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1 The ATP7A gene encodes a copper-transporting ATPase.
2 e Menkes disease gene (MNK), which encodes a copper-transporting ATPase.
3 to these disorders encode highly homologous copper transporting ATPases.
4 bolic disorder caused by inactivation of the copper-transporting ATPase 2 (ATP7B) and copper (Cu) ove
5 sh the key catalytic properties of the ATP7B copper-transporting ATPase and provide a foundation for
6 ochaperone Atox1 directly interacts with the copper-transporting ATPases and plays a critical role in
11 of-function mutations in the gene encoding a copper-transporting ATPase (Atp7a) on the X chromosome.
16 Primary sequence analysis suggests that the copper-transporting ATPase ATP7B or the Wilson's disease
18 myces cerevisiae deficient in the homologous copper-transporting ATPase CCC2, suggesting that this pr
23 nding domain of WNDP and is conserved in all copper-transporting ATPases from bacteria to mammals; ho
25 idating the function and localization of the copper-transporting ATPases in mammalian cells and provi
27 The Wilson's disease protein (WNDP) is a copper-transporting ATPase regulating distribution of co
28 protein, alpha-catenin, tubulin alpha-chain, copper-transporting ATPase, salivary gland protein SGS-3
30 escued by knockdown of ATP7A, a trans-Golgi, copper-transporting ATPase that traffics to the plasma m
31 mutant of Escherichia coli lacking the CopA copper-transporting ATPase was hypersensitive to killing
32 is caused by mutations in a gene encoding a copper-transporting ATPase (Wilson's disease protein, WN
33 reshold for copper-dependent movement of the copper-transporting ATPases within the secretory compart