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1 H2-R439H mutation and without this mutation (control mice).
2 (EAE), an animal model of MS, as compared to control mice.
3 tumors, as compared with 100% of age-matched control mice.
4 y increased sCr versus the Txp group and the control mice.
5 s of colonized mice in addition to germ-free control mice.
6 of top clonotypes from persistently infected control mice.
7 rginine, in tumor-bearing mice compared with control mice.
8 orly differentiated cancer compared with KPC control mice.
9 )F-FAC in the brain at 180% of the levels of control mice.
10 eal fat depots compared to non-tumor bearing control mice.
11 G2a and higher levels of IgA antibodies than control mice.
12  with C rodentium, compared with none of the control mice.
13 day 7 and 14: 60%, respectively) compared to control mice.
14 -inducible knockout mice compared to that in control mice.
15 gs of vitamin D-deficient mice compared with control mice.
16 gdala of Tg2576 mice compared with wild-type control mice.
17 of venous thrombogenicity, compared with the control mice.
18 ctedly developed larger tumors compared with control mice.
19 cytes were unaffected post-I/R compared with control mice.
20 b was primarily taken up by BM and spleen in control mice.
21 en colon tumors from neutrophil-deficient vs control mice.
22 es were lower in Flna-deficient mice than in control mice.
23 ey developed regenerating nodules similar to control mice.
24 -inducible knockout mice compared to that in control mice.
25  of vascular tone, compared with aortas from control mice.
26 teric neurons from TPH2-R439H mice than from control mice.
27 ng carcinoma progressed more quickly than in control mice.
28 colon tumors compared with colon tumors from control mice.
29  sustain longer survival upon infection than control mice.
30 ocnemius homogenates from Acsl1(M) (-/-) and control mice.
31 nistered for 7 or 25 weeks to transgenic and control mice.
32 ice with orthotopic U87MG glioma and healthy control mice.
33 or size between Itgb8(flox/flox);Alb-Cre and control mice.
34 roteome profiles in dKO-Hom mice compared to control mice.
35 onary edema and reduced function compared to control mice.
36 s in the cornea and TG compared to levels in control mice.
37  was reduced by 90% compared to the level in control mice.
38 oxa2 conditional knockout mice compared with control mice.
39 ld male AbetaPP/PS1 and age-matched C57BL/6J control mice.
40 -LSN3316612 was reduced by 82% compared with control mice.
41 d RE levels in these mice were lower than in control mice.
42 e small intestine of Hnf4alphagamma(DKO) and control mice.
43 DSS- or T-cell transfer-induced colitis than control mice.
44 ed Hep3B cell xenografts compared to that of control mice.
45 ytic cells compared to exosomes derived from control mice.
46 avior and had antidepressant-like effects in control mice.
47 d more hepatocellular adenomas and HCCs than control mice.
48 ical and functional analysis in Ric(EKO) and control mice.
49  Itgb8(flox/flox);Alb-Cre mice compared with control mice.
50 ficantly differ from that in the non-injured control mice.
51 ulation, compared to that in vehicle-treated control mice.
52 is transcripts were increased, compared with control mice.
53 iated with increased autophagy compared with control mice.
54 acid (DHA; 22:6omega-3) (33%) in relation to control mice.
55 le luteinizing hormone secretion observed in control mice.
56 +) cells compared with Apoe(-/-) Malat1(+/+) control mice.
57 f HDM-sensitized mice compared with those in control mice.
58 AP increased the severity of liver damage in control mice.
59 ificant difference in oviduct pathology from control mice.
60 les of amifostine-treated and saline-treated control mice.
61 ere higher in OVA-sensitized WT mice than in control mice.
62  colonized by PMSS1 to a greater extent than control mice.
63 burden was higher in Nmur1(-/-) mice than in control mice.
64 hylnitrosamine and carbon tetrachloride than control mice.
65 ice compared with Ah(Cre)/Met(+/+)/Apc(fl/+) control mice.
66 mmunity composition compared to non-stressed control mice.
