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1 lular lipid layers of the SC relative to the corneocytes.
2 ted binding of individual S. aureus cells to corneocytes.
3 acteristic strong binding of S. aureus to AD corneocytes.
4 ed cell-derived markers that persist on shed corneocytes.
5 ganelles and convert into anucleate cells or corneocytes.
6 ells lose their nuclei and become desiccated corneocytes.
7 at a specific depth below the surface of the corneocytes.
8 ocalized to the extracellular spaces between corneocytes.
9 ls that can exceed the size of water-swollen corneocytes.
10 hat promote the adherence of S. aureus to AD corneocytes.
11 corneum showed premature loss of cohesion of corneocytes.
12 ix is largely compartmentalized within fetal corneocytes.
13 lular envelopes indicative of differentiated corneocytes.
14 in compound envelopes of mammalian epidermal corneocytes.
15 phi) and cellular replacement rates are 52.9 corneocytes/69.3 keratinocytes per mm2 per h approximate
16 le-corneocyte resolution to demonstrate that corneocytes actually undergo differentiation to develop
17 ter 6 h, and nearly complete dissociation of corneocytes after 24 h.
18 le N-terminal region of corneodesmosin on AD corneocytes, allowing S. aureus to take advantage of the
19                          This dysadhesion of corneocytes and its contribution to increased transepide
20 ule for the interaction of S. aureus with AD corneocytes and represents a target for intervention.
21  characterized by dysmorphic and pleomorphic corneocytes and the absence of vesicular bodies in trans
22       It is comprised of dead keratinocytes (corneocytes) and is known to maintain its thickness by s
23 s embryos, enlarged granular layer cells and corneocytes, and a morphologically abnormal cornified la
24 of a live mouse, including sebaceous glands, corneocytes, and adipocytes, with unprecedented contrast
25 neal and intraepithelial pustules, nucleated corneocytes, and dilated superficial dermal blood vessel
26 corneocytes in adult stratum corneum, vernix corneocytes appeared swollen, the density of the keratin
27                                      Because corneocytes are also required for barrier function, we h
28 hodology, the number of VPs per investigated corneocyte area was assessed and expressed as the Dermal
29  projections that occur on the surface of AD corneocytes as a result of low levels of skin humectants
30 n that is important for the formation of the corneocyte, as well as the generation of its intracellul
31 portunity to detect biomechanical changes in corneocytes because of, e.g., environmental factors, agi
32 e, PNPLA1-deficient mice lacked a functional corneocyte-bound lipid envelope leading to a severe skin
33 npla1-mutant mice promoted rebuilding of the corneocyte-bound lipid envelope, indicating that supplem
34 xyceramide, and the appearance of prominent, corneocyte-bound lipid envelopes, whereas cornified enve
35 a plausible organization of the SC lipids at corneocyte boundaries, the ratio D( )(lip)/D( )(lip) det
36 e show that FnBPB promotes adhesion to human corneocytes by binding strongly to loricrin, highlightin
37        Whereas the structure responsible for corneocyte cohesion has been visualized at the microscop
38 allel reductions in extracellular lipids and corneocyte compaction in all ichthyoses except epidermol
39 ogenic ClfB-deficient mutant to adhere to AD corneocytes compared to that of its parent clonal comple
40 tial role of vibrational imaging to evaluate corneocyte composition and molecular structure in the tr
41  We applied atomic force microscopy to study corneocyte conformation in patients with AD stratified b
42                                        These corneocyte conformational changes shed further insight i
43 m corneum (SC), is comprised of keratin-rich corneocytes, connected by proteinaceous corneodesmosomes
44  are used to determine whether the lipids or corneocytes constitute the main permeation barrier.
