ライフサイエンスコーパス: core temperature

1 fferential increases in skin temperature and core temperature.

2 hetic nerve activity to CVA are dependent on core temperature.

3 bo trial, despite the attainment of a higher core temperature.

4 inhibition of motor activity and lowering of core temperature.

5 skin blood flow (dry heat loss) for a given core temperature.

6 n, roughly in proportion to the elevation in core temperature.

7 ronary vasospasm, arrhythmias, and increased core temperature.

8 at 30 degrees C and an inability to protect core temperature.

9 f Fe-rich planetary cores could overestimate core temperature.

10 ac arrest and resuscitation without changing core temperature.

11 onitor provides a non-invasive estimation of core temperature.

12 The EQ02 on average overestimated core temperature.

13 mice while monitoring drinking behaviour and core temperature.

14 ctivation of thermogenesis to maintain their core temperature.

15 leep latency with a simultaneous decrease in core temperature.

16 ensitive to central BDNF-induced increase in core temperature.

17 nnection between cardiovascular function and core temperature.

18 ed to absolute or delta changes in muscle or core temperature.

19 ehavioral activation is also known to change core temperature.

20 More severe NE was associated with lower core temperatures.

21 mone, and norepinephrine concentrations, and core temperatures.

22 be associated with the lowering of nocturnal core temperatures.

23 rgery, current practices lean towards higher core temperatures.

24 % VO2, peak for a period sufficient to raise core temperature 0.8 degrees C.

25 cited significant increases from baseline in core temperature (+1.3+/-0.3 degrees C), locomotion (+50

26 icrog, ivt) induced significant decreases in core temperature (-1.8 degrees C) 3-30 min following inj

27 At core temperature + 2 degrees C with respiratory alkalosi

28 ely, using a warm water-perfused suit, up to core temperature + 2 degrees C.

29 Core temperature 24-h profiles did not differ significan

30 lar [ivt]) elicited significant decreases in core temperature 3-30 min following injection with a max

31 e, the induction of therapeutic hypothermia (core temperature 32 degrees C-35 degrees C) has been sho

32 kPa), PaCO2 40 +/- 5 torr (5 +/- 1 kPa), and core temperature 33 degrees C +/- 1 degrees C.

33 with or without endovascular cooling (target core temperature 33 degrees C).

34 , "hypothermia" (standard resuscitation, but core temperature 34 degrees C), or "combi" (hyperoxia pl

35 hypothermic response in 62% (13/21) of mice (core temperature: -4.6 +/- 0.5 degrees C, P = 0.001 vs.

36 The neuromuscular and body-core temperature abnormalities, which were otherwise mil

37 he primary outcome was time-weighted average core temperature adjusted for baseline temperature.

38 Mean core temperature after surgery was lower in the hypother

39 ts with SAD, (1) the reductions in nocturnal core temperatures also were correlated with the reductio

40 d not find a correlation between the EEG and core temperature alterations.

41 study was to determine if increasing murine core temperature altered cytokine expression and improve

42 Delta increase in core temperature and absolute core temperature in HOT we

43 The strongest relationship between core temperature and behavioral activation occurred at 1

44 on ('heat therapy') results in elevations in core temperature and changes in cardiovascular haemodyna

45 systems that regulate metabolism, including core temperature and energy balance, we examined the eff

46 , median (interquartile range) elevations in core temperature and heart rate from baseline to end-exp

47 Methamphetamine (METH) changes core temperature and induces behavioral activation.

48 the DMH prior to MDMA prevented increases in core temperature and locomotion and attenuated increases

49 planted data loggers, we also monitored body core temperature and locomotor activity in some of the a

50 ng the high-dose METH treatment were maximum core temperature and minimum chamber temperature.

51 We recorded EEG, core temperature and motor activity before and after exp

52 C showed hyperactivity accompanying reduced core temperature and obesity when housed at 30 degrees C

53 (Prlr) as a thermal switch lowering maternal core temperature and promoting offspring survival in war

54 ding human beings, to maintain a stable body core temperature and respond to the ambient environment.

55 on the theoretical relationship between the core temperature and surface temperature distribution on

56 (BAT) thermogenesis, physical activity, and core temperature and that ERalpha(vlVMH) neurons provide

57 In actual operation, the core temperature and the surface temperature of the lith

58 The standard-of-care is to monitor core temperature and to maintain normothermia during gen

59 38 degrees C, 35 degrees C, or 32 degrees C core temperature and underwent 4 hours of hemorrhage (re

60 Core temperature and vital signs were measured at baseli

61 High calcium diets also increase core temperature and white adipose tissue uncoupling pro

62 nment that poses a risk of increases in body core temperature and/or perceived discomfort.