67 d in CVB3-induced myocarditis versus healthy control mice.
68 olitis after cohousing or feeding feces from control mice.
69 n arms of the elevated plus maze relative to control mice.
70 nd mRNA levels returned to similar levels as control mice.
71  had fewer organisms in their lungs than the control mice.
72 lenged leukotriene C4 synthase-deficient and control mice.
73 ion was not different in ROCK2(CD)(4Cre) vs. control mice.
74 ucose metabolism in atERalphaKO- compared to control mice.
75 lasma pro-inflammatory cytokines compared to control mice.
76 severe colitis in response to oxazolone than control mice.
77 mpared with isogenic L. monocytogenes-primed control mice.
78 le outer and inner regions resembled that of control mice.
79 KO) mice lost ISCs compared with crypts from control mice.
80 c contractile function after AMI compared to control mice.
81 al joints that were not observed in infected control mice.
82 ause and sinus arrhythmias) when compared to control mice.
83 eras, but not in irradiated, nontransplanted control mice.
84 er significantly between IL12B-deficient and control mice.
85 antly fewer IgE Abs to peanuts compared with control mice.
86 from Hnf4alphagamma(DKO) mice, compared with control mice.
87 , and defeated-resilient mice more resembled control mice.
88 layed enhanced viral clearance compared with control mice.
89  diethylnitrosamine and CCl(4) compared with control mice.
90  excretion were similar between knockout and control mice.
91 -treated cKO mice responded similarly to the control mice.
92 x) of susceptible, resilient, and unstressed control mice.
93 cells in the secretory lineage compared with control mice.
94 staining scores compared with saline-treated control mice.
95 ell loss, compared with those from Apoe(-/-) control mice.
96 d excitation-to-inhibition ratio compared to control mice.
97 t B cell-specific Traf3 (-/-) and littermate control mice.
98 del of Duchenne muscular dystrophy (mdx) and control mice.
99 rved differences during LTP in comparison to control mice.
100  survival rate and comparable body weight to control mice.
101 in Th2 cell-mediated responses compared with control mice.
102 ) rescued myogenic tone in high-fat diet-fed control mice.
103  in regenerating crypts compared to those of control mice.
104 ted HSCs from Mecp2(-/y) mice and wild-type (control) mice.
105  high-fat diet (HFD)-fed L13a KO and intact (control) mice.
106 let insulin secretion from diabetic, but not control, mice.
107 ly earlier (13 +/- 1 d) than their wild-type control mice (106 +/- 26 d).
108 ly smaller tumors (34.6 +/- 11.9 mm(3)) than control mice (161.6 +/- 79.4 mm(3), p < 0.036).
109  longer survival, and smaller scar size than control mice 28 days post-MI.
110 cortical and hippocampal regions only of IgG control mice accompanied with post-traumatic neuroinflam
111 ry, inflammation, and fibrosis compared with control mice, accompanied by increased pro-fibrogenic cy
112  warming in yellow fluorescent protein (YFP) control mice achieved.
113                            Interestingly, in control mice, acidified drinking water caused brain regi
114  and 1.5-fold greater, respectively, than in control mice (Acsl1(flox/flox) ), indicating muscle dama
115 -deficient mice were decreased compared with control mice after consuming an HFD.
116 ure and livers had less fibrin compared with control mice after pIVCL and bile-duct ligation; neutrop
117 cells overly quiescent in both epileptic and control mice again disrupted behavioural performance.
118  isolating muscle spindles from ssTnT-KO and control mice aiming to examine the composition of myofil
119 l level in the former was lower than that of control mice, although triglyceridemia in the two groups
120 bited a muted proximal-to-distal decrease in control mice and a smaller loss after VAGX (45-48%).
121 te localization were increased compared with control mice and associated with nuclear factor-kappaB a
122 Mgat1KO mice died significantly younger than control mice and demonstrated chamber dilation and systo
123 ed to examine their terminal fields in adult control mice and in adult mice in which the alpha-subuni
124 (1) mAChRs in conditioned fear extinction in control mice and in the stress-enhanced fear-learning mo
125 hrombosis in the inferior vena cavae of both control mice and mice administered aPL Abs.
