ライフサイエンスコーパス: connective tissue

1 inflammatory infiltrate on the subepithelial connective tissue.

2 ined by aberrant accumulation of collagenous connective tissue.

3 transport to the development of skeletal and connective tissue.

4 lls were detected both in the epithelium and connective tissue.

5 and is the most common inherited disease of connective tissue.

6 ral proteins that are prevalent in mammalian connective tissue.

7 y determines the properties and functions of connective tissue.

8 trix and provide biomechanical properties to connective tissues.

9 hole, governs ectopic mineralization in soft connective tissues.

10 sparsely in the stroma of subcutaneous loose connective tissues.

11 anding control of collagen mineralization in connective tissues.

12 ate, highlighting the effect of succinate on connective tissues.

13 tion of multiscale force transmission within connective tissues.

14 ition of calcium phosphate complexes in soft connective tissues.

15 ms a distinct microfibrillar network in most connective tissues.

16 tebral disc and potentially other pathogenic connective tissues.

17 he bulk of the extracellular matrix (ECM) in connective tissues.

18 cle cell-rich fibrous tissue (12%) and loose connective tissue (9%).

19 elevates plasma Hcy-thiolactone and leads to connective tissue abnormalities that affect the cardiova

20 ould be evaluated routinely in patients with connective tissue abnormalities, which will enable a bet

21 homa in addition to a number of skeletal and connective tissue abnormalities.

22 sis, a disease in which excessive amounts of connective tissue accumulate in response to physical dam

23 l epithelium length; epithelium on the root; connective tissue adaptation; and soft tissue thickness

24 ssues, including muscles, blood vessels, and connective tissues, adapts to mechanical strains in the

25 lance and wound-healing response of gingival connective tissues affected by diabetes mellitus or agin

26 clinically cause significant destruction of connective tissue and bone.

27 derived from the liver extracellular matrix, connective tissue and epithelium, and proteins related t

28 smoplastic reaction through the synthesis of connective tissue and extracellular matrix, inducing loc

29 ed by inflammatory infiltrates, necrosis and connective tissue and fat replacement of the affected mu

30 Type I collagen is a key protein of most connective tissue and its up-regulation is required for

31 1.2-1.5]), liver disease (RR 2.2 [1.6-2.9]), connective tissue and rheumatic diseases (RR 1.5 [1.3-1.

32 ZB was predominantly localized to endomysial connective tissue and to a lesser extent muscle membrane

33 files of mast cells isolated from peripheral connective tissues and basophils isolated from spleen an

34 w that 3D collagen gels, major components of connective tissues and extracellular matrix (ECM), are s

35 w that 3D collagen gels, major components of connective tissues and extracellular matrix, are signifi

36 re is to link a fundamental understanding of connective tissues and their multiscale properties to be

37 At 6 days p.i, virus was found in connective tissues and vasculature of the gastrointestin

38 nimmunological features, including skeletal, connective tissue, and vascular abnormalities, poor post

39 ese mechanisms may be of importance in other connective tissues, and the key to unraveling cell-matri

40 Since mechanosensory cells in connective tissue are surrounded by a disordered network

41 The structure and mechanics of many connective tissues are dictated by a collagen-rich extra

42 mine the biomechanics of collagen fibrils in connective tissues are still poorly understood.

43 tisic acid (HGA) is elevated and deposits in connective tissues as ochronotic pigment.

44 When taking 1.0 mm connective tissue attachment into account, 60% 3D BA-RSA

45 er bone porosity, trabecular separation, and connective tissue attachment loss (CTAL) as well as redu

46 D100 presented greater bone volume and lower connective tissue attachment loss (CTAL) than group EP (

47 ise effect of collagen glycation on gingival connective tissue biology is not fully understood.

48 e a cartilaginous endoskeleton surrounded by connective tissue, but lack skeletal muscle.

49 s in the collagen and glycoprotein phases of connective tissues by exploiting the spatially distinct

50 fan syndrome (MFS) is a systemic disorder of connective tissue caused by pathogenic variants in the f

51 an syndrome (MFS) is a heritable disorder of connective tissue, caused by mutations in the fibrillin-

52 Mechanical injury to connective tissue causes changes in collagen structure a

53 deposition, inflammatory cell retention, and connective tissue cell differentiation, respectively.