63 s it stable in a solid form at the outermost core temperatures and less dense than the surrounding li

64 mergency department systolic blood pressure, core temperature, and greater metabolic acidosis (analys

65 inuous, real-time measurement of heart rate, core temperature, and mobility.

66 does however increase heart rate, cumulative core temperature, and perceived exertion.

67 hase acutely increased sleep, decreased body core temperature, and prevented high-fat diet-induced we

68 lf-dousing and foot immersion on heart rate, core temperature, and thermal discomfort of healthy volu

69 a significant affect on MDMA neurotoxicity, core temperature, and thermoregulation in rats.

70 ere normotheric and heat stressed (change in core temperature approximately 0.75 degrees C).

71 toxin-induced alveolitis, coexposure to FRT (core temperature approximately 39.5 degrees C) doubled t

72 then exposed to febrile range hyperthermia (core temperature, approximately 39.5 degrees C), Hsp70 l

73 ythms of sleep, melatonin secretion and body core temperature are thought to be generated by the supr

74 15 animals, physiologic data (urine output, core temperature, arterial pressure, heart rate, cardiac

75 demonstrate good agreement of the predicted core temperature as a function of time with actual core

76 handling and related to an increase in body core temperature as a result of higher levels of uncoupl

77 f b.c.c.-iron able to model its behaviour at core temperatures as well as pressures, using ab initio

78 ared to the gold standard rectal thermometer core temperature assessment, as well as the potential in

79 It was projected that core temperatures associated with heat stroke (40.2 degr

80 ed mouse model, we showed that maintaining a core temperature at FRH (39 degrees C to 40 degrees C) r

81 ld intravenous Hartmann's solution decreases core temperature at hospital arrival and decreases the t

82 Keeping core temperature at least 35.5 degrees C in surgical pat

83 ial head injury less than 10 days earlier; a core temperature at least 36 degrees C; and an abnormal

84 umption, carbon dioxide production, and body core temperature before, during, and after hydrogen sele

85 ined persistent tachycardia, well before the core temperature begins to rise.

86 All achieved a core temperature below 34 degrees C (mean target tempera

87 e outcome require less cooling to maintain a core temperature between 33 and 34 degrees C during TH c

88 cardiac arrest care related to management of core temperature, blood pressure, oxygenation/ventilatio

89 was measured by radiotelemetry monitoring of core temperature, brown adipose tissue (BAT) temperature

90 FO did not significantly alter core temperature but decreased the postoperative rise in

91 mSMEI had seizures induced by elevated body core temperature but wild-type mice were unaffected.

92 on indeed becomes entropically stabilized at core temperatures, but in its pure state h.c.p.-iron sti

93 The intervention decreased mean core temperature by 1.20 degrees C (95% CI, -1.33 degree

94 Although use of prehospital cooling reduced core temperature by hospital arrival and reduced the tim

95 Regulated hypothermia produces a decrease in core temperature by lowering the brain's temperature set

96 In contrast, forced hypothermia lowers core temperature by overwhelming the body's capacity to

97 The daytime regulation of core temperature by serotonin 1A receptors appears norma

98 ther lower the core temperature or raise the core temperature by, respectively, producing negative he

99 At 0.5 degrees C increments in core temperature, CMRO(2) was calculated from the produc

100 of energy expenditure, substrate oxidation, core temperature, cold and hunger scores, and plasma par

101 No differences in substrate oxidation, core temperature, cold and hunger scores, or plasma para

102 able, non-invasive alternative for assessing core temperature compared to rectal temperature monitori

103 12, RE: 160 +/- 13 bpm; p < 0.01), estimated core temperature (CON: 37.6 +/- 0.4, AE: 38.7 +/- 0.3, R

104 Elevated core temperature constitutes an important biomarker for

105 oninvasive cutaneous device to monitor their core temperature continuously.

106 owed temperature-sensing capsules to measure core temperatures continuously for >/=48 h and kept acti

107 Survival, core temperature, cytokine production, and bacterial cle

108 icity and did so independently of effects on core temperature, DA transporter function, or METH brain

109 Using this model we analysed core temperature data from an experiment designed to exh

110 ker we propose a stochastic-dynamic model of core temperature data that contains both stochastic and

111 uch as plasma cortisol, plasma melatonin and core temperature data, currently methods are not availab

112 k for the energy storage system based on the core temperature detection is developed in this paper.

113 Core temperature did not differ significantly between th

114 01], but no interaction effect [p = 0.70] on core temperature drift.

115 increased energy expenditure, and attenuated core-temperature drop during acute cold exposure.

