ライフサイエンスコーパス: corneal haze

1 patient's risk to developing post refractive corneal haze.

2 ntiation and the development of fibrosis and corneal haze.

3 al corneal biopsy in 1 affected patient with corneal haze.

4 fy a novel role for desmin overexpression in corneal haze.

5 recruitment to the cornea and development of corneal haze.

6 epithelial thickness, stromal thickness, and corneal haze.

7 to the corneal stroma and the development of corneal haze.

8 ced neutrophil activation and development of corneal haze.

9 failure of glaucoma filtration surgery, and corneal haze.

10 n alphaVbeta3 expression with a reduction in corneal haze.

11 Eighteen patients showed a reduction in corneal haze 1 month after CXL.

12 showed a significant (P < 0.01) decrease in corneal haze (1.3 +/- 0.3) compared with the no-decorin-

13 Previous studies suggest that corneal haze after injury involves changes in the light-

14 e and effective in inhibiting development of corneal haze after PTK in rabbits.

15 ion is accompanied by continued reduction in corneal haze and aberrations, suggesting ongoing remodel

16 It also decreased corneal haze and fine-grained irregularities in ocular w

17 Most of the eyes had corneal haze and JOAG was associated with a higher prese

18 Backscattering of light, a function of corneal haze and opacification, was determined regionall

19 Ophthalmic pathology in patients includes corneal haze and progressive retinal and optic nerve atr

20 The most common complications were corneal haze and striae.

21 py provides a high-resolution measurement of corneal haze, and Amco Clear provides a means of standar

22 itment to the corneal stroma, development of corneal haze, and chemokine production were measured.

23 that LASEK may reduce postoperative pain and corneal haze associated with PRK.

24 Corneas were graded for corneal haze at 0, 1.5, 7, 21, 42, and 91 days after PRK

25 C treatment significantly (P < 0.05) reduced corneal haze at 2 weeks and was essentially normal by 12

26 of surgery (P < 0.001), and higher grade of corneal haze at baseline (P = 0.03) were associated with

27 7 to 9 days after transfer, characterized by corneal haze, conjunctival and episcleral injection, cor

28 gested that the development of postoperative corneal haze could be due to an increase in light scatte

29 Severe corneal haze developed by day 42 and persisted to day 91

30 Significant corneal haze developed in the null and aG1 vector-treate

31 spherical equivalent refractive accuracy, or corneal haze development between tPRK and conventional P

32 Reported complications include keratitis, corneal haze, endothelial cell loss and failure of treat

33 The incidence of corneal haze following tPRK was generally low across the

34 d that UBM particulate substantially reduced corneal haze formation as compared to the saline-treated

35 The results imply that MMC treatment for corneal haze has both short term and long term adverse e

36 ubjected to -9 D PRK significantly decreased corneal haze in vivo.

37 The corneal haze incidence (Grade >= 1) at 12 months was 1.3

38 The corneal haze incidence (Grade 1) at 12 months was 1.35%

39 The main outcome measures were corneal haze incidence, subjective SE, uncorrected dista

40 t can lead to serious complications, such as corneal haze, infectious and non-infectious keratitis, s

41 her, LPS-injected Lum(-/-) mice had elevated corneal haze levels compared with that of Kera(-/-) and

42 No case in the AMT group demonstrated corneal haze, limbal stem cell deficiency, symblepharon,

43 anterior surface irregularities and anterior corneal haze may be the most important limiting factors

44 cant reduction of keratometry, postoperative corneal haze may limit final visual acuity.

45 Among eyes in the MT group, corneal haze occurred in 44% (11/25; P = 0.001), corneal

46 ystine crystal density, iris visibility, and corneal haze on a scale from 0 to 5.

47 In addition, 62 eyes (77.5%) had corneal haze on examination.

48 Corneal haze post refractive surgery is prevented by mit

49 In the primary congenital glaucoma group, corneal haze showed a significant relationship with most

50 Quantitative measurement of corneal haze showed that the postnatal cornea was hazy a

51 s administered LHA510 developed a reversible corneal haze that resolved with cessation of treatment a

52 reakup time (TBUT), conjunctival congestion, corneal haze, vascularization, conjunctivalization, and

53 Corneal haze was evaluated before surgery and at 2, 3, a

54 In contrast, development of corneal haze was inhibited in the dnG1 vector-treated gr

55 Corneal haze was measured by in vivo confocal microscopy

56 Less corneal haze was observed in LASIK-treated eyes at 1 to

57 Corneal haze was present in 52 of 56 eyes 1 hour after t

58 Corneal haze was quantified with corneal densitometry (P

59 Corneal haze was the most frequently reported crosslinki

60 Corneal haze was the most frequently reported CXL-relate

61 The first signs of corneal haze were apparent shortly after reepithelializa

62 ve relationship was noted with C/D ratio and corneal haze, whereas for secondary congenital glaucoma

63 tration into the corneal stroma and elevated corneal haze, which is an indicator of loss of corneal t

64 (SAHA) however has been proposed to prevent corneal haze without any adverse effects.