ライフサイエンスコーパス: core body temperature

1 (i.e., metabolites, cardiac activities, and core body temperature).

2 g naturalistic seizures induced by increased core body temperature.

3 mmals allowing optimal spermatogenesis below core body temperature.

4 experimental conditions that maintain normal core body temperature.

5 .3 degrees to 0.5 degrees C reduction of the core body temperature.

6 stration are temporally linked to changes in core body temperature.

7 f acute pain and for their ability to affect core body temperature.

8 he circadian rhythms of plasma melatonin and core body temperature.

9 ge of 2.0 degrees F (1.1 degrees C) over the core body temperature.

10 be affected by both ambient temperature and core body temperature.

11 s and an intraperitoneal microchip to record core body temperature.

12 exposure resulted in an overall increase in core body temperature.

13 mans, is their ability to adapt to mammalian core body temperature.

14 utaneous vasodilation and subsequent drop in core body temperature.

15 I, 0.44-0.69 C) were associated with greater core body temperature.

16 In humans, perspiration regulates core body temperature.

17 tional heat production is required to defend core body temperature.

18 diminish inflammatory pain without affecting core body temperature.

19 loggers to obtain continuous measurements of core body temperature.

20 regulation of locomotor activity, sleep, and core body temperature.

21 y of mice to regulate brown fat and maintain core body temperature.

22 histology, qPCR, HPLC, LC/MS and measures of core body temperature.

23 uiet wakefulness at baseline and at elevated core body temperature.

24 ith radiotelemetry probes for measurement of core body temperature.

25 trols had similar resting metabolic rate and core body temperature.

26 ed data loggers to obtain direct measures of core body temperature.

27 ine reduction in Ucp1-deficient mice reduces core body temperature.

28 thout altering respiratory exchange ratio or core body temperature.

29 pecific Et2 knockout mice displayed a normal core body temperature.

30 Circadian phases were derived from core body temperature.

31 y is not generally associated with a reduced core body temperature.

32 growth factor 1 (IGF-1) in the regulation of core body temperature.

33 tes, including hepatic glycogen, to maintain core body temperature.

34 d by generalized seizures caused by elevated core body temperature.

35 and apparently uncoupled from the rhythm of core-body temperature.

36 petite responses while increasing thirst and core-body temperature.

37 ed as assessed by metabolic cage studies and core body temperatures.

38 displaying elevated resting heart rates and core body temperatures.

39 ignificantly reduced WAT and slightly higher core body temperatures.

40 ln(-/-) mice were not able to maintain their core body temperature (37 degrees C) and developed hypot

41 n the nasal cavity (33-35 degrees C) than at core body temperature (37 degrees C).

42 ] vs 11.3 mM [95% CI, 9.0-14.1], p = 0.004), core body temperature (39.3 degrees C [95% CI, 39.0-39.5

43 t illness characterized by the rapid rise of core body temperature above 40 degrees C and central ner

44 duced browning, FGF21 BKO mice had preserved core body temperature after cold exposure.

45 osterone and ACTH responses, heart rate, and core body temperature after the 6th exposure in male Spr

46 The estimated core body temperature aligns, to within 0.05 degrees C,

47 We assessed core body temperature amplitude and the rhythmicity of 9

48 Individuals with higher core body temperature amplitude had a greater number of

49 Higher core body temperature amplitude was also associated with

50 d with MiniMitter transmitters that recorded core body temperature and activity (12 h LD cycle).

51 laced in a restricted feeding schedule, both core body temperature and activity entrained to the feed

52 dark phase feeding accelerated adaptation of core body temperature and activity rhythms, however, did

53 dia was observed briefly, but only after the core body temperature and blood pressure began to decrea

54 haracterized by increases in metabolic rate, core body temperature and cardiac performance.

55 Anaphylactic response was assessed by core body temperature and clinical scoring.

56 BAT thermogenic programs, leading to reduced core body temperature and cold intolerance.

57 into the POA induced prolonged elevation of core body temperature and decreased respiratory exchange

58 ons does not result in arrhythmicity because core body temperature and exploratory activity rhythms p

59 ther 37 degrees C or 0.5 degrees C below the core body temperature and followed up for 1 year.

