ライフサイエンスコーパス: corneal edema

1 is and fibrosis, corneal epitheliopathy, and corneal edema.

2 infection, concurrent with the resolution of corneal edema.

3 t of hyperosmolar eye drops on early morning corneal edema.

4 , twenty-three with and thirty-eight without corneal edema.

5 rneal edema or predisposing risk factors for corneal edema.

6 te to keep cornea dehydration which leads to corneal edema.

7 ific features believed to be consistent with corneal edema.

8 d a particular pattern of bullous epithelial corneal edema.

9 de the cornea and regulate the resolution of corneal edema.

10 impflug imaging were done in view of diffuse corneal edema.

11 ssions were estimated to compare the risk of corneal edema.

12 o perform DSEK in patients with pseudophakic corneal edema.

13 Imaging are useful tools in cases with dense corneal edema.

14 sed to calculate the cumulative incidence of corneal edema.

15 No eyes had clinically apparent central corneal edema.

16 njury model and significantly diminished the corneal edema.

17 be necessary to minimize the risk of chronic corneal edema.

18 phema, IOP spikes, and transient microcystic corneal edema.

19 who underwent CXL treatment with subsequent corneal edema.

20 rvention reduced the likelihood of permanent corneal edema (0.5 days [from diagnosis of migration to

21 indings included keratic precipitates (22%), corneal edema (13%), conjunctival injection (10%), chemo

22 h incidence of persistent diplopia (12%) and corneal edema (20%), although half of the corneal edema

23 edema (CME) (172 eyes; 0.496%), significant corneal edema (28 eyes; 0.081%), persistent inflammation

24 a (7 eyes, 29.2%), cataract (5 eyes, 20.8%), corneal edema (4 eyes, 16.7%), and intraocular inflammat

25 evated intraocular pressure (8 eyes, 33.3%), corneal edema (6 eyes, 25%), intraocular inflammation (5

26 phy (94% of eyes) or pseudophakic or aphakic corneal edema (6% of eyes).

27 Of the 690 eyes (60%) that presented with corneal edema, 622 eyes (90%) had clear cornea at last f

28 255 eyes [94.4%]) or pseudophakic or aphakic corneal edema (75 eyes [5.6%]).

29 potony (12.9%), ocular hypertension (12.9%), corneal edema (8.9%), cystoid macular edema (6.9%), and

30 For bullous keratoplasty and/or corneal edema, 8.6% and 22% of grafts underwent repeated

31 were infection of the right eye, initial VA, corneal edema, a hypopyon larger than 1.5 mm, detection

32 gnificant differences between PPC and CCC in corneal edema, AC inflammatory reaction, capsular fibros

33 than 400 um cannot guarantee the absence of corneal edema after corneal collagen cross-linking, whic

34 endothelial damage with visually significant corneal edema after CXL treatment.

35 Given reports of corneal edema after endothelial exposure to platelet act

36 halmitis, suture erosion, haptic erosion, or corneal edema after IOL surgery.

37 main outcome measure: The resolution of corneal edema after surgery.

38 erior chamber cell and flare with or without corneal edema after the initial resolution of perioperat

39 o ECL, including 3 eyes with transient focal corneal edema and 4 eyes that required Micro-Stent trimm

40 corneal thickness due to delayed drainage of corneal edema and a trend towards prolonged corneal opac

41 urves and log-rank test were used to compare corneal edema and clinical progression of eyes in netars

42 Fuchs' endothelial disease and pseudophakic corneal edema and for high-volume surgeons in all diseas

43 netarsudil was associated with reduction of corneal edema and improvement in scotopic CDVA in Fuchs

44 crl(fl/fl)/CAGGCre-ER mice rapidly developed corneal edema and inflammation that was preceded by and

45 occupied the central cornea, accompanied by corneal edema and neovascularization.

46 ne discontinuation, the absence of bilateral corneal edema and opacities were noted at the slit lamp

47 visual acuity ranging from 20/100 to 20/400, corneal edema and opacity, anterior chamber reaction, or

48 tion of alpha-MSH following injury prevented corneal edema and opacity, reduced leukocyte infiltratio

49 Histologic sections showed more corneal edema and polymorphonuclear leukocyte (PMN) infi

50 ions of the anterior segment associated with corneal edema and secondary glaucoma.

51 ered significantly during periods of maximal corneal edema and stromal disease.

52 Examination showed central corneal edema and stromal haziness.

53 Corneal edema and subsequent vision loss ensues when end

54 options in blinding diseases associated with corneal edema and transparency loss.

