotic arr
87 metaphase, started to fade during chromosome congression, and then disappeared at midanaphase.
88 cell fusion is necessary to complete nuclear congression, and there exists a SNARE-mediated, Sey1p-in
89 le dynamics, spindle assembly and chromosome congression, and thus cell cycle progression during mous
90 tic spindle that still manifests the classic congression architecture.
91 hat spindle length regulation and chromosome congression are intrinsic to the spindle and largely ind
92 n-like motor protein required for chromosome congression at prometaphase.
93 chores and resulted in defects in chromosome congression at the metaphase plate.
94  of ald (mps1) meiotic mutants that complete congression at wild-type rates, but have widely varying
95 ter stable attachment, throughout chromosome congression, at metaphase, and throughout anaphase A, CE
96 ons with microtubules, and direct chromosome congression, biorientation, error correction, and anapha
97 K accumulated at leading kinetochores during congression but not during segregation.
98  assembly checkpoint (SAC) and in chromosome congression, but the role of its catalytic activity rema
99              Kinetochores mediate chromosome congression by either sliding along the lattice of spind
100                        Perturbing chromosome congression by other means also resulted in failure to d
101  cerevisiae) was shown to mediate chromosome congression by promoting catastrophe of long kinetochore
102              PEFs are proposed to facilitate congression by pushing chromosomes away from spindle pol
103 roteins, Cin8p and Kip1p, mediate chromosome congression by suppressing kMT plus-end assembly of long
104 tants, suggesting that malorientation during congression commits these chromosomes to eventually nond
105 ciations, however, this effect on chromosome congression could be phenocopied.
106 ndent Plk1 localization induced a chromosome congression defect and compromised the spindle checkpoin
107 ts, disoriented mitotic spindles, chromosome congression defects and delayed mitotic progression.
108 ora B kinase activity, and lethal chromosome congression defects in cancer cells.
109 nents, impaired KT-MT attachment, chromosome congression defects, and whole-chromosome instability.
110 ng of Class I proteins and causes chromosome congression defects, but does not perturb spindle format
111        Loss of Shot also leads to chromosome congression defects, cell cycle progression delay, and d
112 chment, chromosome misalignment, chromosomal congression defects, lagging chromosomes, and chromosome
113 the mitotic spindle, resulting in chromosome congression defects, mitotic cell accumulation, and cell
114  disorganized spindles and severe chromosome congression defects.
115 e CPC from chromosomes and caused chromosome congression defects.
116  loss of CENP-E and consequently, chromosome congression defects.
117 ore fiber microtubules to support chromosome congression, defining for the first time a repurposing o
118           We conclude that efficient nuclear congression depends on the cooperation of two minus end-
119 nucleus-centrosome attachment and pronuclear congression during fertilization.
120 e report a dual mechanism underlying nuclear congression during fission yeast karyogamy upon mating o
121 uiting a pool of PP2A involved in chromosome congression during meiosis I.
122              Net1 is required for chromosome congression during metaphase and generation of stable ki
123 polar spindles, causing abnormal chromosomes congression during metaphase and separation during telop
124 otein that has been implicated in chromosome congression during mitosis, and we found that this form
125 e polar ejection force needed for chromosome congression during mitosis.
126 tor protein that is essential for chromosome congression during mitosis.
127 trosome separation but also their chromosome congression errors and mitotic delay.
128 rometaphase and display increased chromosome congression errors.
129         Strong mitotic arrest and chromosome congression failure occurred after Pp1-87B downregulatio
130 amylation and interpolar spindle, as well as congression failure, mitotic arrest and cell death.
131 heless, chromosomes show typical patterns of congression, fast poleward motion, and oscillatory motio
132  perturbing spindle formation and chromosome congression following maturation.
133 tivity as an essential feature of chromosome congression from poles and localized PP1 delivery by CEN
134  KIF4A and thus actively promotes chromosome congression from the spindle poles to the metaphase plat
135 equired to establish and maintain chromosome congression have distinguishable requirements.
136  These data support a model in which nuclear congression in budding yeast occurs by plus end MT captu
137                                   Chromosome congression in cells also requires positive charge on th
138 d to facilitate Bub3 activity and chromosome congression in higher eukaryotes.
139  cell lines and were required for chromosome congression in mitotic HeLa cells, the gradients were st
140  known as pronuclear migration or as nuclear congression in Saccharomyces cerevisiae.
141 a fundamental physical limit for maintaining congression in small spindles in the face of assembly no
142 ffects spindle pole formation and chromosome congression in vivo.