45                                    Anucleate corneocytes contain keratin filaments bound to a periphe
46 ed that the N-terminal region was present on corneocytes containing low levels of NMF and that blocki
47                    Stratum corneum comprises corneocytes, derived from outer stratum granulosum durin
48 re was no evidence of tracer accumulation in corneocytes, despite the fragility of nucleated keratino
49                                              Corneocyte desquamation has been ascribed to the followi
50 pth, but are not affected by depth-dependent corneocyte diffusivity; and the follicular contribution
51 (many of which failed to be secreted); i.e., corneocytes displayed retained cytosolic lamellar bodies
52 idermal permeability barrier is comprised of corneocytes embedded in a lipid enriched matrix.
53 atum corneum is composed of protein-enriched corneocytes embedded in an intercellular matrix of nonpo
54 that 12R-LOX contributes specifically to CLE-corneocyte envelope cross-linking, which appears to be a
55 te lipid envelope (CLE) to the proteinaceous corneocyte envelope, thereby providing a scaffold for th
56  1/desmocollin 1 redistribute uniformly into corneocyte envelopes (CEs) in the outer SC (shown by pro
57 d for barrier function, we hypothesized that corneocyte formation could also be regulated by barrier
58 ause cornification was blocked by occlusion, corneocytes formed specifically in response to barrier,
59                Our studies revealed abnormal corneocyte fragility and basal permeability barrier func
60 rmal lamellar body secretion, rather than to corneocyte fragility or an abnormal cornified envelope/c
61 ncreased transepidermal water loss, enhanced corneocyte fragility, decreased dendritic epidermal T ce
62 keratosis of the ear and the tail epidermis, corneocyte fragility, increased transepidermal water los
63    Adherence of single S. aureus bacteria to corneocytes from AD patients ex vivo was studied using a
64                                              Corneocytes from AD skin are structurally different from
65 t variations in the mechanical properties of corneocytes from different volunteers.
66                                              Corneocytes from fingermarks were successfully detected
67 from AD skin are structurally different from corneocytes from normal healthy skin.
68  have a reduced ability to adhere to low-NMF corneocytes from patients.
69                             In both tissues, corneocytes have normal-looking cell envelopes, despite
70 ophisticated filter constituted by layers of corneocytes in a lipid matrix.
71                         Compared with mature corneocytes in adult stratum corneum, vernix corneocytes
72 neum, whereas the intracellular space of the corneocytes in mid-stratum corneum (25 microm diameter)
73 lease of proinflammatory IL-1 cytokines from corneocytes in patients with atopic dermatitis (AD) with
74 terial adhesion to the cornified envelope of corneocytes in the outer layer, the stratum corneum.
75 c force microscopy showed that it shrunk the corneocytes in the stratum corneum (p<0.001) and the ima
76                     Adhesion of S. aureus to corneocytes in the stratum corneum is a key initial even
77  colonisation is adhesion of the bacteria to corneocytes in the stratum corneum where the cornified e
78 er cells were all stained uniformly, but not corneocytes in the stratum corneum.
79 nd the transformation of granular cells into corneocytes, in an SP- and Casp-14-dependent manner, sig
80 iven these empirical data: (1) the number of corneocytes is a mean proportional between the sum of th
81 yer and the more rigid internal structure of corneocytes is apparent, which is consistent with the cu
82 e intracellular mechanical behavior of human corneocytes is determined using "nanoneedle" AFM probes.
83 t measures the relative NMF concentration in corneocytes is introduced.
84  the ratio of nucleated epidermal cells over corneocytes is phi proportional, 75,346/17,778 approxima
85                            The maturation of corneocytes isolated from different depths in healthy hu
86 in the concentration of NMF was observed for corneocytes isolated from superficial compared to deeper
87 cyte differentiation, decrease the number of corneocyte layers, significantly increase the number of
88 ption of the outermost and inner two to four corneocyte layers, which swell less.
