ライフサイエンスコーパス: corneal opacity

1 raniofacial and ocular anomalies (colobomas, corneal opacities).

2 cularization but displayed similar levels of corneal opacity.

3 mits posterior segment visualization through corneal opacity.

4 notypes such as irido-corneal adhesions, and corneal opacity.

5 nfectious keratitis (IK) is a major cause of corneal opacity.

6 n cooperatively target genes associated with corneal opacity.

7 revention and treatment of infection-induced corneal opacity.

8 n 1 patient (2%), and there were no cases of corneal opacity.

9 escued pathological phenotypes and prevented corneal opacity.

10 educe the burden of corneal blindness due to corneal opacity.

11 e in matrix disorder at 8 weeks, but minimal corneal opacity.

12 etween successful epilation and less central corneal opacity.

13 -initiated epilation and its relationship to corneal opacity.

14 d bilateral trichiasis and 21% had bilateral corneal opacity.

15 immunohistochemistry for the development of corneal opacity.

16 ndication is visually significant congenital corneal opacity.

17 sits that correlated with the development of corneal opacity.

18 HSV-1 strain RE and subsequently graded for corneal opacity.

19 ups: congenital (72.5%) and acquired (17.5%) corneal opacity.

20 erning the LSC fate and its association with corneal opacity.

21 CSKs as a potential therapeutic solution for corneal opacities.

22 roves the surgical planning of children with corneal opacities.

23 is and follow-up of patients with congenital corneal opacities.

24 , autoimmune ocular disorders, and pediatric corneal opacities.

25 t, superficial, bilateral, diffuse, anterior corneal opacities.

26 e patients worsened significantly because of corneal opacity (0.46 +/- 0.74 logarithm of the minimum

27 rment due to glaucoma (0.71 [0.57-0.86]) and corneal opacity (0.54 [0.43-0.66]) were more common amon

28 SV-1 KOS-63 demonstrated higher incidence of corneal opacity (1.5 x) and neovascularization (2.6 x ;

29 corneal stromal dystrophy (23.3%), traumatic corneal opacity (10%), chemical corneal opacity (6.7%),

30 es were refractive errors (47.1%), keratitis/corneal opacity (16%), amblyopia (14.3%), ocular trauma

31 At 7 dpi Hd infected mice showed more severe corneal opacity (2.23 vs. 0.87; p = 0.0003), neovascular

32 ), traumatic corneal opacity (10%), chemical corneal opacity (6.7%), and Fuchs endothelial dystrophy

33 orkhead-domain missense variants, had severe corneal opacity (90%; sclerocornea in 47%), aphakia (83%

34 nalysis of woe2 adult eyes identified severe corneal opacities, abnormalities of the anterior segment

35 spension outperformed Vigamox(R) by reducing corneal opacity, achieving lower clinical scores (indica

36 report a case of a patient showing bilateral corneal opacities after amantadine chronic treatment for

37 sent here a case with traumatic cataract and corneal opacity after laser-assisted in situ keratomileu

38 corneas emerges as a promising treatment for corneal opacities, aiming to overcome limitations of tra

39 ted circumferential peripheral subepithelial corneal opacities and adjacent abnormal limbal vasculatu

40 This disease is characterized by corneal opacities and vision impairment.

41 urgical condition that presents with central corneal opacity and a significant hyperopic shift.

42 HSK resulted in significant reduction in the corneal opacity and angiogenesis.

43 eles at the mouse Cat4 locus, causes central corneal opacity and anterior polar cataract in heterozyg

44 especially with regard to the development of corneal opacity and blindness.

45 on of IL-6 at the time of infection restored corneal opacity and chemokine levels to that of wild-typ

46 lta41Delta29 and BGS41 vaccination decreased corneal opacity and delayed-type hypersensitivity respon

47 ox/flox);Le-Cre(+) eyes invariably displayed corneal opacity and developed spontaneous corneal neovas

48 syltransferase B3GLCT, leading to congenital corneal opacity and diverse extra-ocular manifestations.

49 Most mutant mice aged 3-8 months develop corneal opacity and eye lesions due to irritation and co

50 l cataract-microcornea with mild to moderate corneal opacity and in a consanguineous Cambodian family

51 Because of progressive corneal opacity and neovascularization, the patient unde

52 ithelial wound healing effect, the extent of corneal opacity and neovascularization.

