戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1                                          The conidial anastomosis tube (CAT) functions in forming net
2 , DOC-1 oscillated with MAK-2 to the tips of conidial anastomosis tubes, while DOC-2 was statically l
3 to specialized cell fusion structures termed conidial anastomosis tubes.
4 1) developmental gene involved in the fungal conidial and appressorium formation, to restrict fungal
5 ene previously shown to control light driven conidial and ascospore development.
6        Eosinophil recruitment in response to conidial aspiration was correlated with the level of chi
7 on of circadianly regulated spore formation (conidial banding), has remained an integral tool in the
8 ised, with microscopic examination revealing conidial-base germ tube degeneration and cell lysis occu
9 rgeted mutation of the MgATG8 gene, arrested conidial cell death but rendered the fungus nonpathogeni
10 layer or dihydroxynaphthalene-melanin in the conidial cell wall amplified the epithelial transmigrati
11 res, reduced conidia production and abnormal conidial cell wall architecture.
12       During this morphotype transition, the conidial cell wall undergoes dynamic remodeling, which p
13  triglyceride lipids in the mobile matrix of conidial cell walls.
14 nalysis of whole-lung samples at day 2 after conidial challenge in neutrophil-depleted CCR4-/- and CC
15                          Also at day 2 after conidial challenge, significantly greater numbers of CD1
16                      Using weekly intranasal conidial challenges, mice developed robust pulmonary art
17 nificant decrease occurring in eosinophils), conidial clearance was enhanced, and the early transient
18 al pigmentation was confirmed by the altered conidial color phenotypes that resulted from disruption
19                             We now show that conidial colour mutants exhibit significant increases in
20  preferentially to hyphal (rather than yeast/conidial) components of Candida albicans, Microsporum au
21 ressorium formation occurred because of high conidial density or addition of exogenous self-inhibitor
22                  Decreased expression of the conidial development regulators brlA and Medusa, genes t
23  that define key physiological events during conidial development.
24 erated reproductive cells (metulae), delayed conidial differentiation and frequent reinitiation of se
25 lators of the core genetic pathway directing conidial differentiation.
26     We describe a new form of interleukocyte conidial exchange called "shuttling." In Talaromyces mar
27  arterial remodeling in response to repeated conidial exposure.
28    Next, we used the oatmeal cereal agar for conidial formation along with the optimal conditions for
29 sues: (i) selection of an optimal medium for conidial formation by dermatophytes and (ii) validation
30 enicillin production, hyphal morphology, and conidial formation.
31                  Furthermore, our dataset on conidial germination allowed comparisons to transcriptio
32 fensins and their cognate peptides inhibited conidial germination and accompanying cell fusion with d
33 dependent protein degradation is involved in conidial germination and appressorial differentiation.
34 gonist, compound 48/80, completely inhibited conidial germination and appressorium formation at a con
35 ne disruption of MaGGPPS5 results in earlier conidial germination and faster greater vegetative growt
36 he respiratory tree is sufficient to prevent conidial germination and invasive disease.
37   RS foliar treatments significantly reduced conidial germination and PM severity on both an intermed
38    In contrast CD14(+)CD16(+) do not inhibit conidial germination and secrete large amounts of TNF.
39  extremely restricted hyphal growth, delayed conidial germination and uncontrolled activation of sexu
40  and physiological processes associated with conidial germination and will facilitate functional pred
41 n of the drug required for the inhibition of conidial germination but not necessarily that required f
42 rom C57BL/6 and gp91(phox)(-/-) mice inhibit conidial germination equally in vitro.
43 owever, such cell wall reorganization during conidial germination has not been studied so far.
44                        This study shows that conidial germination in A. nidulans requires protein syn
45              Moreover, DeltargsA resulted in conidial germination in the absence of a carbon source.
46 s, we performed transcriptional profiling of conidial germination in the filamentous fungus, Neurospo
47                                              Conidial germination into tissue-invasive hyphae can occ
48 us versus A. fumigatus; however, the rate of conidial germination of A. terreus was slower.
49  the velB deletion mutant exhibits increased conidial germination rates in the presence of glucose, a
50 nd UV stresses, and accelerated and elevated conidial germination regardless of the presence or absen
51 ouble mutant, suggesting that RIC8 regulates conidial germination through both GNA-1 and GNA-3.
