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1 nfection), and 15-60 days of a positive PCR (convalescence).
2  markers occur during the first 12 months of convalescence.
3  and if secondary transmission occurs during convalescence.
4 amples obtained during the acute illness and convalescence.
5 tients who provided multiple samples through convalescence.
6  virus (EBV) during both acute infection and convalescence.
7 le, and similar patterns of clearance during convalescence.
8 ut the entire course of COVID-19 disease and convalescence.
9 ined cytotoxic transcriptional programs into convalescence.
10 t not heterologous, isolates were boosted in convalescence.
11 ular impairment and myocardial scar in early convalescence.
12 ease pathogenesis, response to therapies, or convalescence.
13 for the majority of observed phagocytosis at convalescence.
14 r to functional memory CD8(+) T cells during convalescence.
15 in reactivity to IgG1 and IgG3 antibodies at convalescence.
16 f individual memory B cells induced early in convalescence.
17 reased frequencies during acute COVID-19 and convalescence.
18 rocess regarding acute appendicitis, and its convalescence.
19 ness duration and gut microbiome profiles in convalescence.
20  PCR were performed at enrollment and during convalescence.
21 ed up to 8% during acute infection and early convalescence.
22 n (2.3%) or in early (26.6%) or late (58.6%) convalescence.
23 the late stages of acute disease or in early convalescence.
24 und to have severe unilateral uveitis during convalescence.
25 han those based on ecological competition by convalescence.
26 ral blood B cells during acute infection and convalescence.
27 n deposition and protected heart function in convalescence.
28 on methods are best utilized during presumed convalescence.
29 ed when IL-10 levels returned to baseline by convalescence.
30 t showed significant morbidity and prolonged convalescence.
31 roved intraoperative morbidity and decreased convalescence.
32 ldren (>=18 mo to 5 yr) during AVB and after convalescence.
33 eatment is effective but entails significant convalescence.
34  concentrations returning to baseline during convalescence.
35 ins associated with subclinical infection or convalescence.
36 ion decline in number and proportion by late convalescence.
37 F excretion, which returned to normal during convalescence.
38 the systemic angiogenic process during early convalescence.
39 rgence of neutralizing antibodies long after convalescence.
40 od loss, a shorter hospital stay, and faster convalescence.
41 des briefer, less intense, and more complete convalescence.
42 sion; bactericidal activity developed during convalescence.
43 d that IFA is useful for diagnosis of HGE in convalescence.
44 te disease (0.29 +/- 0.02 g/L) compared with convalescence (0.40 +/- 0.04 g/L), although not statisti
45 acute disease (36.9% +/- 2.5%) compared with convalescence (18.8% +/- 1.5%; p =.0003).
46  COVID-19 patients from initial diagnosis to convalescence (2-3 months later), integrated with clinic
47  (43.5+/-14.1 vs. 45.7+/-25.3 hours, P =0.1; convalescence 23.5+/-5.3 vs. 20.2+/-4.1 days, P =0.5).
48 on i.e., at disease presentation compared to convalescence, 28 days post-infection defined by a follo
49 sonance imaging acutely (24-72 hours) and at convalescence (3 months).
50                                           By convalescence, 65% of patients had positive IgM or IgG a
51 acute patients (53.3 +/- 4.4%) compared with convalescence (67.8 +/- 3.2%; p =.015).
52                                           At convalescence (7 weeks +/- 1 week post-ARTI), specimen c
53  a persistent type I interferon signature at convalescence across immune compartments.
54 l injury have a large negative effect on the convalescence after abdominal surgery.
55                                       During convalescence after severe COVID-19 infection with tropo
56 ions that include uveitis can develop during convalescence, although the incidence and pathogenesis o
57 ple aspects of CCHFV pathogenesis, including convalescence, an important aspect of CCHF disease that
58                                       During convalescence, an increasing proportion of pp65-specific
59 -adjusted SR cutoffs of 0.6 mumol/L for late convalescence and 0.5 mumol/L for early convalescence, t
60 hospitalization, postoperative pain, time to convalescence and activity, while providing an optimal c
61  must be considered in predicting protracted convalescence and allocating medical resources.