60 , GC-1--treated mice also failed to maintain core body temperature and had reduced stimulation of BAT

61 iet or LPS were both associated with reduced core body temperature and heat release.

62 Primary outcomes were core body temperature and incidence of moderate to sever

63 We made time-series recordings of core body temperature and locomotor activity in 19 elder

64 olerance to an otherwise lethal reduction of core body temperature and metabolism.

65 olerance to an otherwise lethal reduction of core body temperature and metabolism.

66 Circadian rhythms of core body temperature and motor activity were measured i

67 the pharmacological activity of improgan on core body temperature and nociceptive (tail flick) respo

68 enditure and impairment in maintaining their core body temperature and not because of hyperphagia, de

69 er, mortality can be minimized by monitoring core body temperature and preventing MA-induced hyperthe

70 eased pro-social behavior without decreasing core body temperature and selectively enhanced nucleus a

71 cuits involved in opposing behaviors such as core body temperature and sleep compute conflicting info

72 n the brain are typically increased over the core body temperature and the jugular bulb temperatures.

73 els, given the strict programmes controlling core body temperature and the physiological stress that

74 try devices enabled real-time acquisition of core body temperatures and changes in heart rates and el

75 significant phase-delay in their rhythms of core-body temperature and activity compared with patient

76 movement (REM) latency, increased nocturnal core body temperature, and abnormal hormone secretion pa

77 ia, anxiolytic response, locomotor activity, core body temperature, and blood ethanol concentration,

78 e endogenous circadian rhythms of melatonin, core body temperature, and cortisol in healthy young and

79 Sleep EEG, core body temperature, and locomotor activity were recor

80 3 mg/kg subcutaneous) on locomotor activity, core body temperature, and social behavior (social inter

81 daily rhythms in blood pressure, heart rate, core body temperature, and spontaneous physical and neur

82 ammals, testicular temperature is lower than core body temperature, and the vulnerable nature of sper

83 y demonstrated increased energy expenditure, core body temperature, and UCP1 expression.

84 Women had a mean core body temperature approximately 0.23 degrees C great

85 Changes in core body temperature are fundamental and universal fact

86 wever, the impact of subtle changes of human core body temperature are only beginning to be acknowled

87 monitoring the rate of O(2) consumption and core body temperature as indicators of torpor.

88 temperature >=32 degrees C to maintain their core body temperature at 33.5 degrees C had a high likel

89 pid droplets in BAT and fail to defend their core body temperature at 4 degrees C, despite elevated s

90 nction, it is important to maintain constant core body temperature at ~37 degrees C.

91 determine the effects of IV acetaminophen on core body temperature, blood pressure, and heart rate in

92 Alterations in core body temperature, bronchial constriction, plasma hi

93 r data indicate that histamine modulates the core body temperature by acting at two distinct populati

94 l cardiac rhythm at baseline, but increasing core body temperature by as little as 3 degrees C causes

95 Thus, TRPV1 drugs alter core body temperature by modulating sensory input to the

96 rown adipose tissue (BAT) controls mammalian core body temperature by non-shivering thermogenesis.

97 ch play a critical role in the regulation of core body temperature (CBT) and energy balance.

98 Mammals normally maintain their core body temperature (CBT) despite changes in environme

99 testosterone on the circadian regulation of core body temperature (CBT) in adulthood.

100 Average core body temperature (CBT) in mice is 37 C.

101 ur antagonists on TRPV1 polymodal gating and core body temperature (CBT) in Trpv1(+/+), Trpv1(-/-), a

102 Humans' core body temperature (CBT) is strictly controlled withi

103 dence suggests that the circadian decline of core body temperature (CBT) triggers the initiation of h

104 ts of FEV(1), FEVC, PEF, blood cortisol, and core body temperature (CBT) were performed every 2 h.

105 e responses (APRs) that include increases in core body temperature (CBT), increases in hypothalamic-p

106 Core body temperature (CBT), salivary melatonin, subject

107 tive function requiring coordination between core body temperature (CBT), the central nervous system,

108 s in physical activity, sleep disruption, or core body temperature (CBT).

109 e metabolic signal insulin in the control of core body temperature (CBT).