52 lization of the three Galpha proteins during conidial germination was probed through analysis of cell
53 a, but only CD14(+)CD16(-) monocytes inhibit conidial germination yet secrete little TNF.
54 C2 single-deletion mutants display wild-type conidial germination, a double-deletion mutant is delaye
55 t, as were conidiation, conidial morphology, conidial germination, and in vitro appressorium formatio
56 ei largely clustered in the spore body after conidial germination, and the septum was often assembled
57 d vegetative growth, hyphal tip development, conidial germination, appressorium formation, and, ultim
58 ssary for proper septation, conidiation, and conidial germination, but only myoB is required for coni
59 at RgsA downregulates pigment production and conidial germination, but stimulates asexual sporulation
60 lli-CgCOM1i lines revealed highly suppressed conidial germination, germ tube development, appressoria
61 d normal growth or development of T. virens, conidial germination, production of gliotoxin, hyphal co
62 ngal biomass by 94% and completely inhibited conidial germination, retaining activity even after exte
63 f a wide array of processes occurring during conidial germination, translation is essential if germin
64 levels and changes in gene expression during conidial germination, we analyzed a circuit design of co
65          To analyze the molecular details of conidial germination, we developed a genetic screen in w
66 oss of GNA-3 leads to a drastic reduction in conidial germination, which is exacerbated in the absenc
67 wth and nuclear division in a process called conidial germination.
68 chanism, inducing translation and leading to conidial germination.
69 ting an important role for rasA signaling in conidial germination.
70 ubtle defects in aerial hyphae formation and conidial germination.
71  reduced vegetative growth, conidiation, and conidial germination.
72 n aerial hyphal growth, hyphal branching and conidial germination.
73 c8 mutant leads to a significant increase in conidial germination.
74 e and vacuoles, and also to septa throughout conidial germination.
75 y, focal trehalose biogenesis and control of conidial germination.
76 n conidiation, brlA and vosA expression, and conidial germination.
77 mechanisms in AM contribute to inhibition of conidial germination.
78  expression profiles over the time course of conidial germination.
79 e utilization correlated with suppression of conidial germination.
80  tube (CAT) functions in forming networks of conidial germlings during colony initiation.
81   To select which agar medium best supported conidial growth, representative isolates of dermatophyte
82 esults, some showing that PMN do not inhibit conidial growth, with others showing that they do, most
83 able in their abilities to support T. rubrum conidial growth.
84                                 In contrast, conidial hydrophobin masks relevant structures, because
85 hagocytosis and was markedly enhanced by the conidial hydrophobin RodA.
86 dia, and ECs with CFTR mutation undergo more conidial-induced apoptosis.
87                                              Conidial inocula of an optical density at 530 nm (OD(530
88 esulted in a requirement for relatively high conidial inocula to achieve 100% mortality in both BALB/
89              Genetic analysis of reaction to conidial inoculations in a segregating wheat population
90      Preliminary testing with a 0.11-OD(530) conidial inoculum of the slower-growing Aspergillus terr
91 -CSFRbeta(-/-) leukocytes exhibited impaired conidial killing compared with GM-CSFRbeta(+/+) counterp
92 ulated neutrophil NADPH oxidase activity and conidial killing.
93 ound inactive form, inhibits osmotin-induced conidial lysis.
94 for developing new and easier procedures for conidial mass production as well as improve the pathogen
95 is a new key regulator of conidiogenesis and conidial maturation and survival, and plays a crucial ro
96 osA and velB, the key regulators involved in conidial maturation.
97                                              Conidial melanin and hydrophobin as well as hyphal galac
98  here a flow cytometric assay that relies on conidial metabolism of the viability dye FUN-1.
99 he regulation of RAS activation required for conidial morphogenesis and septin-mediated plant infecti
100 in vegetative hyphae is sufficient to induce conidial morphogenesis, complete reconstitution of devel
101         It shows a significant change in the conidial morphology and displays dramatic polar growth d
102 uption of alb1 causes pleiotropic effects on conidial morphology and fungal virulence.
103  for vegetative growth, asexual development, conidial morphology and plant infection.