62 or post-EVD symptoms are common early during convalescence and decline over time along with severity.
63        We monitored primary infection during convalescence and during the establishment of persistent
64 e and impacts of SARS-CoV-2 infection during convalescence and for their role in immune host defense
65  such as fatigue or exertional dyspnea after convalescence and healthy control participants underwent
66 ical processes such as a temporary period of convalescence and pathogen-induced mortality have led to
67  postoperative complications lead to shorter convalescence and possibly improved 30-day survival.
68 nyan children with severe malaria and during convalescence and related these parameters to the adhesi
69 ere higher at hospital admission than during convalescence and that EPCR-rs867186 G allele was associ
70 ed substantially in sensitivity during early convalescence and time to seroreversion.
71 mmediately after blood-stage infection or at convalescence, and for most subsets was directly associa
72 gher in severe than uncomplicated malaria in convalescence, and higher in children with blood group O
73 d to understand how the acute course of EVD, convalescence, and host immune and genetic factors may p
74  P lt 0.001) higher in the incubation, early convalescence, and late convalescence subgroups, respect
75 condary endpoints were morbidity, mortality, convalescence, and pathology.
76 ine month period encompassing acute disease, convalescence, and recovery.
77 s during the transition from peak illness to convalescence, and these distinct inflammatory patterns
78 of acute pneumonia, to estimate mortality at convalescence, and to analyse mortality risk-factors.
79           Clinical sequelae during early EVD convalescence are common and sometimes sight threatening
80 d SAM, and targeted interventions to improve convalescence are needed.
81 e during the transition from peak illness to convalescence are not yet well understood.
82 s a result of prolonged hospitalizations and convalescence associated with antibiotic treatment failu
83 e pain, cosmetic concerns, and the prolonged convalescence associated with the donor operation.
84                                       During convalescence, at the conclusion of antibiotic therapy,
85 m-neutralizing Abs against SARS-CoV-2 during convalescence between individuals.
86  APP), incubation (elevated CRP only), early convalescence (both APP and CRP elevated), and late conv
87     Because of shortened hospitalization and convalescence, CAS challenges CEA as the preferred treat
88 gly popular owing to decreased morbidity and convalescence compared with open procedures.
89 L, and transfusion rates as well as improved convalescence compared with open-radical cystectomy.
90 h a briefer, less intense, and more complete convalescence compared with the open surgical approach.
91 obtained from three of these patients during convalescence contained antibodies that reacted with E.
92 om acute infectious mononucleosis (AIM) into convalescence (CONV).
93 osis [AIM]) and again 6 months later (during convalescence [CONV]).
94 ck (day0) and returned to normal values upon convalescence (day28).
95 ic surgeries offer added benefits of earlier convalescence, decreased blood loss, as well as decrease
96                                       During convalescence, dengue-specific T cells differentiated in
97 ability to neutralize type 1 interferon into convalescence despite lower levels of binding immunoglob
98 g stability or decline of antibody titers in convalescence, efficacy of different vaccination regimen
99 scence (both APP and CRP elevated), and late convalescence (elevated AGP only).
100  understanding of CCHF, and investigation of convalescence following severe acute CCHF has been limit
101 supply while minimizing pain and duration of convalescence for donors.
102 ewer wound-related complications, and faster convalescence for patients who underwent laparoscopic ga
103 ring the acute phase of infection and during convalescence for WHcAg and WHxAg expression by immunohi
104 uroinflammatory process persists weeks after convalescence from acute respiratory infection.
105 es ranging from acute ZIKV infection to late convalescence from individuals with evidence of prior DE
106 d funding was also provided by the NIHR 606 (CONVALESCENCE grant [COV-LT-0009]).
107  The Ab levels significantly declined during convalescence (> 90 days since onset of illness), compar
108     As a result, visual outcomes and patient convalescence have improved significantly.
109 ude reduced postoperative pain and shortened convalescence; however, there are few data with respect
110 of plasma endothelial markers acutely and in convalescence in Malawian children with uncomplicated or
111 ctor-GNLY and NKT CD160, was associated with convalescence in moderate patients.