54 SOX2 induced proliferation of epithelial and connective tissue cells and promoted angiogenesis.

55 tumors (aggressive fibromatosis) arise from connective tissue cells or fibroblasts.

56 to create 3D open scaffolds for adhesion of connective tissue cells through well-defined adhesion pl

57 ces appeared as fluid filled sacculi between connective tissue compartments accountable for distendin

58 sis is defined as an excessive deposition of connective tissue components and can affect virtually ev

59 etal muscle and instead consisted of fibrous connective tissue composed of type I and type III collag

60 The palatal donor site of the epithelialized connective tissue (CT) graft significantly influences th

61 rcentages of vital bone, residual graft, and connective tissue (CT)/other were determined via histomo

62 s, modulation of infrastructural components (connective tissue, CT; glycocalyx) of donor organs, and

63 a heterogenous group of diseases that cause connective tissue defects.

64 Mature connective tissues demonstrate highly specialised proper

65 es cellular proliferation, angiogenesis, and connective tissue deposition, suggesting vessel healing

66 HC revealed disrupted junctional epithelium, connective tissue destruction, bacterial invasion, incre

67 related to cell cycle progression as well as connective tissue development and function.

68 f a 3D human lymphatic network within native connective tissue devoid of any exogenous material such

69 Loeys-Dietz syndrome (LDS) is a systemic connective tissue disease (CTD) associated with a predis

70 All had connective tissue disease (CTD) serologies, spirometry,

71 biopsies from 30 patients with idiopathic or connective tissue disease (CTD)-related ILD and 13 contr

72 using multivariable Cox regression analysis: connective tissue disease (hazard ratio [HR] 2.94, 95% c

73 Mixed connective tissue disease (MCTD) is a rare and complex a

74 ral bridges may be pathologically altered in connective tissue disease as evidenced by the ultrastruc

75 Bruck Syndrome is a connective tissue disease associated with inactivating m

76 Systemic sclerosis is an autoimmune connective tissue disease in which T cells play a promin

77 r Ehlers-Danlos syndrome is a rare inherited connective tissue disease secondary to mutations within

78 s-Danlos syndrome (vEDS) is a rare inherited connective tissue disease secondary to mutations within

79 was 49+/-14 years, 78% were female, 33% had connective tissue disease, 52% were in New York Heart As

80 od for IPF (kappaw=0.71 [IQR 0.64-0.77]) and connective tissue disease-related interstitial lung dise

81 olesterol biosynthesis, liver cirrhosis, and connective tissue disease.

82 limited specificity for a single autoimmune connective tissue disease.

83 Inflammatory connective tissue diseases (CTD) like lupus and rheumato

84 Autoimmune connective tissue diseases are clinically variable, maki

85 Autoimmune connective tissue diseases arise in a stepwise fashion f

86 These mutants may be useful in treating connective tissue diseases associated with increased met

87 anti-SSA/Ro-positive sera from patients with connective tissue diseases showed high reactivity to the

88 s (55 healthy, 54 with spondyloarthritis and connective tissue diseases) were studied.

89 highest cause-specific mortality of all the connective tissue diseases, and the aetiology of this co

90 ease that presents mixed features with other connective tissue diseases, such as systemic lupus eryth

91 flammation in transplant organ rejection and connective tissue diseases.

92 thic PAH and are rare in PAH associated with connective tissue diseases.

93 digestive diseases; (6) musculoskeletal and connective tissue diseases; and (7) genitourinary diseas

94 temic sclerosis is a systemic autoimmune and connective tissue disorder associated with the human leu

95 enesis imperfecta (Col1a2(oim)), a heritable connective tissue disorder caused by abnormalities in th

96 los syndrome (vEDS) is an autosomal-dominant connective tissue disorder caused by heterozygous mutati

97 Marfan syndrome (MFS) is a heritable connective tissue disorder caused by mutations in FBN1,

98 Marfan syndrome (MFS) is a connective tissue disorder caused by mutations in fibril

99 syndrome (MFS) is a highly variable genetic connective tissue disorder caused by mutations in the ca

100 Marfan syndrome (MFS) is a connective tissue disorder caused by mutations in the FB

101 Bruck syndrome (BS) is a connective tissue disorder characterized at the molecula

102 pathogenesis of systemic sclerosis (SSc), a connective tissue disorder characterized by autoimmunity

103 e alter mitochondrial function and cause the connective tissue disorder cutis laxa.