126 mice given 2,4diHB was comparable to that of control mice and significantly higher than that of untre
127  15%, 70%, and 75%, respectively compared to Control mice and that GH treatment significantly increas
128 ytoplasm of islets cells than in non-treated control mice and this finding was corroborated by immuno
129 lected bone and skeletal muscle samples from control mice and three muscular dystrophic mouse models
130 ates through the blood of Muc2(-/-) (but not control) mice and causes immune activation of Ag-specifi
131                          Compared to healthy controls, mice and glioma patients demonstrated signific
132 portion of IgA-coated bacteria compared with control mice, and a reduced luminal concentration of sec
133 f Cln3(-/-) mice was markedly different from control mice, and acidified water differentially changed
134   We detected VEGF expression in JG cells of control mice, and cAMP agonists regulated VEGF expressio
135 drogen receptor knockout (SCARKO) mutant and control mice, and demonstrate that SCARKO mutant spermat
136 ction in blood ejection fraction relative to control mice, and eventual lethality in the absence of c
137 overexpressing mice were polyploid than from control mice, and had losses or gains of whole chromosom
138 d greater numbers of type 2 macrophages than control mice, and primary A5(+/-);Kras tumor cells had u
139 12D P53(flox/flox) mice with PDACs, C57/BL6 (control) mice, and 5 patients and analyzed by histology
140 nglia of infected mice was less than that in control mice; and (iii) that latency was significantly r
141 that, compared with isotype-treated infected control mice, anti-PD-1-treated mice had improved surviv
142  analysis of the spinal cord of Ppt1(-/-)and control mice at these timepoints revealed a significant
143  of littermate H2b(fl/fl) or H2b/p53(fl/fl) (control) mice at young and old ages.
144        Tissues from 4-month-old Ccne1(T) and control mice (at that age were free of tumors) were anal
145                   In intestinal tissues from control mice, ATIs induced an innate immune response by
146 significant decrease was observed in healthy control mice (baseline and posttreatment mean k(PL), 0.0
147                      Compared with uptake in control mice, BM (64)Cu-alphaCD11b uptake in mice with e
148  HGP and gluconeogenesis in hepatocytes from control mice but failed in hepatocytes from L-F1KO mice,
149 iception and persistent inflammatory pain in control mice but had little effect in Oprm1-cKO mice.
150  and myosin heavy chain IIa in quadriceps of control mice but not in ATF3-KO mice.
151 ve autophagy in livers of calorie-restricted control mice but not in L-Ghr (-/-) mice.
152 U/kg) for 10 days reduced trabecular bone in control mice but not in Tg mice.
153  that received EPA-rich oil when compared to control mice but there was no effect on the total time r
154 tg7, Beclin-1 and LC3A/B-II, seen in HFD-fed control mice, but enhanced their levels after 12 weeks o
155 tion in the above optical ICSS in VgluT2-cre control mice, but not in VgluT2-CB1 (-/-) mice.
156 Tail bleeding time was prolonged in DREAM KO control mice, but not in WT or DREAM bone marrow chimeri
157 e, and male TashT(Tg/Tg) mice, compared with control mice, but not Ret(9/-) mice (which had mycopheno
158 ermal thickness was observed in both MFS and control mice, but only dermal thinning in MFS mice was r
159  mucosal IgA repertoire of I-Ab(DeltaIEC) vs control mice, but opsonization of cultured C rodentium b
160  PH (priming phase of liver regeneration) in control mice, but this effect was delayed in interleukin
161  wild-type littermates (LysMCre;Mcl1(wt/wt), control mice) by administration of azoxythmethane and/or
162                                  Compared to control mice, circadian clock disruption significantly e
163 ontrol in CaMKIIcre:IFNAR(fl/fl) relative to control mice coincided with sustained Cxcl1 and Ccl2 mRN
164 dient of osmo-responsive genes compared with control mice, consistent with their physiologic phenotyp
165 4Q-CCC restored respiration rates to that of control mice correlating with higher electron transport
166 tem, where the home cage behavior of ELS and control mice could be monitored over a continuous 5-10 d
167       Additionally, manipulating activity in control mice could recapitulate the memory deficits in a
168  striking reductions in body fat compared to control mice (Ctrl).