112         IgG levels rose significantly during convalescence in patients with bacteremic pneumonia, rea
113 hat MPXV was cleared from most organs during convalescence, including healed skin lesions, but could
114 erentiate in a coordinated fashion well into convalescence into a state characteristic of long-lived,
115 he potential decrease in hospitalization and convalescence may also prove to be financially advantage
116                                 In contrast, convalescence may be prolonged by other immunologic reac
117 =.001) and decreased 1.5-fold (P=.01) during convalescence (median, 15 days).
118 tracked their response through infection and convalescence.METHODSParticipants were injected intraven
119   Over the approximately six month period of convalescence monitored, we observed a slow and progress
120 IC) and euthanized acutely (n = 43) or after convalescence (n = 22).
121  of acute EVD with the following outcomes in convalescence: new ocular symptoms, uveitis, auditory sy
122 re arising, and may enable us to improve the convalescence of larger numbers of our patients.
123                                       During convalescence, only IgG (and no IgA) RSV-specific memory
124 s, serum neutralizing antibody titers during convalescence, or duration of mechanical ventilation, in
125 elae which may occur during acute infection, convalescence, or recovery.
126  present for months, or indefinitely, in the convalescence phase of critical illness.
127 y cohort, 10 in the acute phase and 8 in the convalescence phase.
128 the onset of severe clinical symptoms to the convalescence phase.
129                                              Convalescence-phase sera from humans were similarly inef
130 e the serum neutralizing antibody responses, convalescence-phase serum samples from people previously
131  features normalized over the resolution and convalescence phases.
132 y, there was a strong, distinct signature of convalescence present at day 9 after infection that rema
133  neutralizing antibodies that develop during convalescence prevent reinfection in animal models.
134 d loss, length of hospital stay, pain score, convalescence, quality of life, and costs.
135 o admission, hospital stay and postoperative convalescence (reflecting the consequences of delay in d
136 g the acute phase and 72, 44, and 32% during convalescence, respectively.
137                              Vaccination and convalescence resulted in the highest SARS-CoV-2-specifi
138 eral blood that changed during treatment and convalescence, returning in the majority of cases to the
139 isode and compared them with findings from a convalescence sample.
140 he incubation, early convalescence, and late convalescence subgroups, respectively, with correspondin
141  undergo coronary angioplasty have a shorter convalescence than those who undergo coronary bypass sur
142                                       During convalescence the phenotype switches to small and large
143 late convalescence and 0.5 mumol/L for early convalescence, the adjusted prevalence of SR deficiency
144 ecurrence, as well as postoperative pain and convalescence, the treatment of inguinal hernias underwe
145                                       During convalescence, this NHP developed neurological signs and
146 n important role in decreasing postoperative convalescence through maintaining preoperative mydriasis
147 -donor nephrectomy, as it results in a short convalescence time and increased quality of life.
148 oncologic control, low morbidity and shorter convalescence time.
149 viduals shortly after infection and later in convalescence to determine the impact of maturation over
150  determination of live viral shedding during convalescence to inform decisions to end isolation.
151 phages expressed pro-repair molecules during convalescence to promote the restoration of tissue integ
152 t were collected during acute EVD and during convalescence up to day 361 following illness onset.
153  admission and during hospital admission and convalescence (up to 30 days after onset of illness).
154       Ebola-specific memory B cells early in convalescence were low in frequency, and the antibodies
155    The titers both at presentation and after convalescence were not associated with the HRV genotype
156 f disease and 100% of the dogs tested during convalescence were seropositive for E. equi antigens.
157                     Donor length of stay and convalescence were similar in both groups (43.5+/-14.1 v
158 n important role in decreasing postoperative convalescence with fewer side effects.
159 cumulate to strikingly high frequencies into convalescence without continued proliferation.
160 his approach in regards to complications and convalescence without evidence of compromise to early an

 
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