55 ectedly beneficial role in the regulation of corneal edema and transparency.

56 ll monolayer of the cornea and can result in corneal edema and vision loss in severe cases.

57 Ultimately, these changes lead to corneal edema and vision loss.

58 disruption of the corneal epithelial layer, corneal edema, and a significant decline in conjuctival

59 nflammation (13%), IOP spikes (6%), hyphema, corneal edema, and BCVA loss (all 4%).

60 measured by increased viral load, decreased corneal edema, and decreased inflammatory cell infiltrat

61 al resistance to HSV-1 and alter viral load, corneal edema, and immune cell infiltration.

62 tion at 10(10) vp and moderate inflammation, corneal edema, and increased intraocular pressure at 10(

63 and photorefractive keratectomy for ectasia, corneal edema, and infectious keratitis.

64 pathology inclusive of epithelial erosions, corneal edema, and neutrophil infiltration.

65 bit increased mortality, growth retardation, corneal edema, and tooth enamel defects.

66 Performing earlier DSEK for pseudophakic corneal edema appears to be associated with improved vis

67 c treatment for Parkinson's Disease (PD) and corneal edema associated with intra-epithelial and -endo

68 orneas were clinically assessed for FECD and corneal edema by using slit-lamp biomicroscopy, and cate

69 Longstanding corneal edema can lead to anterior stromal scarring that

70 nd corneal edema (20%), although half of the corneal edema cases were likely due to pre-existing caus

71 tion of keratoconus, characterized by marked corneal edema caused by a break in Descemet membrane, al

72 ion according to the Mayo Clinic subclinical corneal edema classification.

73 Corneal edema cleared completely in 1 week.

74 Corneal edema cleared in 12/30 eyes and cleared with sca

75 There were no Grade 3 or higher corneal edema, corneal neovascularization, corneal stain

76 defined primary graft failure as nonclearing corneal edema despite a well-attached lenticule on anter

77 In 14 patients, corneal edema developed.

78 mong the 14 patients with corneal edema, the corneal edema did not resolve in 10 patients (71%), 6 (4

79 if-IOL (case 2) in order to treat secondary corneal edema due to pseudophakic bullous keratopathy.

80 ens have seen a reduction in the duration of corneal edema during acute hydrops, and have improved th

81 NBCe1, or CA activity was disrupted in vivo, corneal edema ensued and was associated with significant

82 kness may be useful in detecting preclinical corneal edema, especially in patients with FECD undergoi

83 B/c mice demonstrated both PMN migration and corneal edema, eyes of infected C57BL/6 mice failed to s

84 On examination, the left eye showed corneal edema, focal stromal infiltrates, hypopyon, and

85 120 eyes of patients who underwent DSEK for corneal edema following cataract surgery (CE); 87% of ey

86 with bilateral decreased vision secondary to corneal edema from endothelial dysfunction underwent Des

87 r of eyes with glaucoma (IOP >= 21 mmHg with corneal edema, Haabs striae, optic nerve cupping or buph

88 To minimize the risk of permanent corneal edema, immediate removal of the implant with a 2

89 Corneal edema improved in 4 patients and resolved in 1 p

90 ften secondary to long-standing preoperative corneal edema in 14 of 178 eyes (7.9%), or (partial) gra

91 At presentation, there was corneal edema in 30 eyes (45.4%), enlarged corneal diame

92 Fuchs' endothelial disease, or pseudophakic corneal edema in a 7-year period from 1999 in (1) high-v

93 Amantadine increases the risk of corneal edema in a dose-dependent manner.

94 Visually significant corneal edema in affected individuals was successfully m

95 Subclinical corneal edema in FECD can be detected by Scheimpflug tom

96 The extent of diurnal variation in corneal edema in Fuchs dystrophy is unknown.

97 This paper reviews the pathogenesis of corneal edema in Fuchs' dystrophy, the preoperative and

98 EK is a valid procedure for the treatment of corneal edema in hypotonic eyes after glaucoma procedure

99 A total of 4 of 5 patients had a history of corneal edema in the affected eye, and the fifth patient

100 The incidence of corneal edema in the Parkinson group (123 patients; 1.50

101 The incidence ratio for corneal edema in the Parkinson group vs the controls was

102 ld man with non-medical-responding bilateral corneal edema in treatment with systemic Amantadine for

103 and CA activity, disruption of which causes corneal edema in vivo and indicates that facilitation of