53 hthalmia, congenital cataracts, microcornea, corneal opacity and nystagmus.

54 hen a decrease in tear volume, together with corneal opacity and ocular lesions.

55 B/c mice treated with rmST2 showed increased corneal opacity and perforation (at 5 days PI) when comp

56 Results from a bovine corneal opacity and permeability test demonstrated sligh

57 proteinase with 15 TSRs, leads to congenital corneal opacity and Peters anomaly (persistent lens-corn

58 ation of GAG in corneal stromal cells causes corneal opacity and reduced vision.

59 rVIP and subsequently demonstrated decreased corneal opacity and resistance to corneal perforation co

60 220 382 patients who received a diagnosis of corneal opacity and scars using H17 (ICD, Tenth Revision

61 achlorhydria, and visual impairments such as corneal opacity and strabismus.

62 ul tool in selected patients with concurrent corneal opacity and traumatic cataract.

63 ipopolysaccharide (LPS), is a major cause of corneal opacity and vision loss.

64 y of this myopic shift resulted from central corneal opacity and was successfully managed with supra-

65 Increased corneal opacity and worsened disease were displayed afte

66 diseases including skin blistering diseases, corneal opacities, and neurological degenerations.

67 pressure (30.8 +/- 12.5 mm Hg; mean +/- SD), corneal opacity, and enlarged anterior chambers.

68 t gene, is a novel candidate associated with corneal opacity, and it regulates genes implicated in co

69 in the thickness of the corneal epithelium, corneal opacity, and modest disorganization in the corne

70 ha-4 HDL present in their plasma and develop corneal opacities, anemia, proteinuria, and kidney failu

71 2.91, 95%CI:1.46,5.80), per-existing central corneal opacity (AOR = 3.83, 95%CI:1.52,9.69), pseudoexf

72 In SD-OCT scans, corneal opacities appeared as diffuse stromal hyperrefle

73 s, and in pediatric patients with congenital corneal opacities are disappointing.

74 It is important to recognize corneal opacity as a distinct etiology of central cornea

75 tinfection, were grouped on the basis of the corneal opacity as mild (</=2) or severe (>2).

76 of heterozygous TGFBI-R124C mice developed a corneal opacity at 40 weeks of age.

77 ted in significantly decreased virus-induced corneal opacity between 7 and 21 days after UV-B exposur

78 evious reports have demonstrated superficial corneal opacities, cataracts, increased intraocular pres

79 ce imaging (MRI) in children with congenital corneal opacities (CCO).

80 mice showed a significantly smaller area of corneal opacity compared to PBS-treated mice, thus impro

81 ccelerated reepithelialization and decreased corneal opacity compared with B6 mice after alkali wound

82 exhibited enhanced fungal killing and lower corneal opacity compared with unimmunized mice.

83 Corneal opacities (COs) are among the top 5 causes of bl

84 The case frequency of corneal opacity during the study period was 6535 cases p

85 ic disorder characterized by cardiomyopathy, corneal opacities, encephalopathy, hypotonia, and seizur

86 re of 4 with compromised visibility due to a corneal opacity (estimate = 0.45; P=0.039), and prolonge

87 d 4 in C57BL/6 mice, resulted in significant corneal opacity for up to day 10.

88 PV to extract dropped lens fragments through corneal opacity had similar final outcomes compared with

89 eads to an ocular phenotype characterized by corneal opacities identical to CSCD in humans.

90 in 9, nonglaucomatous optic neuropathy in 3, corneal opacities in 3, retinal disease in 3, and undete

91 Posterior keratoconus is a rare cause of a corneal opacity in an infant.

92 patchy changes in the stroma accompanied by corneal opacity in both eyes.

93 Corneal opacity in IL-6(-/-) mice was substantially dimi

94 ced corneal epithelial healing and decreased corneal opacity in murine corneal alkali burn model by m

95 ations (39.8% vs. 26.5% rural) compared with corneal opacity in rural areas (38.0% vs. 25.5% urban).

96 reased inflammation, neovascularization, and corneal opacity in the C3 KO mice.

97 ern blot analysis) determined that decreased corneal opacity in the galectin-3 inhibitor-treated corn

98 phies (64.66%) were the most common cause of corneal opacity in the study cohort.