116 Core temperatures dropped without triggering observable

117 hese findings call for careful monitoring of core temperature during anesthesia in laboratory animals

118 active compounds was obtained with the lower core temperature during boiling, as well as higher tempe

119 ogen TNF-alpha is regulated by increments in core temperature during fever, generating an enhanced ea

120 l affective disorder, light treatment lowers core temperature during sleep in proportion to its antid

121 The regulation of the level of core temperature during sleep is linked with a proportio

122 lation, responsible for maintaining a stable core temperature during wide fluctuations in external an

123 r are primarily responsible for the observed core temperature effects during the initial post-treatme

124 mice revealed that systemic thermal therapy (core temperature elevated to 39.5 degrees C +/- 0.5 degr

125 RPV1 antagonist significantly increased body core temperature following oral administration in rodent

126 degrees C rather than 23 degrees C increased core temperature from 36.5 to 37.5 degrees C to 39.2 to

127 , increased invasiveness, and require stable core temperatures, good operator technique, and a compet

128 atients with a final recorded intraoperative core temperature >36 degrees C) and hyperglycemia (defin

129 Forty critically ill adults with fever (core temperature, >/= 38.3 degrees C).

130 Core temperature, heart rate, mean arterial pressure and

131 Core temperature, hemodynamics, serum glucose and electr

132 Core temperature, however, decreased only in animals rec

133 FEN at 30 degrees C displayed a significant core temperature hyperthermia for 4 h after the first dr

134 with a peak low of 33.8 degrees C, and this core temperature hypothermia lasted for 20 h after FEN a

135 neurotoxicity in rats that display either a core temperature hypothermia or hyperthermia, although h

136 FEN-treated rats at 24 degrees C displayed a core temperature hypothermia with a peak low of 33.8 deg

137 , neurotoxicity was seen and correlated with core temperature in all regions examined.

138 e that passive heating of up to +2 degrees C core temperature in healthy young men is not enough to c

139 e that passive heating of up to +2 degrees C core temperature in healthy young men is not enough to c

140 ta increase in core temperature and absolute core temperature in HOT were correlated to total game di

141 es in ambient temperature produce changes in core temperature in MDMA-treated rats, but the same chan

142 his study, we show that passively increasing core temperature in mice from the basal (36.5 to 37.5 de

143 n selective manner and to elevate rectal and core temperature in rats without accompanying cardiovasc

144 -HT efflux, neuromuscular activity, and body-core temperature in response to challenge injection of c

145 thermometry is commonly used to measure body core temperature in rodents because of its ease of use.

146 nsitivity (P<0.05), and transiently elevated core temperature in the transgenic mice, but was without

147 dynamics after dopamine infusions at various core temperatures in a pig model of surface cooling and

148 ic pathways allow organisms to protect their core temperatures in response to cold exposure.

149 iencing whole body contractions and elevated core temperatures in response to isoflurane exposure or

150 This study compares core temperatures in swine after central catheter infusi

151 gh thermal conductivity) requires high outer-core temperatures in the early Earth that complicate mod

152 After fire simulation training, core temperature increased (1.0+/-0.1 degrees C) and wei

153 ) healthy adults were passively heated until core temperature increased by 1.5 degrees C.

154 ine, 12 subjects were passively heated until core temperature increased by approximately 0.6 degrees

155 n after treatment for all doses and preceded core temperature increases; the onset of METH-induced hy

156 range, 37.1-37.9 degrees C) with the median core temperature increasing by 0.2 degrees C (interquart

157 In addition to increasing core temperature, infection can lead to changes in the d

158 protocol to estimate the T(wb) threshold for core temperature inflection in 12 volunteers.

159 derived from the T(wb) corresponding to the core temperature inflection point in participants expose

160 thermal niche through their interaction with core temperature, insulation, and environmental conditio

161 Additionally, the validity of the estimated core temperature is not impacted by the use turnout gear

162 Core temperature is reduced spontaneously after asphyxia

163 Of the three rhythms core temperature is the only reliable variable that can

164 Median time to core temperature less than 34 degrees C was 105 minutes

165 Spots for detecting WHO-defined hypothermia (core temperature <35.5 degrees C or peripheral temperatu

166 Reduced maternal core temperature may protect offspring from the teratoge

167 Mean skin (T(skin) ) and core (T(core) ) temperatures, mean skin wetness (W(skin) ) and n

168 s procedure affects brain, muscle, skin, and core temperatures measured with chronically implanted th

169 Core temperature measurement using a pulmonary artery (P

170 emperature as a function of time with actual core temperature measurement using an embedded thermocou

171 udies of DAT function were selected based on core temperature measurements in animals exposed to METH

172 oral and core equivalence modes) against PA core temperature measurements to determine which method

173 Continuous telemetric core temperature measurements were made during a 7h test

174 with both the levels of the previous nights' core temperature minima (P=.002) and the amounts of noct

175 lated by both melatonin and the level of the core temperature minimum.