104 cluding loss of ZO-1 junctional contacts and corneal edema, in female than male mice, characteristic

105 Inferior corneal edema, in particular, should alert the practitio

106 , and the fifth patient had risk factors for corneal edema including a history of anterior uveitis an

107 is related to the risk of complications like corneal edema, intraocular pressure spikes, cystoid macu

108 Ocular examination showed bilateral mild corneal edema, iris atrophy, and presence of pigment in

109 potony, elevated intraocular pressure (IOP), corneal edema, iritis, IOL dislocation, cystoid macular

110 Corneal edema is a significant component of the various

111 Corneal edema is common in the setting of retained lens

112 ad complications which included failed bleb, corneal edema, keratoconjunctivitis sicca, filamentary k

113 Fifty-six percent of eyes had associated corneal edema, most often located inferiorly.

114 ll corneal transplants performed), a primary corneal edema mostly affecting elderly individuals; kera

115 rative AEs included iritis (n = 330, 1.53%), corneal edema (n = 110, 0.53%), and retinal tear or deta

116 (n = 5), corneal epithelial defect (n = 4), corneal edema (n = 3), and glaucoma (n = 1).

117 Complications included transient corneal edema (n = 4) and transient anterior chamber inf

118 orneal graft rejection accompanied by severe corneal edema, neovascularization and opacity that occur

119 No significant differences in CCT, ECD, corneal edema or clinical progression were observed betw

120 Most patients had a history of corneal edema or predisposing risk factors for corneal e

121 angiogenesis was notable for the absence of corneal edema or substantial inflammation.

122 ings of keratic precipitates with or without corneal edema, or anterior chamber cell and flare with o

123 a surgery or medication, refractive surgery, corneal edema, or corneal dystrophy, IOP and CCT reading

124 ahead of systemic symptoms (p < 0.001), and corneal edema (p < 0.001).

125 y Fuchs dystrophy or pseudophakic or aphakic corneal edema (PACE).

126 y and stabilization of refractive changes in corneal edema patients after cataract surgery, using vis

127 Corneal edema patients had a significant reduction of CD

128 nt anterior chamber inflammation, persistent corneal edema (PCE), rebound inflammation, and cystoid m

129 ferential diagnosis in cases with late-onset corneal edema post-cataract surgery.

130 Predicting the extent of corneal edema resolution after Descemet membrane endothe

131 ed the efficacy of hyperosmolar eye drops on corneal edema resolution.

132 Since corneal edema resulting from scrape injury was similar,

133 neal graft failure was defined as persistent corneal edema resulting in irreversible loss of optical

134 ients with increased intraocular pressure or corneal edema seen at Kaohsiung Chang Gung Memorial Hosp

135 s included conjunctival injection, chemosis, corneal edema, severe iritis, fibrin accumulation, and a

136 xtremely high intraocular pressure (IOP) and corneal edema similar to toxic anterior segment syndrome

137 Five patients had corneal edema that did not resolve following fragment ex

138 Among the 14 patients with corneal edema, the corneal edema did not resolve in 10 p

139 non-use of amantadine, the hazard ratio for corneal edema was 1.79 times higher in the amantadine su

140 se revealed that the 30-day hazard ratio for corneal edema was 2.05 higher in patients given moderate

141 The corneal edema was completely resolved during 2 months wi

142 Postoperative corneal edema was identified in 10 (2.9%) of 350 patient

143 The probability of corneal edema was increased 7-fold with Delta5-2mm SN <

144 Stromal disease with corneal edema was induced in rabbits by intrastromal inj

145 Corneal edema was induced, and the percent recovery per

146 Significant corneal edema was not observed.

147 In 4 of 6 episodes, corneal edema was present, typically in the corneal stro

148 Mouse models of pRTA exhibit acidemia, corneal edema, weak dental enamel, impacted colons, nutr

149 Six episodes of reticular bullous epithelial corneal edema were identified in 5 eyes of 5 patients tr

150 Rates of corneal edema were significantly higher in ACIOL cases (

151 Fuchs' dystrophy or pseudophakic or aphakic corneal edema, were enrolled by 105 surgeons from 80 cli

152 l epithelium acts intracellularly to promote corneal edema, whereas 12-HETrE acts in a paracrine mann

153 of 8 eyes with reinversion had postoperative corneal edema, which resolved at 48 hours.

154 cation (X(2) = 10.7, P = 0.001), presence of corneal edema (X(2) = 11.7, P < 0.001), and worse VA (U