104 ibromuscular dysplasia was present in 62.7%, connective tissue disorder in 4.9%, and systemic inflamm

105 ers-Danlos syndrome (vEDS) is a rare genetic connective tissue disorder secondary to pathogenic varia

106 Marfan syndrome (MFS) is a connective tissue disorder that results in aortic root w

107 MTSL2 mutations cause an autosomal recessive connective tissue disorder, geleophysic dysplasia 1 (GPH

108 r cases across the 2 cohorts had a diagnosed connective tissue disorder.

109 s, with the exception of musculoskeletal and connective tissue disorders (992 [8.69% per annum] vs 83

110 myocarditis, and myocarditis associated with connective tissue disorders and may be beneficial in chr

111 s crucial for elucidating pathomechanisms of connective tissue disorders characterized by ECM deficie

112 with respiratory, musculoskeletal system and connective tissue disorders is more prevalent in Whites.

113 molecularly heterogeneous group of inherited connective tissue disorders that share similar skeletal

114 ed with MCVD (cardiomyopathies, arrhythmias, connective tissue disorders, and familial hypercholester

115 gthen the overlap between SCAD and renal and connective tissue disorders, and we highlight several ne

116 ants in several genes, such as those causing connective tissue disorders, have been implicated; howev

117 B-cell lineage hematologic malignancies and connective tissue disorders.

118 eatment of several major systemic autoimmune connective tissue disorders: systemic lupus erythematosu

119 nt a series of genetic evaluations for known connective-tissue disorders, but the evaluations were un

120 Connective tissue displays a large compositional and str

121 ew variant of Ehlers-Danlos syndrome causing connective tissue disruptions in multiple organs.

122 ease of serum amyloid P associated with lung connective tissue during acute respiratory distress synd

123 factors, aging, and trauma all contribute to connective tissue dysfunction and motivate the need for

124 In mammals, the tendon connective tissue experiences and resists physical force

125 s in the collagen and glycoprotein phases of connective tissue extracellular matrix (ECM).

126 ation in wdSCCs; yet tenascin C retention in connective tissue extracellular matrix suggests the rigi

127 ts from the maxillary tuberosity are rich in connective tissue fibers, with minimal presence of fatty

128 Here we isolate and culture connective tissue fibroblasts from highly regenerative m

129 ize and enhanced tissue repair (strengthened connective tissue formation, improved microvascular form

130 he complexities of cell migration in fibrous connective tissues from three separate but interdependen

131 Connective tissue graft (CTG) and enamel matrix derivati

132 The present study shows 2-year results of a connective tissue graft (CTG) associated with LLLT in th

133 of this trial was to assess the outcome of a connective tissue graft (CTG) in the esthetic zone of si

134 redictability of the free gingival graft and connective tissue graft (CTG) techniques are highlighted

135 uccal gingival recession (bREC), effect of a connective tissue graft (CTG) when combined with a bucca

136 lly advanced tunnel flap (TUN) combined with connective tissue graft (CTG) when compared to the trape

137 d patient-centered outcomes of subepithelial connective tissue graft (CTG) with and without enamel ma

138 mes of MAGRs following CAF with or without a connective tissue graft (CTG).

139 r dermal matrix [ADM], collagen matrix [CM], connective tissue graft [CTG]) are able to significantly

140 ques in combination with soft tissue grafts (connective tissue graft [CTG], collagen matrix [CM], and

141 of clinical long-term results 20 years after connective tissue grafting (CTG) or guided tissue regene

142 Connective tissue grafting has a beneficial effect on th

143 sutures in the palatal donor sites following connective tissue grafting via the SIT.

144 Combination therapy of papilla preservation, connective tissue grafting, and coronally advanced flaps

145 tions at palatal donor area of subepithelial connective tissue grafts (CTG) between cyanoacrylates ti

146 this study was to compare thick versus thin connective tissue grafts (CTG) for the treatment of ging

147 Connective tissue grafts (CTGs) and coronally advanced f

148 ted a greater GR reduction for subepithelial connective tissue grafts (SCTG) + coronally advanced fla

149 ompared two different types of subepithelial connective tissue grafts (SCTG) considering clinical par

150 ound healing of donor sites of subepithelial connective tissue grafts (SCTG), harvested by the single