99 ion outcomes in patients with a diagnosis of corneal opacity in the United States.

100 ds, and six months later, there was residual corneal opacity, iris atrophy and peripherical cataract

101 However, corneal opacity is considered a relative contraindicatio

102 ma changes including neovascularization; and corneal opacity, leading to plaque formation.

103 Corneal opacity may delay dropped lens fragment retrieva

104 ent with lysosomal storage disease including corneal opacities, multifocal central nervous system dis

105 Six patients had persistent corneal opacities, necessitating optical iridectomies.

106 TSG-6 markedly decreased corneal opacity, neovascularization, and neutrophil infi

107 No significant corneal opacity, neovascularization, blepharitis, and ep

108 ain of HSV-1, and HSK was monitored based on corneal opacity, neovascularization, leukocytic infiltra

109 ther ophthalmic examination revealed central corneal opacity occupying the optical axis with steepeni

110 uccessful epilation was associated with less corneal opacity (odds ratio [OR], 0.61; 95% confidence i

111 e and comprises neurodegeneration as well as corneal opacity of infantile-onset with epithelial autof

112 an-American male presented with a congenital corneal opacity of the right eye.

113 l tool in cases when a child presents with a corneal opacity of unknown or unclear etiology.

114 No evidence of corneal opacities or renal insufficiency was detected in

115 lude the following: ocular anomalies such as corneal opacity/Peters anomaly, coloboma, and microcorne

116 tion with P. aeruginosa and showed increased corneal opacity, PMN infiltration, bacterial counts, and

117 treated with TLR9 siRNA showed decreases in corneal opacity, polymorphonuclear leukocyte number, IL-

118 ut a posterior chamber, pre-existing central corneal opacity, pre-existing age-related macular degene

119 line, nummular, patch-like, and lattice-like corneal opacities, prominent corneal vascularization was

120 Cataract corneal opacities, refractive error and amblyopia, globe

121 icroscopic findings suggest that the central corneal opacities represent gradual deposition of extrac

122 and keratocyte differentiation in mediating corneal opacity resolution and visual function recovery.

123 isions, codes of 371 (corneal scar) and H17 (corneal opacity), respectively.

124 00% of patients had bilateral and unilateral corneal opacity, respectively.

125 Four weeks postinfection (p.i.), the mean corneal opacity score of -/- mice was 1.1 +/- 0.3 while

126 endogenous IL-6 in IL-6(+/+) animals reduced corneal opacity scores and MIP-2 levels to that of IL-6(

127 ophil infiltration and significantly reduced corneal opacity scores.

128 b over control-treated mice showed increased corneal opacity, stromal damage, and bacterial load.

129 .31 are associated with X-linked ichthyosis, corneal opacities, testicular maldescent, cardiac arrhyt

130 ctively reviewed for the primary etiology of corneal opacity, time of onset, duration of opacity, pre

131 capacity to suppress infectious inflammatory corneal opacity using a new model of LPS-induced keratit

132 We identified patients with corneal opacity using International Classification of Di

133 feld-Rieger anomaly, including iris defects, corneal opacity, vacuolar cataracts, significant iris/co

134 Our study showed that corneal opacity was diagnosed in 6.5% of patients in the

135 Corneal opacity was measured using optical coherence tom

136 Also, the frequency of corneal opacity was not significantly different in the s

137 Similarly, corneal opacity was significantly higher in Hd McKrae-in

138 In the remaining animals corneal opacity was studied and digital photographs were

139 The commonest etiology of corneal opacity was trauma (62.5%), followed by resolved

140 a highly sensitive imaging method to measure corneal opacity, we identify a novel role for desmin ove

141 The corneal opacities were associated with increased exfolia

142 Although no overt changes in corneal opacity were detected by slit-lamp examination,

143 The final vision outcomes in patients with corneal opacity were significantly worse compared with b

144 Most of the patients affected by corneal opacity were White (69.13%), followed by Black o

145 The 2 youngest patients had complete corneal opacity with features of corneal staphyloma and

146 ence tomography (ASOCT) revealed superficial corneal opacity with minimal stromal scarring.

147 erpes keratitis, nonulcerative keratitis, or corneal opacity without an obvious cause, 12 (0.6%) carr