110 Order effects, core body temperature changes due to menstruation, and c

111 diet, more quickly realigned NREM sleep and core body temperature (ClockLab) diurnal rhythms to the

112 duced a continuous and more rapid decline in core body temperature compared to low rebound [LR] mattr

113 e and amplitude of the rhythms of melatonin, core body temperature, cortisol, alertness, performance

114 Valid core body temperature data were available in 17/23 (mean

115 on of sake yeast with locomotor activity and core body temperature decreases under the stressful envi

116 Mean (+/-SE) daily core body temperature did not differ significantly betwe

117 unclear, but the shift of ~0.5 degrees C in core body temperature does not appear to be sufficient t

118 ly, implant recipients demonstrated elevated core body temperature during cold challenges, enhanced r

119 lower exercise capacity, failure to maintain core body temperature during cold stress, and reduced ab

120 eotherms use thermogenesis to maintain their core body temperature, ensuring that cellular functions

121 Patients who dialyzed at 0.5 degrees C below core body temperature exhibited complete protection agai

122 These mice maintain a lower core body temperature, fail to respond to a cold challen

123 in a novel physiological pathway regulating core body temperature, feeding behavior, and obesity in

124 ycles, hormone release, bioluminescence, and core body temperature fluctuations.

125 Reduction of core body temperature has been proposed to contribute to

126 Reserpine pre-treatment caused reductions in core body temperature; heating the rats to normal body t

127 Inducible Ucp1 knockout did not alter core body temperature; however, BAd-CRISPR Ucp1 mice had

128 ses in cerebral blood flow (CBF), as well as core body temperature; however, the isolated influence o

129 from the WT, but they showed an increase in core body temperature in anticipation to the meal time s

130 To maintain core body temperature in cold conditions, mammals activa

131 hydrocapsiate) to induce a sustained fall in core body temperature in conscious mice.

132 To maintain core body temperature in mammals, CNS thermoregulatory n

133 sodium nitrite can further decrease daytime core body temperature in mice via nitric oxide (NO) sign

134 he compounds disclosed herein do not elevate core body temperature in preclinical models and only par

135 of FGF21 and its site of action to regulate core body temperature in response to cold.

136 the in vivo release of histamine and drop in core body temperature in vivo using a MC-dependent model

137 ses in energy expenditure and maintenance of core body temperature in WT and FL-PGC-1alpha(-/-) mice.

138 in terms of the similarity in 24 h rhythm of core body temperature, in weeks when food was only avail

139 e transponder microchips, we showed that the core body temperature increased approximately 2 degrees

140 ng PPE was 65.7 (13.5) minutes; and the mean core body temperature increased by 0.46 degrees C (0.20

141 P) to mice induced a TRPV1-dependent drop in core body temperature into the mild hypothermia range (3

142 Homeostatic control of core body temperature is essential for survival.

143 Core body temperature is normally tightly regulated by a

144 Core body temperature is normally tightly regulated to w

145 s were survival at 90 days and time to reach core body temperature less than 34 degrees C.

146 (-8), 3 h before (-3) or 3 h after (+3) the core body temperature minimum (CBTmin) measured on the b

147 s centred prior to the critical phase at the core body temperature minimum, phase advances occurred w

148 a were aligned according to circadian phase (core body temperature minimum; CBTmin) and averaged.

149 logic monitoring of blood pressure, EEG, and core body temperature monitoring and intermittent arteri

150 c anaphylaxis, the symptoms and decreases in core body temperature observed in wild-type mice were re

151 Four HCWs reached or exceeded a mean core body temperature of >/= 38.5 degrees C.

152 ol) mice entered deep torpor, with a minimum core body temperature of 24 degrees C, 2 degrees C above

153 atients randomized to hypothermia achieved a core body temperature of 34.7 degrees C before reperfusi

154 mes, defined as the time required to reach a core body temperature of 38.5 degrees C, and cardiovascu

155 Core body temperature of Mc4r-null mice was normal, and

156 Core body temperature of PVG rats was maintained at eith

157 such as the regulation of blood pressure and core body temperature, oncogenesis, and immune function(

158 ates and pneumatic pressures and maintaining core body temperature, optimal patient outcomes can be a

159 als also exhibited no significant changes in core body temperature or cardiovascular rhythm, whereas

160 ods when determinations were based on either core body temperature or plasma melatonin measurements,

161 We measured core body temperature over most of the gestation period

162 The mean increase in core body temperature over time (from baseline to 150 mi

163 Core body temperature, PET imaging of glucose uptake and

164 nt in rats was not accompanied by changes in core body temperature, physical activity, or heart rate.

