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1 IL-1beta polyclonal Ab significantly reduced corneal disease.
2 eal PMN number and significantly exacerbated corneal disease.
3               Both groups developed invasive corneal disease.
4 utic vaccine against recurrent HSV-1-induced corneal disease.
5 AS4 significantly decreased postreactivation corneal disease.
6                        Heterozygotes have no corneal disease.
7 a major role in the pathobiology of diabetic corneal disease.
8 athogenesis of herpes simplex virus-mediated corneal disease.
9 ted altering KC status, such as inflammatory corneal disease.
10  reactivations result in progressively worse corneal disease.
11 h implications for the treatment of blinding corneal disease.
12 n tear fluid derived from virally associated corneal disease.
13  loss of neurotrophic support contributes to corneal disease.
14  growth of fungal hyphae and the severity of corneal disease.
15  F. solani keratitis, a potentially blinding corneal disease.
16 located posterior chamber IOL in 1 case with corneal disease.
17 lenses of patients with contact lens-related corneal disease.
18 successful cataract surgery in patients with corneal disease.
19  be used with caution in patients with prior corneal disease.
20 ulation, followed by cataract, glaucoma, and corneal disease.
21 d exciting possibilities in the treatment of corneal disease.
22 nal ganglia were inversely proportional with corneal disease.
23 nd underappreciated role in the treatment of corneal disease.
24 creasing number and variety of patients with corneal disease.
25 ore were at higher risk of developing severe corneal disease.
26 rneal development and to study the causes of corneal disease.
27  increased amounts of MIP133 and more severe corneal disease.
28 stimulates MIP133 secretion, and exacerbates corneal disease.
29 rnea and have been shown to be biomarkers of corneal disease.
30 and its absence in MMP-9(-/-) mice decreased corneal disease.
31 in a significantly reduced capacity to cause corneal disease.
32  m or more should be evaluated for potential corneal disease.
33 ctions in triggering immune complex-mediated corneal disease.
34 s, healthy subjects, and patients with other corneal diseases.
35 us, normal subjects, and patients with other corneal diseases.
36 PKP is still being performed for more severe corneal diseases.
37 pacity, and it regulates genes implicated in corneal diseases.
38 ent a new option as an LVA for patients with corneal diseases.
39  QDs in A-CXL for corneal ectasias and other corneal diseases.
40 EW to enhance the vision of individuals with corneal diseases.
41 hod may also be applicable to other heredity corneal diseases.
42  and explains how two SNPs may contribute to corneal diseases.
43 rategy for the pathogenesis and treatment of corneal diseases.
44 itical defect tightly linked to common human corneal diseases.
45  surgical management of patients with severe corneal diseases.
46 mental models and therapeutic strategies for corneal diseases.
47  dry eye after refractive surgery, and other corneal diseases.
48 four previous reported methods in identified corneal diseases.
49 s a significant problem associated with many corneal diseases.
50                                              Corneal disease 1 (Dstn(corn1)) mice are homozygous for
51              C57BL/6 mice showed significant corneal disease 1 and 3 days after infection, which was
52                                              Corneal disease-1 (corn1) and corn1(2J) are spontaneous
53 d normal subjects (n = 12) and patients with corneal diseases (12 patients each: dry eye syndrome [DE
54 cal protection against primary HSV-1-induced corneal disease, a local ocular vaccine may prove more e
55      Lymphangiogenesis (LG) accompanies many corneal diseases after inflammatory, infectious, or chem
56 f the current strategies of treatment of the corneal disease and glaucoma in patients with Iridocorne
57 role that T cell costimulation plays both in corneal disease and in controlling the ability of the vi
58  MIP-133 production is necessary to initiate corneal disease and plays an important role in the subse
59 LPS before challenge reduced the severity of corneal disease and protected challenged mice against pe
60 peutic topical application of CD ameliorated corneal disease and reduced the bacterial count in the e
61 vation are separable, (ii) the phenotypes of corneal disease and spontaneous reactivation are separab
62 33 production might be necessary to initiate corneal disease and that it may play an important role i
63             The rabbits were followed up for corneal disease and viral loads in tears for 28 days.
64 ions are surgeries performed for irreparable corneal diseases and damage.
65 cia KPro offers functional success in severe corneal diseases and excellent retention in the short to
66             Clinical findings relate them to corneal diseases and to outcomes of refractive surgery.
67 h as arthritis, inflammatory bowel diseases, corneal disease, and periodontitis.
68                         Its efficacy against corneal disease appeared to be longer lasting than its e
69 and the use of popular current vital dyes in corneal disease are reviewed.
70 cal processes central to the pathogenesis of corneal disease, as well as other conditions including t
71  we found that two SNPs linked previously to corneal diseases, astigmatism, and Stevens-Johnson syndr
72 ce keratitis mimicking the onchocercal human corneal disease, BALB/c mice preimmunized with the bindi
73 d facilitates ophthalmologic examinations of corneal diseases based on the altered tissue stiffness.
74  function when visual impairment caused by a corneal disease becomes too severe.
75  motility contributed to the role of pili in corneal disease but was not involved in the role of pili
76  the leading causes of blindness of infected corneal diseases, but the pathogenesis of fungal keratit
77 ease, compared with the production of severe corneal disease by 2 x 10(5) PFU of marker-rescued virus
78                  Abrogation of virus-induced corneal disease by anti-cytokine antibodies suggests tha
79                                     Invasive corneal disease can be established with a surface inocul
80               PURPOSE OF REVIEW: Preexisting corneal disease can be exacerbated by cataract surgery a
81  most patients with visual impairment due to corneal diseases can be treated successfully with surger
82 (8) PFU per eye, d34.5 produced virtually no corneal disease, compared with the production of severe
83 were age- and sex-matched with no history of corneal disease (control group).