151 tes have been proposed to replace autogenous connective tissue grafts, therefore the aims of this stu

152 Palatal biopsies, in the form of connective tissue grafts, were obtained from periodontal

153 Because secretion of the profibrotic connective tissue growth factor (CCN2) increased signifi

154 Pathways of liver fibrosis are controlled by connective tissue growth factor (CCN2).

155 Connective tissue growth factor (CTGF or CCN2) is a matr

156 Recent studies revealed that connective tissue growth factor (CTGF) aggravates the fi

157 cytosol, increase expression of YAP and TAZ connective tissue growth factor (CTGF) and Cyr61 target

158 he protein levels of 2 profibrosis proteins, connective tissue growth factor (CTGF) and TGF-beta1, in

159 ed the signaling of Smad3 and p53 to produce connective tissue growth factor (CTGF) and then resulted

160 Connective tissue growth factor (CTGF) and transforming

161 Delivery of connective tissue growth factor (CTGF) into full-transec

162 Connective tissue growth factor (CTGF) is a secreted gly

163 Connective tissue growth factor (CTGF) is known to regul

164 Connective tissue growth factor (CTGF) up-regulation ind

165 We identified connective tissue growth factor (CTGF), a matricellular

166 Connective tissue growth factor (CTGF), a matrix-associa

167 Increasing evidence indicates that connective tissue growth factor (CTGF), a multifunctiona

168 expression of YAP-regulated genes, including connective tissue growth factor (CTGF), and cysteine-ric

169 in-1, collagen I, alpha-smooth muscle actin, connective tissue growth factor (CTGF), and plasminogen

170 TGF-beta), TGF-beta receptor 1 (TGF-betaR1), connective tissue growth factor (CTGF), E-cadherin, SRY-

171 jury markers such as interleukin-18 (IL-18), connective tissue growth factor (CTGF), neutrophil gelat

172 for expression of the subplate neuron marker connective tissue growth factor (CTGF).

173 targeting cell cycle regulator cyclin A and connective tissue growth factor (CTGF).

174 aturation through the regulated secretion of connective tissue growth factor (CTGF).

175 fter injury requires fine-tune regulation of connective tissue growth factor (Ctgf).

176 and ameliorated by blocking YAP1/TAZ target connective tissue growth factor (CTGF).

177 ERK1/2 acts on connective tissue growth factor (CTGF/CCN2) expression t

178 Several lines of evidence indicate that connective tissue growth factor (CTGF/CCN2) stimulates c

179 collagen VI, myocilin), matricellular genes (connective tissue growth factor [CTGF], secreted protein

180 expression of two TEAD-dependent genes, the connective tissue growth factor and amphiregulin genes.

181 in the levels of alpha-smooth muscle actin, connective tissue growth factor and fibronectin, and dec

182 collagen/fibronectin deposition, as well as connective tissue growth factor and matrix metalloprotei

183 ic RHOA effector, and elevated expression of connective tissue growth factor and smooth muscle actin.

184 mRNA levels of profibrotic genes, including connective tissue growth factor and tissue inhibitor of

185 e myosins, NotchR and Wnt pathway genes, and connective tissue growth factor by Pofut1 in skeletal mu

186 ro-fibrotic targets including ICAM-1 and the connective tissue growth factor CTGF.

187 at were capable of dose-dependently reducing connective tissue growth factor gene expression in vitro

188 that cysteine-rich protein 61 (CYR61)/CYR61 connective tissue growth factor nephroblastoma overexpre

189 -fibronectin, alpha smooth muscle actin, and connective tissue growth factor) and stabilized or impro

190 (transforming growth factor)-beta and CTGF (connective tissue growth factor) levels.

191 P1(-/-)) in mice disinhibited TGFbeta1-CTGF (connective tissue growth factor) signaling, leading to s

192 transforming growth factor-beta-1) and CTGF (connective tissue growth factor), reduced fibroblast pro

193 rine protease inhibitor, clade E, member 1), connective tissue growth factor, and collagen I].

194 luding overexpression of fibronectin 1 (FN), connective tissue growth factor, and other genes.