169 ns-signaling mice was indistinguishable from control mice, demonstrating that IL-6 trans-signaling fu
170            We found evidence that littermate control mice develop D1R hypersensitivity after they are
171             Accordingly, CD11c-Ecad(del) and control mice developed comparable contact hypersensitivi
172                            Four hours later, control mice developed higher bacterial burden in blood
173           Gastric microbiomes from PPARD and control mice did not differ significantly.
174 sed serum antibody titers (P < .01), whereas control mice did not.
175  lose weight after leptin treatment, whereas control mice do not.
176 rosclerotic plaques in their aortas than the control mice due to reduced cholesterol uptake and synth
177 pressure difference between the knockout and control mice during high salt intake.
178 sure and urine secretion ability compared to control mice especially at 6 months posttransplant.
179 LC numbers remained stable and similar as in control mice, even in aged animals.
180                                              Control mice exhibited a large proximal-to-distal decrea
181                                              Control mice exhibited multiple IHRs, and a limited incr
182                            Compared with the controls, mice exposed chronically to As (10 ppb in drin
183 AL) gained less weight and body fat than did control mice fed a high-fat diet, resulting in ameliorat
184 /-) hearts 2.3-fold compared with littermate control mice fed a ketogenic diet, yet it did not improv
185                                              Control mice fed ATIs for 1 week were gavaged daily with
186 rgic stimulation when compared with those of control mice fed normal chow.
187 liver function on the DDC diet compared with control mice fed the DDC diet.
188                           Liver tissues from control mice given CCl(4) had significant increases in p
189 s, and plasma amylase activity compared with control mice given cerulein injections.
190                                  Livers from control mice given VPA and APAP accumulated cholesterol
191 iratory capacity in ischemic muscle, whereas control mice had normal mitochondrial function.
192                 Gp91(phox) knockout (KO) and control mice had similar GAS numbers at skin infection s
193 istal bowel tissues that were aganglionic in control mice, had a significant increase in colon motili
194                                  In normoxic control mice, HIF-1alpha deletion in the CNS or NTS did
195  autophagy genes were up-regulated in HD vs. control mice; however, IKKbeta knockout partially reduce
196 mulus of NELF-B UtcKO was similar to that of control mice; however, subsequent full decidual response
197 n was seen in ocularly infected mice than in control mice; (ii) that expression of various HSV-1 gene
198 ficantly higher survival rate than wild-type control mice in response to Candida albicans infection,
199 mor growth between conditional Raptor KO and control mice in the s.c. tumor models, although depletio
200            Inhibiting the alpha5-GABA(A)R in control mice in vivo improved spatial learning but also
201 es were observed between postnatal-OIRKO and control mice in: body composition (lean or fat mass); fa
202  hiPSC-derived cardiomyocytes, mdx mice, and control mice (in the presence or absence of propranolol
203                Sex differences were noted in control mice, in which female islets showed 5 selenoprot
204                             HFD treatment of control mice induced release of L13a from 60S ribosome,
205 tin, while vimentin levels were decreased in control mice injected with synthetic miR-144.