84         Inherited, iatrogenic, and metabolic corneal disease could be potentially treated by supplyin
85 d histopathologically, the human onchocercal corneal disease, demonstrating the antigenic specificity
86 lgorithm may be useful for computer-assisted corneal disease diagnosis.
87 t in the absence of costimulation with CD28, corneal disease does not take place.
88                      Patients with incurable corneal diseases experience visual impairment (VI) despi
89 t difference was that of more severe central corneal disease in DIF-negative patients.
90                  This idiopathic spontaneous corneal disease in dogs shares clinical features with ch
91 or recurrent HSK as evidenced by the lack of corneal disease in mice treated with anti-CXCL1 Ab.
92 ins, and especially alpha-toxin, can mediate corneal disease in mice.
93 m solani and Fusarium oxysporum cause severe corneal disease in the United States and worldwide and w
94                                              Corneal disease in this strain was significantly reduced
95 sents a powerful tool to study leukocytes in corneal diseases in context.
96 eople worldwide are bilaterally blind due to corneal diseases including infectious etiologies, trauma
97 eal physiology and in the pathophysiology of corneal disease, including modulation of keratocyte apop
98 g-h3) and furthermore that some of the human corneal disease-inducing substitutions identified in bet
99 red as a cost-effective treatment for severe corneal diseases internationally.
100 ever, delaying oral immunization until after corneal disease is established fails to mitigate keratit
101                                              Corneal disease is the most common cause of bilateral bl
102               Keratoconus(KC) is an ecstatic corneal disease leading to corneal-thinning and the form
103 as mock-immunized mice developed more severe corneal disease leading to stromal keratitis.
104 chs corneal dystrophy (FCD) is a progressive corneal disease marked by the development of guttae, foc
105                      Mild ocular surface and corneal disease may be treated effectively with aggressi
106                                  Preexisting corneal disease must be managed appropriately to get the
107 s (n = 177) and adults with non-transplanted corneal disease (n = 40).
108 h the changes observed in previously studied corneal diseases nor did they correlate with genes encod
109 ival, or it can promote diseases such as HSV corneal disease (or herpes stromal keratitis [HSK] in hu
110  important implications for the treatment of corneal disease, particularly corneal blindness due to L
111 n P. aeruginosa epithelial cell invasion and corneal disease pathogenesis was explored.
112  biology, serve as a reference to understand corneal disease pathology, and provide potential insight
113 may be useful therapeutic targets in several corneal disease processes.
114             To have a solid understanding of corneal diseases' progression, it is important to unders
115                                     In vivo, corneal disease response grades in the heat-killed antig
116 ic neutralization of IL-2 after the onset of corneal disease resulted in a rapid regression of inflam
117                          Treatment decreased corneal disease severity and reduced significantly the n
118                                              Corneal disease severity differed significantly among wi
119 d that extended to the cornea, such that HSV corneal disease severity was reduced in susceptible BALB
120 ct on the cornea also depends on preexisting corneal disease, severity and chronicity of intraocular
121                                     Signs of corneal disease should be identified preoperatively to a
122                    Researchers investigating corneal diseases should carefully consider these factors
123  efficacious against HSV-1-induced recurrent corneal disease than against recurrent HSV-1 ocular shed
124  infected Il1b-/- mice developed more severe corneal disease that is associated with impaired bacteri
125 tively uncommon, underdiagnosed degenerative corneal disease that is caused by damage to the ophthalm
126 ng elderly individuals; keratoconus (27%), a corneal disease that slowly deforms the cornea in young
127 ng transplant outcomes for infection-related corneal diseases that are common in the developing world
128          MDDN is a novel diagnostic tool for corneal diseases that can be used to automatically diagn
129 identify many candidate loci in the study of corneal diseases that lead to severe visual impairment.
130 ralization exacerbated P. aeruginosa-induced corneal disease, TIMP pAb- and normal rabbit serum (NRS)
131 nder the ROC curve of the algorithm for each corneal disease type was over 0.910 and in general it ha
132         To demonstrate the identification of corneal diseases using a novel deep learning algorithm.
133 val scarring (using Tauber staging), central corneal disease (vascularization, scarring, ulceration,
134                                              Corneal disease was categorized daily for 8 days with qu
135                                              Corneal disease was evaluated, and 7 to 378 days after i
136 entral nervous system (CNS) inflammation and corneal disease was evaluated, and responses in genetica
137 PAK strains in the cornea and development of corneal disease was impaired in DeltaexoS, DeltaexoT, an
138 a above 10(5) pfu the course and severity of corneal disease was not significantly different.
139                                              Corneal disease was quantified at regular intervals for
140                              Recurrent HSV-1 corneal disease was significantly reduced by therapeutic
141 s for cataract, vitreoretinal, glaucoma, and corneal diseases were included.
142  the homotypic gD1 vaccine against recurrent corneal disease, whereas the homotypic vaccine was much
143 role for endosymbiotic Wolbachia bacteria in corneal disease, which is characterized by neutrophil in
144 restore the reading ability in patients with corneal diseases, which is a crucial part of visual reha
145 age-related macular degeneration, as well as corneal diseases with abnormal angiogenesis.
146 edicated towards the use of MSC for treating corneal diseases with very promising outcomes.
147                           Only patients with corneal diseases without other ocular diseases were incl

 
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