176 Rectal core temperature monitoring can help fire services mitig

177 ent cold stress, which might make monitoring core temperature more important in this context ABSTRACT

178 rsal medulla produces an increase in BAT and core temperature more than 300% greater than TRH alone (

179 hypothermia (13+/-3% vs. 7+/-1% decrease in core temperature, n=6), and peritoneal exudate mediator

180 nsumption, resting metabolic rates, and body core temperatures occurred, but only after 7 days of tre

181 hetic sodium pentobarbitone, a small rise in core temperature occurs following ketamine.

182 lantic salmon were either super-chilled to a core temperature of -1.5 degrees C or directly chilled o

183 This resulted in a mean decrease in core temperature of 1.4 degrees C compared with 0.2 degr

184 een cooled and underwent OLT with the median core temperature of 33.4 degrees C (92.1 degrees F) (ran

185 Hypothermia with a core temperature of 35 degrees C was instituted for 24 h

186 eceive either aggressive warming to a target core temperature of 37 degrees C (aggressively warmed gr

187 sive warming had a mean final intraoperative core temperature of 37.1 degrees C (SD 0.3) whereas the

188 compared to SAL-treated groups, with a peak core temperature of 38.6 degrees C.

189 venting air exposure and oven-heated until a core temperature of 75 degrees C was reached.

190 We previously reported that increasing core temperature of bacterial endotoxin (LPS)-challenged

191 Furthermore, increases in core temperature of MDMA-treated animals increase neurot

192 changes in ambient temperature do not affect core temperature of saline-treated animals.

193 This thermal early warning network takes the core temperature of the energy storage system as the jud

194 real-time measurement to predict whether the core temperature of the lithium-ion battery energy stora

195 to target tumors with ts p53 mutations, the core temperature of tumor-bearing mice was lowered to 32

196 They reached peak core temperatures of 42.2 +/- 0.3 degrees C.

197 ass, administration of pre-CPB vasopressors, core temperature on CPB, pre- and post-CPB hematocrit, t

198 ute infections, but the effects of increased core temperature on host defenses are poorly understood.

199 temperature was adjusted to either lower the core temperature or raise the core temperature by, respe

200 Core temperature over 80 h for surviving infants was 35.

201 and exhibited a 0.81 degrees C lower initial core temperature (P<0.0005), an approximately twofold in

202 At the end of the TT in the heat, both core temperature (pla 39.7 +/- 0.3 degrees C, bup 40.0 +

203 f engineering systems where the time-varying core temperature plays a key role.

204 s also disrupted after CNO triggering of Di; core temperatures plummeted, then recovered.

205 We constructed daily, 24-h core temperature profiles for analysis.

206 ionship between (1) endogenous melatonin and core temperature profiles, (2) the proportional control

207 ) and were highly correlated with changes in core temperature (r = .9, P < .001).

208 While T(below) was also uncompensable, the core temperature rate of rise was considerably lower tha

209 In the prospectively cooled patients, core temperature reached target range less than or equal

210 l control cases that had not had activity or core temperature recordings previously.

211 Normal core temperature (rectal) was regulated in all animals.

212 ividuals with function-altering mutations in core temperature-regulating genes to determine whether d

213 at 20 and 22 degrees C showed a hypothermic core temperature response.

214 h the homeothermy (the stable maintenance of core temperature), resulting in hypometabolism and hypot

215 ed schedule, the endogenous component of the core temperature rhythm of subjects who were exposed to

216 In contrast, the core temperature rhythm of subjects who were not exposed

217 Chronic stress lowered the amplitude of core temperature rhythms, and lesions of the pPVTh block

218 and is a nexus that differentially regulates core temperature rhythms/HPA activity/specific white adi

219 Core temperature rose continuously in T(above).

220 in diverse physiological processes including core temperature sensing, apoptosis, and immune response

221 ed due to turnout gear [ps < 0.025], but not core temperature skin temperature, or ventilatory rate [

222 tigating hypoxia and hypercapnia or reducing core temperature so that neuroprotection occurs before t

223 Core temperature stability is the result of a dynamicall

224 overage in vivo but caused increases in body core temperature, suggesting that peripheral restriction

225 t of thermoregulation and the maintenance of core temperature, sympathetically-mediated control of th

226 side, in anaesthetized male Wistar rats at a core temperature (T(b)) of 37 degrees C, during acute se

227 Acute moderate-intensity exercise increases core temperature (T(c) ; +0.7-0.8 degrees C); however, s

228 -004, an EP1 receptor agonist, increased the core temperature (T(c)) in a dose-dependent manner (1.6+

229 e animals did not develop fever, whereas the core temperature (T(c)) of CVF vehicle-treated controls

230 ent temperature of 33 degrees C, the average core temperature (T(CORE)) of ovariectomized (OVX) contr

231 brile response without affecting basal body (core) temperature (T(c)).