206 tor protein Alfy/Wdfy3 has no consequence in control mice, it significantly accelerates age of onset
207                                           In control mice, IT-type PNs targeted neighboring CGE INs,
208                                Compared with control, mice lacking the sialidase neuraminidase 3 have
209                    Compared with livers from control mice, livers from Cyld(DeltaLPC) mice (but not C
210 ortex, P < .05 for all regions compared with control mice; mean olfactory bulb signal intensity ratio
211                           When compared with control mice, mice exposed to stress displayed increased
212                                  Relative to control mice, MSDB-specific knockout of CB1 or MAGL bidi
213 ompletely between days 8 and 9 in littermate control mice (n=12), but were still present at day 9 in
214                                           In control mice not exposed to HF diet in utero, VPA/hydral
215 file from global-COX-1-ko or aspirin-treated control mice, notably significantly higher levels of PGI
216                        PDE10A was studied in control mice (NT) and in mice carrying human wild-type t
217 nificantly lower lung metastatic burden than control mice or mice injected with SUM149 shERK1 cells.
218 fl)CD11c-Cre mice with colitis compared with control mice or mice without colitis.
219 linical allergy scores to 1 from 3.5 in sham control mice (P < .001) after 6 treatments accompanied b
220 decreased in NTS-HIF-1alpha(-/-) compared to control mice (P < 0.0001).
221 3 compared to 7.8 +/- 0.5 for the uninfected control mice (P < 0.05).
222 r muscle (78.1 +/- 14.5% vs. 2.5 +/- 0.5% in control mice, P < 0.0001, N = 5-10/group) and decreased
223 61 +/- 134 mum(2) vs. 2221 +/- 100 mum(2) in control mice, P < 0.01, N = 5-10/group).
224 r-deficient (db/db), diet-induced obese, and control mice; pancreatic islets were isolated 7 days lat
225                                Compared with control mice, Pkd1 knockout mice exhibited reduced arter
226 6 to 40 hours after infection as compared to control mice, pointing at a role for LKB1 in macrophages
227        Administration of N-acetylcysteine to control mice prevented VPA- and APAP-induced ER stress a
228 ministration of tauroursodeoxycholic acid to control mice, prevented upregulation of STARD1 in liver
229                                              Control mice received an equivalent dose of nitrate-depl
230                                              Control mice received complete Freund adjuvant alone.
231                                              Control mice received no treatment or had acute osmotic
232                                              Control mice received phosphate-buffered saline (PBS).
233 ted as 25-50% T1 reductions in comparison to control mice receiving CSF saline.
234  rates in mice recipients in comparison with control mice receiving CTLs pre-cultured with TMPs from
235         Compared to mice on WDSW and vehicle controls, mice receiving WDSW + saroglitazar had lower w
236                 We gave T7K24R and C57BL/6N (control) mice repeated injections of cerulein to induce
237 pletion of glutathione from liver tissues of control mice, resulting in ER stress and the upregulatio
238              Intravital imaging of livers in control mice revealed sinusoidal complexes of neutrophil
239 s of carotid arteries from Pcsk6(-/-) versus control mice revealed suppression of contractile SMC mar
240 for hyperhomocysteinemia, and sibling CBS+/+ control mice revealed that deficiency of CBS upregulates
241                    Conversely, compared with control mice, SAP knockout mice fed on a normal diet had
242 ty compared to that of persistently infected control mice (Shannon indices of 2.1 and 2.6, respective
243                             Tumor bearing HF control mice showed a significant increase in serum free
244 inal uptake in the treated mice, whereas the control mice showed only urinary excretion.
245 iotic-induced microbiota-depleted (AIMD) and control mice showed significant variations in the metabo
246                                Compared with control mice, slc26a6(-/-) mice (previously shown to hav
247                                           In control mice, spindles were phase-coupled with DWs, and
248                  In both male and OVX female control mice, subcutaneous E(2) implant improved insulin
249 stributions of villus and crypt afferents as control mice, suggesting surgery did not contribute to t
250      Compared with resilient and nonstressed control mice, susceptible mice exhibited a higher reacti
251             Results showed that, compared to controls, mice that had their circadian rhythms disrupte
252          Our hypothesis was that compared to controls, mice that had their circadian rhythms disrupte
253                 When compared with sedentary controls, mice that performed a four-week treadmill exer
254 ough Trim33 knockdown mice weighed less than control mice, their skeletal muscles were histologically
255 y and hyperglycemia comparable with diabetic control mice, they exhibited significantly improved heal
256                                       Unlike control mice treated with 17beta-estradiol alone, cervic
257 nd liver were collected from Mdr2KO and FVBN control mice treated with Ghr, des-octanoyl-ghrelin (DG)
258 by the sites of pre-OVX spines than those in control mice treated with vehicle.