165 Anaphylactic symptom scores, core body temperatures, plasma histamine levels, basophi

166 Thus, modest, sustained reduction of core body temperature prolonged life span independent of

167 average brain temperature increase over the core body temperature ranged from -0.5 degrees to 3.8 de

168 othermy wherein metabolic rates are reduced, core body temperatures reach ambient levels, and key phy

169 ons to their whole-body metabolic rhythm and core-body-temperature rhythm.

170 tressor exposure, including disturbed sleep, core body temperature rhythmicity, and gut microbial dys

171 We studied the activity and core-body temperature rhythms in a cohort of 38 male pat

172 enes to determine whether differences in the core body temperature set point affect the regulation of

173 A lower core body temperature set point has been suggested to be

174 nd nocturnal periods of circadian rhythms in core body temperature, sleepiness, power in the theta ba

175 ed the telemetric monitoring of activity and core body temperature (T(b)) and bilaterally implanted w

176 Mammals maintain a nearly constant core body temperature (T(b)) by balancing heat productio

177 We measured core body temperature (T(b)) daily rhythms of Cape groun

178 Chow), and measured the influence of diet on core body temperature (T(b)), brown adipose tissue (BAT)

179 milar cardiovascular changes and increase in core body temperature (T(co)) can be elicited.

180 f these recordings as a proxy for continuous core body temperature (T(core)) measurements has not bee

181 es are scarce, endothermic animals can lower core body temperature (Tb).

182 Defense of core body temperature (Tc) can be energetically costly;

183 i.c.v.) suppressed LPS-induced increases in core body temperature (Tc), whereas a lower dose (300 ng

184 meters studied provided a closer estimate of core body temperature than equilibrated rectal temperatu

185 , 2, and 3 had significantly (p < .05) lower core body temperatures than animals that received no tre

186 fects are partly mediated by the lowering of core body temperature that occurs during CR.

187 e ectothermic strategy, maintaining elevated core body temperatures that presumably confer physiologi

188 he physiological effect of norepinephrine on core body temperature, the fast increase of iBAT tempera

189 luding better noninvasive methods to measure core body temperature, the use of diagnostic imaging, ad

190 mild heat treatment that temporarily raised core body temperature to approximately 39.5 degrees C.

191 anticipatory locomotor activity and rise in core body temperature under the influence of the FEO.

192 iously shown to cause fever and viremia, and core body temperature, viremia, and blood cell and chemi

193 Core body temperature was 1 masculineC higher after high

194 Core body temperature was controlled using an intravascu

195 Core body temperature was measured by using ingested tel

196 Core body temperature was monitored with a rectal thermo

197 Core body temperature was not significantly altered in t

198 Core body temperature was recorded continuously for 10 d

199 Core body temperature was reduced in the calorie restric

200 emeters were implanted in 1-year-old GS, and core body temperature was remotely monitored.

201 After 3 days of cold exposure, core body temperature was significantly reduced in ad-li

202 Diurnal patterns of activity and core body temperature were progressively disrupted in R6

203 in PKR(-/-) mice during the dark period, and core body temperatures were lower during the light perio

204 ake levels were unchanged, but surprisingly, core body temperatures were lower.

205 Core body temperatures were measured through radioteleme

206 Jugular vein and core body temperatures were similar.

207 nd that leucine enkephalin directly controls core body temperature when exogenously injected into the

208 thermogenesis) and used by mammals to defend core body temperature when exposed to cold.

209 Transgenic mice are unable to maintain a core body temperature when placed in a cold environment,

210 to maintain elevated energy expenditure and core body temperature when subjected to hypercaloric die

211 Lowering core body temperature with cold saline infusion and cool

212 eterm neonates were associated with improved core body temperature (with moderate certainty of eviden

213 PV1 determines whether an antagonist affects core body temperature, with promising implications for a

214 hese rhythms correlate with daily rhythms in core body temperature, with temperature lowest when metH

215 investigated the effects a lower ambient or core body temperature would have on damage to striatal d