232 ined blood pressure levels and abnormal body core temperature (Tb) regulation.

233 or the EP3 receptors and measured changes in core temperature (Tc) by using telemetry.

234 tress induced a transient hypothermia, where core temperature (Tc) declined immediately and then rose

235 The mean drop in core temperature (Tc) following hemorrhage was 1.5 degre

236 Heating stopped when maximum core temperature (Tc) of 42.4 degrees C was attained (Tc

237 Core temperature (Tc), heart rate (HR), activity, and bl

238 To this end, the core temperatures (Tc) of COX-1 and COX-2 gene-ablated m

239 eas the increases in preoptic PGE2 and body (core) temperature (Tc) following the intravenous (i.v.)

240 CO2 (+0.7%), core temperature (Tcore, +0.5 degrees C) and HR (+54 bpm

241 Core temperatures (Tcs) were recorded using thermocouple

242 sized that PFO+ subjects would have a higher core temperature than PFO- subjects due, in part, to abs

243 hypothesis that obese individuals have lower core temperatures than those in normal-weight individual

244 Hom mice maintain higher core temperatures than WT in the home cage, have chronic

245 These data suggest that the increase in core temperature that occurs during bacterial infections

246 at temperatures at the body surface than at core temperature, thereby favoring superficial hemostasi

247 If heat shock models elevated core temperature, these results suggest that fever may p

248 bient temperature of 4 degrees C, decreasing core temperature to 30 degrees C within 120 min.

249 used cooling blankets to lower the patients' core temperature to 32-33 degrees C.

250 Heat stress increased core temperature to a similar extent in both groups (Y:

251 the probabilistic prediction of a patient's core temperature to fall below 35.5 C during surgery.

252 , but is dependent on an induced decrease in core temperature to produce the effect.

253 h allowed the heart-rate derived estimate of core temperature to remain consistent with changes in Da

254 The core temperature triggering each response defines its ac

255 , FGF21 levels were related to the change in core temperature upon acute cold exposure, indicating a

256 n blood flow via laser-Doppler flowmetry and core temperature via ingestible telemetric pill were mea

257 Infant core temperature was 34.2 +/- 1.2 degrees C over the fir

258 The mean (+/- SD) final intraoperative core temperature was 34.7 +/- 0.6 degrees C in the hypot

259 Average patient core temperature was 36.7 degrees C (36-37.3 degrees C),

260 In studies employing a telemetry apparatus, core temperature was also increased by CRF(1-41) (1 nmol

261 Core temperature was analysed for parametric, circadian

262 e was controlled to +/-0.5 degrees C and rat core temperature was continually measured using a non-in

263 Core temperature was higher in HT group for first hour a

264 when a correction factor for differences in core temperature was included in the calculation.

265 However, when core temperature was increased by 2 degrees C with CO(2)

266 Estimated core temperature was marginally different from that meas

267 Foam core temperature was measured and increased to 47 degree

268 Core temperature was measured by a rectal probe attached

269 Core temperature was reduced to 32 degrees C-33 degrees

270 In all pups, core temperature was similar to chamber temperature, wit

271 serotonin 1A receptor activation also lower core temperature, we investigated the relationship betwe

272 ceived exertion, and area under the curve of core temperature were significantly elevated due to turn

273 Muscle and core temperatures were approximately 1 degrees C higher

274 For all groups, core temperatures were monitored rectally every 30 min f

275 ynamic data, catecholamine requirements, and core temperatures were recorded at 10-min intervals; blo

276 Simultaneous core temperatures were recorded from 1 to 5 days.

277 mitted to the hospital for a night of sleep (core temperatures were recorded), followed by infusions

278 The estimation method of the core temperature, which can better reflect the operation

279 ted with significant reductions in nocturnal core temperatures, which were correlated with similarly

280 as feasible and safe, and it rapidly reduced core temperature with minor reperfusion delay.

281 ear relationship, which allows 3D mapping of core temperatures with high accuracy and fast response.

282 s an early myocardial depression and rise in core temperature, yet reduced oxygen consumption and res