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1 iation, and a specialized form of apoptosis (cornification).
2 and de novo epidermal lipid synthesis during cornification.
3 sslinking enzymes essential for keratinocyte cornification.
4 process that appears as an important step in cornification.
5 sions of the oviduct; and persistent vaginal cornification.
6 g little or no sebum and undergoing abnormal cornification.
7 parently brought about by early keratinocyte cornification.
8 cts on infundibular keratinocytes to promote cornification.
9 ln/Lys-rich domains, and is likely to impair cornification.
10 s a dominantly inherited genetic disorder of cornification.
11 with acantholysis and Mendelian disorders of cornification.
13 strated that Cl-amidine treatments slow down cornification and alter autophagy in the granular layer.
14 gmenting JNK activity could be used to delay cornification and enhance wound healing, whereas attenua
15 ed with execution of the genetic programs of cornification and epidermal barrier formation, to a full
16 ed by ARNT, which have critical roles in the cornification and epidermal barrier function of the skin
17 ogical analysis showed that all EFNAs induce cornification and keratin genes while suppressing wound
18 CDH10/11/12/2; P < 0.05), whereas epidermal cornification and proliferation measures were upregulate
19 imination by shedding, keratinocyte death by cornification, and cell-cell cannibalism by entosis.
20 er, the pan-Casp inhibitor Z-VAD-FMK delayed cornification, and corneocytes were structurally aberran
22 sensorineural hearing loss and disorders of cornification, and screening of several connexin genes w
23 ,15-EET epoxygenases in regulating epidermal cornification, and they have important implications for
25 by two unrelated methods initiated a wave of cornification, assessed as TdT-mediated dUTP nick end-la
28 -mediated hydrolysis of phospholipids during cornification, contributes to stratum corneum acidificat
31 ital ichthyosis (ARCI) is a diverse group of cornification diseases associated with severe clinical c
32 eciphering of the molecular basis of various cornification disorders, less is currently known about t
34 perproliferation of the epidermis, disturbed cornification, fragile cornified envelope (CE, a skin ba
36 cross linking leads to defects in epidermal cornification in lamellar ichthyosis and acral peeling s
37 rders characterized by erythema and abnormal cornification, including erythrodermic psoriasis, atopic
38 target of Blimp-1 repression indicates that cornification involves suppression of normal osmotic reg
41 to increase neutrophil activation, cellular cornification/keratinization, and antimicrobial peptide
42 al differentiation in vivo: transcription of cornification markers, inhibition of motility, withdrawa
46 the breakdown and degradation of DNA during cornification of lingual keratinocytes and aberrant DNA
47 -mediated transport, and reflected increased cornification of stratified epithelium and thus mirrored
48 tial therapeutic implications for preventing cornification of the corneal epithelium in response to t
51 ition to blistering and a severe disorder of cornification, patients typically display an abnormality
56 ed differentiation with abnormally increased cornification, reduced thickness of non-corneal layers,
57 that inhibit JNK production downregulate the cornification response of PCHCE cells to osmotic stress.
58 ocess is dysregulated in genetic diseases of cornification that are driven by impairments in keratino
61 ase inhibitor z-VAD, suggesting that reduced cornification was not entirely because of premature apop
62 f a complex differentiation process known as cornification, which culminates with the formation of th
63 roup of rare autosomal dominant disorders of cornification with overlapping features including hyperk