143 light chain, may be important for chromosome congression, in addition to having a role in the transpo
144 t defects are due to a failure of chromosome congression independent of kinetochore-microtubule attac
145 hase A but is not responsible for chromosome congression, interkinetochore tension, or the establishm
146  kinetochore, and in its absence, chromosome congression is defective.
147               Thus, CENP-E-driven chromosome congression is guided by microtubule detyrosination.
148                                      Nuclear congression is inhibited by drugs that perturb the actin
149 1F activity contributes to normal chromosome congression, kinetochore spacing, and anaphase A rates.
150  passenger complex (CPC) controls chromosome congression, kinetochore-microtubule attachments, and sp
151                                         This congression mechanism depended on the kinetochore-associ
152                         We find that nuclear congression occurs through the interaction of MT plus en
153 cteristic feature of mitotic spindles is the congression of chromosomes near the spindle equator, a p
154 bres were destabilized, leading to defective congression of chromosomes to the metaphase plate and pe
155 spindle microtubules by kinetochores and the congression of chromosomes to the metaphase plate.
156 tosis in yeast, dramatically interferes with congression of chromosomes to the metaphase plate.
157 ombination with polar ejection forces, drive congression of chromosomes to the metaphase plate.
158 material during cell division depends on the congression of chromosomes to the spindle equator before
159                                          The congression of chromosomes to the spindle equator involv
160 bilizes fibres of the mitotic spindle to aid congression of chromosomes.
161 d mitotic division, AIR-2 may coordinate the congression of metaphase chromosomes with the subsequent
162                                We found that congression of pole-proximal chromosomes depended on spe
163 -end-directed kinetochore motor required for congression of pole-proximal chromosomes.
164 e have studied this process by following the congression of pole-proximal kinetochores and their subs
165 evere defects in chromosome condensation and congression on the metaphase plate result in delayed ana
166 t from mitosis without detectable chromosome congression or anaphase movements.
167 pole organization and separation, chromosome congression or segregation, and anaphase spindle elongat
168 tly revised by the discovery that chromosome congression precedes metaphase I arrest.
169                       Klp2-dependent nuclear congression proceeds at constant speed, whereas dynein a
170 which delay chromosome individualization and congression, putting the oocyte at risk of aneuploidy.
171 understanding of how prometaphase chromosome congression relates to anaphase chromosome segregation.
172                 It is generally assumed that congression requires that chromosomes are connected to t
173                                   Chromosome congression requires the stable attachment of microtubul
174                           The discovery that congression returns nonexchange chromosomes to the metap
175 hore is critical for facilitating chromosome congression, segregation, and checkpoint signaling.
176 oncomitant increase in defects in chromosome congression, separation, and segregation.
177 These cells exhibited defects in chromosomal congression, sister chromatid cohesion and spindle posit
178 isopeptidases, causes a defect in chromosome congression that depends on its precise kinetochore targ
179 wn of DUSP7 also led to defective chromosome congression that resulted in a prolonged mitosis.
180                                       During congression, the kinetochore moving away from its spindl
181  homologous chromosomes, promotes chromosome congression through the action of the chromokinesin KLP-
182 e centrosome/nucleus complex, for chromosome congression to a well ordered metaphase plate, and for t
183 support a model in which CENP-E functions in congression to tether kinetochores to dynamic microtubul
184 upports a model in which CENP-E functions in congression to tether kinetochores to the disassembling
185 and the inability of chromosomes to complete congression to the equatorial plane for proper mitotic s
186 ast kinesin-8 contributes both to chromosome congression to the metaphase plate and to the coupling o
187 alian cells at prophase inhibited chromosome congression to the metaphase plate with many chromosomes
188 e capture at prometaphase, timely chromosome congression to the metaphase plate, and proper interkine
189 is essential for proper centromere motility, congression to the metaphase plate, and subsequent anaph
190 oblasts (MEFs) display defects in chromosome congression to the metaphase plate, severe chromosome mi
191 ssential for neither microtubule capture nor congression to the metaphase plate.
192             During cell division, chromosome congression to the spindle center, their orientation alo
193 dynactin inhibition did not block chromosome congression to the spindle equator in prometaphase, or s
194  a strong bilateral defect, in which nuclear congression was completed but fusion did not occur.
195  are followed by reattachment and successful congression, whereas in Ska-depleted cells, detached kin
196 s in Nnf1R-depleted cells prevent chromosome congression, whereas those in Mcm21R-depleted cells inte

 
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