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1 ucible progression from hyperexcitability to convulsive seizures.
2  were hypersusceptible to chemically induced convulsive seizures.
3 inguishing between epileptic and psychogenic convulsive seizures.
4 ng of treatment relative to the cessation of convulsive seizures.
5 acute group I mGluR-dependent propensity for convulsive seizures.
6 gy through blood-brain barrier breakdown and convulsive seizures.
7 lsant pentylenetetrazole-induced generalized convulsive seizures.
8 ns ranging from mild cognitive impairment to convulsive seizures.
9  (AD)-generating stimulations and individual convulsive seizures.
10 rosis), a history of secondarily generalised convulsive seizures (2.3; 95% CI 1.7 to 3.0 for these se
11 1) a strong IEGP response to kainate-induced convulsive seizures, (2) no IEGP response after prolonga
12 arlier were video-monitored for spontaneous, convulsive seizures 9 hr/day every day for 24-36 days.
13 e hypersynchronization and nonconvulsive and convulsive seizures, accompanied by expression changes i
14 ysis over 9 days revealed convulsive and non-convulsive seizure activity in rats infused with LGI1-mA
15 se is a strain that is highly susceptible to convulsive seizures after repeated sensory stimulation.
16 eatment was initiated after observation of a convulsive seizure and repeated at 4 to 6 week intervals
17 ed per 100 000 person-years) for generalised convulsive seizures and intussusception were similar bet
18 tion of p.Arg1872Trp by EIIa-Cre resulted in convulsive seizures and lethality at 2 weeks of age.
19 e detect ictal discharges that coincide with convulsive seizures and myoclonic jerks.
20 ctal arrhythmias were mostly found following convulsive seizures and often associated with (near) SUD
21 ication which may be impaired in autism, non-convulsive seizures and other conditions.
22 eizure phenotype associated with spontaneous convulsive seizures and profound deficits in hippocampus
23 on of consciousness occurs after generalized convulsive seizures, and includes analgesia, lasting for
24 sorder that includes both convulsive and non-convulsive seizures, and is dependent upon Celf4 gene do
25 seizures (lightning-like jerks), generalized convulsive seizures, and varying degrees of neurological
26 esults suggest that local anesthetic-induced convulsive seizures are mediated by excitatory glutamate
27                                       Lethal convulsive seizures are observed beginning at postnatal
28 h sexes, two models of SUDEP, and found that convulsive seizures caused a postictal decrease in venti
29 essive increase in the number and density of convulsive seizure clusters.
30 milial Alzheimer's disease transgene-induced convulsive seizures did not occur in mice lacking PrP(C)
31                                              Convulsive seizures during ethanol withdrawal (ETX) in r
32  was the change in mean monthly frequency of convulsive seizures during the treatment period compared
33 rvation period to establish baseline monthly convulsive seizure frequency (MCSF; convulsive seizures
34  provided significantly greater reduction in convulsive seizure frequency compared with placebo and w
35            Together with solitary living and convulsive seizure frequency, peri-ictal apnoea (>14 s f
36      The primary end point was the change in convulsive-seizure frequency over a 14-week treatment pe
37 nnabidiol resulted in a greater reduction in convulsive-seizure frequency than placebo and was associ
38 patients who had at least a 50% reduction in convulsive-seizure frequency was 43% with cannabidiol an
39  that were validated in EEG recordings of 48 convulsive seizures from 48 subjects with refractory foc
40  (SUDEP) that exhibit audiogenic generalized convulsive seizures (GCS), ending in death due to respir
41 ere we found that both focal and generalized convulsive seizures (GCSs) in epilepsy patients caused a
42 o the bilateral motor cortex, which mediates convulsive seizures, have not been delineated.
43 One serious adverse effect of these drugs is convulsive seizures; however, the mechanisms underlying
44 se models of SUDEP we found that generalized convulsive seizures impaired CO(2) chemoreception, and i
45    Certain anti-convulsant drugs, as well as convulsive seizures impede recovery when administered du
46 l symptoms, as well as spontaneous recurrent convulsive seizures in 45% and epileptiform spikes in 10
47 umented to exert a protective action against convulsive seizures in animal models, when administered
48 in the mouse hippocampal CA1 reliably evoked convulsive seizures in awake mice.
49 potential as an acute abortive treatment for convulsive seizures in emergency situations.
50 ic activities and neural dynamics leading to convulsive seizures in patients and mouse models of Drav
51 5% CI 3.94-14.71), three or more generalised convulsive seizures in the previous year (3.1, 1.64-5.87
52 5-year seizure freedom (eg, a normal MRI and convulsive seizures in the previous year has a probabili
53  spinal cord in several forms of generalized convulsive seizures, including audiogenic seizures (AGS)
54                                  Generalized convulsive seizures increase glucose utilization within
55           Generalised-particularly nocturnal-convulsive seizures, longstanding epilepsy, and solitary
56 accompanied by forebrain accumulation of the convulsive seizures mediating miR-134.
57 all animals as did the total duration of non-convulsive seizures (NCS) in the alpha-chloralose-anesth
58 or older, had been treated for a generalised convulsive seizure of longer than 5 min duration with ad
59  was to elucidate the effects of generalized convulsive seizures on distinct and separate corticotrop
60 ce (n = 9) had increased numbers of observed convulsive seizures (P = 0.004), a higher total seizure
61                      The median frequency of convulsive seizures per month decreased from 12.4 to 5.9
62 e minutes or more) or repetitive generalized convulsive seizures received intravenous diazepam (5 mg)
63 poexcitation of cortical circuits leading to convulsive seizure resistance, and (2) hyperexcitation o
64  FPI-treated rats developed nonconvulsive or convulsive seizures that could be distinguished electrog
65                                    Focal and convulsive seizure thresholds were evaluated 10-12 min p
66                                      A brief convulsive seizure was associated with marked changes in
67 es, with or without progression to bilateral convulsive seizures, was the most common seizure type.
68  monthly convulsive seizure frequency (MCSF; convulsive seizures were defined as hemiclonic, tonic, c