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1 hh develop a dorsal telencephalic midline, a cortical hem, and two cortical hemispheres.
2 ssed in the cortical hem, and in its absence cortical hem progenitors contribute excessively to the a
3 G neuronal progenitor domain or the adjacent cortical hem (CH).
4 xpression of transthyretin mRNA, the CPe and cortical hem are linked by shared expression of members
5 phalic structures, including hippocampus and cortical hem, while the ventral telencephalon appears to
6  functions to repress the generation of both cortical hem- and PSB-derived CR cells.
7  fate mapping studies, as a novel marker for cortical hem- and septum-derived CR cells.
8  transcription factor (TF)-binding sites for cortical hem-patterning TFs.
9 lation of TGFbeta signaling in explants from cortical hems of wild-type mice altered p21 expression a
10 ephalic dorsal midline structures, including cortical hem, hippocampus and choroid plexus, either fai
11  Wnt signaling profile of the dorsal midline cortical hem, which in turn causes gyrification of the d
12 orsal midline signaling in the production of cortical hem- and PSB-derived CR cells.
13 onstrate that, although the specification of cortical hem-derived CR cells is dependent on signaling
14                                 The Wnt-rich cortical hem is a transient, neuron-containing, neuroepi
15  in forebrain signalling centres-the septum, cortical hem and the pallial-subpallial boundary-known t
16 The cerebellar rhombic lip and telencephalic cortical hem are dorsally located germinal zones which c
17 cerebellar rhombic lip and the telencephalic cortical hem.
18                                          The cortical hem is an embryonic signaling center that gener
19                                          The cortical hem, a source of Wingless-related (WNT) and bon
20                    In addition, although the cortical hem does not show features of differentiated CP
21  source of fibroblast growth factors and the cortical hem, a medial structure expressing winglessint
22 ate, which expresses Fgf8 and Fgf17, and the cortical hem, which expresses Bmps and Wnts.
23 expresses fibroblast growth factors, and the cortical hem, which expresses bone morphogenetic protein
24 that Wnt gene expression is deficient at the cortical hem in XtJ/XtJ mice, but that the expression of
25 close developmental relationship between the cortical hem and the CPe, Wnt gene expression is upregul
26       We identified interactions between the cortical hem, rich in Wingless-Int (WNT) proteins and bo
27 e dorsal telencephalic signaling center, the cortical hem.
28 alic and diencephalic signaling centers, the cortical hem and zona limitans intrathalamica (ZLI), are
29 alon, WNT2b expression appears to define the cortical hem, a dorsal signaling center previously chara
30 gions of the telencephalon (for example, the cortical hem (CH)) to populate the entire cortical surfa
31 in both Cajal-Retzius cells derived from the cortical hem that guide migration of progenitors and neu
32         FGF8 coordinates with Wnt3a from the cortical hem to regulate graded expression of transcript
33 tion of neocortical CR cells arises from the cortical hem.
34 sion might be a consequence of growth in the cortical hem (medial patterning center), which produces
35 t decreased Wnt3a and Lef1 expression in the cortical hem and adjacent hippocampal promordium and con
36 e, Wnt gene expression is upregulated in the cortical hem both before and just as the CPe begins to f
37 s were required for enhancer activity in the cortical hem in vivo Mis-expression of Lmx1a in hippocam
38  plexus in XtJ mice is due to defects in the cortical hem that include Wnt gene misregulation.
39 ping telencephalon Lmx1a is expressed in the cortical hem, and in its absence cortical hem progenitor
40 cal surface due to a migratory defect in the cortical hem, and is accompanied by upregulation of Ebf3
41 expression pattern of Frizzled10 mRNA in the cortical hem, dorsal thalamus and dorsal neural tube.
42                                       In the cortical hem, expression of LacZ mRNA was confined to th
43 itulates Cux2 expression specifically in the cortical hem.
44 rol the migration of CR cells arising in the cortical hem.
45 dial telencephalic structures, including the cortical hem, which normally expresses a number of Wnt m
46  of the Frizzled10 gene in order to mark the cortical hem (the most caudomedial edge of the telenceph
47  the previous finding that in these mice the cortical hem is expanded leading to increased production
48 rchestrate the formation and function of the cortical hem (CH), a critical signaling center that cont
49  source, positioned as a mirror image of the cortical hem, along the lateral margin of the cortical p
50           In the absence of Nf2, NPCs of the cortical hem, hippocampal primordium and neocortical pri
51 omolog 3) mutants results in the loss of the cortical hem-derived CR character but does not affect th
52  mutants, even prior to the formation of the cortical hem.
53   Effects of diminished BMP signaling on the cortical hem were at least partly responsible for these
54 crease in Cux2 enhancer activity outside the cortical hem.
55                           In particular, the cortical hem, a region of high bone morphogenetic protei
56  the explants were evaluated by removing the cortical hem or presumptive extrahippocampal cortex from
57 whose normal expression is restricted to the cortical hem are completely absent in Gli3(Xt/Xt) embryo
58 elencephalic neuroepithelium adjacent to the cortical hem.
59 ly, we compared several human enhancers with cortical hem-restricted activity and found that recurren