259                      Compared with infection controls, mice treated with anti-PD-1 and anti-PD-L1, bu
260 king memory performance, which was absent in control mice under l-DOPA treatment only.
261                             When compared to control mice, VGX did not significantly affect the devel
262 recreating granule cell hyperexcitability in control mice via excitatory chemogenetic receptors, with
263 iKO, villin-Cre+, Sirt1(flox/flox) mice) and control mice (villin-Cre-, Sirt1(flox/flox)) on a C57BL/
264 y of bladder structure between recipient and control mice was confirmed by histology.
265 urine voiding behavior of recipient mice and control mice was monitored by cystometry.
266 t reduction of wild type HTT (wtHTT) in Hu21 control mice was tolerated up to 7 months post-injection
267 ds from colon cells of CRT-knockout mice and control mice were analyzed by qRT-PCR, immunoblot, and t
268 sts (MEFs) and hepatocytes from Ccne1(T) and control mice were analyzed to determine the extent to wh
269 s differentially RNA-edited between case and control mice were enriched for functional terms highly r
270 id fructose (WDF) to induce steatohepatitis; control mice were fed normal chow.
271                                              Control mice were given either entecavir or non-HBV-spec
272                          Nr1d1(-/-) mice and control mice were given intraperitoneal injections of LP
273 10) or 110 MBq (82-116 MBq, n = 10), whereas control mice were injected with vehicle (n = 10).
274                                              Control mice were intratracheally instilled with sterile
275             Ground control (GC) and vivarium control mice were maintained on Earth in flight hardware
276                                       Ground control mice were maintained on Earth under identical en
277 ubbles and a labeled model drug, while three control mice were not treated (May-November 2017).
278 n this replication attempt, while 70% of the control mice were reported to be tumor-free after 9 week
279                    SCD and their littermates control mice were treated with antibiotics to deplete gu
280 l" mice were tk-positive, treated with GCV; "control" mice were either tk-negative treated with GCV,
281 , developed more tumors and metastases, than control mice, whereas A5;Kras mice did not develop any t
282 on of only contralateral forelimb muscles in control mice, whereas it induces activation of both cont
283 k phase) in the blood and brains of surgical control mice, whereas this temporal pattern was absent (
284 d to increased HA accumulation compared with control mice, which also coincided with decreased hyalur
285 led significant differences between PDX- and control mice, which could be due to differences in diet
286 , and extensive midzonal hepatocyte death in control mice, which were strongly minimized in HSC-deple
287 b mice exhibited lower species richness than control mice, while E2-treated mice had reduced evenness
288                                  Compared to control mice with a single V558Delta Kit mutation, mice
289 bolic profile and beta-cell phenotype as the control mice with an intact Akt1 gene.
290                                              Control mice with B16F10 orthotopically inoculated in th
291 lic stress was induced by HFD, beta cells in control mice with intact Akt1 proliferated as a compensa
292 exhibited anemia and hypoferremia similar to control mice with intact Ncoa4 regulation but showed a m
293 teriorating LV ejection fraction occurred in control mice with large infarcts (>/=25% LV).
294 l deprivation (MD) were compared to those in control mice with normal visual experience.
295 We also performed studies with C57BL/6 mice (controls), mice with deletion of neutrophil elastase (NE
296 e were 2-fold thicker than mucus layers from control mice, with increased proportions of Akkermansia
297 sive pancreatitis in T7K24R mice, but not in control mice, with typical features of chronic pancreati
298 ctive as antiepileptics at the same doses in control mice without colitis.
299 ling (hWtEPOR) were compared with littermate control mice (WT) to test the role of EPOR signaling und
300 -out mice [PON1KO], and wild type littermate control mice [WT].

 
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