104 lg2p) had altered appressorium formation and conidial morphology and produced fewer, smaller lesions
105 etically related to Geomyces spp. but with a conidial morphology distinct from characterized members
106 reas the Clg2p-ClUrase interaction regulated conidial morphology without affecting fungal pathogenici
107 ation of Clg2p in appressorium formation and conidial morphology, and its role in pathogenicity, seve
108  Tox2(+) ccsnf1 mutant, as were conidiation, conidial morphology, conidial germination, and in vitro
109                           Except for altered conidial morphology, the phenotypes of hdc1 mutants were
110 an also influence U. botrytis colony growth, conidial number and size, and have a strong effect on th
111  correlated with anamorphic features such as conidial ontogeny and morphology than with teleomorph fe
112 and disruption of alb1 resulted in an albino conidial phenotype.
113 dition to alb1, arp1, and arp2 suggests that conidial pigment biosynthesis in A. fumigatus is more co
114 anning 19 kb, were identified as involved in conidial pigment biosynthesis in A. fumigatus.
115 ins a six-gene cluster which is required for conidial pigment biosynthesis in A. fumigatus.
116 ptaketide synthase (Alb1p) to synthesize its conidial pigment through a pentaketide pathway similar t
117 d was sufficient to restore the bluish-green conidial pigment.
118 howed that it is essential for multicellular conidial pigmentation and development in a plant endophy
119                     By analysing a series of conidial pigmentation mutants, a new fungal pigmentation
120                                              Conidial pigmentation of A. fumigatus significantly infl
121              Involvement of the six genes in conidial pigmentation was confirmed by the altered conid
122              The visual fading of A. terreus conidial pigmentation with the subculturing, revealing t
123 entified a gene, alb1, which is required for conidial pigmentation.
124 ward and side scatter characteristics of the conidial population and by mean fluorescence intensity (
125         The Delta odeA strain was reduced in conidial production and mycelial growth; these effects w
126 ) accelerates the shift leading to increased conidial production and reduced sclerotial production un
127 tages and is required for vegetative growth, conidial production and sexual development.
128 re, the DeltaFgvps27 mutant was defective in conidial production and was unable to generate peritheci
129  mycelial growth and, unexpectedly, enhanced conidial production.
130 se two pathogens, shuttling augments initial conidial redistribution away from fungicidal neutrophils
131  elaboration of its infection structures and conidial rodlet protein production.
132 ential roles for controlling asexual traits (conidial size and number).
133 ne thousand times more single-celled asexual conidial spores, three times sooner than the ancestral g
134                In Aspergillus fumigatus, the conidial surface contains dihydroxynaphthalene (DHN)-mel
135      These results suggest that A. fumigatus conidial surface proteins are important for evasion and
136                         Here we analysed the conidial surface proteome of A. fumigatus, two closely r
137  melanin biosynthesis resulted in an altered conidial surface with masked surface rodlet layer, leaky
138 ins exclusively detected on the A. fumigatus conidial surface, we assessed null mutants for 42 genes
139  that the alb1 disruptant exhibited a smooth conidial surface, whereas complementation of the alb1 de
140  melanin and RodA hydrophobin on the dormant conidial surface.
141 itating proper formation of the A. fumigatus conidial surface.
142 d in a significant increase in C3 binding on conidial surfaces, and the conidia of the alb1 disruptan
143 t is facilitated by complement deposition on conidial surfaces.
144 n of a novel regulator of conidiogenesis and conidial survival called MybA because the predicted prot
145 that AfBIR1 expression and activity underlie conidial susceptibility to NADPH (reduced form of nicoti
146                                       During conidial swelling and mycelial growth, the expression of
147 ameters determined were: rates and extent of conidial swelling, production of differentiated germinat
148 trehalose amount, long-term spore viability, conidial tolerance to oxidative and UV stresses, and acc
149 mber of the bZIP/CREB family, helps regulate conidial tolerance to oxidative stress.
150  of AfuvosA or AfuvelB causes a reduction in conidial trehalose amount, long-term spore viability, co
151  which is likely associated with the lack of conidial trehalose and increased beta-(1,3)-glucan level
152             L-ficolin opsonization increased conidial uptake and enhanced killing of A. fumigatus by
153 dly and reproducibly determined by measuring conidial viability.
154 ungal burden, most likely due to the reduced conidial viability.
155 l germination, but only myoB is required for conidial viability.
156 ression affected conidiophore morphology and conidial yields quantitatively but did not alter the tem

 
Page Top