89 2B, has been proposed to covalently bind the corneocyte lipid envelope (CLE) to the proteinaceous cor
90                                          The corneocyte lipid envelope (CLE), a monolayer of omega-hy
91 tein-bound omega-hydroxyceramides and of the corneocyte lipid envelope and die shortly after birth fr
92 ion, and suggest further that acylCer and/or corneocyte lipid envelope are required elements in perme
93 hed lipids, showed numerous foci with absent corneocyte lipid envelope in ABT- versus vehicle-treated
94 er) are the predominant lipid species of the corneocyte lipid envelope in the epidermis.
95 ke a defect in the formation of a functional corneocyte lipid envelope linked to impaired omega-O-acy
96 ovalent binding to protein, thus forming the corneocyte lipid envelope, a key component of the epider
97  omega-hydroxyl of the ceramide, forming the corneocyte lipid envelope, a scaffold between lipid and
98                Mutants showed lamellar body, corneocyte lipid envelope, and cornified envelope abnorm
99 ical structure of the epidermal barrier, the corneocyte lipid envelope.
100 tter constituting the major component of the corneocyte lipid envelope.
101 nces the concept that the oxidations disrupt corneocyte membrane lipids, promoting release of free om
102                             Abnormalities in corneocyte morphology have been observed in patients wit
103 cts (natural moisturizing factor [NMF]), and corneocyte morphology in patients with AD.
104        NMF levels are highly correlated with corneocyte morphology in patients with AD.
105                    Extensive dissociation of corneocytes occurs after 24 h of water exposure.
106 LK5 causes excessive proteolysis, leading to corneocyte overdesquamation and barrier compromise.
107  900-microm2 corneocyte surface area, 17,778 corneocytes per mm2, 14-d (SC) turnover time, and 93,124
108 n, and the weight of evidence shows that the corneocyte phase of the SC is considerably more permeabl
109 hydrophobic lipid matrix with embedded fetal corneocytes possessing unique biomechanical and water-bi
110 on in transepidermal water loss or lipid- or corneocyte-related gene expression.
111 Here, we use intravital pH imaging at single-corneocyte resolution to demonstrate that corneocytes ac
112 (SC) and Malpighian epidermis, the number of corneocytes results from subtraction of a cellular fract
113 e SC interstices in RXLI could contribute to corneocyte retention, by increasing CD and interlamellar
114 iciently to delay CD degradation, leading to corneocyte retention.
115 ikely facilitating exfoliation by increasing corneocyte rigidity.
116 cent cornified-bound lipid envelope, i.e., a corneocyte scaffold abnormality does not explain the bar
117 ratum corneum surface that results in proper corneocyte shedding in desquamation.
118 ipped, yielding the characteristic polygonal corneocytes shown by scanning electron microscopy as wel
119 information on fixed human epithelial cells, corneocyte skin flakes, and polymers used for bioimplant
120 nocytes per mm2, 16 (SC) layers, 900-microm2 corneocyte surface area, 17,778 corneocytes per mm2, 14-
121           Natural moisturizing factor (NMF), corneocyte surface protrusions, cytokines, free sphingoi
122 n children who developed AD, whereas NMF and corneocyte surface texture were similar.
123 cognized ligands distributed over the entire corneocyte surface.
124 tinocytes, which transform into protein-rich corneocytes surrounded by extracellular lamellae of uniq
125                                              Corneocytes swell uniformly with the exception of the ou
126 yer is a compacted lattice of lipid-embedded corneocytes that provides an organism's barrier to the e
127  comparable with the thickness of a layer of corneocytes, this technique can be used to follow the di
128 enhancing its partitioning into keratin-rich corneocytes through concurrent binding of SPP with kerat
129 y important if one considers the intervening corneocytes to be impermeable "bricks." However, the lat
130 er, although increased fragility of isolated corneocytes was demonstrated.
131  IR and Raman spectral quality of individual corneocytes was high and revealed depth-dependent variat
132                                              Corneocytes were also labeled with an anti-corneodesmosi
133 hibitor Z-VAD-FMK delayed cornification, and corneocytes were structurally aberrant in Casp14(-/-) mi
134 e lipid multilamellae region surrounding the corneocytes within the stratum corneum.

 
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