195 ding fibronectin, alpha-smooth muscle actin, connective tissue growth factor, collagen I, and TGF-bet

196 cation network factor 2, previously known as connective tissue growth factor, CTGF) using a specific

197 t plasminogen activator inhibitor-1 (PAI-1), connective tissue growth factor, fibronectin, and p21 in

198 ents within individual target mRNAs, such as connective tissue growth factor, induced hyper-adhesive,

199 ransforming growth factor-beta1 (TGF-beta1), connective tissue growth factor, matrix metalloproteinas

200 agonists of transforming growth factor-beta, connective tissue growth factor, several tyrosine kinase

201 decorin, important for the interaction with connective tissue growth factor, was detected predominan

202 wnstream effectors SMAD3 phosphorylation and connective tissue growth factor.

203 stoma overexpressed/cysteine-rich protein 61/connective tissue growth factor/nephroblastoma overexpre

204 Our work shows that muscle and cartilage connective tissues harbor progenitor cells capable of fu

205 thelial cells play a key role in stimulating connective tissue healing through a FOXO1-dependent mech

206 ted possible involvement of keratinocytes in connective tissue healing.

207 These novel data redefine adaptation in connective tissue, highlighting the fundamental importan

208 nflammatory classical complement pathway and connective tissue homeostasis.

209 decreased alveolar bone levels and a loss of connective tissue homeostasis.

210 In the intramuscular connective tissue (IMCT) of both muscles, a decrease in

211 g early stages, which likely translates into connective tissue impairment in DS patients.

212 e same processes, which specify the basis of connective tissue impairment observed in DS patients, po

213 , raising the possibility of broad roles for connective tissue in adult positional information.

214 Collagen forms the structural scaffold of connective tissues in all mammals.

215 SPGs) are important structural components of connective tissues in essentially all metazoan organisms

216 se sclerotome resulted in defects in fibrous connective tissues in the spine.

217 With recent advances in the engineering of connective tissues, including arteries, we are on the cu

218 emical evaluation to determine the extent of connective tissue inflammatory infiltrate (CTII) using t

219 Dense connective tissue injuries have limited repair, due to t

220 Maintenance of connective tissue integrity is fundamental to sustain fu

221 redefined the cellular origins of periocular connective tissues interacting with the EOMs, which do n

222 asal epithelium positioned at the epithelial-connective tissue interface (ECTI), separating the basal

223 er, the majority of the bone, cartilage, and connective tissue is derived from the neural crest.

224 Connective tissue is involved in driving regenerating fi

225 Connective tissue is one of the four major types of anim

226 peutic avenue for functional rejuvenation of connective tissue is reprogrammed fibroblast replacement

227 As connective tissue is ubiquitously distributed throughout

228 r invasive malignant epithelial cells in the connective tissue, keratin pearls in the OSCC, and ducts

229 ults revealed less gingival inflammation and connective tissue loss in the L+H groups compared with t

230 a family with a congenital-onset disorder of connective tissue, manifesting as early-onset joint hype

231 ived from animal sources, predominantly from connective tissue mast cells in pig mucosa.

232 ls were used as "surrogates" for mucosal and connective tissue mast cells, respectively, and their re

233 e number of blood vessels and sub-epithelial connective tissue matrix components within the wound bed

234 hosphorylation, neuropeptide biogenesis, and connective tissue maturation.

235 osphate/phosphate ratio is the cause of soft connective tissue mineralization in these disorders.

236 dered in patients presenting with congenital connective tissue/myopathy overlap disorders with joint

237 by Menninghaus et al. fails because its main connective tissue - "negative emotions" - is beyond the

238 roblastic properties and sprouted to form 3D connective-tissue networks.

239 Tuberous sclerosis complex-related connective tissue nevi are not limited to the lower back

240 cation, size, and histological appearance of connective tissue nevi in patients with TSC.

241 To describe the clinical characteristics of connective tissue nevi on the trunk and extremities of p

242 Connective tissue nevi were categorized per anatomic loc

243 complex (TSC) frequently develop collagenous connective tissue nevi.

244 We observed a decrease in a cerebriform connective tissue nevus and a reduction in pain in child

245 [19-70] years) with TSC (56%) had at least 1 connective tissue nevus on the trunk or thighs; of these

246 The juxtacanalicular connective tissue of the trabecular meshwork together wi

247 a precursor protein that is expressed by the connective tissues of the craniofacial skeleton, namely,

248 SIL, without significant down-regulation of connective tissue or transforming growth factor-beta sig

249 ed stromal progenitor performance related to connective tissue organization as well as muscle develop

250 ne = 20.47%, porcine = 19.52%, P = 0.82) and connective tissue/other (bovine = 43.32%, porcine = 49.2

251 bone formation, residual graft material, and connective tissue/other when ridge preservation is accom

252 The main expansion then occurs in the connective tissue outside the islet, which remains more

253 ining revealed a greater proportion of dense connective tissue (P=0.02) in women compared with men.

254 doexfoliation (PEX) syndrome exhibit various connective tissue pathologies associated with dysregulat

255 In rare pathologic conditions, connective tissue phenotype mast cells (MC(TC)s) can be

256 Nonimmunological connective tissue phenotypes in humans are common among

257 l delay, visceral and cardiac malformations, connective tissue presentations with arterial involvemen

258 apamycin (90.3% and 82.9%), respectively, 2) connective tissue progenitor cells, 3) platelet-derived

259 Connective tissue progenitor cells, platelet-derived gro

260 formed blastema, the spatial coordinates of connective tissue progenitors are predictive of their ul

261 ogether with kidney, smooth muscle, and limb connective tissue progenitors from the lateral plate mes

262 protease hydrolysis of meat myofibrillar and connective tissue protein extracts produces bioactive pe

263 r ability to hydrolyse meat myofibrillar and connective tissue protein extracts to produce bioactive

264 and thermal denaturation temperature of the connective tissue proteins (second peak in the DSC therm

265 with excessive accumulation of extracellular connective tissue proteins.

266 However, connective tissue proteome dynamics remain largely undef

267 nd the outer, orbital layer inserts into the connective tissue pulley.

268 ndicate that muscle functions as a planarian connective tissue, raising the possibility of broad role

269 pic delivery of Tgfbeta1 protein into nipple connective tissue reduced epidermal proliferation.

270 growth (growth hormone binding protein), and connective tissue remodeling (MMP3).

271 ms by diminishing inflammation and promoting connective tissue repair.

272 Fibroblasts are then cultured to produce connective tissue rich in extracellular matrix (stage 2)

273 The overproduction of connective tissue shares features with mammalian wound h

274 Fibrosis of the subsynovial connective tissue (SSCT) is a pathognomonic change in ca

275 hat selective breeding has resulted in lower connective tissue stability of Atlantic salmon fillets.

276 lasts lose contractility, leading to reduced connective-tissue stiffness.

277 es have been described in various species as connective tissue structures "bridging" small cranio-cer

278 as greater in the alveolar septa than in the connective tissue surrounding the airways or vessels.

279 The adhesion of soft connective tissues (tendons, ligaments, and cartilages)

280 ease in Sox9 expression in muscle-associated connective tissues, tendons, and bones led to hypoplasia

281 ndon and bone are attached by a transitional connective tissue that is morphologically graded from te

282 Fibrosis, the progressive accumulation of connective tissue that occurs in response to injury, cau

283 Tendon is a functionally important connective tissue that transmits force between skeletal

284 e localized fibrotic disorders of the penile connective tissues that can substantially impair a patie

285 nce the majority of the bones, cartilage and connective tissues that comprise the head and face are d

286 ucture-function relationships in native soft connective tissues that may be used to guide material de

287 d, subsequently, differentiate into bone and connective tissue to form the mandible.

288 nd clinical management of three intermediate connective tissue tumors: desmoid tumor (DT) or aggressi

289 & Microbe, Pundir et al. (2019) report that connective-tissue-type mast cells spy on bacterial commu

290 Our findings indicate that injury to dense connective tissues under prestrain alters boundary const

291 tly increased new bone formation and reduced connective tissue volume after 42 days demonstrating tha

292 tissue as either skin, fat, glands, ducts or connective tissue was demonstrated with an overall accur

293 ecently to genetic diseases characterized by connective tissue wasting (Penttinen syndrome) or overgr

294 this feat by means of elastic compliance of connective tissue, which passively redistributes the ove

295 essential for inducing the formation of new connective tissue, which requires the generation of sign

296 ssed that inspire the need to replace native connective tissue with biomaterials.

297 SSc results in dysfunctional connective tissues with excessive profibrotic signaling,

298 terogeneous group of hereditary disorders of connective tissue, with common features including joint

299 Connective tissues within the synovial joints are charac

300 ight at harvesting, softer texture and lower connective tissue yield compared with the WP group.